• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/43

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

43 Cards in this Set

  • Front
  • Back
ADPKD

Associated with:
Intracranial aneurysms (15%)
Hepatic (75%), pancreatic (10%), splenic, epididymis, seminal vesicle cysts
Same risk of RCC as general population
Presents 3rd to 4th decades
Progress to renal failure, requires dialysis
TS with AML/LAM

Associated with:
AML in 80% of TS patients (bilateral, multifocal)
Renal cysts
CNS: giant cell astrocytoma, subependymal/cortical tubers
LAM (a/w chylous effusion), cardiac rhabdomyoma
Adenoma sebaceum, shagreen patches

Risk of hemorrhage (> 4cm), treat with arterial embolization
Triad:
Adenoma sebaceum, mental retardation, seizures
XGP

Types: diffuse and focal
DDX: RCC
Secondary to chronic UTI (Proteus, E. coli)
Features:
Renal enlargement
Dilated calyces, thin cortex
Staghorn calculi (renal pelvis)
US: loss of corticomedullary differentiation

Stones: composed of magnesium ammonium phosphate (struvite)  sequelae of UTI
Proteus mirabilis: MC organism associated with urolithiasis
Papillary necrosis

Result from ischemia of the medulla, results in papillary necrosis

DDX (POSTCARD):
Pyelonephritis, obstruction, SCD, TB, cirrhosis/pancreatitis, analgesics (phenacetin), RVT, DM
Unilateral process: POTR

Features: 
Sloughing
Papillary necrosis

Result from ischemia of the medulla, results in papillary necrosis

DDX (POSTCARD):
Pyelonephritis, obstruction, SCD, TB, cirrhosis/pancreatitis, analgesics (phenacetin), RVT, DM
Unilateral process: POTR

Features:
Sloughing of papilla
Lobster claw deformity
Signet ring sign

Forniceal abnormalities (arrows)
Sloughed papilla (arrow)

DDX extracalyceal contrast

Papillary necrosis
Medullary sponge kidney
Renal tuberculosis,
Calyceal diverticulum
Post-obstructive atrophy
Congenital megacalyx
Adrenal adenoma

Intracellular lipid not present in metastasis, pheochromocytoma, carcinoma

CT: < 10 HU virtually diagnostic
Enhance vigorously, rapid washout (slower washout in metastasis)
Adrenal adenoma: washout > 50% contrast on 15-minute delayed

MR: loss of signal on opposed-phase imaging
Retroperitoneal fibrosis

Etiology:
Idiopathic (Ormond’s)
Secondary RPF: medication (methysergide), radiation, hematoma

Features:
Encases aorta and IVC, level L4-5
Ureteral obstruction, medial deviation of ureters

DDX:
Lymphoma: ST proliferation behind the aorta (elevates aorta)
Oxalosis

Case findings:
Coarse medullary nephrocalcinosis (MC finding)

Hereditary hyperoxaluria:
Rare AR, diffuse oxalate deposit in the kidneys, heart, blood vessels, lungs, spleen, and bone marrow
Cortical nephrocalcinosis

Secondary hyperoxaluria:
Associated with disturbances of bile acid metabolism

Medullary nephrocalcinosis

Skeletal deossification
Primary hyperparathyroidism
Paraneoplastic syndromes associated with lung and renal carcinomas (ectopic parathyroid hormone production)
Metastatic carcinoma to bone
Multiple myeloma
Cushing’s syndrome/steroid therapy

Increased absorption of calcium from GI tract
Sarcoidosis
Hypervitaminosis D
Milk alkali syndrome

Hereditary causes
RTA type 1 (distal convoluted tubules)
Hyperoxaluria (95% of cases)
Medullary sponge kidney (renal tubular ectasia)

Cortical nephrocalcinosis

Chronic membranous glomerulonephritis
Acute cortical necrosis
Congenital oxalosis (5% of cases)
Alport’s syndrome: deafness and hereditary nephritis
Rejected renal transplants
Unilateral delayed/persistent nephrogram

DDX:
Renal artery stenosis
Renal vein thrombosis
Urinary obstruction
Hypotension
Schistosomiasis

Case findings:
Diffuse bladder wall thickening with some irregularity
Curvilinear calcification of the bladder wall

