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399 Cards in this Set
- Front
- Back
Cardiac Output
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- the volume of blood ejected from the left ventricle each minute
- in children, the CO is almost completely dependent on HR until the heart muscle is fully developed at 5yo CO = HR x SV Adult = 5-8L/min Kids = 2-3l/min |
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Transitional Circulation
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- Changes at birth
! Gas exchange is transferred from placenta to the lungs. ! Fetal shunts: Ductus venosus, Ductus arteriosus, Foramen ovale - blood from mom, umbi, placenta, will dump into ductus venosis which bypasses the liver - dumps into inferior VC into atrium - then to LA via foramen ovale, bypassing lungs - if some blood basses from RA - VA, blood goes to aorta via pulm art via ductus arteriousis if these shunts remain open after birth, trouble |
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Causes of Heart Defects
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Multifactorial Causes:
- teratogenic - chance - Familial link - chromosomal abnormalities -deleted chrom 22 - trisomy 21 - turners syndrome -trisomy 13 |
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Congenital Cardiac Anomalies
Signs & Symptoms of CHD |
- Cyanosis
- peripheral - central - resp distress - CHF - decreased CO - abnormal cardiac rhythms - cardiac murmur - FTT (failure to thrive) ==================== s1 = closure of AV valves s2 = closure of semi- lunar (pulm/aortic) ==================== - hearing trickling/ movement of blood between s1/s2 = murmur - indicative of congenital heart defects |
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Nursing Diagnosis for Ped Cardaic Anomalies
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- dec CO
- excess fluid volume - ineffective breathing pattern - imbalanced nutrition - compromised fam coping |
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Nursing Interventions for Ped Cardiac Anomalies
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- meds
- maintain cardiac and resp function - foster dev - promote nutrition - emotional support |
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Echocardiogram
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! Ultrasound – to generate an image of the heart.
! Assesses: location and relationship of intracardiac and extracardiac structures, cardiac function, measures sizes of cardiac chambers, valve functions, size of defects, estimates gradient and blood flow direction. |
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Valve insufficiency leads to:
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valve insufficiency leads to:
1. valvular stenosis (stiff- won't open/close properly) 2. valvular regurgitation (valves open up in two directions, allowing for backflow) |
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Classification of Congenital Heart
Defects Increased Pulmonary Blood Flow |
! an increased pulmonary blood flow
exists. ! Blood is shunted from Left side of heart to the right side. ! Major complication: CHF |
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Patent Ductus Arteriosis
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PDA
- patent means open therefor communication between aorta and pulmonary artery - remember this shit should close soon after birth ======================= ! Occurs in premature infants (< 28 weeks) – 80% ! 5-10% of all defects (2002), 1/2000 of term infants ! Can be present with all other heart defects. |
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Management for Patent Ductus Arteriosis
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! Indomethacin
! Fluid restriction X 2 days then gradually increase ! Diuretics if needed ! Oxygen ! Transcatheter closure is sometimes attempted in children greater than 18 months of age. ! Surgical ligation |
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Atrial Septal Defect. ASD
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- a communication btw RA and LA, resulting in increased pulmonary blood flow
- inability of foramen ovale to close - means the heart will ahve to pump extra blood to get sufficient blood out to aorta and to tissues |
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ASD Management
|
! Closure of this defect depends on type of ASD
! Some will close spontaneously ! Cath Lab Closure versus open heart surgery ! Adulthood discovery. ! Asymptomatic are monitored without medications. ! Symptomatic are treated with diuretics and digoxin. |
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Ventricular Septal Defect
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- a communication btw LV/AV, again putting more blood into pulm art
|
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Summary of Management of
Lesions |
! Complete assessments
- decreased perfusion. - congestive heart failure. ! Medications: digoxin, diuretics, oxygen ! Elevate HOB ! Nutrition ! Cardiac catheterization and / or Surgery, as needed |
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Cyanotic Defects with
Decreased Pulmonary Blood Flow. |
A decrease of pulmonary blood flow with the delivery of unoxygenated blood to the body results in hypoxia or a decreased volume of flow.
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Tetralogy of Fallot
Four Components to defect: |
! Ventricular septal defect (large)
! Pulmonary stenosis ! Overriding aorta ! Right ventricular hypertrophy (secondary to pulmonary stenosis) |
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! TET SPELLS:
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! at risk for spells of
extreme hypoxemia ("tet spells") which may be fatal ! Children will squat or move into a fetal position to make themselves feel better. WHY? - increases venous return |
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Treatment of TOF
|
- b blocker
- morphine - prostaglandin E1 (rarely used) - +/- BT Shunt (Blalock-Taussig shunt) - surgical repair |
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General Management of
Decreased Pulmonary Blood Flow Defects |
! Prostaglandin E1
! Adequate Hydration ! Supplemental oxygen ! Surgery |
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Nursing Care of Pediatric
Congenital Heart Defect Patients |
complete physical assessessments (head to toe
- signs of decreased perfusion / decreased ventilation - monitor bp\- listen to heart sounds (rate, rhythm, qualiity) - pulses - characterisics - fluid status: edema - activity - growth |
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Cardiac Catheterization
|
! Involves the insertion of a catheter
through an artery/vein into the heart. ! Performed under fluoroscopy (cath-lab) |
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Cardiac Catheterization
What information is obtained? |
- pressures iwhtin the heart
- 02 sats - blood flow patterns - structural info (valves, chambers, great vessels) |
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Complications of Catheterization
|
- arrhythmias
- bleeding - cardiac perforation - CVA - contrast agent reactions - hypercyanotic spells - local vascular complications - infections |
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Nursing Care Pre Catheterization
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- NPO
- Sedation family and child anxiety - orientate family and child to unit, cath lab information - pre cath prep info - procedure - post cath care info |
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Nursing Care Post Catheterization
Extremity perfusion compromise: |
Arterial perfusion compromise of extremity
Signs: - pallor, mottling, diminished strength of extremity pulse (PPP is important), cool temp, delayed cap refill |
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Venous obstruction:
|
signs:
- edema of extremity, duskiness with normal cap refill - venous congestions interferes with arterial circulation |
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Nursing Care Post Catheterization
|
- monitor for bleeding
- fam/child anxiety and knowledge deficit - resp compromise - renal function alterations - pain - hypothermia |
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Obesity
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Classification of Overweight and Obesity in Adults
|
BMI = kg/m2
underweight <18.5 Normal weight 18.5 - 24.9 overweight 25-29 Obesity >30 |
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Childhood obesity?
|
- for kids, BMI >85 - 95% = overweight
- so we consider age, gender, height and weight - WHO defines as abnormal fat accumalation that presents a risk to health |
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Prevalence rates of obesity:
|
- increases, from 10.6% to
32.6% in boys and from 13.1% to 26.6% in girls for overweight, and from 2.0% to 10.2% in boys and from 1.7% to 8.9% in girls for obesity (Tremblay, Katzmarzyk & Willms, 2002). ! Over a 15-year period in Canada, the prevalence of overweight and obesity has tripled among boys (i.e., from 10.6% to 32.6%) and doubled among girls (i.e., from 13.1% to 26.6%). ! This suggests that not only have children become more overweight in the past few decades, but overweight children have been getting heavier. |
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Complications of obesity
|
Slide #10 of obesity presentation...
Think. Psychosocial pulmonary GI renal musculoskeletal neurological CV endocrine |
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Health promotion in Children and Youth
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- Health of children is a product of genetics, the role models they copy, their environments, the skills and attitudes of
parents, and the adequacy of health services. - Requires advocating and accomplishing changes in behavior of the adults significant to the child. " Support people to make healthy decisions " Education in schools " Policy and program development " Advocacy groups " Online resources " Pediatric obesity clinics |
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Key messages in decision making tools
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- Fully mobilizes client centred care, clients need support to participate in decision making.
- Usual approaches to decision support are not enough. - Decision coaching tailors support to the need of the client. |
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Decision support analysis tool. Knowledge
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! Do you know the benefits and harms of each option?
! Present Options: Benefits and Harms - make it client centered - don't put anything into his mouth or his mind |
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Decision support analysis tool.
Value |
" Are you clear about which benefits and harms matter to you the most?
" Discuss importance of benefits and harms. - how important is this shit to you? |
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Decision support analysis tool.
Support |
" Do you have enough support and advice to make a choice?
" Discuss preferred role in decision making, does the client want to involve family? " Does the client feel pressure or support from others? |
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Decision support analysis tool.
Certainty |
- do you feel sure about the best choice?
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Stroke
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! Stroke strikes 3/10000 children each year in Canada
! Recent trend in pediatric strokes caused by: -untreated ear infections, -chicken poxs -too much cow’s milk. ! Can also be caused by trauma: car accident ! Unknown reason….. ======================= ! A stroke is when the blood supply to any part of the brain is interrupted, resulting in tissue death and loss of brain function. ! 2 kinds: 1. Ischemic stroke & 2. Hemorrhagic |
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Stroke Misdiagnosed
|
" Bells Palsy (facial paralysis)
" Sore or injured leg " Injury that occurs in infancy and are only diagnosed when the child not meeting developmental milestones. |
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An ischemic stroke
|
may be caused by a blood clot that forms in the brain (a
thrombus) or blood clot or plaque that travels to the brain from another location (an embolism). |
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Hemorrhagic stroke
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Hemorrhagic stroke
! A hemorrhagic stroke is bleeding that occurs within ischemic brain tissue. ! Hemorrhagic stroke occurs when a blood vessel that is damaged or dead from lack of blood supply (infarcted) ruptures. |
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Signs and symptoms of pediatric stroke
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! Blindness in one eye or hearing problems in one ear
! Confusion ! Dizziness or loss of balance/coordination ! Nausea and/or vomiting ! Numbness or weakness on one side of the body – the side opposite from where the stroke has occurred ! Seizures ! Severe headache ! Trouble speaking or understanding speech |
|
Diagnosis methods for pediatric
stroke |
! Physical exam
! CAT scan ! MRI ! Ultrasound ! EEG |
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TREATMENT stroke
|
! Aspirin
! Heparin and Low Molecular Weight anticoagulants. ! Warfarin ! Thrombolytic Agents |
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Factors affecting childhood obesity
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! Access
! Socioeconomic Factors ! Lifestyle ! Technology ! The nature of the food supply ! The food industry ! Portion sizes |
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Premature Rupture of Membranes
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• PROM
– Premature rupture of membranes – Before labour begins • Preterm PROM (PPROM) – Before 37 weeks • Associated with – Infection – Polyhydramnios – Incompetent cervix – Trauma – Bleeding problems |
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Risks
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• Maternal
– infection increases (Chorioamnionitis) – malpresentation – abruptio placenta • Fetal-newborn – prolapse of umbilical cord – respiratory distress syndrome – sepsis • ↑↑ perinatal morbidity and mortality |
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(P)PROM
|
Confirm
• -nitrizine, check the acid base comp of the fluid - monitor infection of the mom (temp, increased HR • Sterile speculum – Ferning on slide as seen through microscope If term… – Monitor maternal and fetal vital signs – If prolonged • antibiotics – Induction |
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Management If preterm
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• Fetal assessment
• Maternal vital signs – Baseline, q 4h • Fix underlying problem and infection • Avoid vaginal exams • Monitor wbc • Conservative management • Corticosteriods to increase lung maturity (betamethasone) |
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Corticosteroids in PTL
|
• 24 and 34 weeks’ gestation, at risk of preterm delivery within 7 days should be considered for antenatal treatment with a single course of corticosteroids.
