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92 Cards in this Set

  • Front
  • Back

ECG chart


Too fast

- Increased myocardial O2 demand


- Decreased ventricular filling time


- Decreased coronary perfusion




Resulting in decreased CO

ECG chart


Too slow

- Slow ventricular rate




Resulting in decreased CO



ECG chart


No P wave

- Loss of atrial kick causing decreased preload




Resulting in decreased CO




- Risk for clot formation

ECG chart


ST changes

- Potential ischemia/injury causing decreased contractility




Resulting in decreased CO

Pacemakers of the heart


Rate of firing


SA node

60-100/min



Pacemakers of the heart


Rate of firing


AV junction

40-60/min

Pacemakers of the heart


Rate of firing


Purkinje fibers

20-40/min

Stroke volume assessment


Preload (5)

- JVD


- CVP


- Crackles


- Edema


- Mucus membranes



Stroke volume assessment


Afterload (3 Vs)

- Vessel diameter


- Viscosity


- Valves

Stroke volume assessment


Contractility (3)

- Hx


- EF (from ECHO)


- Preload (Starling's law)

ECG complex


P wave

- Represents atrial depolarization

ECG complex


PR interval

- Represents the time it takes for the electrical impulse to leave the SA node, depolarize the atria, and pass through the AV junction


- Measured from the beginning of the P wave to the beginning of the QRS complex


- Normal range is 0.12-0.2 sec

ECG complex


QRS complex

- Represents ventricular depolarization


- Normal range is 0.04-0.1 sec

ECG complex


J point

- The junction between the QRS complex and the ST segment

ECG complex


ST segment

- Represents the time where the ventricles have depolarized and repolarization begins


- Located from the end of the QRS complex to the start of the T wave

ECG complex


ST depression

- Represents myocardial ischemia



ECG complex


ST elevation

- Represents myocardial injury

ECG complex


T wave

- Represents the time it takes for the ventricles to repolarize


- Represents ventricular repolarization

ECG complex


QT interval

- Represents ventricular activity (depolarization and repolarization


- Prolonged QT may lead to life threatening dysrhythmias (R on T phenomenon leading to ventricular tachycardia)

ECG chart


Interventions


Too slow (2)

- Atropine


- Pacemaker

ECG chart


Interventions


Too fast (4)

- CSM (carotid sinus massage)


- Vagal maneuvers


- Chemical (beta blocker, amiodarone, adenosine)


- Electrical (cardioversion)

ECG chart


Interventions


No P wave (2)

- Chemical (amiodarone)


- Electrical (cardioversion depending on rhythm)

ECG chart


Interventions


ST changes (3)

- MONA (morphine, oxygen, nitro, aspirin)


- ? Thrombolytic


- ? Cath lab

Properties of Electrical Cells


Automaticity

- Ability to generate an electrical impulse spontaneously, without external stimulation



Properties of Electrical Cells


Excitability

- Ability of cell to depolarize in a response to an electrical stimulus

Properties of Electrical Cells


Refractoriness

- The period of time during which cardiac electrical cells are unresponsive to any stimulus, regardless of strength



Properties of Electrical Cells


Rhythmicity

- Ability of cardiac pacemaker cells to fire at regular intervals





Properties of Electrical Cells


Conductivity

- Spread of electrical activity from one specialized cardiac cell to another

Junctional arrhythmias


Rate


Junctional escape rhythm



40-60 bpm

Junctional arrhythmias


Rate


Accelerated junctional rhythm



60-100 bpm

Junctional arrhythmias


Rate


Junctional tachycardia

>100 bpm

Junctional arrhythmias


Criteria (3)

1) No P wave, normal QRS, regular


2) Inverted P wave, normal QRS. regular


3) Normal QRS, inverted P wave, T wave

Premature terms


Unifocal

- From one abnormal focus

Premature terms


Multifocal

- From more than one abnormal foci

Premature terms


Bigeminy

- Every second beat is abnormal





Premature terms


Trigeminy

- Every third beat is abnormal

Premature terms


Quadrigeminy

- Every fourth beat is abnormal

Premature terms


Couplet

- Two abnormal beats occurring together

Premature terms


Triplet

- Three abnormal beats occurring together

Premature terms


Run

- More than three abnormal beats occurring together

First degree heart block

- PR interval >0.2 sec


- P wave to QRS complex ratio is 1:1


- Regular

Second degree heart block type I

- PR interval consecutively lengthens


- More P waves than QRS complexes


- Irregular

Second degree heart block type II

- PR interval constant (normal or >0.2 sec)


