The aging brain goes through a multitude of alterations in neuronal functioning which in turn results in the worsening in motor and cognitive abilities (Willis et al., 2008). Dementia, more specifically, Alzheimer’s disease results in the deterioration of language, memory and the ability to reason in aging individuals over the course of 5 to 20 years (Muthaiyah et al., 2014). The changes that occur neuropathologically with AD evolve before symptoms are shown in individuals who are affected (Muthaiyah et al., 2014). The weakening of the brain through age encompasses a variety of linked processes, modern treatments have targeted numerous facets of neurodegeneration all together (Willis et al., 2008). A modern treatment that has shown potential
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For example, one study examined three varying doses of walnuts (2, 6, and 9%) for effectiveness in reversing age-sensitive deficiencies in motor ability as well as cognitive ability (MWM) in Fisher 344 rats (Willis et al., 2008). Results suggested that moderate consumption of walnuts in aged rats could reverse the outcomes on cognitive as well as motor performance due to age (Willis et al., 2008). Additionally, discoveries from a laboratory proposed that cognitive ability in aged rats enhanced through the consumption of walnuts (6%) in diets that were equal to the suggested intake of 1oz/day for humans (Poulose et al., 2012). Intakes of 6% or 9% walnuts in diets (equal to 1 oz or 1.5 oz walnuts intake each day for humans) remediated memory loss, enhanced motor skills along with learning, as well as refined anxiety behaviors in AD mouse model (Muthaiyah et al., 2014). Performance was fairly alike to the control diet Wt mice (Muthaiyah et al., 2014).
In this study, I examined how properties of walnuts in general (without targeting a specific phytochemical) can reverse or improve memory loss in C57BL/6J control model mice with c.548GTKI strain. I studied three doses of walnuts, corn, and a mixture of both corn and walnuts (3g walnuts, 3g corn, 1.5/1.5 walnuts & corn). I hypothesized that consumption of walnuts reduces memory loss in dementia mouse model. Consumption of walnuts reduces memory in dementia mouse model, which in turn means that mice fed a diet including walnuts complete the maze sooner than mice fed a control
For example, one study examined three varying doses of walnuts (2, 6, and 9%) for effectiveness in reversing age-sensitive deficiencies in motor ability as well as cognitive ability (MWM) in Fisher 344 rats (Willis et al., 2008). Results suggested that moderate consumption of walnuts in aged rats could reverse the outcomes on cognitive as well as motor performance due to age (Willis et al., 2008). Additionally, discoveries from a laboratory proposed that cognitive ability in aged rats enhanced through the consumption of walnuts (6%) in diets that were equal to the suggested intake of 1oz/day for humans (Poulose et al., 2012). Intakes of 6% or 9% walnuts in diets (equal to 1 oz or 1.5 oz walnuts intake each day for humans) remediated memory loss, enhanced motor skills along with learning, as well as refined anxiety behaviors in AD mouse model (Muthaiyah et al., 2014). Performance was fairly alike to the control diet Wt mice (Muthaiyah et al., 2014).
In this study, I examined how properties of walnuts in general (without targeting a specific phytochemical) can reverse or improve memory loss in C57BL/6J control model mice with c.548GTKI strain. I studied three doses of walnuts, corn, and a mixture of both corn and walnuts (3g walnuts, 3g corn, 1.5/1.5 walnuts & corn). I hypothesized that consumption of walnuts reduces memory loss in dementia mouse model. Consumption of walnuts reduces memory in dementia mouse model, which in turn means that mice fed a diet including walnuts complete the maze sooner than mice fed a control