• reduces perinatal mortality, respiratory distress syndrome, and intraventricular hemorrhage • Betamethasone 12 mg IM q 24 hr x 2 doses or • Dexamethasone 6 mg IM q 12 hr x 4 doses |
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Preterm Labour / Birth
|
• Rate of PTB Canada ~ 8.1%
• Rate of PTB increasing! • Leading cause of mortality & morbidity (~75-85%) • Preterm infant • Stress to family • Costs to system – $12,354 compared to $1,084 (2005-6) • The earlier gestation of previous PTB, inc risk of even earlier PTB next time |
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Risk Factors for PTL/B
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• Previous preterm delivery/fetal loss
• ART • Hypertension (X6) • Uterine / cervical abnormalities • Multiples • Polyhydramnios • Antepartum bleeding (abruptio placenta) • Leading cause of mortality and morbidity (~75-85%) • Substance use – Cocaine / tobacco use (>11 cig./day) • Intra-abdominal surgery • Medical conditions - diabetes, obesity • Physical trauma – domestic abuse • Psychosocial – anxiety, depression, stress |
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Preterm Labour
Common symptoms: |
– Low abdominal pain /cramps / backache
–Bleeding / spotting / show / ROM –Pelvic pressure - baby pushing down – Increased amount / changes in vaginal discharge –Contractions q 10 min or more often |
|
Assessment
|
• Maternal vital signs / FHR / contractions
• Sterile speculum exam – Membranes intact/ruptured? – obtain vaginal cultures – infection? • CBC / Urinalysis - infection • Ultrasound: – verify gestation – placental location / fetal presentation – transvaginal scan - cervical length, incompetent cervix |
|
Diagnosis
|
• Uterine contractions
(4 in 20 minutes or 8 in 1 hour) • Cervical – Length <25mm – Dilatation of greater than 1 cm – Effacement of 80% or more • + Fetal fibronectin |
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Population-based strategies include:
|
• raise public awareness of problem of prematurity
• preconception preparation • nutrition, avoidance of drugs / tobacco • patient education on risk avoidance & recognition of early symptoms • Early antenatal care and early detection of risk factors - bacterial vaginosis, cervical incompetence. |
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Management of PTL
|
• Should labour be stopped?
• Monitor VS, contractions, fetus – Bedrest – Magnesium Sulfate – Sedatives • Avoid stimulation – Vaginal exams – Sexual intercourse – Nipple stimulation – Keep bladder empty • Hydration - IV? • Antibiotics RoM or infection • Corticosteroids • Tocolytics: – Indomethacin anti-prostaglandin inhibits uterine activity • Postpone delivery x 48 hours • Not recommended for long term use - PDA – Calcium channel blockers - Nifedipine (Adalat) – Beta adrenergic agonists (terbutaline, ritordrine) • Progesterone – Weekly IMs, helpful to prevent and reduce PTB when Hx of PTB, and other risk factors |
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If labour can’t be stopped…
|
• The more preterm, the greater the risk and adverse outcomes
• Where should she deliver? • Very little difference in length of labour • may not need to be 10 cm to deliver VLBW infant • Narcotics rarely used – epidural • May use forceps; not vacuum |
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Incompetent cervix
|
• Premature, painless dilatation of cervix
– 20-28 weeks – 2nd trimester abortions – Anomalies of cervix (invasive cervical biopsy, DES) – Infections – Multiple gestation, polyhydramnios • Diagnosis – Heaviness in pelvic area, membranes rupture – Ultrasound, delivery |
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Cervical Cerclage/Suture
|
Shirodkar, McDonald Insertion associated with
– Infection, – Hemorrhage – PPROM, Preterm labour – Damage to cervix • Permanent narrowing or closure • Tearing of the cervix or uterus if labour progresses with the stitches still in place – Not appropriate if • vaginal bleeding • uterine contractions • membranes have ruptured • C-section or suture removed |
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Multiple Gestation
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- >1 fetus
- 1974: 18.2/1,000 births - 1997: 25/1,000 births - 340% increase in triplet births - 25 per 100,000 births (1974 ) = 109 in 1997 2 factors: 1. assisted reproduction (ART) - clomid treatment, superovulation, invitro fertilization, intrauterine insemination 2. increasing maternal age |
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Identical-
Monozygotic |
• Develop from single fertilized ovum
• Same sex and same genotype • Increased risk of anomalies • Identical twins usually have common placenta • # of amnions, chorions, and placentas – depends on timing of cell division cells in 1st 2 wks |
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Fraternal-Dizygotic
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• 2 separate ova fertilized
by 2 separate sperm – 2 placentas, 2 chorions, 2 amnions – Sometimes placentas fuse and appear to be one – No more similar to each other than other siblings – May be of same or different sex |
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• Singleton
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– 6.3% weigh <2500g
– 7% < 37 weeks – Mortality 4.1 per 1000 |
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Twins
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– mean age 35.2 weeks
– 56.6% weigh <2500g – Mortality 25.7 per 1000 – 97% < 37 weeks |
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Triplets
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– mean age 32.1 weeks
– 94.1 % weigh <2500g – Mortality 62.2 per 1000 |
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Quadruplets
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– mean age 29.7 weeks
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Education, DX, nutrition of a mom carrying multiples
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• Educate
– prevention and symptoms of pre-term labor • Diagnosis – ideally by 5th month • Nutrition – counselling and dietary resources to support a weight gain of 18-27 kilos (40-60 pounds) • when health of the mother or family situation warrants: – extended work leave – bed rest support – child care for siblings |
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Effects on pregnancy/mother
|
- increase intensity of all pregnancy related symptoms/complaints
.Shortness of breath .Edema . Nausea, vomiting, heartburn . Insomnia, fatigue .weight gain • Increased risk for: – Preterm labour – Anemia, diabetes, hypertension, cardiac, renal problems – Abnormal presentation – Twin-to-twin transfusion syndrome – Vanishing twin - Fetal reabsorption – Uterine dysfunction – Abruptio placenta / placenta previa / pph – Prolapsed cord • Safest way to deliver? – vaginal or C/S |
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Management
|
• Prenatal
– Intermittent or continuous EFM – simultaneous auscultation / recording of fetal heart rates – Ultrasound – Monitor blood sugar, blood pressure etc. – Awareness of complications – Rest • Head of bed elevated, left lateral • Delivery – Complications • Placenta previa, preeclampsia etc. } C/S • IUGR, malpresentation, fetal distress, anomalies: } – time from birth of 1st twin to birth of others – U/S confirm presentation of 2nd twin –Careful ID of infants / blood specimens |
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Effects on family
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• PTB
– NICU • Instant family – Financial • Space, equipment • Social –marital • Sleep deprivation • Time • Loss of one fetus –Selective terminations |
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Precipitous Labour & Delivery
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- < 3 hours
Mother . Lacerations . PPH Infant . Stress or hypoxia from inc contractions . Facial bruising . Airway . excessive secretions . Cerebral trauma Will try to prevent in future . induction |
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Macrosomia
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> 4000grams
• Male • Gestational Diabetes • Grand Multipara • Postdates (>42 weeks) • Ethnicity |
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• Mother/pregnancy/delivery at risk for
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– Cephalopelvic disproportion
– Shoulder dystocia – Dysfunctional labour – Vaginal lacerations • 3rd 4th degree tears – Postpartum hemorrhage |
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• Fetus at risk for
|
– Meconium aspiration
– Asphyxia – Brachial plexus injury, fractured clavicles |
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Shoulder Dystocia
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• Fetal shoulders impact against the mother’s symphysis pubis
• Head delivers normally, but then pulls back – Turtle sign • Macrosomia • Cephalo-pelvic disproportion • Mismanaged delivery • Risks to baby – Brachial plexus injury, fractured clavicles, fetal anoxia from cord compression |
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Obstetrical Emergency
Turtle Sign |
• Get help – obs (NICU)
– Techniques: • Open pelvis (McRoberts, hands-knees) • Dislodge 1 shoulder (Suprapubic pressure) • Rotate the fetal shoulder girdle into the wider oblique pelvic diameter (Woods screw) • Posterior arm sweep – Ideally anterior shoulder is delivered IMMEDIATELY after head to minimize risk of cord compression • Support patient/support person |
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McRobert's Manoeuvre
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- pelvis tilts, orienting symphysis more horizontally to facilitate shoulder delivery
|
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Suprapubic Pressure
|
• NOT fundal pressure
• Lateral pressure in direction of fetal nose • Protect your back • Support/inform patient +support person |
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Shoulder Dystocia
Complications Maternal |
- episiotomy
- extended lacerations - hematomas - uterine atony - hemorrhage - bladder injury - rectal injury |
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Shoulder Dystocia
Complications Fetal |
- # of clavicle or humerus
- bachial plexus injury or spinal nerve damage - Erb's palsy - asphyxia - mentla retardation - death |
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Persistent Occiput Posterior
(POP) |
- 25% of pregnancies at term
- Must rotate 135 o ROP -> ROT -> ROA->OA Maternal Risks - intense pain in small of back - often longer labour - if delivers „³ 3o or 4o laceration |
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POP - Nursing Implications
|
• Encourage change of position
• Knee-chest or all 4’s position • Comfort measures –Back pain, counter-pressure, shower • Encourage to void Q2h • Assess stress & coping – information about what’s happening |
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Breech
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• 3-4% of births
– 25-26 weeks – 21% incidence – 40 weeks – 3-4 % incidence • Etiology: – prematurity – uterine or pelvic structural abnormaility – placenta previa – polyhydramnios – multiple gestation – fetal anomaly or abnormality – chance |
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Diagnosis of Breech
|
• Maternal perception of movement
• Leopold/s Maneuver’s – hard, ballotable part in uterine fundus • FH auscultated above umbilicus –pre-term or small fetus - may be below umbilicus. • Vaginal examination • U/S • Thick meconium (if ROM) |
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External Cephalic Version
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• 36+ weeks
• Ultrasound • Reactive NST • Can’t be engaged • Tocolytic to relax uterus • Risk of cord around neck/body • Fetal Monitoring • Monitor for SOL |
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Term Breech Delivery -
What is the best way to deliver? |
• U/S
–Confirm position, size • Best method of delivering an uncomplicated term frank or complete breech singleton, >2500 and <4000g with flexed head = vaginal birth (2009) • Footling=c/s –Unless advanced labour |
|
Fetal Risks
|
- Cord prolapse
- Traumatic injury to aftercoming head leads to intracranial hemorrhage or anoxia - Preterm breech - footling & body may deliver before full dilatation = entrapment |
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Vaginal Delivery of Breech
|
• Assessment of FH / vaginal exam with
ROM • Bladder empty • Practitioner skilled in breech delivery, anesthetist, NICU • Do not pull or tug on body as delivering – maintain flexion |
|
Cord Prolapse
|
Obstetrical Emergency
• Sudden, severe, variable decelerations or no FHR – Feel or see cord Causes • Malpresentation )Breech, transverse) • Polyhydramnios • Premature rupture of membranes • Amniotomy before engaged vertex • Long cord • Always check fetal heart after membranes rupture!! • Get help • Prepare for c-section • Keep pressure off cord – Knee-chest position -- Keep hand in vagina – 02 – Warm wet sterile cloth if outside vagina • Vaginal delivery if fully dilated |
|
Cephalopelvic disproportion
(CPD) |
• Fetus is larger than pelvic diameters
– Size of baby – Type of pelvis – Other anomalies • Dystocia – Painful, prolonged labour – Delayed engagement |
|
Management
|
• Change positions
– Squat or sitting – Increases outlet – Knee chest, all fours, side to side • Pain relief • Forceps – May make it worse • C-section |
