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127 Cards in this Set

  • Front
  • Back
Maxillary primary teeth are 2-3 mm ___ than their permanent successors
Smaller
Primate space are ___ % prevalence
70
Where are the primate spaces
Mesial to primary maxillary canines
Distal to primary mandibular canines
Ugly duckling stage common in children ___ to ___.
9-13
Ugly Duckling stage:
Eruption of laterals tips the centrals ____ from each other
away

Space corrects itself when canines erupt
Localized genetic factors
Maxillary laterals are the ___ most commonly congenitally missing teeth after the 3rds
2nd
Localized Genetic factors:
Mandibular second bicuspids are also often
congenitally missing
Localized genetic factors:Impacted permanent canine may cause a _____ when it causes posterior and anterior teeth to drift
Diastema
Generalized genetic factors:
1. Microdontia
2. Normal-sized teeth in a large arch
Generalized genetic factors:
Tooth size discrepancies affect ___% of the population
5
To determine if a tooth size discrepancy exists:
Bolton analysis
Diagnostic Wax-up
Inferiorly displaced labial frenum
When it doesn't migrate apically like it normally does
Distalizing appliance (Distalizing upper molars will create space)
PM will drift distally
Anterior diastemata appear
Rotated teeth will create ___ in adjacent sites
space
Supernumerary teeth: Mesiodens may cause ___
spacing
Close the diastema ___ excision of the inferiorly displaced frenum
Before


Cuz frenectomy results in scar tissue formation
Implants ARE or ARE NOT appropriate for cleft areas
ARE NOT
2 options for congenitally missing laterals
1. Create/maintain space for implants
2. "Lateralize" canines (canine substitution)
Diastema closure removable appliance
Hawley with finger springs
- Small, localized diastema closure
- Tooth are tipped into space
- Good choice if roots are well-aligned
Diastema closure Fixed appliance
Generalized and/or large spaces
Teeth are moved bodily
Root alignment achieved/maintained
Localized genetic factors
1. Peg laterals
2. Congenitally missing/impacted teeth (maxillary laterals and mandibular second bicuspids)
3. Impacted permanent canine (posterior and anterior teeth many drift, causing a diastema)
Generalized genetic factors
1. Microdontia
2. Normal-sized teeth in a large arch (BOlton tooth size discrepancies)
Other factors creating diastemas
1. Iatrogenic discrepancy from ortho extractions
2. Racial factors
3. Inferiorly displaced labial frenum
4. Clefts
5. Pathological conditions like caries and perio disease and cysts
6. Ortho treatment
7. Rotated teeth
8. Supernumerary teeth
9. Habits
10. Tongue size/posture
Should u do something about a <2 mm diastema in an older child?
No

Will spontaneously close when permanent canines erupt
Microdontia or generalized spacing
Usually requires ortho care
Class I Malocclusion is what percent
54%
Normal occlusion is what percent
30%
Class II malocclusion is what percent
15%
Class III malocclusion is what percent
1%
Convex profile can be caused by:
1. Mandibular retrognathism
2. Maxillary prognathism
3. Combination of both
Sunday bite
Posturing mandible forward to mask the skeletal and dental malocclusion

Make sure mandible is in CENTRIC RELATION during veal of profile and skeletal relationship
Mouth breather does what kind of dental and skeletal changes?
1. Face height increased
2. Posterior teeth supra-erupts
3. Mandible would rotate down and back
4. Open the bite anteriorly
5. Increase OJ
6. Narrowing of the maxilla due to stretched cheeks
Mouth breather does what do posture?
Lower the mandible
Lower the tongue
Tip back the head
Almost all Class II skeletal patients have a ____ molar relationship
Class II
Class II Div 1
Maxillary centrals proclined (flares out)
Class II Div 2
Max centrals may be retroclined
Class II div 1 has ___ mandibular plane angle, ____ lower face height.
Higher/steep mandibular plane angle

Longer lower face height

Incisors typically proclined
Class II Div 2 has ___ mand. plane angle, ___ lower face height, ___ bite
Class II skeletal

Low mandibular plane angle

Short lower face height

Retroclined maxillary incisors

Deep bite
Class II: ANB is what?
>4
Treating Class II
1st phase- late mixed dentition with eruption of 1st permanent molars

2nd phase- eruption of all permanent dentition (full ortho treatment)
Orthopedic correction of a skeletal class II may only be attempted in a ____
Growing patient

Prior to peak height velocity

12 in girls
14 in boys
Patients get only __ opportunity in life for orthopedic corrections
1
Necessary EARLY TREATMENT:
Distal step in primary 2nd molars

Relationship of the 1st permanent molars are determined by that of the primary molars

