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78 Cards in this Set

  • Front
  • Back
• Tooth #3 has a history of spontanteous pain that lasted three weeks, then subsided for several months. Teeth #2, 4, 5 & 6 are normally responsive to cold, percussion and palpation. #3 is sensitive to percussion and palpation but nonresponsive to cold. A periapical view of the area shows an intact lamina dura on all teeth. The diagnosis for this presentation is:
1. ? Irreversible pulpitis with acute apical periodontitis
2. ? Necrotic pulp with acute apical periodontitis
3. ? Reversible pulpitis with acute apical periodontitis
4. ? Healthy pulp with acute apical periodontitis
2. ? Necrotic pulp with acute apical periodontitis
• In pulpal and periapical disease, the occurrence of pain:
1. ? Is the common presentation
2. ? Is the exception rather than the rule
3. ? Is diagnostic of an irreversible pulpitis
4. ? Indicates nonodontogenic etiologies
2. ? Is the exception rather than the rule
• If a patient reports sharp radiating pain that is diffuse it is likely not the result of:
1. ? A neuralgia
2. ? Acute exacerbation of chronic apical periodontitis
3. ? An irreversible pulpitis
4. ? A cracked tooth
2. ? Acute exacerbation of chronic apical periodontitis
• Clinical sign and symptoms will correlate with histology in pulpal disease.
1. ? True
2. ? False
2. ? False
• A negative response to vitality tests indicates:
1. ? Pulpal necrosis
2. ? Pulpal hyperemia
3. ? Acute apical periodontitis
4. ? Lesions of nonendodontic origin
1. ? Pulpal necrosis
The correct terminology for a channel that forms between an area of pathology and an anatomic space is a:
1. ? Fistula
2. ? Perforation
3. ? Operculum
4. ? Sinus tract
4. ? Sinus tract
• The radiographic view most often employed in endodontic diagnosis and treatment is the:
1. ? Bitewing view
2. ? Water's view
3. ? Occlusal view
4. ? Periapical view
4. ? Periapical view
Pulpal diagnosis that fall under the category of "Vital Pulp" include:
1. ? Reversible pulpitis
2. ? Irreversible pulpitis
3. ? Necrotic pulp
4. ? A & B
4. ? A & B
• When the patient presents with multiple teeth that are painful the clinician should consider:
1. ? That multiple teeth need root canal therapy
2. ? Conditions that affect multiple teeth
3. ? Conditions that cause referred pain
4. ? B & C
4. ? B & C
A standard method for determination of the origin of a patent sinus tract is:
1. ? A periapical film
2. ? Periodontal probing
3. ? Threading of the sinus tract with a gutta percha point and making a radiograph
4. ? Percussion
3. ? Threading of the sinus tract with a gutta percha point and making a radiograph
2. Referred pain from a pulpitis:
A. ? Can affect teeth in the same quadrant
B. ? Can affect teeth in the opposing quadrant
C. ? Never crosses the midline
D. ? All of the above
D. ? All of the above
The vitality test chosen for a particular case should aim to:
A. ? Reproduce the chief complaint
B. ? Inflict pain
C. ? Avoid what hurts the patient the most
D. ? Stimulate nerve endings in the Plexus of Raschow
A. ? Reproduce the chief complaint
5. A negative response to vitality tests indicates:
A. ? Pulpal necrosis
B. ? Pulpal hyperemia
C. ? Acute apical periodontitis
D. ? Lesions of nonendodontic origin
A. ? Pulpal necrosis
8. Standardized sensor/film holders are used for initial and final radiographs because they:
A. ? Are easier to use
B. ? Maintain the same angulation each time
C. ? Minimize radiation to the patient
D. ? Fit with the rubber dam better
B. ? Maintain the same angulation each time
11. A reversible pulpitis does not demonstrate:
A. ? Thermal sensitivity
B. ? An onset related to a recent restoration
C. ? Association with a carious lesion
D. ? True spontaneous pain
D. ? True spontaneous pain
15. Percussion sensitivity in a tooth indicates:
A. ? An irreversible pulpitis
B. ? Focal abscesses in the pulp
C. ? Inflammation in the periodontal ligament space
D. ? A ranula
C. ? Inflammation in the periodontal ligament space
16. Palpation sensitivity indicates:
A. ? A sinus tract
B. ? Inflammation of the periosteum
C. ? A reversible pulpitis
D. ? Chronic apical periodontitis
B. ? Inflammation of the periosteum: Correct! Yes- it indicates that the disease process has eroded the facial cortical plate and the inflammation has spead into the overlying periosteum
19. Referred pain from a pulpitis:
A. ? Can affect teeth in the same quadrant
B. ? Can affect teeth in the opposing quadrant
C. ? Never crosses the midline
D. ? All of the above:
D. ? All of the above: Each of these points is true- referred pain can make teeth in the same and opposing quadrants feel painful but it will not cross the midline.