|
Assisted / Operative Deliveries
Vacuum, Forceps, C-section |
• Indications
– Fetal distress – Maternal distress • Exhaustion • Anesthesia - Inability to push • Lack of rotation Interventions during – Support, tell her what is happening – Fetal heart (Oxygen) |
|
Prevention
|
• Support throughout labour
• Mobility • Position changes (walking) • Rest • Keep bladder empty • Well hydrated and nourished |
|
Forceps
|
Types
– Outlet – Low – Mid pelvis • Rotate or traction Must be – Completely dilated – Membranes ruptured – Adequate pelvis - no CPD – Bladder empty - May need episiotomy |
|
Forceps - Complications
|
• Newborn
– Bruising – Edema – Facial lacerations – Cephalhematoma – Transient facial paralysis – Cerebral hemorrhage • Woman – Vaginal or perineal lacerations – Infection secondary to lacerations – Increased bleeding • Risk for PPH – Bruising and edema |
|
Vacuum Extractor ~14%
|
- Suction to fetal head
(occiput) - Should be progressive descent with first two pulls (with contractions) - ^ risk - cephalhematoma - jaundice due to reabsorption of bruising - breastfeeding |
|
Caesarean Section~26%+
|
• Fetal distress
• Active genital herpes • Multiple gestation (3+ fetuses) • Umbilical cord prolapse • Tumors that obstruct birth canal • Lack of progress • Maternal infection • Pelvic size disproportion • Placenta previa • Previous cesarean section |
|
Preparation
|
• Establishing IV lines
• Indwelling catheter • Abdominal prep • Teaching – What to expect before, during, and after delivery – Why is it being done – What sensations will the woman experience – Role of significant others – Interaction with newborn (NICU @ delivery) |
|
Vaginal Birth after C-section-VBAC
|
• ~75% success - depends on
– Cause of 1st section – Type of 1st section – Previous vaginal birth – Maternal physical and mental health • Trial of labour (TOL) • Most common risks are – Hemorrhage – Uterine rupture 0.6% up to 9% in classical scar – Infant death or neurological complications |
|
Care
|
. Continuous EFM
- internal monitor . Monitor uterine contractions . IV fluids in progress . Avoid oxytocin if possible - inc risk of rupture . Avoid cervical ripening - Increases risk for rupture X4 . Support . Prepared for C-section short notice - Would have had pre-op work-up - Type and Cross |
|
3 types of women…those who…
|
1. prefer to give birth without any pain relief
2. sure they will want pain relief 3. unsure of their pain relief options, and how they will affect their labour and delivery • All need support person/labour companion – Doula, husband, someone there for her • Your role – support their efforts to have a natural childbirth, if that is their goal or facilitate other plans they may have – Optimal outcome |
|
Methods of Childbirth
Preparation |
• Grantly Dick-Read – childbirth without fear
– Breathing, psychoeducation, relaxation • Lamaze – Uses specific breathing patterns – Controlled muscular relaxation techniques – Disassociation relaxation – Use of gentle touch and verbal cues – Relaxation may also be promoted by cutaneous stimulation (Gate control theory) – Abdominal effleurage • Bradley – Partner-coached childbirth – Slow deep breathing – 12 week education |
|
purpose of all methods
|
– promote relaxation and save energy
– “fight or flight” • Diverts energy away from uterus and gut – Helps contractions and labour – Maximum oxygen to mother and babe – Helps to maintain alertness and presentness • Maintain control – Relax face, hands, bottom – Calmly talk through each contraction – Take over in helping with breathing or relaxation when support person needs a break • Ask them what you can do |
|
Positions
|
• Impeded by monitor
– Telemetry – regional anesthesia – IV-induction, augmentation – shyness • Promote normal circulation and relaxation • Promote labour and relieve discomfort – Especially if baby posterior • Keep active in early labour – Stay home until contractions well established – Walking • Facilitate any position she is comfortable in – pelvic rocking – pillows – slow dancing with partner – sitting on birth ball and swaying – Sitting on toilet – lifting up the abdomen **Look at pics slide #11** |
|
Birthing Balls
|
• Helps to increase balance in pregnancy
• Eases back labour • Sitting in a supported squat widens pelvis outlet to the maximum |
|
Environmental considerations
|
• Dim lights
• Peaceful surroundings – moaning and groaning • Privacy – Manage visitors • Warmth or coolness, fan • Music • Talk quietly • Avoid interruptions • Clean – Keep bed clean and dry, smooth wrinkles – Peri care • Safe |
|
Water - hydrotherapy
|
• Safety getting in and out
• Buoyancy of water and warmth-relaxing • Positions - been used for a long time throughout the world |
|
• Heat use for labor
|
– deep tub bath, birthing baths, especially in difficult labours
• Can do FHR and Vital signs • Hard to know if membranes slow leak – Shower • Distract, calm, refresh – DO NOT LEAVE UNATTENDED – labour companion or nurse • Emergency buzzer – heated rice sock on groin or back |
|
Cold use for labor
|
– ice packs on lower back
– cool cloth to wipe face, lips |
|
Massage
|
• Heel of hand to press firmly on the bottom of spine, massaging in small circles
• Thumbs to massage on either side of spine • Massage shoulders, taking care to keep the movement slow and firm. – helps keep breathing relaxed • Use long stroking massage down spine – first with one hand and then the other • Counterpressure during contractions – Ask exactly where to push and how hard to push – Back, hips – Method of communication |
|
Imagery for labor
|
– visualize picture a peaceful place
• Beach or mountain stream • Any place she likes to go • Womb opening and baby moving down pelvis |
|
Hypnosis for labor
|
– low-risk way of managing labour pain and anxiety that works for some women
|
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Distraction
|
– early labour, walk, play cards, knit, watch TV, shower
|
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Non-focused awareness
|
– paying attention to everything a person sees, hears, feels, smells without focusing on any
|
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• Acupuncture for labor
|
– Few small studies - low-risk, effective way of managing
labor pain for some women |
|
• Acupressure for labor
|
– Number of specialized sites
|
|
Aromatherapy
|
– emenogogues, promote menstrual bleeding may induce
miscarriage, oils not to be used include: • Basil, clary sage, cedarwood, cypress, fennel, jasmine, juniper, lemongrass, marjoram, myrrh, origanum, parsley, peppermint, rose, rosemary and thyme. . • chamomile and lavender must be avoided during the first trimester, but after that can be very beneficial for certain conditions • Labour comfort and relaxation – 2 drops of lavender, 2 drops of geranium and 2 drops of lemon or bergamot added to a bowl of hot water |
|
TENS.
|
Transcutaneous Electrical Nerve Stimulation
• Electrical Analgesia • Pulses prevent the pain signals from uterus and cervix from reaching brain – stimulate release of endorphins • Can take up to one hour to work effectively - therefor practice and use beforehand |
|
Pharmacologic use
|
• Provide maximum pain relief with minimum risk to mother and fetus
• Remember – All systemic drugs used in labor for pain relief cross placental barrier by simple diffusion – Drug action in body depends on rate at which substance is metabolized by liver – Fetus has inadequate ability to metabolize analgesic agent |
|
Medications
|
• Labour established or may establish!
• Demerol, Fentanyl, and Morphine – 1st stage of labour – Patches, IM, or IV at the peak of the contraction • Side effects – sedative effect and makes women drowsy and their breathing shallow. – nausea – cross the placenta to the baby may cause fetal respiratory depression at birth if given too late in labor • Narcan, opoid receptor antagonist, IV – Caution use in addicted mother….. |
|
Nitrous Oxide N2O
|
• “Laughing gas”
• ENTONOX – 50% oxygen/ 50% nitrous oxide • Self-administered • Quick relief • At onset of contraction not pain • Rapidly eliminated • Short effect to fetus • All stages of active labour and postpartum • Claustrophobia, mask fear |
|
Epdidurals
|
• May delay or speed up labour
– Timing of administration • > 4cm • May impede breastfeeding • Mothers may spend more time in bed and less time with baby in the hospital • Pushing during second stage of labour may be impaired due to lack of sensation • anesthetic (bupivacaine) or analgesic (morphine or fentanyl) injected into epidural space • Active labour >4cm and <6cm • Produces little or no feeling to area from uterus downward • May preload with crystalloid solution bolus – NS or RL • May need urinary catheterization due to loss of bladder sensation • Assess sensation motor control and orthostatic blood pressure • Assess level of effect |
|
Continuous Epidural Analgesia
“walking epidural” |
- Allows for ambulation and mobility promote contractions
- ^ control over body compared to classic anesthesia epidural - Cocktail of medications - Narcotic e.g. fentanyl, morphine, a local anesthetic and epinephrine used in smaller amounts than the regular epidurals - Designed to provide pain relief still be aware of contractions - will not mask extraordinary pain that we need to be aware of - e.g. abruptio - Not all women will be able to walk - refuse (15-25%), leg weakness "not feeling normal", hypotension |
|
Combination spinal-epidural
|
• before the epidural line is installed, medication is injected into the spinal fluid around the spinal cord
– This spinal injection acts more quickly than epidural – Then the epidural line is placed and used for continuous anesthesia |
|
Spinal Block
|
• Local anesthetic (analgesic) injected directly into spinal canal
• Quick acting • Level of anesthesia dependent upon level of administration • May be administered higher for cesarean birth or lower for vaginal birth • Onset of anesthesia is immediate – Takes time to set up • Protect legs from injury for 8 to 12 h postpartum due to decreased movements and sensation • Total spinal anesthesia – Respiratory and cardiac arrest |
|
Potential Complications with Anesthesia
|
- Regional anesthesia
- spinal or epidural, similar possible complications - Maternal hypotension - bolus IV fluid and notify anesthetist - Bladder distension - Inability or reduced ability to push during 2nd stage - Elevated temperature with epidural anesthesia - Possible neurological damage - Wet tap: needle inadvertently enters the subarachnoid space = rapid leakage of CSF „_ headache. - Epidural Blood Patch (EBP). 15-20 ml of patient¡¦s own blood injected into the epidural catheter after epidural block had worn off completely |
|
Pudendal Block
|
• Local anesthesia
– inject pudendal nerve – anesthesia to lower vagina, vulva, perineum • End of labour • No effect on fetus or progress of labour • Potential complications – hematoma, perforation of rectum, trauma to sciatic nerve |
|
Local Anesthesia
|
• Anesthesia (lidocaine) injected into perineum prior to episiotomy
• Provides pain relief only for episiotomy incision • There is no effect on maternal or fetal vital signs – If done correctly • Requires large amounts of local anesthetic agents |
|
General Anesthesia
|
Urgent or Emergency situations
- C-section, fetal distress, retained placenta, failed regional Challenges in the pregnant woman - Increased mortality and morbidity - Changes in oral mucosa, edema, obesity - Higher Mallampati Classification - amount of the posterior pharynx visible - Increased esophageal reflux - Delayed gastric emptying in labour - Not NPO - ^ O2 consumption, „` functional respiratory capacity - Positioning |
|
Nursing Care
|
• Monitor fetal heart EFM
– Disconnect from wall (also unplug bed!) • Assess when last ate or drank • Administer prescribed pre-medication – Antacid (sodium citrate) – Antihistamine ? Impact on breastfeeding • Avoid supine hypotension • Oxygenate |
|
CRICOID Pressure
|
CRICOID only upper airway cartilaginous structure that is
complete ring, prevents passive regurgitation and aspiration of gastric contents during induction of anaesthesia FIGURE 20–7 Proper position for applying cricoid pressure “Sellick’s manoeuvre” until a cuffed endotracheal tube is inserted. The cricoid cartilage is depressed 2 to 3 cm posteriorly so that the esophagus is occluded |
|
IV Considerations
|
• Ensure IV access is established and maintained
– Large bore #18 • Urinary catheter • EKG pads • Tell them what is happening • Reassure, touch… |
|
Major Complications
|
• Fetal depression
– If mother receives general anesthesia = infant respiratory depression – Avoid when infant is considered high risk • Uterine relaxation – Most general anesthetic agents cause some uterine relaxation – Synoticinon • Monitor pp • Vomiting / Aspiration – Stomach may not be emptied prior to anesthesia – Anesthetic agents may also cause vomiting and aspiration |
|
Many ways to help the woman in
labour |
• Many things can happen not according to their plan or their ideal
– Fetal distress – Physicians or other care providers – Respect their decisions • Always reassure them they have done well – Allay anxiety • Explain • Support • Assess – Be there for and with her |
|
Definitions of Pain
|
1.IASP (International Association for the Study of Pain) - genesis of pain –mind, body, spirit
2. McCaffery- Expert on pain is the person experiencing it. |
|
History of Pain
|
Interventions date back and are recorded in the book of Genesis.