- More P waves than QRS complexes


- Regular

Third degree heart block

- No consistent relationship between P wave and QRS complex


- More P waves than QRS complexes


- Regular

Pacemaker definition

- An electronic device that regulates HR and electronically stimulates the myocardium to depolarize, which then starts a contraction

Indications for Pacemaker (3)

- Pt with dysrhythmias


- Decrease in CO


- Unresponsive to drug therapy (due to acidosis, heart block)



Stat Pacemakers

- Can be either internal or external and are used for short term and or temporary "rescue" tx

Other reasons for a pacemaker

Single chamber pacemakers

Dual chamber pacemakers

Types of Temporary Pacers (3)

- Epicardial


- Transcutaneous


- Transvenous

Epicardial pacemaker

Transcutaneous pacemaker

Transvenous pacemaker

Pacemaker codes (3)

Pacemaker codes


Letter 1

Pacemaker codes


Letter 2

Pacemaker codes


Letter 3

Atrial spike

Ventricular spike

Pacemaker malfunctions (3)

- Failure to pace


- Failure to capture


- Failure to sense

Failure to pace


Definition

Failure to pace


Causes (3) and Interventions (4)

Failure to capture


Definition

Failure to capture


Causes (4) and Interventions (4)

Failure to sense


Definition

Failure to sense


Causes (2) and Interventions (3)

R on T phenomenon

Wandering atrial pacemaker

3 different P waves in one rhythm

Heart sounds


S1

- Closing of mitral and tricuspid valves

Heart sounds


S2

- Closing of aortic and pulmonic valves

Heart sounds


Murmur (2)

Whooshing sound


- Regurgitation (leaky valve)


- Stenotic (narrowed valve)

Heart sounds


S3

- Systolic failure


- Fails to eject

Heart sounds


S4

- Diastolic failure


- Fails to relax

Wolff–Parkinson–White syndrome (WPW)

- Electrical abnormality


- Abnormal pathway between the atria and ventricles through the bundle of Kent


- Narrow PR interval (<0.12 sec)


- Widened QRS complex

Antiarrhythmic Classification


Class Ia

- Inhibits fast Na channels


- Prolongs repolarization time


- Ex. quinidine, procainamide

Antiarrhythmic Classification


Class Ib

- Inhibits fast sodium channels


- Shortens repolarization time


- Ex. lidocaine

Antiarrhythmic Classification


Class Ic

- Inhibits fast sodium channels


- Repolarization time unchanged


- Ex. flecainide, propafenon

Antiarrhythmic Classification


Class II

- Beta blockers


- Repolarization time unchanged


- Ex. propanolol, atenolol

Antiarrhythmic Classification


Class III

- Markedly prolongs repolarization time, usually by K channel blockade


- Ex. amidarone

Antiarrhythmic Classification


Class IVa

- AV node and calcium channel blocker

- Repolarization time unchanged


- Ex. verpamil, diltiazem


Antiarrhythmic Classification


Class IVb

- Calcium channel openers


- Repolarization time unchanged


- Ex. adenosine

Alpha receptor

- Located in the vessels of the skin, muscles, kidneys, and intestines


- Stimulation causes vasoconstriction of peripheral arterioles

Beta 1

- Located in the cardiac tissue


- Stimulation causes increased HR, conduction, and contractility

Beta 2

- Located in vascular and bronchial smooth muscle


- Stimulation causes vasodilation of peripheral arterioles and bronchodilation

Beta blockers

- Decreases HR, BP


- Slows conduction through the SA and AV nodes


- Decreases force of contraction


- Decreases myocardial O2 consumption

Calcium channel blockers

- Negative inotropic effect


- Slows AV node conduction


- Dilates coronary and peripheral arterioles


- Decreases O2 demand

ACE inhibitors

- Inhibits the conversion of angiotensin I to angiotensin II


- Prevents sodium and water reabsorption


- Prevents vasoconstriction



Numbers method of calculating HR

300, 150, 100, 75, 60, 50

Sinus pause

< 3 sec

Sinus arrest

> 3 sec

Sinus exit block

If the regular rhythm can fit perfectly within the block