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46 Cards in this Set

  • Front
  • Back
How are the microorganisms destroyed?
The cilia push a thin layer of mucus & entrapped particles from the nasal cavity toward the pharynx where the mucus is swallowed
Gastric juices in the stomach destroy the microoganisms in the mucus
Give the location and role of the vocal cords
House in the larynx
2 pairs of horizontal vocal folds extending inward from walls of larynx
Made of muscle tissue, connective tissue, covered by mucous membrane
False vocal cords: Upper folds that do not produce sound (the muscle closes airway during swallowing)
True vocal cords: Lower folds of muscle tissue and elastic fibers. Air forced between the cords cause vibration from side to side/sound waves
What and where is the epiglottis and how does it function?
A flap-like cartilage structure at the back fo the tongue near the entrance to the Trachea
Allows air to enter the Larynx
During swallowing the Larynx rises and the epiglottis presses downward to partially cover the opening into the larynx
Helps food and liquids from entering air passages
Waht is the role of surfacant?
Substance produced by the lungs and secreted into alveolar spaces
Reduces the surface tension within the aveoli
Reduces the alveoli's tendency to collapse, especially when air volume is low
Eases inspiratory efforts to inflate the alveoli
What is the role of atmospheric pressure?
Inhalation: Pressure inside the lungs and alveoli decreases, atmospheric pressure forces are into the airways:
Diaphragm muscles below lungs contract moving downward
Thoracic cavity enlarges
What is a diaphragm?
A sheetlike structure largly composed of skeletal muscle and connective tissue that separates thoracic and abd. cavity
What is the role of the diaphragm?
Inhalation: Diaphragm contracts, moves downward, making space for inflow of air with inspiration

Exhalation: Diaphragm contracts upward, pushing air out with expiration
Intercostal muscles
Inhalation: External intercostal muscles. Between the ribs contract and move downward, decreasing pressure (raises ribs, elvates sternum, enlarges thoracic cav. movethe thoracic wall up and outward, the parietal and visceral pleura move too, lungs expand in all directions
Exhalation: Internal intercostal muscles
Posterior
For forced inhalation
contraction pulls ribs and sternum down & inward, increasing pressure in lungs
What is the role of the abdominal muscles?
Inhalation:
For deeper than normal breath
Pull thoracic cage up and outward, enlarging thoracic cav., decreasing pressure

Exhalation:
Squeeze abd. organs inward and diaphragm upward against lung forcing air out
Increases pressure in the abdominal cavity
Spirometry
Measure respiratory Air Volumes and capacities
Spirometry
Measures respiratory air volumes and capacities
Tidal Volume
TV
500 mL
Volume of air that enters (or leaves) during a single respiratory cycle
Inspiratory Reserve Volume
IRV
3000 mL
Volume that can be inhaled during forced breathing in addition to tidal volume
Expiratory Reserve Volume
ERV
1100 mL
Volume that can be exhaled during force breathing in addition to tidal volume
Residual Volume
RV
1200 mL
Volume that remains in the lungs even after maximal expiration
Vital Capacity
VC
4600 mL
Maximum volume of air that can be exhaled after taking the deepest breath possible
VC=TV+IRV+ERV
Total Lung Capacity
TLC
5800 mL
Total volume of air that the lungs can hold
TLC=VC+RV
Laryngitis
Infection causing mucous membrane of the larynx to become inflamed and swollen
Swelling can obstruct airway and interfere with breathing
Tonsillitis
Infections of tonsils and mucous membranes causing swelling
Can block air passageway interfering with breathing and swallowing to pharynx
Mucous membranes of pharynx, auditory tubes and middle ear are connected and infection can travel too all these areas
Visceral Pleura
Layer of serous membrane attaches to each lung's surgace and folds back to become the parietal pleura
Parietal Pleura
The ouside membrane lines the outside of the lung, the mediastinum and the inner wall of the thoracic cavity
Plural Cavity
The potential space between the visceral and Parietal pleura
Serous Fluid
Lubricates adjacent pleural surfaces, reducing friction as they move against one another during breathing
Holds the pleural membranes together
Name the potential spaces in the pleural membrane
No significat space exists between the visceral and parietal pleura, buthe potential space between them is called the pleural cavity
What hold the visceral and parietal membranes together?
Serous fluid
Air pressure
What are the differences between the right and left lungs?
Right:
Larger, three lobes
Left:
Smaller due to space shared with mediastinum and heart, two lobes
Nose
Frontal Head
on the face superior to mouth
Nasal Cavity
Hollow behind nose
Paranasal sinuses
Air-filled, maxillary, frontal, ethnoid & sphenoid bones of skull
Pharynx
Dorsal of oral cavity, nasal cavity and larynx
Where is the respiratory center? Describe its location in relation to other organs
Two groups of neurons in brain stem extending the length of the Pons and the Medulla Oblongata
Respiratory Center of the Medulla Oblongata
Dorsal Respiratory group:
Stimulates diaphragm muscles of inspiration
Basic rhythm of breathing