Historically how people viewed the mechanisms of pain influenced the strategies to treat pain. |
|
Theories of Pain
|
Specific Theory of Pain
Pattern Theory **Gate-Control Theory** ================= - theories are testable but not the absolute truth |
|
Factors that Influence the Gate
|
Open gate increases nociceptive impulses
Partially or closed gate decreases nociceptive impulses |
|
Assessment & Measurement of Pain
|
Consider=
- Developmental level of child - Child’s own assessment of pain - What is the stimulus for pain - Parents assessment of their child’s pain -The child’s language for pain |
|
ABC’s of Pain Assessment in children
|
1. Always evaluate the child’s
2. Be careful to assess the physical aspects of the child’s pain, as an integral part of the physical exam 3. Consider the impact of family, health care, and environmental factors on the child’s pain. 4. Document the child’s pain severity on a regular basis. Use a pain scale that is appropriate. 5. Evaluate the effectiveness of pain interventions regularly and modify the treatment plan as necessary until the child does not have pain. |
|
Practice Tips
|
1. Be honest
2. Use appropriate language 3. Clinicians should be competent with children (no students doing shit!!!) 4. When possible procedures conducted in a designated treatment room 5. Parents should be present if they wish 6. Parents should not be asked to restrain their child. 7. Child should be given the opportunity to ask questions. 8. Appropriate drug therapy 9. Treatment should always be holistic |
|
Modern pain management
|
- prevent pain
- utilize new drugs and combinations - titrate with individual pts - use timed dosing |
|
Traditional pain management
|
- palliate pain
- stay with a few tried & true drugs - stay with low dose - use PRN dosing |
|
Adjuvant drug classes
|
- antihistamines
- anticonvulsants - antidepressants - anxiolytics |
|
Number 1 side effect of opiates is...
|
- CONSTIPATION!!!
- resp depression only happens if you give the wrong dose |
|
Demerol / Mperidine
|
- synthetic drug
- supposed to have less addictive qualities then the real shit - supposed to replace morphine - metabolism creates nor - mperidine - builds up on receptor sites - clings to the sites, therefor more drug = more bi- product = ppl start to get twitchy = seizures - and over time, you get less pain relief because it keeps binding up on sites *** A dirty drug *** |
|
Bleeding During the First and
Second Trimester |
• Abortion
– Expulsion of the fetus before 20 weeks’ gestation – < 500g – Spontaneous: • Occur naturally • Uncertain number up to 30% –50% due to abnormal chromosomes – Induced • medical or surgical • Genetic, social – Threatened, imminent, incomplete |
|
3 Types of Abortion Cases
|
1. Threatened
2. Imminent 3. Incomplete |
|
Care for a Bleeding Lady
|
Threatened abortion or miscarriage
Bed rest ?abstinence from sex Support , explain Persistent bleeding: - Hospitalization - IV therapy or blood transfusions - Dilatation and curettage (D&C) or suction evacuation |
|
Ectopic pregnancy ‘tubal pregnancy”
|
Implantation of fertilized ovum outside uterus
- 0.4-1.9 % pregnancies, prevalence increases Initially symptoms of pregnancy - Positive hCG Chorionic villi - grow into tube wall or implantation site Rupture and bleeding into the abdominal cavity occurs - Result is sharp unilateral pain and syncope - Referred shoulder pain - Lower abdominal pain Vaginal bleeding Medical: IM methotrexate (stops growth of rapidly growing cells) if future pregnancy desired Monitor: hCG levels Surgical: Salpingostomy or salpingectomy |
|
Bleeding During the Second
Part of Pregnancy |
Two main types
–Placenta previa –Abruptio placentae |
|
Abruptio Placentae
|
1 in 1000 births
• is increasing Marginal placental abruption. Placenta separates at its edges. Blood passes between fetal membranes and uterine wall. Blood escapes vaginally Slides #8-9 |
|
Causes of Abruptio Placentae
|
• Decreased placental blood flow
– Hypertension, pre-eclampsia – Smoking – Cocaine – Alcohol • Trauma – MVA, IVP • Multiple gestation • Increased Parity • Maternal age |
|
Maternal Implications
|
• Intrapartum and postpartum hemorrhage
• DIC (Disseminated intravascular coagulation) • Hypofibrinogenemia • Ruptured uterus – overdistention • Hemorrhagic shock • Renal failure |
|
Fetal-Neonatal Implications
|
• Sequelae of prematurity
• Hypoxia • Anemia • Brain damage • Fetal demise |
|
Immediate Care--one-one care for a Bleeder
|
Assess maternal
– Vital signs Q 5-15 minutes – Monitor blood pressure and pulse frequently – Observe signs of shock • Colour, skin, consciousness – Assess type & amount of bleeding over time period – NPO Prepare 2 large bore IV sites – Fluids and blood products as ordered Monitor fetus and uterine activity electronically – Assess resting tone Q 5-15 minutes – Continuous EFM • Assess fetal status Q 5-15 minutes |
|
Nursing Care for a Bleeder
|
• Oxygen per mask/prongs
• Review and evaluate diagnostic tests – Hgb, PT/PTT, Type and Cross match • Prepare for cesarean, as needed • Assess coping mechanisms of woman and family – Crisis – Provide information and emotional support • Neonatal resuscitation team, as needed • Pain relief, as needed |
|
Placenta Previa
|
• 4 in 1,000 births
I LOW LYING – implanted in lower segment close to os baby can still be born vaginally II MARGINAL – lower edge touches cervical os but does not cover it, baby may be born vaginally III PARTIAL • partially covers os. C/S IV TOTAL • completely covers os. C/S |
|
Placenta previa
• Detection |
– Routine ultrasound
– Must be monitored • 80+% migrate during pregnancy – 34 weeks+ with painless vaginal bleeding as lower uterine segment forms • Goal is to get to 36-7 weeks gestation |
|
Placenta previa associated with
|
• Previous placenta previa
• Abnormal uterus shape – Fibroids - age • Multiparity • Previous C-section or other uterine surgery • Smoking, cocaine use • Unknown |
|
Implications for Placenta Previa
|
• Maternal psychological stress
• Transverse lie common • Changes in FHR • Meconium staining • Fetal compromise (hypoxia) • Cesarean birth • Neonatal anemia |
|
Nursing Care
Placenta Previa |
• Bed rest
• Avoid Constipation • Avoid sexual activity • No vaginal exams! • Objectively and subjectively assess – blood loss, pain, uterine contractility • With active hemorrhage - Continuous external monitoring of FHR and uterine activity - NO internal monitoring |
|
If contractions never go away... Most likely have
|
- Abruptio
|
|
Succenturiate Placenta
|
– One or more accessory lobes of fetal villi develop on placenta = PPH
|
|
Circumvallate Placenta
|
– Double fold of chorion and amnion form ring around umbilical cord on fetal side of placenta
– increased risk of late abortion, APH, preterm labor – May also cause IUGR, prematurity, fetal death |
|
Battledore Placenta
|
– Umbilical cord is inserted at or near placental margin
– This leads to increased chance of preterm labour and bleeding – May also cause prematurity and nonreassuring fetal status |
|
Velamatous Insertion
|
– Vessels of umbilical cord
divide some distance from placenta in placental membranes – If one vessel is torn= hemorrhage – leads to nonreassuring fetal status and/or hemorrhage in fetus |
|
Retained Placenta
|
• Retention of placenta
beyond 30 minutes after birth – Mismanaged 3rd stage • Occurs in 2% to 3% of all vaginal births • If not expelled – Manually removed – if woman does not have an epidural anesthesia in place, general anesthetic |
|
Placenta Accreta
|
• Chorionic villa attach directly to
myometrium of uterus • May result in maternal hemorrhage and failure of placenta to separate from uterus • May result in need for hysterectomy at time of birth • Incidence of placenta accreta is 10% to 25% in presence of placenta previa |
|
Postpartum Hemorrhage
|
• >500ml
• >1,000ml – severe, woman will be compromised, BP may be stable • Bleeding affects 5-15% births • 4 Ts 1.Tone (70%) 2.Trauma (20%) 3.Tissue (10%) 4.Thrombin(<1%) • Immediate – 3rd stage • Later – 4th stage up to 6 weeks |
|
1. TONE - Uterine Atony
|
• Lack of uterine muscle tone
– Overdistended uterus • Twins, polyhydramnios, macrosomia – Grandmultipara – Preeclampsia and MgS04 – Over use of syntocinon during labour – Infection • Fundal expression & massage – check for clots and tissue • Severe – Bimanual expression • Administer uterine stimulants – Oxytocin IV – Hemabate IM |
|
Subinvolution
|
• Usually occurs 1 to 2 weeks after birth
– Failure of uterus to return to normal size after pregnancy – lochia rubra of greater than 2 weeks duration • Provide mother with discharge instructions and information about possible complications |
|
2. TRAUMA - Lacerations
|
• Cervical or vaginal lacerations suspected when bright-red bleeding in presence of contracted uterus
– Precipitous delivery – Forceps – Vacuum – Prepare for • Exploration • Surgery – IV, etc…. • Usually repaired immediately after delivery Hematomas – May not severe hemorrhage but bleed, pain, ecchymosis Risk for: •Infection •Blood loss •Pain management •Keep bladder empty |
|
3. TISSUE
- Retained Placental Fragments |
• 3rd stage mismanagement common cause
– “Fundal fiddling” – Bleeding • firm fundus and no lacerations – Inspect placenta thoroughly after its delivery • Missing lobes – Insertion of vessels – Infarcts |
|
4. THROMBIN
|
• DIC
• Other bleeding disorders |
|
Signs of PPH
|
- excessive or bright red bleeding
- a boggy fundus that does not respond to massage - abnormal clots - any unusual pelvic discomfort or backache - persistend bleeding in the presence of a firmly contracted uterus - rise in the level of the fundus of the uterus - increased or decreased pulse/bp - hema toma formation or bulging/shiny skin in the perineal arean - decreased level of consciousness |
|
General care for PPH
|
Bleeding won’t stop
– IV • Oxytocin • Hemobate • Blood transfusions – Vital signs • Signs of shock – Pack uterus – Cauterization – Hysterectomy |
|
the main cause of PPH PP day 15 is...