MC in Africa and Middle East
Schistosoma haematobium: trematode (fluke) family
Increased risk of squamous cell carcinoma of the bladder
Classic: calcification of bladder and ureters with possible strictures and obstruction
DDX bladder wall thickening and calcification:
TB: involvement of the kidneys prior to the ureters and bladder making differentiation possible
Other chronic infection, including parasites
Post-radiation cystitis
Post-cyclophosphamide cystitis
Transitional cell and squamous cell carcinoma (rare)
Renal lymphangiectasia

Case findings:
US:
Anechoic multiloculated collections containing thin septae surrounding both kidneys
Echogenicity of the renal parenchyma is normal without evidence of focal renal mass, shadowing renal calculus, or hydronephrosis
CT:
Bilateral perirenal fluid collections with attenuation measurements consistent with fluid
Kidneys are well perfused and there is no delay in function

DDX:
Urinoma
Seroma
Perinephric abscess
Other names: renal lymphangiomatosis, renal lymphangioma, peripelvic lymphangiectasia, and renal peripelvic multicystic lymphangiectasia
Rare disorder of the lymphatic system that occurs in all ages
Characterized by ectatic perirenal, peripelvic, and intrarenal lymphatic vessels
MC bilateral
Associated with: renal venous thrombosis, hypertension
Diagnosis: aspiration of chylous fluid
Cervical stenosis

Case findings:
Uterus is enlarged and endometrial cavity is distended with low density fluid
Fallopian tubes are distended with fluid of the same density

Hematometra: accumulation of blood in the uterus
Hematosalpinx: accumulation of the blood in the fallopian tubes
Hematocolpos: accumulation of blood in the vaginal vault
Renal cell carcinoma

Case findings:
IVP: no function of right kidney with a partially visible right renal mass
CT: large, partially necrotic, enhancing mass in the right kidney

DDX low-density or necrotic renal mass:
RCC, non-Hodgkin’s lymphoma
Abscess
Angiomyolipoma
Oncocytoma
Metastatic disease
Renal Adenoma
Complex renal cyst, renal infarct
Sarcoma, TCC

Renal cell carcinoma (Robson staging system)

Stage I: confined within capsule of kidney

Stage II: invading perinephric fat but still contained within Gerota’s fascia

Stage III:
III-a: invading the renal vein or IVC
III-b: regional lymph-node involvement
III-c: both nodes and IVC/renal vein

Stage IV: invading adjacent viscera (excluding ipsilateral adrenal) or distant metastases
Undescended testicle

In RLQ, there is a soft tissue density
Only a left testicle is seen in the scrotum 
80% of all undescended testes naturally descend to the scrotum during the first year of life 

DDX:
Neoplasm 
Lymphadenopathy 
Focal, dense
Undescended testicle

In RLQ, there is a soft tissue density
Only a left testicle is seen in the scrotum
80% of all undescended testes naturally descend to the scrotum during the first year of life

DDX:
Neoplasm
Lymphadenopathy
Focal, dense fluid collection (hematoma)
Congenital abnormality
Medullary nephrocalcinosis

CT: faint calcifications within the renal medulla 
MC etiology:
Hyperparathyroidism 
Medullary sponge kidney (renal tubular ectasia)
RTA type I (distal convoluted tubules)

LC etiology:
Hypervitaminosis D 
Milk-alkali
Medullary nephrocalcinosis

CT: faint calcifications within the renal medulla
MC etiology:
Hyperparathyroidism
Medullary sponge kidney (renal tubular ectasia)
RTA type I (distal convoluted tubules)

LC etiology:
Hypervitaminosis D
Milk-alkali syndrome
Nephrotoxic drug use (e.g., amphotericin B)
Any hypercalcemic or hypercalciuric state (including sarcoidosis)
Chronic pyelonephritis
Renal lymphoma

NHL >> HD
Patterns of involvement:
MC direct extension from RP disease
Hematogenous extension (common)
Primary renal lymphoma (rare)

Features:
Multiple hypoechoic, hypodense masses
Diffuse involvement
Adenopathy
Oncocytoma

Rare type of adenoma
Arise from oncocytes (epithelial cells) of proximal tubule
Classic: (DDX of central scar: adenocarcinoma)
Central stellate scar composed of fibrous tissue
Angiography: spokewheel appearance, homogeneous blush
Well-defined sharp borders
Treatment: renal-sparing surgical resection
Renal tuberculosis

