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40 Cards in this Set

  • Front
  • Back
What does the Cardiovascular system do?
transports oxygen, nutrients, and other substances to body's tissue and carries metabolic waste product to kidneys and lungs
How many chambers in the heart?
4; 2 chambers on each side, atrium and ventricle
Right side of heart
receives blood from superior and inferior venae cava and pumps it through pulmonary arteries to pulmonary circulation
Left side of heart
receives blood from pulmonary veins and pumps it through aorta into systemic circulation
Three layers of the heart wall
pericardium, myocardium and endocardium
Pericardium
heart encased in pericardium, which has a fibrous later and two serous layers
Myocardium
middle layer, thick muscular tissue that contracts to eject blood from ventricles
Endocardium
lines inner chambers and valves
Tricuspid valve
right side of heart
Mitral valve
left side of heart; atrioventricular (AV) valves -separate atria from ventricles
Aortic valve
opens from left ventricle into aorta (semilunar valve)
Pulmonic valve
opens from right ventricle into pulmonary artery (semilunar valve)
Diastole
ventricles relax and fill with blood from left and right atria; higher atrial pressures passively open AV valves, allowing blood to fill ventricles (80% of blood from atria flows into relaxed ventricles) Contraction forces remaining 20% through (atrial kick) at the end, ventricles are filled with blood
Systole
ventricles contract creating pressure to close AV valves (prevents backflow into the atria); semilunar valves open, eject blood to aorta (left) and pulmonary arteries (right). As blood is ejected, ventricular pressure decreases causing semilunar valves to close; ventricles relax...diastole
Electronic Conduction
SA (cardiac pacemaker) node through internodal tracts to AV node travel through branches and Purkinje fibers in myocardium which results in ventricular contraction.
AV node compensation
If SA node fails to discharge, AV node can generate ventricular contraction at slower rate of 40 to 60 impulses per minute.
Bundle branches compensation
If both SA and AV nodes are ineffective, bundle branches may contract, but at very slow rate of 20 to 40 impulses per minute.
Ventricular systole
1st phase of Cardiac cycle
(S1 -"lub") closing of MV and TV
Ventricular diastole
(S2 -"dub") closing of the AV and PV
Factors that affect the heart
Aging, Lifestyle, Disease
5 points of Auscultation in Heart Assessment
1. 2nd Right ICS (aortic valve)
2. 2nd Left ICS (pulmonic valve)
3. 3rd Left ICS (Erb's point)
4. 4th Left ICS (Tricuspid valve)
5. 5th Left Midclavicular Line (Mitral valve) -Apical
Murmur Intensity Grades
Grade I barely audible in a quiet room
Grade II quiet but clearly audible
Grade III moderately loud
Grade IV loud and associated with a thrill
Grade V very loud; thrill easily palpable
Grade VI very loud; thrill palpable and visible
Aortic stenosis (Systolic) [heard over aortic valve area; radiates to neck, down left sternal border]
S1-III-S2
(heard between normal heart sounds) -medium pitch, coarse with crescendo-decrescendo pattern; pitch low
Pulmonic stenosis (Systolic) [heard over pulmonic valve; radiates left to neck; thrill at 2nd & 3rd ICS]
S1-III-S2
heard over pulmonic valve; (heard between normal heart sounds) medium pitch, coarse with crescendo-decrescendo pattern
Aortic regurgitation (Diastolic) [patient sitting and leaning forward, 2nd R ICS, radiates to L sternal border]
S1 S2 III---
Blowing in early diastole; pitch high
Pulmonic regurgitation (Diastolic) [patient sitting or leaning forward; 3rd & 4th ICS]
S1 S2 III---
Blowing; pitch high or low
Mitral Stenosis (Diastolic) [bell at apex with pt in L lateral decubitus position]
S1 S2
Low rumble more intense in early and late diastole; pitch low
Tricuspid Stenosis (Diastolic) [Bell over tricuspid area]
S1 S2
Similar to mitral stenosis but louder on inspiration; pitch low
Mitral regurgitation (Holosystolic) [diaphragm at apex, radiates to left axilla or base]
S1 IIIII S2
Harsh blowing quality; pitch high
Tricuspid regurgitation (Holosystolic) [5th ICS, left sternal border]
S1 IIIII S2
Blowing; pitch high
P
Atrial depolarization and contraction
QS
Ventricular depolarization and contraction
T
Ventricular repolarization and relaxation
Ken-tuck-y
S1 S2 S3
Ten-ness-ee
S4 S1 S2
Pulse Amplitude Ratings
0+ Absent
1+ Diminished, barely palpable
2+ Normal
3+ Full volume
4+ Full volume, bounding hyperkinetic
What is "Bruit"?
a rushing, blowing sound created by the blood passing through the narrowed section of the vessel. (Carotid pulse)
Normal findings on Auscultation
S1 loudest at mitral area
S2 loudest at pulmonic area
Abnormal findings
Splitting (valves aren't synchronized) S1 or S2
Third heart sound (S3) -CHF, fluid overload
Fourth heart sound (S4) -CAD, cardiomyopathy
Opening snap
Summation gallop
Midsystolic click
Pericardial friction rubs
Murmurs
Inspect upper and lower extremities for
Symmetry
Skin integrity
Color and temperature
Capillary refill
Hair distribution
Sores
Varicose veins
Peripheral Edema chart
1+ barely perceptible pit - 2mm
2+ Deeper pit; rebounds in a few seconds - 4mm
3+ Deep pit; rebounds in 10 -20 seconds - 6mm
4+ Deeper pit; rebounds in >30 seconds - 8mm