|
- retained products of conception
|
|
Polyhydramnios/Hydramnios
|
• Excessive amniotic fluid > 2000ml
• Associated with –Maternal • Diabetes • Rh sensitization – Large placenta |
|
Fetal Anomalies
|
– Hydrops fetalis
• fluid accumulation in the fetus from congestive heart failure, obstructed lymphatic flow – Anencephaly – Cardiac anomalies – Esophageal or duodenal atresia – Neural tube defects • Exposed meninges – Twins |
|
Assessment for amniotic fluid
|
• Fundal height
– disproportionately large for dates • Difficulty palpating fetus and auscultating FHR • Tense, tight abdomen on inspection – Straie gravidarum • Large spaces between fetus and uterine wall on ultrasound |
|
Implications of PolyHydraminos
Maternal |
- sob
- edema - problems with mobility, sleep - greatle increased cesariean rate - uterine dysfunction - abruptio placentae - PPH |
|
Implications of PolyHydraminos
Fetal - Neonatal |
- preterm birth
- increased mortality rate - prolapsed cord - malpresentation - premature rupture of membranes |
|
Care for Polyhydraminios
|
• Provide information
– Ruptured membranes, cord prolapse • Emotional support • Maintain absolute sterility during amniocentesis • Social work/perinatal loss team if fetal defect identified |
|
Oligohydramnios
|
• <500ml
• Postmaturity • IUGR – secondary to placental insufficiency • Major renal malformations – Renal agenesis – Dysplastic kidneys – Lower urinary tract obstructions |
|
Assessment
Oligohydramnios |
• Fundal height
• Fetus easily palpated and outlined • Fetus not ballotable • Variable decelerations (- increased risk of compressing the cord, not enough fluid around to buffer it) • Reduced amniotic fluid volume – AFV on ultrasound |
|
Implications
Oligohydramnios |
• Dysfunctional labor with slow progress
• Fetal deformation defects – Adhesions – Skin and skeletal abnormalities – Pulmonary hypoplasia – Dysmorphic facies – Short umbilical cord • Umbilical cord compression • Head compression |
|
Amniotic bands
|
• Folds of amniotic sac or premature rupture of membranes that have sealed over
– Anomalies – Associated with increased risk of club foot, cleft palate and lip • Can lead to – amputation of digits – permanent bands – malpresentation-breech |
|
Care for oligohydraminos
|
• Provide information and encourage questions
• Evaluate EFM tracing for _________ _________ • Reposition mother to relieve cord compression • Assist with amnioinfusion – 500ml, sterile normal saline • Evaluate newborn – Anomalies – Pulmonary hypoplasia – Postmaturity |
|
Amniotic Fluid Embolism
|
• Amniotic fluid, fetal cells, hair, or other debris enter maternal circulation
• Symptoms – Sudden onset respiratory distress – Acute hemorrhage – Circulatory collapse – Hemorrhagic shock – Coma and maternal death* – Fetal death if birth not immediate • Dyspnea • Cyanosis • Frothy sputum • Chest pain • Tachycardia • Hypotension • Mental confusion • Massive hemorrhage ***Emergency!!!*** |
|
Care
Amniotic Fluid Embolism |
• Emergency team
• Positive pressure oxygen delivery • Large bore IV • CPR as needed • Prepare for cesarean birth • Prepare for CVP line insertion • Administer blood |
|
Grand Multipara
|
>5 pregnancies to viable age 20 weeks
– age-related anomalies • Anemia • Malpositions and malpresentations – Operative delivery • Antepartum hemorrhage - 2 fold increase • Postpartum hemorrhage - 4 fold increase |
|
Obesity
|
Toronto-1994 and 2000
# overweight mothers rose by 37% over the seven years # obese women went up 70% # severely obese jumped 80%, to 1/ 200 women • Associated with – Fertility problems – Increased malformations – Complications – increased difficulty with anesthesia – C-sections (>50%) • Infections • Mobility – Maternal and fetal morbidity, mortality – Fetal anomalies, risk linked to amount overweight |
|
Adolescent
pregnancy |
Increased risk Factors
- substance, poverty, education - less prenatal visits - increased complications - increased hospitalizations Psychosocial - support - school - intergeneratinoal household |
|
Older Gravida
|
• >35
• Decline in fertility • Increase in chronic diseases – Hypertension, cardiac, thyroid, cancers etc. • Increased difficulties in pregnancy – May be 1st episode of these problems or exacerbate them • Increased risk of c-section • Increased anomalies • Increased risk in general • Keeping up with baby! |
|
INFECTIONS
|
• TORCH
–TOxoplasmosis –Rubella –Cytomegalovirus –Herpes Simplex Some resources substitute: –Other (Syphilis, HIV, Hepatitis B) |
|
Toxoplasmosis: Protozoan
|
• Transmission – raw meat, cat feces
• ~23% people are seropositive – If infected before pregnancy no problems, but if infected during pregnancy then risks to fetus – CNS damage, hydrocephalus, microcephaly… • Treatment – Sulfadiazine and pyrimethamine – Given after the 1st trimester |
|
Rubella: Virus
|
Transmission: Across placenta to fetus
– Rubella syndrome • Cataracts/congenital glaucoma, congenital heart disease (patent ductus arteriosus or peripheral pulmonary artery stenosis), loss of hearing, pigmentary retinopathy • Purpura, splenomegaly, jaundice, microcephaly, mental retardation, meningoencephalitis, radiolucent bone disease • Treatment: Prevention – Vaccination of all children – Vaccination of women of reproductive age |
|
A woman's rubella titre is <1:8 at 8wks gestation.
would her doctor order a rubella vaccine postpartum? |
-Yes
-Then after that, tell her to not get pregnant for at least 3 months or else you could eff up the baby |
|
Cytomegalovirus
|
• >40% of people are seropositive
• If acquire primary CMV in pregnancy - fetal neurological, eye, ear, and liver damage • No treatment, prevent with good handwashing and avoid crowds of young children |
|
Herpes
|
• Transmission congenital or intra or postpartum
• ↑ 70% transmitted during asymptomatic shedding • increased spontaneous abort, preterm, lbw, microcephaly, CNS complications • Asymptomatic at birth ~ 2 weeks may develop fever, vesicles • Suppressive antivirals ~36 wks – acyclovir • ROM - 40% vertical transmission • Vaginal birth unless active or prodromal symptoms • Breastfeeding check (unless active lesions on breast) • Hand washing++ |
|
Bacterial Vaginosis -- BV
|
• 10-25% of all women
• 50% asymptomatic – lactobacilli and more Gardnerella vaginalis, anaerobes, and mycoplasmata – itching, burning, copious white discharge • Infections = – Spontaneous abortions – Preterm delivery – Maternal and fetal morbidity and mortality |
|
Group B streptococcal
infection-GBS |
• 15-40% women colonized
– Urine, uterus=pain – Rectum, vagina = asymptomatic • 40-70% pass on to baby Risk • Preterm labour (with or without ROM) • PROM --PPROM • Unexplained T in labour • Previous baby with GBS infection • Bladder or kidney infection – GBS bacteria – Culture |
|
Baby 1-2% babies affected by GBS
|
1. Early onset: especially preterm, 1st week
– Temperature dysregulation, fever, seizures, respiratory distress, unusual behaviour, stiffness, extreme limpness – Up to 25% mortality 2. Late onset: 2-4 weeks – Problems feeding, meningitis, grunting resp, fever, seizures, extreme limpness – 10% mortality – Up to 20% of the babies who survive GBS related meningitis - permanently handicapped |
|
Treatment for GBS
|
• Prevention and early identification
– Screen ~ 35-37 weeks • IV antibiotics – IV -- penicillin G, erythromycin, clindamycin • Neonatal infection treated with antibiotics |
|
Hepatitis C - HCV
|
• 0.2-0.5% pregnant women in Canada
• Vertical transmission 5-6% • Acute and chronic liver disease – HELLP • Universal screening for HCV is not recommended – should be offered to all women falling into any at-risk category • Treatment – Interferon and ribavirin therapy – teratogenic • Breastfeeding check |
|
Hepatitis B
|
• Vertical transmission
• Breastfeeding – avoid if cracked or bleeding • May not be symptomatic – Be a carrier for life – Permanent liver damage, death • Immunization – (Birth), 2, 4, 6 month |
|
Human Immunodeficiency Virus
(HIV) 0.1% of pregnancies |
Fetal/neonatal risk
– Passes through placenta (25%) 8 weeks, blood transfer at birth – Antiretroviral therapy has decreased infection rates – Following birth • Positive antibody titer • Passive transfer of maternal antibodies rather than HIV infection – Passes through breastmilk • NO Breastfeeding (~ 3rd world countries - ok) |
|
HIV Care
|
• No tx=25% risk of HIV
• Three-part Zidovudine (AZT,ZDV,Retrovir) prophylaxis regimen –Pregnancy - Oral ZDV daily – Labor - Intravenous ZDV during labor and until birth – Infant - Oral ZDV • Start 8 to 12 hours after birth • Continue for 6 weeks |
|
H1N1 flu virus
|
• May or may not more likely to get the flu
– But less likely to take flu shots – Decreased lung capacity – Systemic suppression of cell-mediated immunity - immunologic shift • If pregnant woman does catch H1N1, especially in 3rd trimester – pneumonia and severe respiratory distress – complications = abortion or early delivery • Tamiflu and Relenza within 48hrs of symptoms • Decrease chance of exposure |
|
Postpartum
Infections |
• C-section - wound infections
• Perineal cellulitis – pain • Mastitis – fever, engorement • Respiratory complications from anesthesia • Retained products of conception – Foul lochia • UTIs • Endometritis – lower abdominal tenderness one or both sides, tenderness elicited with bimanual examination, T>38.3°C |
|
a woman who is 31 wks gestation akss you about taking seasonal flu vaccine, how would you reply?
|
- make sure your husband gets it
- its recommended for preggos |
|
Risks for puerperal infection
|
Bacterial infections
• C-section - 1 • Frequent pelvic exams -2 • Prolonged rupture of membranes • Pre-existing infection • Hemorrhage, anemia • Obesity • Diabetes – UTI, Vaginal infections • Nutritional deficits • Delivery room cleaning practices |
|
There are two types of injuries:
|
1. Intentional – the result of violence such as suicide attempts, rape, and assaults
2. Unintentional – result from falls, burns, drowning, motor vehicle incidents, and other situations commonly referred to as ‘accidents’ |
|
Pediatric injuries
|
Injuries thought of as “Accidents” or “freak accidents” implies that nothing can be done.
