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87 Cards in this Set

  • Front
  • Back

Vascular System

Disorders of the arteries (oxygenated blood), veins (deoxygenated blood) and lympathic vessels.

Peripheral Artery Disease Risk Factors

Tobacco use, diabetes, hyperlipidemia, elevated CRP, and uncontrolled htn

What is the leading cause of atherosclerosis?

PAD

Pulses, Edema, Hair, and Ulcer Location PAD

Peripheral pulses decreased/absent, no edema, loss of hair on legs/feet/toes, and ulcers on tips of toes, foot/lateral malleous

Margin/Drainage of PAD

Rounded, smooth looks punched out, and minimal drainage

Tissue of PAD Ulcer

Black, eschar or pale pink granulation gangrene. Amputation may be required if blood flow not restored.

Pain of PAD

Intermittent claudication, pain in muscles (lack of O2), PAD of femoral/popliteal cause calf pain, PAD of iliac arteries cause buttock/thigh pain

Nails, Skin color/texture and temp of PAD

Nail thickened/brittle, skin color pallor develops w/ leg elevation, foot redness appers w/ limb in dependent position, skin texture is thin shiny, and taut, and temp is cool gradient down the leg. dermatitis and pruritus barely occur

Dx studies of PAD

Doppler ultrasound, angiography, ankle brachial index (divide ankle systolic by branchial systolic)

Venous disease risk factors

Venous stasis (age, a fib, immobility), endothelial damage (surgery, IV meds, fractures), hypercoagulatbility of blood (genetics, high altitudes, hormone therapy)

Pulses, edema, hair and ulcer location of VD

Peripheral pulses are present, lower leg edema, hair present/absent, ulcer near medial malleolus

Margin/drainage of VD

Irregularly shaped and moderate to large drainage

Tissue of VD

Yellow slough/dark red "ruddy" granulation,

Pain w/ VD

Dull ache/ heaviness in calf/thigh, ulcer often painful

Nails, skin color, skin texture, temp and dermatitis and pruritus of VD

Normal/thickened nails, skin color is bronze/brown pigmentation, varicose veins may be visible, skin thick hardened and indurated, skin is warm w/ no temp gradient, dermatitis/pruiritus occur often.

Atherosclerosis skin color/temp/peripheral pulses
Hyperemia in depedent position and pallor w/ elevation, skin texture is thin, shiny, taut, cool temp. Decreased/absent peripheral pulses

If gangrene on foot where do you place Xs?

On Dorsalis pedis and posterior tibial

Nursing Planning/Intervention for PAD

Assess pain, monitor extremeties for color, motion/sensation/pulses, obtain BP, assesss s/s of ulcer/gangrene.

Teaching for PAD

Stop all tobacco, avoid caffeine, aggressive tx of hyperlipidemia, BP less than 140/90, diabetes and renal failure 130/80. Walking most effective, avoid crossing legs, bmi and waist circumference. Dietary cholesterol, decreased intake of sat fat, sodium <2 g/day. avoid exposure to cold, never apply direct heat to limb.

Drug therapy for PAD

Antiplatelet agents and ACE inhibitors.

Antiplatelet for PAD

Considered critical. Aspirin 75-100 mg/day for symptomatic, 75-325 for asymptomatic, Clopidogrel (Plavix) daily. Combining Plavix and asa are typically not recommended but is okay for symptomatic PAD

What should Plavix not be used w/ for increased risk for stroke/MI?

Omeprazole (Prilosec) PPI

First line drug treatment for intermittent claudication

Cilostazol (Pletal) and pentoxifylline (Trental)

When are Cilostazol (Pletal) and pentoxifylline (Trental) contraindicated?

HF

Interventional radiologic procedures for PAd

Peripheral artery bypass operation w/ autogenous vein or synthetic graft material to bypass or carry blood around the lesion

What kind of care is very important to teach PAD pts?

Meticulous foot care

Complications of PAD

It progresses slowly. Leads to atrophy of skin and underlying muscles. Minor trauma can result in delayed healing, wound infection, and tissue necrosis. Arterial ischemic ulcers occur most often over bony promineces. Amputtation may be needed if adeqate blood flow is not restored. Uncontrolled pain/severe spreading infection are indicators that an amputation is needed

Acute Intervention of PAD

After surgery/radiologic intervention to extremity: Check extremity Q15M intially for color, temp, cap refill, presence of peripheral pulses, and sensation and movement.

Acute arterial Ischemia

Sudden interruption in the arterial blood supply to a tissue, organ or extremity that if utnreated results in tissue death

Common Cause of acute arterial ischemia

embolism, thrombosis, or trauma

Tx of acute arterial ischemia include?

