Myocardial Infarction Essay

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Mr. Canton displayed early onset symptoms of an acute myocardial infarction according to the nurse’s assessment and an EKG that presented ST elevation. As shown on the EKG, elevation is a sign of immediate concern resulting from depolarization and repolarizing of the left ventricle. “Plaque progression, disruption, and clot formation are the same for myocardial infarction as they are for angina” (Understanding Pathophysiology, 2016, p. 620).
Myocardial Infarction In early stages of congestive heart failure, crackles heard in the lung bases are from accumulating fluid known as transudate. Mr. Canton has a heart rate of 120, with respirations of 24. Since the left ventricle is not pumping blood out of the aorta adequately, this is resulting in his difficulty to breathe. His cooler body
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Brain receptors becomes “confused” from the referred pain coming from the heart that is can be signaled from the arms as well as up into the jaw. As Mr. Canton’s arteries are tightening the fluid is continuing to build up, reflexes stimulating pain from the myocardial infarction may be a reason his gastrointestinal track is irritated causing him to vomit. In diagnosing a myocardial infarction, blood is drawn for labs to look for elevated isoenzymes (myocardial cells). Enzymes are the proteins that aid to the body. Mr. Canton’s isoenzymes are elevated caused by the damage to his heart muscle. CK-MB generally rises after a heart attack, inflammation of the heart muscle, muscular dystrophy, and other problems related to the heart. (University of Rochester Medical Center) One of the main reasons leading to a myocardial infarction is by atherosclerotic plaque buildup caused by a disruption in the blood flow of the heart. As blood is increases its flow rate, this can cause plaque to rupture developing into a thrombus (blood clot). When cells are not getting the amount of oxygen

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