However, injuries are preventable and predictable events…to reduce the risk of the potentially devastating effects of unintentional injury, preventions steps must be taken |
|
accidents/injuries in Saskatchewan
|
! Every year approximately 775 children under 6 are hospitalized and 15 die in Saskatchewan
! Injuries are the leading cause of hospitalization and death in children and youth in SK |
|
When caring for children and youth, keep in mind.
|
! Kids are really not just small adults
! airway and shock mgt paramount ! head injury: increased morbidity & mortality ! forces over small area = multisystem injury ! little or no external injury ! kids die from hypoxia and resp arrest ! increased heat loss, glucose & fluid requirements ! psyche sequelae – you not only have an injured child, but also an injured family! |
|
What makes children more susceptible to accident/injury?
|
Airway:
Anatomy increases obstruction risk ! Large head ! Short neck ! Small mandible ! Large, posteriorly-placed tongue - think about the effects of these anatomical features to incubation ====================== |
|
Pediactric Breathing
|
! Increased respiratory rate
! 30/min = ? normal for small child ! Slowing rate = impending arrest Small thorax ! Transmitted breath sounds ! Misleading findings on auscultation ! Inspection, palpation more reliable ! Diaphragm breathers ! Pliant chest walls ! Weak accessory muscles ! Limited respiratory reserve |
|
Leading Cause of
Pediatric Cardiac Arrest |
Resp Failure
|
|
Pediatric circulation
|
! Small blood volume
! Rapid control of blood loss essential ! Good initial compensation for hypovolemia ======================== BP monitoring ! Poor method -low BP LATE sign: kids compensate well > 25% loss of blood volume (think s/s of hypovolemic shock) *****minimum acceptable systolic BP: 70 + (2 x age)****** |
|
Pediatric abdomen injuries
|
Spleen, liver = Most common injuries
! High, broad costal arch ! Relatively larger organs ! Poor abdominal muscle development = poor protection |
|
C-Spine Injuries
|
! Less common in children, higher mortality
! falls>MVA>sports (trampolines) ! <8 yr: 2/3 above C3 |
|
Spinal Cord Injury
|
! Spinal cord injury causes myelopathy or damage to white matter or myelinated fiber tracts that carry sensation and motor signals to and from the brain.
! It also damages gray matter in the central part of the spinal, causing segmental losses of interneurons and motorneurons. |
|
Each segment of spinal cord innervates...
|
Slide #16 of pediatric trauma lecture
|
|
Complete spinal cord injury
|
! A complete spinal cord injury produces total loss of all motor and sensory function below the level of injury.
! Almost one half of all spinal cord injuries are complete. ! Even in complete spinal cord injuries, the spinal cord is rarely cut or transected |
|
Incomplete spinal cord injury
|
! An incomplete injury means that there is some functioning below the primary level of the injury.
! A person with an incomplete injury may be able to move one limb more than another, may be able to feel parts of the body that cannot be moved, or may have more functioning on one side of the body than the other. |
|
Prognosis
|
! The level of injury is very helpful in predicting what parts of the body might be affected by paralysis and loss of function.
! incomplete injuries there will be some variation in these prognoses. ! Cervical (neck) injuries usually result in quadriplegia. Injuries at C-3 and above level requires ventilator support. ! C-5 injuries often result in shoulder and biceps control, but no control at the wrist or hand. Diaphram function is present when injury at C-5. ! C-6 and C-7: Quadriplegia, some function of upper extremities. ! Sensory level lost below the sternum. ! Injuries at the thoracic level and below result in paraplegia, with the hands not affected. ! T-1 to T-8 there is most often control of the hands, but poor trunk control as the result of lack of abdominal muscle control. ! Lower T-injuries (T-9 to T-12) allow good trunk control and good abdominal muscle control. Sitting balance is very good. ! Lumbar and Sacral injuries yield decreasing control of the hip flexors and legs. ! other changes may occur such as: bowel and bladder dysfunction. ! Sexual functioning also may occur with SCI males may have their fertility affected, while women's fertility is generally not affected. ! Other effects of SCI may include low blood pressure, inability to regulate blood pressure effectively, reduced control of body temperature, inability to sweat below the level of injury, and chronic pain |
|
Spinal injury dx
|
! A neurologic
examination indicates the location of the injury, if it is not immediately evident. ! The reflexes may be abnormal or may be absent in affected areas of the body. Spine X-rays ! A CT scan or MRI |
|
Spinal injury tx
|
! Corticosteroids, such as dexamethasone or methylprednisolone, are used to reduce swelling that may damage the spinal cord.
! Surgery: This may include surgery to remove fluid or tissue that presses on the spinal cord (decompression laminectomy). ! Bedrest may be needed to allow the bones of the spine, which bears most of the weight of the body, to heal. ! Treatment will address muscle spasms, care of the skin, and bowel and bladder dysfunction. ! Extensive physical therapy, occupational therapy, and other rehabilitation interventions are often required after the injury has healed. |
|
Autonomic dysreflexia
|
! Hypertension, severe headaches, pallor below
and flushing above the injury, and seizures occur due to impaired Autonomous Nervous System. ! Can be triggered by a full bladder. |
|
Immobility
|
Central Nervous System
! Depression is a normal response to progressive loss of muscle function and impaired mobility. ! Hospitalization can result in social isolation that may compound feelings of depression. Cardiovascular ! Poor circulation ! Impaired blood flow can also lead to blood clot formation. Respiratory ! secretions in the lungs may accumulate and thicken, causing mucus plugs and increased respiratory difficulty. ! Secretions in the lungs also harbor bacteria that may cause pneumonia. Muscle Skeletal ! atrophy ! Joint stiffness and pain. ! Contractures GI/GU ! Urinary tract infections ! kidney stones. ! Constipation Integument ! Pressure sores or ulcers |
|
Nursing interventions for immobility
|
! Stretching and range-of-motion exercises to each of the joints everyday, and several times a day.
! Proper body alignment, and change positions at least every two hours. ! Deep Breathing and coughing ! Maintain an adequate fluid intake. ! Maintain regular contact with people and do not allow immobility or embarrassment to be a barrier to interacting with others. |
|
Other complications of spinal cord injury.
Dehydration |
Early signs of dehydration include:
! Increased thirst. ! Dry mouth and sticky saliva. ! Reduced urine output with dark yellow urine. - give 100cc/kg/day Symptoms of moderate dehydration include: ! Extreme thirst. ! Dry appearance inside the mouth and the eyes don't tear. ! Low urine output. Less than 1-2 mls per hour. ! Urine is dark amber or brown. ! Lightheadedness that is relieved by lying down. Severe dehydration is life-threatening. ! Altered behavior, such as severe anxiety, confusion, or not being able to stay awake. ! Faintness ! Weak, rapid pulse. ! Cold, clammy skin or hot, dry skin. ! Little or no urine output. ! Loss of consciousness. ! Poor skin turgor ! Low blood pressure ! Delayed cap refill |
|
GU differences
|
Kidneys mature in the first year
Neonates have limited ability to concentrate urine Normal Urine Output: Infant – 2 ml/kg/hr Child = 1 ml/kg/hr Adolescent/Adult = 0.5 ml/kg/hr Kidney function ! determines the secretion of drugs |
|
Urine output
|
! 1-2 mls/kg/hr
! 1ml/hr*45kg=45mls/hr ! 45mls/hr*24 hrs=1080mls/24 hours ! Minimum Output in 24 hours: 1080mls ! Actual output: 800mls |
|
Electrolyte differences
|
Glucose:
! Infants have high glucose needs and low glycogen stores. They often become hypoglycemic during periods of stress ! Hypoglycemia and hyperglycemia may be an early sign of sepsis ! Glycosuria may be an early sign of infection in the child. ! IV solution of choice is a dextrose based solution |
|
IV solutions: Isotonic
|
! Have a concentration of dissolved particles equal to the intracellular pressure.
! osmotic pressure is the same in and out of the cells. ! Examples of Isotonic solutions are: Normal Saline, D5W, Ringers lactate |
|
Hypertonic
|
! Hypertonic solutions draw fluid from the intracellular space, causing cells to shrink and the extracellular space to expand.
