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83 Cards in this Set

  • Front
  • Back
What is CHRONIC HYPERPLASTIC PULPITIS?
Unique pattern of pulpal inflammation where the pulp tissue reacts to injury by undergoing hyperplasia
What is the etiology of PULP POLYP?
- open chronic pulpitis
- ample blood supply
- increased regenerative capacities of young pulpal tissue
PULP POLYPS are generally found in which teeth?
Molars of children/ young adults
What is the treatment for PULP POLYP?
- endodontic therapy
- extraction
Occurence of PULPAL CALCIFICATIONS increases with what two factors?
- aging
- trauma/caries
What are PULP STONES?
Spherical calcifications formed within the coronal portions of the pulp.
Prominent PULPAL CALCIFICATIONS have been associated with what two conditions?
- Dentin Dysplasia Type II
- Ehlers Danlos Syndrome
What is the treatment for PULPAL CALCIFICATIONS?
No treatment required
What is a PERIAPICAL ABCESS?
accumulation of acute inflammatory cells at the apex of a non-vital tooth
What is the etiology of a PERIAPICAL ABCESS?
- often associated with a non-vital tooth
- may be symptomatic or asymptomatic
- spreads along path of least resitance
What is a PARULIS?
- gum boil
- an intraoral opening of a sinus tract that often presents as an erythematous mass of subacutely inflamed granulation tissue
What is the treatment for a PERIAPICAL ABCESS?
- drainage
- elimination of the focus of infection: reduction of occlusion, analgesic and Abx, endodontic therapy
What is a PERIAPICAL GRANULOMA?
a mass of chronically inflamed granulation tissue at the apex of a nonvital tooth
PERIAPICAL GRANULOMA can arise from which two conditions?
- initial periapical pathosis
- progression of a periapical abcess
What are the radiographic features of PERIAPICAL GRANULOMA?
- radiolucency with loss of apical lamina dura
- well circumscribed or ill defined
- root resorption may occur
What is a complication of PERIAPICAL GRANULOMA?
may transform into a periapical cyst
What is a PERIAPICAL CYST?
Inflammation stimulates epithelium at the apex of a nonvital tooth to form a true epithelium lined cyst
What are the two etiological sources of PERIAPICAL CYSTS?
- source of epithelium
- source of inflammation
What are the epithelial sources of PERIAPICAL CYST?
- usually rest of Malassez
- crevicular epithelium
- sinus lining
- epithelial lining of fistula tract
What are the inflammatory sources of PERIAPICAL CYST?
- peridontal disease
- pulpal necrosis with spread through lateral foramen
What is a complication with PERIAPICAL CYST?
Rarely squamous cell carcinoma has been reported within them.
What is a FIBROUS PERIAPICAL SCAR?
Defect created by the periapical lesion is filled with dense fibrous tissue instead of bone (when facial and lingual cortical plates have been lost)
What is OSTEOMYELITIS?
Acute or Chronic inflammatory process in the medullary spaces or cortical surfaces of bone that extend away from the initial site of involvement.
What majority of OSTEOMYELITIS cases are caused by what?
Bacterial infection.
What is ACUTE SUPPURATIVE OSTEOMYELITIS?
Acute inflammation process spreads through the medullary spaces of the bone. There is insufficient time for the body to react to the presence of the inflammatory infiltrate.
What conditions pre-dispose people to OSTEOMYELITIS?
- chronic systemic diseases
- immunocompromised status
- disorders associated with decreased vascularity of bone
Most cases of ACUTE SUPP. OSTEOMYELITIS arise after what two events?
- odontogenic infections
- traumatic fracture of the jaws
Which two bacteria are most frequently involved in cases of OSTEOMYELITIS?
Staphylococci
Streptococci
What are the signs and symptoms of ACUTE SUPP. OSTEOMYELITIS?
- pain
- fever
- lymphadenopathy
- leukocytosis
- significant sensitivity
- soft tissue swelling
- less than 1 MONTH duration
Define SEQUESTRUM"
drainage or exfoliation of a fragment of necrotic bone
Define INVOLUCRUM:
When a fragment of necrotic bone becomes surrounded by vital bone
What are the radiographic features of ACUTE SUPP. OSTEOMYELITIS?