Anticoagulation, thrombolysis, embolectomy, surgical revascularization or amputation.

Thromboangitis obliterans (Buerger's disease)

Occlusive disease of the median and small arteries and veins. Distal and upper and lower limbs affected most commonlny

Occurs mostly in what gender and age group

Young men <45 y/o long hx of tobacco/marijuana use and chronic peridontal infection w/out other CVD risk factors

Dx tests and labs for Buerger's disease

None

Plans/Interventions for Buerger's disease

Smoking cessation, monitor pulses, avoid injury, adminster vasodilators, instruct on med therapy. Surgical options: revascularization, implantation of spinal cord stimulator

Raynaud's Phenomenon may be related to what?

Rheumatoid arthritis

S/S of Raynaud's phenomenon

Vasospasm (Color changes on fingers, toes, ears, or nose) (White blue red). coldness or numbness in vasconstrictive phase, then throbbing aching pain, tinging and swelling in hyperemic phase, episodes last only minutes by increased w/ cold, emtional upset, caffeine and tobacco use.

Dx test of Raynaud's phenomenon

None, based on persistent symptoms for at least 2 years.

Patient teaching Raynau'ds phenomenon

Mintor pulses, tell pt to wear loose warm clothing, avoid temp extremes, stop using tobacco and avoid caffeine, avoid injury to hands/fingers

Most common problem affecting aorta

Aneurysm

3/4 of aneurysms occur in the _____ ______ and 1/4 occur in the ______ ______

Abdominal aorta, thoracic aorta

Risk factors for aortic aneurysms

Age, male, caucasian, family hx, htn, CAD, previous stroke, obestiy and tobacco use

True Aneurysm

Aneurysm in which one of three later vessel of the aneurysm is intact

False aneurysm
Not an aneurysm but a disruption of all arterial wall layers w/ bleeding that is contained by surrounding automic structures

S/S of abdominal aortic aneurysm

Pulsating mass in abdominal systolic bruit over aorta, tenderness of deep palpation abd or lower back pain, changes in bowel habits

Thoracic aneurysms s/s

Pain radiating to neck, shoulders, lower back, syncope, dyspnea, increased pulse, cyanosis, weakness, hoarseness, swelling difficulty

Dx Test of Aneurysms in thoracic/abdominal

Chest xray, ECG (Rule out MI), CT most accurate to determine the length and cross sectional diameter and presence of thrombus in aneurysm.

Nursing Plan/Implementation of Aneursym in Thoracic/Abdominal

Decrease risk factor (smoking cessation, decrease BP, control hyperlipidemia), annual monitoring of size, w/ CT, MRI, ultrasound. Notify physician if severe back/abd pain, fullness, soreness over umbilicus, sudden dev of discoloration in extremeties, or persistent elevation of BP. Aortic: Report SOB, cx/back pain, hoarseness or difficulty swalling

Most severe complication of aneurysm

Rupture. If thoracic or abd more then 90% die. Severe back pain will occur or severe abd pain, lumbar pain that radiates to flank/groin

Surgical therapy for aneurysm

Incising the diseased segment of aorta, removing thrombus/plaque, inserting a synthetic graft, or suture the native aortic wall around graft to act as a protective cover.

Surgical repair of abd aortic aneurysm w/ a Woven Dacron graft
Preop: assess all periphereal pulses as baseline for postop, instruct as cough/deep breathing exercises

Postop surgical repair of abd aortic aneurysm w/ a Woven Dacron graft

Monitor VS, airway, neuro assessment, encourage turning, coughing and deep breathing and splinting, monitor peripheral pulses distal to graft site, monitor for changes in pulse, cool to cold extremeties, severe pain, abd distention, monitor for hypovolemia, monitor urine output/BUN/Creatinine, assess bowel sounds, assess surgical site, instruct pt to avoid straining, medicate for pain, no smoking and avoid heavy lifting.

Risk Factors for Phlebitis
Mechanical irritation from catheter, infusion of irritating meds, and rarely infectious and usually resolves quickly after cath removal.

What to do if edema is present in phlebitis?

Elevate extremeity to promote reabsorption of fluid, apply warm/moist heat and NSAIDs.

Venous thrombosis

Formation of a thrombus in association w/ inflammation of the vein. Classifications of superficial vein thrombosis (SVT) and deep vein thrombosis (DVT)

Etiology of Venous Thrombosis: Clue Three factors called Virchow's triad

1: Venous statsis: age, a fib, obesity and immobility 2: Damage of the endothelium (inner lining of the vein: Surgery, causitive IV meds, fractures, hx of previous venous thromboembolism, IV drug abuse 3: Hypercoagulability of blood: genetics, high altitudes, hormone therapy, malignancies and tobacco

Superficial Vein Thrombosis (SVT)

Formation of a thrombus in a superficial vein

S/S SVT

Palpable firm subcutaneous cordlike vein. May be itchy, tender, or painful to touch, reddened, warm.