! Examples of hypertonic solutions are: D51/2NS, D5NS, D10W |
|
Hypotonic
|
! Hypotonic solutions should be given with caution as fluid moves from the extracellular
space into the cells, causing the cells to swell. ! Example: 0.45 normal saline and 2/3 – 1/3 |
|
UTI's
|
Risks of UTI
! Females are especially susceptible to UTIs. ! Low oral fluid intake ! critically ill patients ! intermittent or indwelling catheter. ! Any abnormality of the urinary tract that obstructs the flow of urine (eg, kidney stones or an enlarged prostate) ! Congenital anomaly of the urinary tract. Types of Infection ! Escherichia coli ( E Coli) is the most common infecting organism in patients with uncomplicated UTIs ! Other gram-negative microorganisms ! Gram-positive pathogens ! Other microorganisms Diagnosis ! Urinalysis: ! Culture and Sensitivity Test ! Voiding cystourethrogram ! Renal ultrasound Nursing Interventions ! Increased fluid intake ! 100% pure Cranberry juice and vitamin C (ascorbic acid) supplements inhibit the growth of some bacteria by acidifying the urine. ! A heating pad and pain relief medication are helpful for pain management. ! antibiotics. |
|
Perinatal Mood and
Anxiety Disorders (PMAD) |
– Pregnancy and 1 year postpartum
- Depression - Anxiety • Obsessive Compulsive Disorder (OCD) • Post Traumatic Stress Disorder (PTSD) –Bipolar Disorder –Psychosis –Pinks and Baby Blues • not disorders, but may indicate vulnerability |
|
Depression
|
•Depressed mood most of the day
•Anhedonia - severely diminished interest or pleasure in activities “What do you like about being pregnant/being a mother?” •Weight changes •Insomnia or hypersomnia •Psychomotor; restless, agitated, slowed •Diminished energy level •Feelings of worthlessness or excessive guilt •decreased concentration, increaesd indecisiveness •Recurrent thoughts of death or suicide |
|
Perinatal
depression symptoms |
• Irritability and anger
• Overwhelmed • Hopeless and helpless • Guilt and shame • Sadness • Lack of feelings toward the baby Sadness, crying • Inability to cope, take care of self or family • Suicidal thoughts • Appetite changes • Loss of interest, joy, or pleasure • Sleep disturbances • Poor concentration/focus • Anxiety • “I don’t feel like me” • Mood swings |
|
Antenatal Depression
|
“Melancholia in pregnancy”
• 1840s • 1970s – ‘protective’ – decreased Hospitalization rates (Kendell, 1976) –Hormones (Kornstein, 2002) –decreased psychosis & suicide (Brockington, 1996) • 40% of depressed women had suicidal thoughts (Bowen et al., 2009; Levey, 2004) • 2010 • ~20% of women |
|
Pinks
|
•11%, 44% high risk
•Elation hours/days after birth (Glover et al., 1994). •Normal to be happy and excited • but extremes in mood in either direction need to be monitored •Early discharge •Sleep •Bipolar •Expectations of pregnancy |
|
Postpartum blues
|
- normal, transient, emotional response
- up to 85% of women spontaneously recover by 2 weeks - Depressed 2-3 days increased PPD 4-8 weeks (Dennis, 2003; Hannah, 1992; Teissedre, 2004) - Social support **Confirms need for increased awareness, early identification, and timely intervention • 60% of women experience their first major depression in PP • Idealization of birth and motherhood (Beck, 2001; Fergerson, 2002) • Hormones, thyroid, cholesterol, folate, anemia, stress |
|
Anxiety in mom
|
• May be equally detrimental to fetus and maternal functioning as depression
• Predominant component in antenatal depression • Comorbid and temporal relationship depression – PPD – Younger women • 4-15% of women. Up to 24% in pregnancy (Sutter-Daly, 2004, Heron, 2004) • inability to relax, persistent arousal • fearfulness, phobic • preoccupied / over-concern with baby • high parenting expectations • panic attacks - tension, sweats, palpitation |
|
Preggo Obsessive
Compulsive Disease (OCD) |
- Intrusive, repetitive thoughts
• harm coming to baby • Guilt, worry, shame • Cleaning • Checking • Baby, house • Hypervigilent • Counting • Behaviours to avoid harm or minimize triggers • Ordering • Obsession with germs, cleanliness • Assure • Thought ≠ action |
|
Post Traumatic
Stress Disorder (PTSD) |
• ~ 3% of births • Risk
~partial -25% • Anxiety disorder • Post terrifying event where grave harm occurred or was threatened • Birth complications - mother or baby • Hemorrhage • Emergency C/S - preterm birth -NICU baby Birth • Powerless • Uncared for • No trust • Poor communication Previous PTSD • Previous Sexual Abuse |
|
Psychosis
|
•1-2 in 1,000 postpartum women
- of those 5% Suicide and 4% Infanticide •First 2-3 weeks after delivery or later postpartum •Risk - History of Bipolar Disorder •Delusions (baby possessed by devil) •Hallucinations (seeing angels) •Insomnia (no sleep for days) •Rapid mood swings (>non-postpartum psychoses) •Waxing/waning (can appear and feel normal between) |
|
Mental health aboriginal preggos
|
. Birth rate 1.5 times higher
. 40% depressed in pregnancy . Experience > adverse social factors increased risk for depression . Important to know more about maternal depression, anxiety, and mood problems in Indigenous women |
|
Qualitive data for mental health preggos
|
• Ambivalence & conflict about motherhood (also in Aboriginal women)
Best of times-worst of times… Themes – Becoming a mother – Being a mother – Partnership: being and feeling alone – Lack of support – Lack of choices – Lack of culture |
|
Mental health preggos Risk Factors
|
Sociodemographic
-Gender, age, income, single, new immigrant, ethnicity History of depression - psychiatric problems - Own and family - Mood reactions to hormonal changes Social - Unplanned, lack of social support, relationship issues, partner and substance abuse Medical Fertility problems, obstetrical difficulties-previous losses, thyroid, anemia, pain Lack of sleep Mother issues 20% OF ALL WOMEN ARE VULNERABLE |
|
Cortisol
|
“the stress hormone”
•“Normalize” worry in childbearing women •Hypothalamic-pituitary-adrenal HPA axis-placental neuroendocrine axis •Maternal stress can affect fetal development •Sustained pattern of stress reaction throughout life •Neuronal death and abnormal development of fetal brain structures - increased stress = increased carbohydrate ingestion - cortisol = blood sugar irregularities = fatigue, immune system deficiencies, anxiety, panic attacks, depression Immune system balance - altered fetal immune function - +++ = shrinking hippocampus decreased memory Interferes with serotonin & dopamine - low mood, cognition Sleep disturbance - Early morning surge and insomnia Blood pressure Other hormones Fertility, estrogens |
|
Mental health effects on mother
|
. Increases and worsens with parity
. GI - Irritable bowel . Cardiac - comorbidities . Cognitive problems - Hippocampus . Sleep = ¨decreased coping, postpartum psychosis . Relationship - Partners 50% more depression . Social - Isolation-substance use-decisions - Relationship - Partners10% of men overall, 75% if wife severely depressed (Goodman, 2004) - Gestational hypertension (Kurki, 2000) - Abruptio placenta (H 1994 K h Hanna, 1994; Kahn, 2002) - increased Epidural, operative, and preterm deliveries (Ch Chung, 2001) - Less prenatal care, prenatal vitamins (Bonari, 2004; Zuckerman, 1989) - increased Alcohol, smoking (Homish, 2004; Zhu, 2003) |
|
Mental health effects on baby
|
. Preterm
. decreased Apgar . decreased birth weight and weight gain . Less and shorter duration of breastfeeding Behaviour . More withdrawn, irritable, less consolable, depressed . Pattern of stress reaction . 3.3 times the risk of Sudden Infant Death Syndrome (Mitchell, 2008) Effects of lifestyle . alcohol ?FASD, smoking, poor diet, prenatal care, etc. |
|
child readiness
|
associated with
Behaviour .anxiety in pregnancy = increased 3 X ADHD in boys and depression in girls . Emergency room visits, somatic complaints . Social and school difficulties . Criminality Mothers less attentive to safety and stimulation .Injuries and hospitalizations |
|
Father role in mental health in preggos
|
–10% depressed perinatally
–Up to 50%↑ paternal depression in PPD • No reason to expect it will be any less prenatally –Similar effects on child (Goodman, 2004; Ramchandani, 2005) –Non-depressed partner important to child development |
|
Parenting effects d/t mental illness
|
• Less responsive to baby’s cues
•Attachment problems begin (Spinelli, 2001) • Less attention to stimulating child and safety issues • Siblings |
|
Feelings in Pregnancy and
Motherhood Study |
Phase 1: October 2004-August 2006
Socially high-risk women - 39% Phase 2: December 2005-Dec 2008 - General population – 14% - Saskatoon and Five Hills Health Regions |
|
Who most at risk of preggo mental illness?
|
•History of depression 5Xs
•Mood swings 2-3 Xs •Stress 2Xs •Unplanned pregnancy – Time 1 •Dissatisfaction with relationship – Time 2 •Smoking •Social support |
|
Mother First
|
Maternal Mental Health Strategy:
2 initiatives to improve maternal mental health 1. Increase awareness in Saskatchewan 2. Building capacity in Saskatchewan |
|
1. Increasing Awareness
|
• Materials
• Available • Saskatchewan Prevention Institute • Healthline online • Healthline 1-877-800-0002 – Fridge magnet • Saskatchewan Drug Information Services – 1-800-665-DRUG (3784) • Visits to regions, groups • Evaluate process |
|
2. Building Capacity
|
PPD ‘Unmasking Postpartum Depression’ conference – Smiling Mask
• October 2009, Regina • Started momentum - priorities for action • Government invited our recommendations Formed Working Group • Regions, First Nations groups, Government, Professions, Prevention Institute, Health Quality Council, Healthline • H1N1 • WebEx q 1-2 weeks • Recommendations started right away May 2010-press conference |
|
RECOMMENDATION #1
|
Education - Increase awareness
• Materials available – Prevention Institute www.skmaternalmentalhealth.com • Ensure in curriculum of health professionals, schools • E-Learning event – DVD - - ongoing learning • Promote positive maternal mental health Promote Positive Mental Health • Exercise ( Bowen, 2009) e c se o e , 00 ) • Walking, swimming • Sleep – 6 hours in 24 • Nutrition • Simplify life • Yoga or other relaxation Support quitting self-medication • smoking, drinking, drugs |
|
RECOMMENDATION #2
|
Universal Screening - normalize – we care about all aspect s of your health – physical
and mental Edinburgh Postnatal Depression Scale >=12 probable depression • 2 times in Pregnancy - 1st visit, 28-34 weeks • 3 times in Postpartum - prior to discharge from early visit program (2-3 weeks) and within public health and immunization visits 2 months (4 if not 2) and 6 months |
|
Why screen?
|
Can’t always tell by looking or may not have time for in-depth mental health assessment
• busy with physical assessment or baby fussy Can help to identify those at high-risk or in immediate distress Inexpensive, quick, and easy Can be educational, opens communication • Review form with woman to promote treatment Treatment can alleviate symptoms • Unethical not to?? |
|
Edinburgh Postnatal Depression Scale
EPDS |
• Perinatal depression screening tool -1987
• Screen for the probability of depression DOES NOT diagnose depression reflects past 7 days • Takes out physical symptoms • Self report, free Validated • pregnant, hospital, and community populations • languages/dialects • Aboriginal women in Regina • Picks up anxiety and poor coping • Rapid identification of self-harm thoughts • Can be used with partner… • 10-11 -- possible mild depression • >12 -- probable depression • Anxiety > 4 on items 3, 4, 5 |
|
What Self harm thoughts….
|
Reassure
“Given that things are so difficult for you right now, it would be surprising if you didn’t have the occasional thought of not wanting to be here” “Are you feeling so angry you might be afraid of harming your baby or yourself?” “We are here to help you” • Don’t leave positive #10 alone • Suicide check • Any thoughts of harming baby or others? |
|
Postpartum suicide
|
• Suicide
–Risk postpartum increased 70-fold –Presence of clinical disorder • Follow-up on all positive responses or even non-positive if there is high score or history - “Just want to check with you. Any thoughts of harming yourself?” |
|
Positive #10….Assess:
|
1. Previous history – ideation and attempts
2. Type of thoughts – present and future 3. Intention/Plan/Level of risk 4. Presence of self-harming behaviours 5. Risk and protective factors 6. Support network 7. Safety planning 8. Emergency contacts |
|
Assess Risk of Harm to Baby
|
• Adapt a “Question 11”
– “Has the thought of harming your baby or others occurred in the last week?” NOTE (does NOT contribute to score) • What kind of thoughts? • Behaviours • Maladaptive and adaptive coping • Social support |
|
When to screen?