20. Heat sensitivity indicates:
A. ? Dentin is exposed in an otherwise healthy tooth
B. ? Sensitization of the deeper C-fibers of the pulp
C. ? Referred pain
D. ? Sinusitis
B. ? Sensitization of the deeper C-fibers of the pulp
21. Thermal tests for vitality elicit a reponse by:
A. ? Stimulating electrical nerve impulses in the Plexus of Rashcow
B. ? Inducing dentinal fluid flow that stimulates nerve endings in the odontoblast layer
C. ? Conducting impulses through the dentinal tubules
D. ? Causing vascular contraction
B. ? Inducing dentinal fluid flow that stimulates nerve endings in the odontoblast layer
23. Chronic apical periodontitis is:
A. ? Symptomatic
B. ? Associated with a vital tooth
C. ? Always associated with a sinus tract
D. ? A radiographic diagnosis
D. ? A radiographic diagnosis
26. periodontal defect can be differentiated from a sinus tract draining through the sulcus by:
A. ? Vitality tests and probing
B. ? Threading of the tract with a gutta percha point
C. ? Palpation
D. ? Percussion
A. ? Vitality tests and probing: If not previously root canal treated vitality tests should be positive; Probing period defects are often broad where sinus tracts are narrow
• The primary function of pulpal dendritic cells is to:
1. ? Phagocytize foreign antigen that makes it to the pulp
2. ? Produce the organic matrix of dentin
3. ? Give rise to fibroblasts or odontoblasts
4. ? Present antigen and initiate an immune response
4. ? Present antigen and initiate an immune response
Dentinal tubules:
1. ? Form "Z" structures as they course pulpally
2. ? Are up to 10 microns in diameter
3. ? Are found 8000 to 57,000 per mm2
4. ? Have no lateral branches
3. ? Are found 8000 to 57,000 per mm2
The first protective response to dentinal irritation is:
1. ? Dentinal sclerosis
2. ? Reparative dentin
3. ? Reactionary dentin
4. ? Dead tract formation
1. ? Dentinal sclerosis
Reparative dentin is formed by:
1. ? Resident odontoblasts
2. ? Endothelial cells
3. ? Newly differentiated odontoblasts
4. ? Perivascular cells in the coronal pulp
3. ? Newly differentiated odontoblasts
• The pain of an irreversible pulpitis qualifies as:
1. ? Allodynia
2. ? Hyperalgesia
3. ? Allodynia and hyperalgesia
4. ? Neither allodynia nor hyperalgesia
3. ? Allodynia and hyperalgesia
• Allodynia is defined as a(n):
1. ? Reduction in the pain threshold so that previously non-noxious stimuli are perceived as painful
2. ? Increasse in the perceived magnitude of a painful stimulus
3. ? Increase in the pain threshold so that perviously non-noxious stimuli are perceived as painful
4. ? Decrease in the perceived magnitude of a painful stimulus
1. ? Reduction in the pain threshold so that previously non-noxious stimuli are perceived as painful
• Hyperalgesia is due to:
1. ? Peripheral mechanisms
2. ? Central mechanisms
3. ? Referred pain
4. ? Peripheral and central mechanisms
4. ? Peripheral and central mechanisms
• Dentinal tubules are inverted cones with the largest diameter:
1. ? In the middle of the tubule
2. ? At the dentino enamel junction
3. ? On the dentin pulp border
4. ? Occurring alternately on pulpal or enamel sides
3. ? On the dentin pulp border
• The odontoblastic layer is located immediately subjacent to the predentin. It contains:
1. ? Cell bodies of fibroblasts
2. ? Dentritic cells
3. ? Cementum
4. ? Ameloblasts
2. ? Dentritic cells
• Which of the following nerve fibers are thought to be primarily responsible for mediating dentinal hypersensitivity?