Ventral Respiratory Group:
Stimulate Intercostal and abd. muscles
More forceful breathing: some increase inspiration/expiration
Pontine Respiratory Group
Connects with the Medullary Rhythmic Center (rhythm of breathing)
Stimulates diaphragm muscles
What cause the rate of breathing to increase and why?
Repiratory Center: Affects breathing rate and depth, and so do certain chemicals in body fluids, the degree to which lung tissues stretch, and a person's emotional state (typically fear and pain)
Chemo-receptors
(chemo-sensitive areas in the medullary respiratory center)
Senses change in cerebral spinal fluid levels of carbon dioxide or hydrogen ions
Stimulation of these receptors increases breathing rate
Peripheral Chemo-Receptors
(In carotid and aoric bodies)
Sense changes in low blood oxygen
Stimulates increased breathing rate
Inflation reflux
Regulates the depth of breathing when stretched lung tissues stimulate receptors in visceral pleura, bronchioles and aveoli
Prevents over stimulation of lungs during forceful breathing
Emotional Upset
Fear and pain increase breathing rate
What's the role of carbon monoxide and how does it bind with hemoglobin?
Carbon Monoxide bind to hemoglobin preventing oxygen from binding
Starves tissues of oxygen
Emphysema
Progressive degenerative disease
Alveolar wall destroyed: small clusters of air sacs merge thus forming larger chambers, decreases surface area of respiratory membrane where gases are exchanged
Alveolar walls lose elasticity
Capillary networks diminish
Lung Cancer
Uncontrolled division of abnormal cells
Cells rob normal cells of nutrients
Normal cells are crowded out metastisizing secondary cancer in other parts of the body
Cystic Fibrosis
Defect in channels leading from glands
Causes formation of extremely thick sticky mucous in lung
Encourages infectinos by micro-organisms not otherwise common in lungs
Athsma
A chronic (long term) lung disease that inflames and narrows the airways
Muscles react and tighten making breathing difficult
The swelling also can worsen, making the airways even narrower
Cells in airways may make more stick, thick mucous further narrowing the airways
Bronchitis
an acute inflammation of the air passages within the lung
Trachea and bronchi within the lungs become inflamed due to infection
The thin mucous lining of airways become irritated and swollen
Chronic Pulmonary Obstructive Disease
Progressive disease the makes it hard to breathe
Less air flows in and out of the airways because of one or more of the following: The airways and air sacs lose their elastic quality, the walls between many of the air sacs are destroyed, become thick and inflamed (swollen), the airways make more mucous than usual, clogging the airways
Causes: Cigarette smoking is the leading cause, Long-term exposure to lung irritants, such as air pollution, chemical fumes, or dust
Book references
Know the alveoli & caopillaries pictured on pg. 450, fig. 16-8, 451 16.10,
p. 453 fig. 16.12
p. 457 fig 16.15
p. 446 fig 16.2