Case findings:
Small densely rim calcified left kidney due to longstanding tuberculosis 

Putty kidney: small calcified end stage kidney resulting from autonephrectomy due to renal TB 
Papillary necrosis
Calyceal dilatation, amp
Renal tuberculosis

Case findings:
Small densely rim calcified left kidney due to longstanding tuberculosis

Putty kidney: small calcified end stage kidney resulting from autonephrectomy due to renal TB
Papillary necrosis
Calyceal dilatation, amputated calyx
Parenchymal scarring
Infundibular stenosis

Classic: corkscrew ureter, with multiple infundibular and ureteral stenosis
Classic:
Multifocal caliectasis due to irregular infundibular strictures
Papillary necrosis
Retrograde pyelogram showing complex stricturing of the right upper pole infundibulum, with associated caliectasis
Corkscrew ureter

Putty kidney
Pelvic lipomatosis

Case findings:
IVP: inverted teardrop bladder caused by extrinsic compression
Ureters are dilated, tortuous, and displaced laterally

DDX tear-drop bladder:
Hypertrophy of the iliopsoas muscles
Retroperitoneal fibrosis
Large pelvic abscess
Large hematoma, usually due to trauma or anticoagulation therapy
Collateral venous circulation from IVC obstruction
Large iliac artery aneurysms
Adenopathy from lymphoma, and prostatic carcinoma
Pyelosinus backflow

Due to forniceal rupture
Backflow is categorized as depending on the pathway taken by the contrast exiting the calyx:

Pyelotubular, pyelointerstitial:
Contrast passing through the collecting ducts and interstitium
Result in a
Pyelosinus backflow

Due to forniceal rupture
Backflow is categorized as depending on the pathway taken by the contrast exiting the calyx:

Pyelotubular, pyelointerstitial:
Contrast passing through the collecting ducts and interstitium
Result in a wedge-shaped striated area or blush extending from a calyx to as far as the renal outline
Pyelosinus
Pyelolymphatic:
Contrast entering the lymphatic channels
Opacification of multiple fine vesicles channels in the hilum of the kidney directed towards the para-aortic lymph nodes
Pyelovenous:
Contrast entering the renal veins (rarely recognized radiographically)
Medullary sponge kidney

Case findings:
Bilateral nephrocalcinosis

Features:
Tubular ectasia with linear collections of contrast identified within medullary pyramids
With time, these will progressively dilate and form cystic cavities
Cavities res
Medullary sponge kidney

Case findings:
Bilateral nephrocalcinosis

Features:
Tubular ectasia with linear collections of contrast identified within medullary pyramids
With time, these will progressively dilate and form cystic cavities
Cavities result in urinary stasis, leading to stone formation

May be sporadic or associated with:
Caroli’s disease
Hemihypertrophy
Ehlers Danlos syndrome
DDX of benign tubular ectasia:
Common finding
Linear paint brush striations arising from the medullary pyramids which fill with contrast on IVU
Unlike medullary sponge kidney, NO evidence of cystic dilatation within the tubules or formation of calcification
Calyceal diverticulum

Outpouching of the collecting system into the corticomedullary region
MC asymptomatic but may develop calculi or milk of calcium
Retrocaval ureter

Normally, right ureter remains lateral to the IVC

Classic: J-shaped ureter
Ureter passes behind the IVC and exits between the aorta and IVC
Medial looping at L2-3
May cause ureteral narrowing and obstruction
Pyeloureteritis cystica

Multiple small subepithelial cysts in the renal pelvis and ureter
Seen in setting of chronic obstruction or infection
NOT premalignant

Classic:
Multiple small, well defined, lucent, filling defects seen in the renal pelvis and ureter
Lesions seen in profile cause scalloping of ureteric margin
Predilection for upper 1/3 of ureter
May be bilateral
Ureteric pseudodiverticulum

MC associated with chronic infectious or inflammatory states
MC seen in older patients
50% develop uroepithelial malignancy, and therefore need close surveillance
Urethral stricture 

Etiology:
MC used to be gonococcal
Periurethritis: indwelling urethral catheters 
Urethritis

Inflammatory strictures in bulbar urethra, which is the most dependent part and contains the greatest number of paraurethral glands
Urethral stricture