- significant resorption of trabecular bone
- diffusely blotchy area
- motted with indistinct margins
- islands of residual bone (sequestra)
What is the treatment of ACUTE SUPP. OSTEOMYELITIS?
- surgical intervention to establish drainage and remove sequestra
- use of antibiotics
-
What is CHRONIC SUPP. OSTEOMYELITIS?
Defensive response leads to the production of granulation tissue which subsequently forms dense scar in attempt to wall off the infected area. Encircled dead space acts as a reservoir for bacteria, and antibiotics can't reach the site. Begins to evolve 1 month after spread of infection.
What are the two etiological patterns of CHRONIC SUPP. OSTEOMYELITIS?
- sequela of acute osteomyelitis
- low grade inflammatory reaction that never went through an acute phase
Which area is affected more frequently in cases of OSTEOMYELITIS?
Mandible
What are the radiographic features of CHRONIC OSTEOMYELITIS?
patchy, ragged, ill-defined radiolucency often with central radiopaque sequestra
What is the treatment for CHRONIC SUPP. OSTEOMYELITIS?
- removal of all infected and necrotic tissue mandatory
- antibiotics but IV in high doses
- hyperbaric oxygen in difficult cases to stimulate vascularization, collagen synth and osteogenesis
- weakened jaw bones must be immobilized
What is DIFFUSE SCLEROSING OSTEOMYELITIS?
obvious infection process directly is responsible for sclerosis of bone
- increased radiodensity develops around sites of chronic infection such as periodontisis, pericoronitis, and apical inflammatory disease.
What is CONDENSING OSTEITIS?
A focal bony reaaction to low-grade inflammatory stimulus, seen at the apex of a tooth with long-standing pulpitis or pulpal necrosis
CONDENSING OSTEITIS occurs most frequently in...
- children and young adults
- at apices of Mn premolars and molars
What are the radiographic features of CONDENSING OSTEITIS?
localized, uniform zone of increased radiodensity adjacent to the apex of a tooth that exhibits a thickened pariodontal ligament space or apical inflammatory lesion.
What is the treatment for CONDENSING OSTEITIS?
- resolution of odontogenic infection, endo or extraction
What is a BONE SCAR?
residual area of condensing osteitis which remains after resolution of inflammatory focus
What is OSTEOMYELITICS WITH PROLIFERATIVE PERIOSTITIS?
Periosteal reaction to presence of inflammation
What is the most common cause of GARRE's OSTEOMYELITIS?
dental caries associated with periapical inflammatory disease
An open apex in CHRONIC HYPERPLASTIC PULPITIS can help reduce what?
Pulpal necrosis
What things have been associated with increased frequency of OSTEOMYELITITS?
- tobacco use
- alcohol abuse
- IV drug abuse
- diabetes mellitus
- malnutrition
- AIDS
- Radiation
Define "BONE SCAR":
Residual area of condensing osteitis which remains after resolution of inflammatory focus
How long does bone remodelling normally take after OSTEOMYELITIS?
6-12 months
Identify the inflammatory lesions:
Identify the inflammatory lesions:
CHRONIC HYPERPLASTIC PULPITIS (pulp polyp)
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CHRONIC HYPERPLASTIC PULPITIS (pulp polyp)
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CHRONIC HYPERPLASTIC PULPITIS (pulp polyp)
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CHRONIC HYPERPLASTIC PULPITIS (pulp polyp)
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CHRONIC HYPERPLASTIC PULPITIS (pulp polyp)
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PULP STONES
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PULP STONES
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PULP STONES
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PULP STONES
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PARULIS
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PARULIS
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PARULIS
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PERIAPICAL GRANULOMA
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PERIAPICAL GRANULOMA
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PERIAPICAL GRANULOMA
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CONDENSING OSTEITIS
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CONDENSING OSTEITIS
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CONDENSING OSTEITIS
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CONDENSING OSTEITIS
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CONDENSING OSTEITIS
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BONE SCAR