Bad cuz the best senario from this is an end-on Class II
Class II treatments (skeletal correction)
1. Orthopedic
2. Orthodontic
3. Surgical
Orthopedic
1. Head gear (restrains maxillary; allows mandible to catch up; no effect on mandibular teeth)
2. Functional appliance (forces patients to position their mandible forward to enhance mandibular growth)
3 types of head gear
1. Cervical pull (short or normal face heights; lower angle)
2. Combination (average face height)
3. High pull (long face height and high angle)
Surgical approaches
1. Mandibular advancement
2. Sliding genioplasty
3. Maxillary setback
4. Maxillary impaction
5. Combination
6. Distraction Osteogenesis
Orthodontic treatment
Extraction of 2 maxillary PM (camouflage)
Get a class II molar at the end of treatment

Best indicated with: maxillary protrusion, excessive over jet, excessive maxillary crowding with minimal mandibular crowding
Classification of cross bites:
Always defined by the relationship of the maxillary teeth to the mandibular
Bilateral posterior lingual crossbite
No mandibular shift
Midlines generally aligned
Look for constricted maxilla and expanded mandibular teeth
Anterior Crossbite
Typically an AP problem

Skeletal
Dental
Dental crossbite
Resulting from malposition of teeth
Can be:
Single or multi tooth problem

Generally due to ectopic eruption or crowding out of the arch
Skeletal crossbite
Can involve the transverse dimension and/or sagittal (A-P) dimension
Functional crossbite = most common cross bite in children
Altered posture of the mandible from CR caused by occlusal interferences or mild maxillary constriction which causes patient to slide in one direction to intercuspate or avoid interference
#1 cause of functional cross bite is:
In the primary canines

Can also be caused by interferences of maxillary buccal cusps related to mild maxillary constriction
In CR, ___ are generally coincident and ___ is normal
Midlines....overjet
In CO, ____ are often ____ and the patient will have some type of cross bite (Pseudo class III)
Midlines...Off
If child has Class I molar occlusion and normal skeletal growth but the appearance of mild maxillary constriction...they may need ____
Palatal expansion
Growth is affected ____
Bilaterally.

Condylar and dentoalveolar remodeling may occur

Early treatment is warranted to avoid development of skeletal asymmetry
Unilateral dental posterior crossbite (without mandibular shift)
Much less prevalent than functional cross bite due to a shift

Midlines coincident

Cause- usually unilateral maxillary dentoalveolar constriction
Relative crossbite
Class II malocclusion

No crossbite in CO
Rare crossbites:
Bilateral buccal crossbite
- "Brodie-bite"
- "Scissor-bite"
- entire maxillary arch telescopes out and over the mandibular arch
- Patient is usually Class II deep bite


Unilateral posterior buccal crossbite
- Unilateral maxillary expansion with or without mandibular constriction
- Supra-eruption of maxillary posterior segment on affected side may produce vertical maxillary excess (VME)
You can only do limited treatment for simple dental cross bite when:
There's:
1. Sufficient space M-D
2. Sufficient overbite to retain the tooth
3. Root in a normal position within the dental alveolus
4. Normal cuspid and molar occlusion

If any of these are not met or if patient has skeletal malocclusion, --> need MORE COMPREHENSIVE treatment
Correct ___ single tooth cross bites ASAP
Anterior
Treatment for simple dental cross bite:
1. Full/limited ortho with cross bite elastics
2. Hawley retainer (corrects 1 or 2 teeth in crossbite)
3. Anterior inclined bite plane
4. Tongue blade therapy (limited uses)
Limited treatment modalities for anterior teeth:
Inclined plane/bite plan
- ideal for interlocked incisors
- should engage tooth at a 45 degree angle
- Cemented with temp cement for easy removal
- close monitoring needed to evaluate supra-eruption of the posterior dentition

Tongue blade therapy (for minor anterior cross bite):
- Requires compliance, encouragement
- Teeth must be in the initial stage of eruption with minimal interlocking
- Tongue blade placed lingual to maxillary teeth and labial to mandibular teeth
- Constant pressure for 10min/hr
Posterior crossbite correction:
1. Maxillary expansion (rationale for max orthopedic skeletal therapy)

2. Rapid palatal expansion (minimizes tooth movement and maximizes skeletal displacement)
- Primarily accomplished with tooth-borne appliances
- Period of 1-6 weeks of activation
- 1 turn/day or more (0.25mm per turn)
- Maximizes skeletal displacement
- Typically lose 25-33% of total expansion to relapse
- Need to retain for 3-6 months to allow suture to reorganize and form new bone
Brands of rapid palatal expanders
1. Hyrax appliance
- Tooth borne and hygienic