Etiology:
MC used to be gonococcal
Periurethritis: indwelling urethral catheters
Urethritis

Inflammatory strictures in bulbar urethra, which is the most dependent part and contains the greatest number of paraurethral glands
Strictures caused by instrumentation occur in naturally narrowed segments of the urethra
Straddle injuries MC in bulbar urethra
Direct blows injure the pendulous urethra
Retrograde urethrogram showing a tight focal stricture (arrow) in the upper urethra
Reflux of contrast into Cowper's duct and gland
Adrenocortical carcinoma

Case findings: 
Large left adrenal cortical carcinoma with tumor extension into IVC (arrow)

MC > 5 cm at presentation
Large heterogenous suprarenal mass with areas of hemorrhage, necrosis, and calcification 
Well circumsc
Adrenocortical carcinoma

Case findings:
Large left adrenal cortical carcinoma with tumor extension into IVC (arrow)

MC > 5 cm at presentation
Large heterogenous suprarenal mass with areas of hemorrhage, necrosis, and calcification
Well circumscribed, despite its large size, and a well defined capsule-like rim
May contain areas of lipid  detected on opposed-phase chemical shift imaging
Classic: IVC extension

Coronal T2: large right adrenal cortical carcinoma with extension of tumor into IVC (arrow)
Gartner's duct cyst

Case findings:
Well circumscribed non-enhancing thin-walled cyst anterolateral to the vagina (paravaginal location)

Retention cyst arising from Gartner’s ducts, occurring along the Wolffian duct remnant
Paravaginal and anterolateral in position

DDX:
Squamous inclusion cyst of the vagina
Ureteric diverticulum
Bartholin’s gland: located more distally and superficially adjacent to the vulva
Nabothian cyst

Case findings:
Multiple clustered small Nabothian cysts

Inclusion cyst formed in the uterine cervix when the opening of a Nabothian duct located within the cervix is covered by epithelial cells and the fluid is then retained
Prostatic utricle cyst

Midline lesion posterior and superior to the verumontanum
Abnormality in the regression of mullerian duct
Can also be acquired due to posterior urethritis and verumontanitis

DDX mullerian duct cyst: larger and extend above the base of the prostate
Ovarian neoplasm

Diagnosis: Ovarian serous cystadenocarcinoma 

Epithelial:
Serous cystadenoma/cystadenocarcinoma
Mucinous cystadenoma/cystadenocarcinoma
Clear cell carcinoma
Brenner tumor

Stromal:
Granulosa cell tumor (estrogen)  Call-Exner
Ovarian neoplasm

Diagnosis: Ovarian serous cystadenocarcinoma

Epithelial:
Serous cystadenoma/cystadenocarcinoma
Mucinous cystadenoma/cystadenocarcinoma
Clear cell carcinoma
Brenner tumor

Stromal:
Granulosa cell tumor (estrogen)  Call-Exner bodies
Sertoli-Leydig (androgen)
Thecoma, fibroma (low T2)
Meig’s syndrome: ovarian fibroma, ascites, pleural effusion
Germ cell tumor:
Dysgerminoma (similar to seminoma): radiosensitive
Yolk sac tumor (endodermal sinus tumor): elevated AFP
Embryonal cell carcinoma
Choriocarcinoma
Teratoma (dermoid cyst)

Metastasis:
Lymphoma
Primary uterine tumor
Stomach, colon, breast
Kruckenberg tumor: ovarian metastasis from stomach or colon primary

Ovarian serous cystadenoma
Axial T2: bilateral cystic adnexal masses with minimal internal complexity
Subcapsular renal hematoma

Case findings:
Right kidney is compressed with a delayed nephrogram relative to the left kidney

Page kidney: chronic long-standing compression of underlying renal parenchyma by subcapsular haematoma can result in hypertension, due to local ischemic change causing excess secretion of renin
Urethral diverticulum

Males: congenital urethral diverticula
Females: urethral diverticula are acquired
Epidermoid cyst of the testis (keratocyst)

Case findings:
Solid mass in the right testis with calcification and acoustic shadowing

Comprise layers of keratin, within a fibrous capsule lined with simple squamous epithelium

US:
Solid mass with echogenic rim and peripheral acoustic shadowing
Onion-ring (lamellated) pattern of the periphery with a target appearance
Treatment: enucleation thereby avoiding an orchiectomy