Risk Factors SVT
Age, pregnancy, obesity, malignancy, estrogen therapy, long distance travel

Tx of SVT

Low molecular weight heparin for 45 days, NSAIDs, compression stockings and mild exercise.

DVT what is the preferred terminology?

VTE

Nursing Plan/Intervention for VTE

Bedrest, elevate affected extremeity above the heart, avoid pillow under affected knee, do not massage, provide antiembolism stocking to reduce venous stasis, monitor for warmth and edema, measure/record circumference of thighs/calves, monitor for SOB and cx pain (can indicate PE, if so rapid O2 and notify physician), adminster heparin, monitor PT and INR, obtain medical alert, pt w/ lower VTE may or may/may not have unilateral s/s

Drug therapy for VTE
Anticoagulants.

Three major classes of anticoagulants

Indirect thrombin inhibitors, Vitamin K antagonist, and direct thrombin inhibitors

Indirect Thrombin Inhibitors

Heparin, Enoxaparin (Lovenox)

Vitamin K Antagonists
Warfarin (Coumadin)
Drug Alert For Anticoagulant Therapy
Avoid aspirin, NSAIDs, fish oil supplements, gingko biloba, and certain antibiotics (sulfamethoxazole and Bactrim). Instruct pt report signs of bleeding (black/bloody stools, bloody urine, coffee ground emesis, and nosebleeds), hypotension, tachycardia, ecchymosis

Surgery for VTE

Venous thrombectomy and placement of vena cava interruption device (Greenfield filter)

Venous Thrombectomy

Removal of thrombus through an incision in the vein, anticoagulation afterwards

Vena cava interruption device
can be inserted percutaneously through right femoral or right internal jugular vein. Filter device is opened and spokes penetrate vessel walls, filter acts as a sieve type device, permitting filtration of clots w/out interruption of blood flow. Filter may need removal/replacement overtime. Recommeneded if anticoagulation is contraindicated.

Complications of Post Thrombotic Syndrome

Chronic venous htn, stiff noncompliant vein walls and persistent venous obstruction. S/S pain, aching, heaviness, swelling, cramps, itching, and tingling.

S/S of complications of PTS

Persistent edema, increased pigmentation, eczema, secondary varicosities, lipodermtoscelrosis, skin on lower leg becomes scarred and the leg becomes tapered like an inverted bottle.

Chronic Venous Insufficiency
Condition that develops when leg veins/valves fail to keep blood moving forward can lead to venous leg ulcers. Not life threatening but painful, defibilitating and costly

S/S of CVI

Skin of lower leg appearing leathery, w/ char. brownish/brawny appearance, edema, eczema, and pruritus

Common location for venous ulcers

Medial malleolus. Wound irregularly shaped and tissue typically ruddy color. Ulcer drainage may be excessive. Pain is present and may be worse when leg is in dependent position.

CVI Plan/Implementation
Elastic or compression stockings, clean pair daily, avoid prolonged sitting/standing, constrictive clothing, and crossing legs, elevate legs 10-20 mins every few hours of the day, elevate legs aboe heart when in bed, cover open ulcers w/ dressings before applying compression stockings.

Dilated vein

Damaged/weakened walls w/ a leaky valve meaning they cannot close and open to control the blood flow in either direction

S/S varicose veins

Enlarged, raised veins, pain, cramps, aches, restless legs, itching/burning.

CVI can progress to these symtpoms?

Leg swelling/pain, discoloration and ulcers.

Varicose Veins

Dilated, tortuous subcutaneous veins most frequently found in the saphenous system

Etiology of varicose veins

Family hx, female gender, use of oral contraceptives, tobacco use

S/S of varicose veins

Heavy/achy feeling or pain after prolonged standing/sitting which is eleviated by walking/limb elevation, itchy/burning/tingling/throbbing/cramplike leg sensation, swelling restless or tired legs

Care for varicose veins

Rest w/ affected limb elevated, compression stocking, exercise such as walking

Tx of varicose veins

Sclerotherapy, laser/light therapy, and surgical procedures such as phlbectomy and vein ligation

Sclerotherapy

Injection of a substance that destroys superficial veins

What is key factor to varicose veins?

Prevention

how to prevent varicose veins?

Avoid sitting/standing for long periods of time, maintain ideal body weight, avoid wearing constrictive clothing and walk daily.