A muscle tear refers to muscle fibers being damage (Virginia 2013). A tear in the gastrocnemius can cause great risk of injury since the gastrocnemius is located between the knee and ankle joint (Virginia 2013). This can be cause by direct trauma to the calf region or sudden landing on the foot (Medial Head Gastrocnemius 2006). This injury is common in tennis players due to the constant motions of jumping and lunging (Medial Head Gastrocnemius 2006). In the first degree of gastrocnemius tear, the individual will experiment pain but the tendon will not be lengthened (Medial Head Gastrocnemius 2006). In the second degree, there is a partial tear and the ligament is lengthened (Medial Head Gastrocnemius 2006). In the third degree, function of
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Injury in the calf can also results from Achilles tendon rupture and thrombophlebitis, which is a blood clot in the leg (Virginia 2013). For treatments, the first step usually involves RICE, which stands for rest, ice, compression, and elevation (Virginia 2013) Ice should be apply in increments of 10 minutes for at least 2 hours (Medial Head Gastrocnemius 2006). In addition to RICE, the individual can also perform some exercises prescribed by the therapist (Virginia 2013). The individual can start with stretching and then build to other range of motion exercises (Virginia 2013). Overtime, the individual should be able to regain agility and strength of the calf again (Virginia …show more content…
In Thomsen's Disease, the cause is due to a mutation in the CLCN1 gene by autosomal dominant pattern (Myotonia congenita 2007). The CLCN1 gene regulates the flow of negatively charged chloride ions into and out of the muscle fibers (Myotonia congenita 2007). This is important in muscle contraction and relaxation (Myotonia congenita 2007). Therefore, when the CLCN1 gene is impaired, it cause myotonia (Myotonia congenita 2007). The levels of myotonia in Thomsen's Disease range from grade 1 to grade 3 (Colding-Jorgensen 2005). Grade 1 means there is only mild myotonia, while grade 3 represents the more severe state of myotonia (Colding-Jorgensen 2005). Individual with Thomsen's Disease will experience muscle stiffness and delayed time in muscle relaxation (Facts about myopathies 2012). This usually interfered with daily activities of walking and eating (Facts about myopathies 2012). However, it can be manage with proper exercise and medications (Facts about myopathies 2012). With proper physical therapy, the effect of myotonia can be reduced (NINDS Myotonia 2011). Some studies theorized that ongoing muscle contractions can cause the sodium-potassium pump to temporary increase helping the muscle cells membrane to be more hyperpolarized (Colding-Jorgensen 2005). Hyperpolarization assists with accelerating muscle relaxation (Colding-Jorgensen 2005). In addition to physical
Injury in the calf can also results from Achilles tendon rupture and thrombophlebitis, which is a blood clot in the leg (Virginia 2013). For treatments, the first step usually involves RICE, which stands for rest, ice, compression, and elevation (Virginia 2013) Ice should be apply in increments of 10 minutes for at least 2 hours (Medial Head Gastrocnemius 2006). In addition to RICE, the individual can also perform some exercises prescribed by the therapist (Virginia 2013). The individual can start with stretching and then build to other range of motion exercises (Virginia 2013). Overtime, the individual should be able to regain agility and strength of the calf again (Virginia …show more content…
In Thomsen's Disease, the cause is due to a mutation in the CLCN1 gene by autosomal dominant pattern (Myotonia congenita 2007). The CLCN1 gene regulates the flow of negatively charged chloride ions into and out of the muscle fibers (Myotonia congenita 2007). This is important in muscle contraction and relaxation (Myotonia congenita 2007). Therefore, when the CLCN1 gene is impaired, it cause myotonia (Myotonia congenita 2007). The levels of myotonia in Thomsen's Disease range from grade 1 to grade 3 (Colding-Jorgensen 2005). Grade 1 means there is only mild myotonia, while grade 3 represents the more severe state of myotonia (Colding-Jorgensen 2005). Individual with Thomsen's Disease will experience muscle stiffness and delayed time in muscle relaxation (Facts about myopathies 2012). This usually interfered with daily activities of walking and eating (Facts about myopathies 2012). However, it can be manage with proper exercise and medications (Facts about myopathies 2012). With proper physical therapy, the effect of myotonia can be reduced (NINDS Myotonia 2011). Some studies theorized that ongoing muscle contractions can cause the sodium-potassium pump to temporary increase helping the muscle cells membrane to be more hyperpolarized (Colding-Jorgensen 2005). Hyperpolarization assists with accelerating muscle relaxation (Colding-Jorgensen 2005). In addition to physical