DDX:
Calcified haematoma
Intratesticular cyst (anechoic centre and through transmission)
Teratoma
Tubular ectasia of the testis (cystic transformation of rete testis)

Case findings:
US: well-defined tubular structures in the region of the mediastinum testis

Anastomotic ducts are located in the mediastinum testis, in which numerous seminiferous tubules drain and connect with the head of the epididymis
Secondary to obstruction of seminiferous tubules following infection or trauma
Associated with: ipsilateral epididymal cyst
DDX:
Intratesticular varicocele: rare, increases in size on the Valsalva maneuver and shows blood flow
Cystic dysplasia of the testis: rare, associated with congenital abnormalities
Cystic malignant testicular tumor: can occur anywhere in the testis
Teratoma: may present as a cystic tumor, although this is very rare
Seminal vesicle cyst

Case findings:
Slight peripheral enhancement of the cyst wall
No internal septations

Aberrations of distal Wolffian ducts lead to atresia or stenosis of the ejaculatory ducts, resulting in obstruction and congenital seminal vesicle cyst formation

DDX testicular cyst:
Bladder or ureteral diverticulum
Seminal vesicle abscess
Müllerian duct cyst
Cystadenoma of the seminal vesicle or of the prostate
Dermoid cyst
Teratoma
Echinococcus cyst (rare)
Axial fat-suppressed T1-weighted 
Coronal T2-weighted post-Gd
Coronal fat-suppressed T1WI post-Gd
Axial fat-suppressed T1-weighted
Coronal T2-weighted post-Gd
Coronal fat-suppressed T1WI post-Gd
Ovarian fibroma

Case findings:
Large multilobulated solid ovarian mass
Heterogenous enhancement
Low T2 signal

Fibroma associated with Meig’s syndrome (ovarian fibroma, ascites, pleural effusion)
DDX low T2 ovarian lesion (fibrous components):
Fibroma
Fibrothecoma
Cystadenofibroma
Brenner’s tumor
Sagittal T2WI
Coronal T1WI
Sagittal T2WI
Coronal T1WI
Pseudomyxoma peritonei
Case findings:
US: complex cystic lesion which is septated with solid areas
CT:
Scalloping of the liver due to the gelatinous nature of the fluid
Fluid in the peritoneal cavity with areas of calcification (arrows)
MR T2WI:
Gelatinous fluid in the pelvis and surrounding the viscera
Also seen invading the anterior abdominal wall and extending into the perianal region

Mucinous implants of the peritoneum
Gelatinous ascites secondary to a benign or malignant tumor of the appendix, ovary or other organs (pancreas, colon, etc.)
Enhancement helps to differentiate from ascites
Walls may show calcification
Multilocular cystic nephroma (MLCN)

Case findings:
Well-marginated, rounded, or polycyclic cortical mass that extends beyond the normal renal outline 

Multilocular cystic renal tumor: term that encompasses two histologically distinct entities
Mult
Multilocular cystic nephroma (MLCN)

Case findings:
Well-marginated, rounded, or polycyclic cortical mass that extends beyond the normal renal outline

Multilocular cystic renal tumor: term that encompasses two histologically distinct entities
Multilocular cystic nephroma (MLCN)
1/3 of MLCN theoretical risk of cystic RCC, MC adult women
Cystic partially differentiated nephroblastoma (CPDN)
CPDN: 2/3 of MLCN, MC young children, theoretical risk of Wilms’ tumor
Imaging cannot distinguish between MLCN and CPDN
Clinical presentation:
Painless abdominal mass with rapid enlargement

Features:
Well-marginated cysts with multiple septa (only septa enhance)
Congenital lesion with large (> 10 cm) cystic lesions
Compared with the poorly defined necrotic cystic appearances of a degenerative Wilms' tumor
Multilocular cystic renal tumor features:
Round, well-encapsulated multilocular cystic mass
Central location
Invasion into renal sinus
Prolapse into collecting system (pathognomonic finding)
Contain multiple non-communicating cysts with thin septa separating the cysts

DDX:
Cystic Wilms' tumor
Wilms' tumor coexisting with CPDN
Current thinking suggests that cystic nephroma, CPDN and Wilms’ tumor represent a spectrum of disease
MLCN: benign end of the spectrum
Wilms' tumor: malignant end of the spectrum
CPDN: intermediate stage where blastemal elements are present within internal septa