2. Haas appliance
- Provides more palatal support during expansion and retention
- Tissue and tooth-borne
- Less hygienic

3. Bonded RPE
- Bite plane effect
- Mixed dentition
-Primary teeth often extracted upon removal
Temporary side effects from maxillary expansion
1. Temporary diastema
- usually closes spontaneously in 1-2 weeks

2. Buccal tipping of teeth
3. Bite opening
4. Excessive dental expansion vs skeletal can cause fenestration of the PM and molar roots through the buccal cortical bone
Skeletal Class III
1. Attempt to treat with an expander and/or face mask in the pre-pubertal years and hope that the correction will be maintained...or

2. Accept that skeletal growth is predetermined and wait for growth to stop, and treat with ortho/orthognathic surgery approach
Treatment for Class III
1. Protraction facemask
- moves maxillar downward and forward
- 8 oz to 1 lb of pressure per side
- Usually preceded by palatal expansion to "loosen" circummaxillary sutures and promote maxillary protraction
Adult Class III surgical intervention
1. dental and/or mild skeletal Class III
- Class III elastics
-Lower incisor extraction

2. Skeletal correction requires
- Maxillary advancement and/or mandibular setback
-May also require maxillary downdraft and transverse surgical expansion or SARPE
Surgically assisted rapid palatal expander
Indicated when more than 10 mm of expansion is needed
-Orthopedic expansion in non-growing patient

Midline osteotomy and Le Fort I (minus down-fracture)
Ectopic eruption: which is most likely
Maxillary 1st molars erupting mesially and resorbs distal of primary 2nd molar so it gets lost early
Causes of premature space loss
1. Caries (most common)
2. Trauma
3. Ectopic eruption
4. Ankylosis leading to extraction
5. Congenitally missing teeth
Congenitally missing teeth most commonly found:
Maxillary lateral incisor
Mandibular second premolar
Band and Loop
Maintains space of prematurely lost 1st or 2nd PRIMARY molar

Primary 2nd molar or permanent 1st molar can be banded

Wire loop has limited strength (can only hold space of 1 tooth)
Nance holding arch
Maxillary palatal arch and an acrylic button

Does not contact the anterior teeth

Uses palatal tissue to provide resistance to anterior movement of posterior teeth

Indicated if primary maxillary molars have been lost bilaterally

Acrylic button can become embedded in soft tissue
If molars are lost bilaterally
Nance or lingual arch is indicated
Functions of primary teeth
1. Mastication
2. Esthetics
3. Space maintenance
Management of arch length deficiencies
1. Maintain leeway space
2. Advance/flare incisors
3. Distal movement of molars
4. Expansion of arch
5. Interproximal enamel reductions
6. Serial extractions
Maintain leeway space
The amount by which the space occupied by the primary C,D,E exceeds that occupied by permanent 3,4,5

Maxillary: 1.5 mm per side
Mandibular: 2.5 mm per side
Distal movement of molars
Maxilla:
1. Pendulum
2. Distal Jet
3. Nance and coil spring
4. Head Gear (distalizes posterior teeth w/out reciprocal protrusion of anterior teeth

Mandible:
1. Lip bumper (remove lip pressure to allow incisors to come facially and flare and tips molars distally)
Serial extractions
1. Severe crowding
2. Primary canines are extracted to provide space for alignment of the incisors
3. Primary 1st molars are extracted when 1/2 to 2/3 of the 1st PM root is formed to speed eruption of the 1st PM
4. When the 1st PM have erupted they are extracted and the canines erupt into the remaining extraction space


REMBER CD4
Influences on vertical development
Specific causes
Muscular function
Respiratory pattern
Skeletal growth
Habits
Specific causes:
1. Disturbances in embryologic development (rare <1%)
2. Fetal molding and birth injuries
3. Childhood jaw fractures
- Condylar process is susceptible to injury
- Tends to regenerate well after early fractures (<5% are problematic)
4. Acromegaly (1/25,000)
- Anterior Pit Tumor:
will lead to High levels of GH leads to excessive mandibular growth in adults and skeletal class III
- Abnormal growth stops once tumor removed but skeletal deformity persists
5. Idiopathic condylar resorption and hemimandibular hypertrophy
Idiopathic condylar resorption
Resorption of one or both condyles leading to asymmetry
Hemimandibular hypertrophy
Unilateral excessive growth of mandible causing variable ants of asym
2 ways facial muscle can affect jaw growth:
1. Formation of bone at the point of muscle attachment
2. Muscle's role as part of the ST matrix whose growth normally carries the jaw growth downward and forward
Decreased facial muscle strength
Mandible drops down and back leads to excessive eruption of posterior teeth and narrowing of maxillary arch leads to anterior open bite
Mouth breather
Lowered mandibular and tongue position
Adenoid facies
familiar facial pattern to obstructing adenoid tissue