1. ? A alpha fibers
2. ? A gamma fibers
3. ? A delta fibers
4. ? C fibers
3. ? A delta fibers
1. Odontoblasts are:
A. ? Post-mitotic cells
B. ? Responsible for sensory input in dentin
C. ? Only responsible for dentinogenesis and are therefore not needed after tooth development is complete
D. ? Formed from the inner enamel epithelium
A. ? Post-mitotic cells: if one dies a progenitor cell must migrate into the area and differentiate into an odontoblast to replace it
2. The granular layer of root dentin found in the root adjacent to the cementum is called:
A. ? Epithelial rests of Mallaisez
B. ? The hyalinized layer of Marsh
C. ? The Granular Layer of Tomes
D. ? Contour Lines of Owen
C. ? The Granular Layer of Tomes: This is also technically secondary dentin
6. Peritubular dentin is more mineralized than intertubular dentin.
A. ? True
B. ? False
A. ? True
9. Pulp lymphatics have not been demonstrated in the dental pulp.
A. ? True
B. ? False
B. ? False
11. A delta fibers are:
A. ? Located peripherally in the pulp
B. ? Located centrally in the dental pulp
C. ? Provide sympathetic innervation to the pulpal vasculature
D. ? Responsisble for dull throbbing pain
A. ? Located peripherally in the pulp: They are concentrated in the Plexus of Raschow and are responsible for the sharp pain caused by an air blast over vital dentin
15. Lateral branches of neuronal bundles in the dental pulp end in a dense plexus of nerves referred to as the:
A. ? Solar plexus
B. ? A-delta afferent plexus
C. ? Plexus of Raschow
C. ? Plexus of Raschow
16. Myelinated nerve fibers in the dental pulp:
A. ? Conduct the action potential slower than unmeylinated nerves
B. ? Conduct the action potential in the same manner as unmyelinated nerves
C. ? Conduct the action potential faster than unmyelinated nerves
D. ? Are predominantly A-beta fibers
? Conduct the action potential faster than unmyelinated nerves
17. Arterio-venous anastomoses and Arterio-venious shunts are pulpal vascular configurations that:
A. ? Predispose the pulp to necrosis secondary to inflammation
B. ? Allow compartmentalization of tissue pressure increases during inflammation
C. ? Perfuse odontoblasts in the pulp horns
D. ? Collapse in response to increase intrapulpal pressure
B. ? Allow compartmentalization of tissue pressure increases during inflammation: Tissue pressure increases if unchecked could result in collapse and hypoxia with certain necrosis for the pulp because it exists in non-compliant hard tissue ; A-V shunts direct blood away from areas of high tissue pressure;
The major difference between 5.25% sodium hypochlorite (NaOCl) and 0.5 % sodium hypochlorite is
1. ? 5.25% NaOCl dissolves vital tissue and 0.5% does not
2. ? 0.5% NaOCl is not bactericidal
3. ? 0.5% NaOCl does not dissolve necrotic tissue
4. ? nothing - they perform the same
1. ? 5.25% NaOCl dissolves vital tissue and 0.5% does not
• Clinical therapies for dentin hypersensitivity center on
1. ? anesthetizing the irritated nerves
2. ? raising the firing threshold of A delta fibers
3. ? decreasing the firing threshold of A delta fibers
4. ? occluding the exposed dentinal tubules
4. ? occluding the exposed dentinal tubules
• Pulpectomy is the
1. ? removal of all pulp tissue from the root canal system
2. ? amputation of the coronal pulp
3. ? treatment of choice for a necrotic tooth
4. ? none of the above
1. ? removal of all pulp tissue from the root canal system
The appropriate vital pulp therapy for a complicated crown fracture in an immature permanent tooth is
1. ? partial pulpotomy (Cvek Pulpotomy)
2. ? pulpectomy
3. ? pulp cap
4. ? indirect pulp cap
1. ? partial pulpotomy (Cvek Pulpotomy)
Apexigenesis is the formation of the root end by medicament stimulation.