Multilocular cystic nephroma
Subepithelial hemorrhage

Case findings:
Findings are masked on CECT
CECT: mild calyceal dilatation and constriction of lumen of renal pelvis
NCCT: high-attenuation thickening of pelviureteric wall caused by hemorrhage

Bleeding may be parenchymal, subcapsular, perinephric, subepithelial (rare)
Subepithelial renal sinus hemorrhage is NC seen in coagulopathic patients
DDX on IVP: focal pelviureteral wall irregularity may be indistinguishable from
Subepithelial hemorrhage
Transitional cell carcinoma
Pyeloureteritis cystica
Submucosal edema
Perinephric hemorrhage

Case findings:
Left perinephric (PN) and anterior pararenal (AP) hematomas
Focal defect in the left renal cortex (arrow) represents renal parenchymal scarring
Selective right renal angiography showed small aneurysms (not shown
Perinephric hemorrhage

Case findings:
Left perinephric (PN) and anterior pararenal (AP) hematomas
Focal defect in the left renal cortex (arrow) represents renal parenchymal scarring
Selective right renal angiography showed small aneurysms (not shown)

Diagnosis: perinephric hemorrhage due to polyarteritis nodosa
DDX on CECT:
Lymphoma
Perinephric hemorrhage
Causes of subcapsular perinephric hemorrhage:
Trauma:
Renal laceration
Avulsion of the renal artery
Neoplasm
Malignant (renal cell carcinoma)
Benign (angiomyolipoma)
Vasculitis: polyarteritis nodosa
AVM
Aneurysm
Anticoagulant therapy
Acute infection
Acute or chronic nephritis

Angiomyolipoma
Intratumoral hematoma (H) with perinephric hematoma (arrow)

Renal cell carcinoma
Hypoattenuating mass (M) in the lower pole, which is displaced anteriorly by a perinephric hematoma with a fluid-fluid level (arrow)
Renal infarct

Case findings:
Global infarct of the right kidney with rim of enhancement in capsule (cortical rim sign)
Thromboembolism
Aneurysm of aorta or renal artery
Atherosclerosis
Subacute bacterial endocarditis (septic emboli)
Dissection of the aorta or renal artery
Vasculitis: polyarteritis nodosa, SLE, drug-induced
Trauma
Avulsion of the renal artery
Occlusion of the renal artery
Penetrating vascular injury
Hypercoagulable state
Acute venous occlusion

Renal vein thrombosis
Hypercoagulable states
Nephrotic syndrome
SLE
Dehydration
Trauma
Neoplasm: RCC, TCC, Wilms’ tumor
Aneurysm
Left ovarian vein thrombosis
Acute cortical necrosis

Case findings:
Lack of enhancement of the renal cortex.
Narrow subcapsular rim of enhancement (arrows)
Rim of subcapsular cortical enhancement from collateral flow of capsular vessels

Results from ischemic necrosis of the renal cortex with sparing of the renal medulla
Ischemia due to vasospasm of small vessels, toxic damage to glomerular capillary endothelium, and primary intravascular
thrombosis
Associated with:
Complications of pregnancy
Sepsis, shock, severe dehydration
Hemolytic uremic syndrome

Cortical necrosis can result from any condition that produces acute, prolonged shock
Endometrioma

Case findings:
T1: mass in the right ovary with high SI
T2: low SI (“shading”, arrow)
US: cystic mass with diffuse low-level echoes

DDX high T1 adnexal lesion:
Hemorrhagic cyst
Endometrioma
Dermoid (ask for fat-saturation sequence
Endometrioma

Case findings:
T1: mass in the right ovary with high SI
T2: low SI (“shading”, arrow)
US: cystic mass with diffuse low-level echoes

DDX high T1 adnexal lesion:
Hemorrhagic cyst
Endometrioma
Dermoid (ask for fat-saturation sequence)
MC ovarian (80%)
US:
Cystic mass with diffuse, low-level echoes (this case)
Cystic to complex
May have a solid appearance
MRI:
High T1, T2 shading
Multiplicity

Mature cystic teratoma (dermoid)
High T1 adnexal mass
Fat-saturation of the lipid omponent of the mass (arrow)