Any chronic respiratory obstruction could lead to this type of facial morphology (Long Face Syndrome)
Anterior open bites
Blacks > whites
Deep bite
Whites> blacks
Vertical excess
Excessive eruption of the maxillary and mandibular posterior teeth
Treatment goals for vertical excess
1. Control vertical dimension through management of posterior eruption and alveolar development

2. Growth redirection through mandibular autorotation
Treatment for vertical deficiency
1. incisor intrusion
2. posterior tooth extrusion
3. surgical intervention
4. combination of ortho and surgical treatment
Treatment options for vertical deficiency:
Dental correction:
1. Archwires
2. Bite plate
3. Bite raiser/turbo

Surgical correction:
1. Lefort Osteotomy
2. Mandibular osteotomy
Etiology of open bite malocclusion
1. Disturbances in embryologic development
2. Skeletal growth disturbances
3. Hemi-mandibular hypertrophy
Muscle dysfunction results in:
1. Increased anterior facial height
2. Excessive eruption of posterior teeth
3. Narrowing of maxillary arch
4. Anterior open bite
Open bites:
Longer anterior facial height, longer lower facial height, increased genial angle
Thumb sucking
Most important determinant is DURATION (>6 hrs can produce malocclusion)

Sequelae of thumb sucking:
1. Increased buccinator muscle contraction
2. Tongue lowers vertically away from maxillary teeth affecting cheek/tongue pressure balance
3. Upper molars move lingually and arch width narrows
4. Maxillary arch becomes V-shaped due to constriction of corners of mouth in canine area
5. Hindered eruption of incisors
6. Displacement of incisors (Flared and spaced max incisors and lingually positioned mandibular incisors)
7. Posterior teeth erupt as jaw is positioned downward
8. Anterior open bite develops
What is least likely to correct spontaneously
The constricted maxillary arch
Take a ceph to:
distinguish between dental and skeletal problems
Infantile swallow
Jaws apart
Lips contracted
Tongue contacts the lower lip
Transition swallow
Lips contracted
Separation of posterior teeth
Forward protrusion of tongue between teeth
Adult swallwo
Teeth together
Lips relaxed
Tongue to palate

In children by age 6
Tongue thrust
too little pressure and frequency to affect tooth position

Not the cause for open bite
Forward resting tongue posture
Can be very influential on teeth position
Functional Appliances are used to address:
1. Habit control
2. Functional re-education
3. Growth stimulation
Functional matrix
Includes all muscles ass with deglutition, breathing, swallowing and speech
Balters' objectives for functional appliances
1. Elimination of the lip entrapment (lip bumper)
2. Elimination of mucosal damage due to deep bite (anterior and posterior bite plates)
3. Correction of mandibular retrusion and the associated malposition of the tongue
4. Correction of the occlusal plane
___ is usually considered a sequellae of an infantile suckle pattern.
Tongue thrust
Underlying cause of a tongue thrust may be ___,___ or ___ problems
Physiological
psychological
or neurological
Treatment for tongue thrust: Tongue crib or lower lingual tongue thrust appliance or tongue rollers/beads
Can be removable or fixed appliance

Fixed are more effective
Removable appliance can be a helpful reminder for ___ children.
Older
When an open bite extends from the anteriors to the posteriors..
you should suspect more serious etiological factors
Other causes of open bites
Enlarged adenoids
Enlarged turbinates
Enlarged tonsils
Macroglossia (enlarged tongue)
Constricted nasal airway
Lateral tongue thrust
Can be either bi-lateral or unilateral

Less obvious

Often overlooked

Usually result in a posterior open bite
Low resting tongue position
Can contribute to the development of mandibular prognathism pushing the mandible forwards
___ and ___ are often related
Digital habits and tongue thrusts
Monobloc type appliance
Includes:
Monobloc, activator, bionator, andresen, woodside and many others

Eliminates deep bite and prevents tongue from going forward
Herbst
Many forms but all share the same basic design of 2 bucally placed piston hinges
WHy is retention necessary?
1. Reorganization of the perio and gingival tissues
2. Soft tissue pressures from cheeks lips and tongue)
3. Occlusal changes related to post-treatment growth
Classic work of John Edwards
Elastic recoil of gingival fibers esp in correction of rotation
Positioners:
may be used as an interim retainer for achieving final occlusion following alignment (used to make a good case better)
Occlusal adjustment/equilibration
may be helpful for improved interdigitation in cases where some teeth have not previously been in occlusion