1. ? True
2. ? False
2. ? False
The quoted success rate for a Cvek pulpotomy is
1. ? 60%
2. ? 70%
3. ? 90%
4. ? 100%
3. ? 90%
• One significant aspect of bacterial penetration into the dentinal tubules in the infected root canal is that
1. ? they become entombed by the filling material and die
2. ? proper disinfection of the root canal must involve removal of infected dentin and not just surface disinfection
3. ? bacterial titers decrease in the main canal
4. ? apical periodontitis regresses
2. ? proper disinfection of the root canal must involve removal of infected dentin and not just surface disinfection
• Evidence of dentin bridge formation on a radiograph is a guarantee of success for a Cvek pulpotomy and is the goal of this therapy.
1. ? True
2. ? False
2. ? False
• Treatment of an irreversible pulpitis relies on
1. ? maintenance of asepsis
2. ? the right type of file system
3. ? disinfecting irrigants like sodium hypochlorite
4. ? the antibacterial capacity of chlorhexidine
1. ? maintenance of asepsis
2. The major goal of vital pulp therapy is to
A. ? prevent pain
B. ? retain as much tooth structure as possible
C. ? prevent apical periodontitis
D. ? save the patient money
C. ? prevent apical periodontitis
5. Requirement(s) for success in pulp capping is (are)
A. ? an uninflamed pulp
B. ? bacteria-tight coronal seal
C. ? an appropriate pulp capping material like calcium hydroxide or mineral trioxide aggregate (MTA)
D. ? all of the above
D. ? all of the above
8. Rubber dam isolation of a tooth during root canal therapy
A. ? is the standard of care
B. ? protects the patient's airway
C. ? prevents contamination of the canals with saliva
D. ? all of the above
D. ? all of the above
10. Complete pulpotomy is recommended in permanent teeth only in the case of
A. ? an elderly patient with calcified canals
B. ? a cariously exposed molar with immature apices
C. ? a necrotic pulp
D. ? acute apical abscess formation
B. ? a cariously exposed molar with immature apices
12. One significant aspect of bacterial penetration into the dentinal tubules in the infected root canal is that
A. ? they become entombed by the filling material and die
B. ? proper disinfection of the root canal must involve removal of infected dentin and not just surface disinfection
C. ? bacterial titers decrease in the main canal
D. ? apical periodontitis regresses
B. ? proper disinfection of the root canal must involve removal of infected dentin and not just surface disinfection
14. Treatment refractory cases in endodontics have been found to harbor Streptococcus faecalis in 30% of the cases sampled.
A. ? True
B. ? False
A. ? True: Nasty bugs and they are resistant to calcium hydroxide alone
16. Inflammation secondary to a reversible pulpitis is effectively and most appropriately treated by
A. ? root canal therapy
B. ? extraction
C. ? removal of the irritating stimulus
D. ? direct pulp capping
C. ? removal of the irritating stimulus
18. In a traumatically-exposed pulp, the top 1-2 mm of tissue is removed because
A. ? more extensive removal of the pulp is painful
B. ? young patients are difficult to control
C. ? it will extrude beyond the wound surface
D. ? inflammation does not extend beyond this level
D. ? inflammation does not extend beyond this level
19. A complicated crown fracture is
A. ? a fracture that involves the crown and the root
B. ? a fracture that involves enamel and dentin and exposes the pulp
C. ? a fracture that involves enamel and dentin but does not expose the pulp
D. ? a fracture that causes loss of the tooth
B. ? a fracture that involves enamel and dentin and exposes the pulp
20. Hard setting calcium hydroxide is preferred as a pulpotomy dressing in Cvek pulpotomies because the zone of necrosis is thinner and the dentin bridge forms more coronally than with calcium hydroxide paste.
A. ? True
B. ? False
A. ? True: The zone of necrosis with calcium hydroxide paste is wider and the resultant bridge forms more apically making vitality testing difficult
• Reactionary dentin is made by:
1. ? Resident odontoblasts
2. ? Newly differentiated odontoblasts
3. ? Fibroblasts
4. ? Undifferentiated mesenchymal cells
1. ? Resident odontoblasts
Dentin sclerosis occurs:
1. ? After tertiary dentinogenesis is completed
2. ? As a result of the precipitation of mineral crystals in the tubule lumen
3. ? As a result of deposition of peritubular dentin
4. ? B & C
4. ? B & C
• Interglobular dentin is that dentin formed:
1. ? Between the dentinal tubules
2. ? In response to a strong stimulus
3. ? In the apical 1/3 of the root
4. ? In response to mild but persistent stimuli
4. ? In response to mild but persistent stimuli
• A superfamily of growth factors known to play a central role in all forms of dentinogenesis is the:
1. ? Angiogenic growth factor family
2. ? Platelet derived growth factor family
3. ? Angiotensin family
4. ? Transforming growth factor family
4. ? Transforming growth factor family
• Bacteria cannot typically invade the dentinal tubules because of size exclusion.
1. ? True
2. ? False
2. ? False
• TGF-beta is a growth factor known to be involved in tertiary dentin formation. One source for this factor in tertiary dentinogenesis is:
1. ? Perivascular cells in the pulp
2. ? Schwann cells on A-delta fibers
3. ? Monocytes and macrophages in the odontoblastic layer
4. ? Degraded dentin matrix
4. ? Degraded dentin matrix
• The neurogenic response to caries includes:
1. ? The release of neuropeptides
2. ? Depolarization of sensory afferents
3. ? Nociceptive input
4. ? Phagocytosis
1. ? The release of neuropeptides
• Tertiary dentin is:
1. ? Physiologic dentin
2. ? Always atubular and fibrous
3. ? Highly permeable
4. ? Further divided into reactionary and reparative dentin
4. ? Further divided into reactionary and reparative dentin
• In carious dentin, the highest proportion of bacteria are:
1. ? Gram (+) Rods
2. ? Gram (+) cocci
3. ? gram (-) cocci
4. ? gram (-) rods
1. ? Gram (+) Rods
• A histologic examination of pulps under dentinal carious lesions shows:
1. ? Nerve sprouting in response to the impinging infection
2. ? Neuronal necrosis
3. ? Little to no inflammation
4. ? A decrease in the numbers of dendritic cells
1. ? Nerve sprouting in response to the impinging infection
18. Pulpal inflammation in response to caries is:
A. Chronic until there is a near or frank exposure and then it becomes acute (why even large lesions can be relatively asymptomatic)
19. Reparative dentin is a reaction to a strong stimulus that is cytocidal.
A. True (made by newly differentiated odontoblasts that are replacing killed odontoblasts)
21. Interglobular dentin is:
A. A form of reparative dentin that has inclusions of organic material, is disorganized and more permeable than primary dentin
22. During caries progession, if the odontoblast is quickly killed, the result can be:
A. Dead tracts (note: can’t kill the odontoblast and still get reactionary dentinogenisis, and must have a vital odontoblast to mediate dentinal sclerosis)
24. The most common etiologic factor for pulpal and periapical disease is:
A. Caries
25. Reactionary dentin is made by:
A. Resident odontoblasts "reacting" to a stimulus
28. The humoral response of the pulp to caries includes:
A. Odontoblast expression of chemokines (a scientist here at UNC-CH (that would be Dr. L) showed that odontoblasts make IL-8, a potent chemitactic agent for neutrophils)
30. Due to the noncompliant environment of the dental pulp, it deals with the increased tissue pressures of inflammation by:
A. Shunting of blood away from inflammed areas (A-V shunts and arteriovenous anastomoses allow inflammation to be compartmentalized in the dental pulp)
35. Lateral branches of neuronal bundles in the dental pulp end in a dense plexus of nerves referred to as the:
A. Plexus of Raschow