|
• First or 2nd prenatal visit
• 28-34 weeks • Within first 2-3 weeks • At immunization visits • Any time you feel there is a need |
|
Golden Triangle of mental illness
|
1. Alcohol
2. Abuse - "How do you handle problems?" 3. Depression/anxiety |
|
RECOMMENDATION # 3
|
• Prioritize pregnant and
postpartum women in MHS Accessible in each region – Inventory of services and supports Increase treatment options • Something… Healthline • Immediate |
|
Maternal Mental Health Program
|
• Psychiatrist, psychologist, nurse therapist
– Clinic once/month – Nurse 4hr/week – Support to GPs – Family physician residents •Advisory Group • PPD group, breastfeeding centre |
|
Interventions
|
Interpersonal therapy
• Cognitive Behavioral Therapy • Significantly > education Groups • Support, psychoeducation Support • Home visitor “listening visits” • Peer • Electroconvulsive Therapy – ECT • Bright Light therapy • Hormones • Alternate treatments - Limited information • Food and supplements • Folic acid • Omega 3 Fatty Acids (EPA - eicosapentaenoic) • Grapefruit juice (when no meds) • Vitamin D? • St. John’s Wort contraindicated in pregnancy • Placenta…. Acupuncture, massage, meditation |
|
Medication of preggo mental illness
|
First thought is to come off
•up to 50-75% will relapse (Cohen, 2007) •35% of pregnant women who are taking medications, are taking psychotropic medications •Up to 50% pregnancies are unplanned 1st month most critical •Teratogenesis – when might not know pregnant *** •preconceptual couselling If start medication during pregnancy •Not likely to begin until after 1st few weeks •Is increased risk of bleeding Neonatal adaptation syndrome - transitory SSRIS - Keep on one that they are stabilized on Sertraline . low transfer to fetus and through breastmilk Wellbutrin, Zyban - helps with depression and quitting smoking Atypical antipsychotics - Anxiety Bipolar . Treatment ca n be problematic . Valproic acid/Epival and other anticonvulsants can ª risk for NTD . increased preconceptual and pregnancy Folic Acid . Lamotragine better tolerated . Lithium, neutral . limit in labour Dosing – Always aim for the lowest, most effective dose •undertreatment – ‘tapering’ (lithium) – Depressive symptoms tend to increase as delivery approaches – hemodilution • if woman has decreased or discontinued medication, closely monitor symptoms • Combination of approaches • Meds may be needed help to stabilize mood so that other approaches can work |
|
Promote Positive
Mental Health |
• Self-esteem
“Look how the baby listens to you” Exercise (Bowen, 2009) • Walking, swimming • Sleep – 6 hours in 24 • Nutrition • Simplify life • Yoga or other relaxation - massage • Support quitting self-medication • smoking, drinking, drugs |
|
RECOMMENDATION #4
|
Sustainability and Accountability
Maternal Mental Health Committees • Provincial • Regional • Intersectoral – Bring supports/treatment services together – Identifiable place to get help with Maternal Mental Health |
|
Symptoms of depression
|
• Less interest in things you usually like
• Crying for no reason • Irritable, angry, or more sensitive • More tired or feeling hyper • Not sleeping or sleeping too much • Problems concentrating • Not able to cope • Anxious or panicked • Thoughts of harming yourself, your baby, or others |
|
Diabetogenic Effect of Pregnancy
|
Glucose mother„_baby
1st trimester - Rise in hormones stimulate insulin production & increase tissue response to insulin - 1st trimester - insulin needs frequently decreases 2nd and 3rd trimester - Human placental lactogen hPL - increased resistance to insulin and decreased glucose tolerance - Insulin needs increase (2-3Xs by end of pregnancy) - increased insulin required |
|
GDM
|
Pregnancy/Maternal Effects
- Polyhydramnios - PROM, premature rupture of membranes - Preterm labour Worsening myopathies - vascular, renal, retinal - Pre-existing diabetes increase Ketosis - increased Gestational Hypertension - increased Type 2 Diabetes later in life |
|
GDM
Fetal effects |
• Macrosomia
• Intrauterine growth restriction • Hypoglycemia • Hyperbilirubinemia • Immature respiratory development = RDS • Congenital Anomalies • Fetal demise Child effects – Increased risk of developing diabetes – Obesity |
|
Effects on fetus
|
• Cardiovascular
– Transposition of great vessels – Septal defects • Skeletal – Cordal regression syndrome – Spina bifida • GI – Tracheoesophaegeal fistula – Imperforate anus • CNS – Meninogmyocele – Anencepahly – Microcephaly • GU – Polycystic or absent kidneys |
|
10 Risk Factors
|
1. Hx
2. Family hx 3. Previous or suspected macrosomia(>4,000 g) 4. Previous unexplained stillbirth, miscarriage 5. Previous hypoglycemia or hyperbilirubinemia 6. Advanced maternal age 7. Obesity 8. Repeated glycosuria in pregnancy 9. Polyhydramnios 10.Culture |
|
Screening for GDM
|
Current CDA guidelines suggest routine screening of all women
• 24–28 weeks of gestation • 50 g glucose challenge test (GCT), using a threshold of 7.8 mmol/L (140 mg/dL) SOGC guidelines • screen only those w risk factors at 24-28 wks |
|
Goal for GDM interventions
|
maintain euglycemic state
Pregnancy • Team approach • Monitor – Glucose – Infections – Fetal development and health • Diet • Insulin as needed • Oral antihyperglycemics – ______indicated • Gentle exercise – Walks after meals |
|
Insulin requirements intra/post - partum
|
Intrapartum
. Balance insulin with need for increased energy in labour . Often maternal insulin requirements decrease in labour Postpartum . insulin requirements decrease significantly . rarely diabetic immediately postpartum |
|
Hypertensive Disorders of
Pregnancy HDP |
AKA
- Pregnancy Induced Hypertension - PIH - Gestational Hypertension – GH - Pre-eclampsia 5 - 7% of pregnancies insidious or sudden onset |
|
Gestational Hypertension Risk Factors
|
• Nullipara or first pregnancy with a new partner
• Personal or family history of hypertension (pregnancy) • Poor nutrition • Obesity • Culture (east indians and filipinos have narrow veins) • Advanced maternal age (> 35) • Multiple gestation (i.e. twins, triplets, etc) • Diabetes |
|
GHTN Classification
|
Pre-Existing 1%
. predates pregnancy or before 20 weeks Gestational 5% - Diastolic . 90 mmHg - after 20 weeks and up to 12 weeks PP *Accuracy in BP measurement* Preeclampsia 1-2% -Proteinuria (>2+) - or 1 or more adverse conditions Severe Preeclampsia - >160/110 mmHg - Heavy proteinuria - 1 or more adverse conditions Eclampsia - seizure |
|
GHTN Adverse Conditions
|
• Headache
• Visual Disturbances • Abdominal Pain • Nausea/Vomiting • Chest pain/SOB • Abnormal maternal lab values • Fetal morbidity • Edema / Weight gain • Reflexes |
|
GHTN Pathophysiology
|
Increased peripheral vascular resistance
Decreased resistance to effects of angiotensin II Decreased levels of vasodilators - nitric oxide Loss of normal vasodilation capability Increased levels of vasoconstrictors (partially produced by placenta) Concurrent vasospasm Decreased renal perfusion and glomerular filtration Decreased intravascular volume = increased blood viscosity = increased hematocrit |
|
GHTN Maternal Risk
|
• Seizures, renal failure and damage
• Abruptio placentae • Ruptured liver and pulmonary embolism • Disseminated intravascular coagulation (DIC) • HELLP syndrome • Cesarean section • Chronic hypertension • Stroke |
|
GHTN Fetal-Neonatal Risks
|
Vasospasm
- decreased blood flow to placenta/baby - decreased oxygen available = fetal distress Preterm delivery/Premature Abruptio Placentae Hypermagnesemia - MgSo4 - Hypotonic Increased morbidity and mortality - Fetal demise - IUGR/SGA |
|
GHTN Management
|
• Home monitoring
– BP, weight, urine, NST~weekly • Stress reduction • Diet – Avoid excess weight gain but no dieting – Calcium, no restrictions (NaCl, protein) • Baseline bloodwork – enzymes, clotting, electrolytes – ekg in pre-existing • Treat nausea & vomiting • Treat blood pressure – Quit smoking – ?low dose ASA |
|
In-patient: severe or preeclampsia tx
|
• Bed rest – Left lateral position - ambulation
• Fetal evaluation (EFM, BPP) • Hourly I&O – 24-hour urine collection –creatinine ratio • Frequent BP, pulse, resp • Blood work – Liver enzymes, electrolytes, clotting • Consider seizure prophylaxis – MgSo4 • Assess for adverse conditions – Abdominal discomfort, visual disturbances, headache, weight, edema • Assess deep tendon reflexes and clonus • Timing / mode of delivery – Ripen and Induce • Ergometrine (not given b/c it will cause bp to increase) – C/section – Anesthesia • Coagulation |
|
GHTN Anti-Hypertensives
|
Labetalol - B adrenergic blocker
Aldomet (Methyldopa)-Centrally-acting sympatholytic Nifedipine (Adalat) - calcium channel blocker Hydralazine (Apresoline) - Arteriolar dilators ACE inhibitors Atenolol (and prazosin) contraindicted - Neonatal renal failure, IUGR, preterm, fetal/neonatal demise |
|
GHTN Anti-Convulsants
|
Magnesium Sulfate MgSO4
4-6 g loading dose followed by 1-2 g q 1hr IV . Can slow labour . Muscle weakness . Lack of energy and drowsiness . Respiratory depression . Lower blood pressure . Tachycardia **Goal - decrease CNS irritability and prevent seizures |
|
Magnesium Toxicity
|
• CNS Depression
– Respiratory rate <12 – Oligouria <30mls/hr – Diminished or absent DTR – Serum magnesium 4.8 - 9.6 mEq • Antagonist – Calcium Gluconate • 1gm over 3 min |
|
Eclampsia = SEIZURE. What to do?
|
Anticonvulsants:
- Bolus of magnesium sulfate Sedation and other anticonvulsants: Dilantin Diuretics to treat pulmonary edema - Furosemide (Lasix) Digitalis (for heart) Intensive nursing care DELIVER - if fetus <34 weeks give corticosteroids to increase fetal lung maturity (Betamethasone) |
|
HELLP Syndrome
|
• 10-20% women with severe pre-eclampsia
• Sudden, without warning – GI pain HELLP • Hemolysis • ELevated Liver Enzymes • Low Platelets – thrombocytopenia – Platelets aggregate at sites of vascular damage – Be ready to administer platelets if < 20 109/L – Epidural anathesia may not be an option if low platelets |
|
DIC – Disseminated
Intravascular Coagulation |
• Can be caused by pre-eclampsia, hemorrhage, intrauterine fetal demise,
amniotic fluid embolism, sepsis, HELLP, etc. • Over-activation of normal clotting mechanism • Mini clots develop • Depletes platelets and clotting factors = EXCESSIVE BLEEDING |
|
DIC Postpartum
|
• Monitor BP, blood work
• Kidney liver function • 6 weeks pp • Monitor BP ongoing • Spacing of next child 2-10yrs pp • Weight management • Increased risk for cardiovascular problems later life |
|
Rh Alloimmunization
|
Rh- mother Rh+ fetus
Fetal rbc = maternal circulation - usually occurs at birth, abortion 1st pregnancy not affected - Unknown abortions Subsequent pregnancies - Maternal antibodies = fetal circ = Hemolysis of fetal rbc = fetal |
|
Rh Fetal and Neonatal Risks
|
• Anemia
• Hemolytic syndrome • Erythroblastosis fetalis – Marked fetal edema, called hydrops fetalis – Congestive heart failure – Marked jaundice – Severe fetal compromise |
|
Rh Prevention
|
• Screen for Rh incompatibility and sensitization
– History – Antibody screen (indirect Coombs’ test) • sensitization Give Rh immune globulin (RhoGam, WinRho) – Pregnant Rh-women with no antibody titer • At 28 weeks’ gestational age – Baby’s father Rh+ or unknown – After abortion & within 72 hours postpartum – Amniocentesis, APH |
|
ABO Incompatibility
|
Mother O blood type -- infant A, B, or AB
– Anti-A and anti-B antibodies occur naturally – During pregnancy maternal antibodies cross placenta – Cause hemolysis of fetal rbc • hyperbilirubinemia – Unlike Rh incompatibility • 1st infant involved • no antepartal treatment or prevention |
|
Anemia
|
Hemoglobin (Hgb)
• < 100g/L • Fatigue, paleness, malaise Implications for woman • Depression • Increased risk for infections • Fatigue – Mothering challenged – Breastfeeding • Further complications – Embolisms – Permanent damage to heart – Future pregnancies Implications for the infant • Low birth weight • Prematurity • Stillbirth Treatment • Dietary • Iron supplements • Avoid transfusions |
|
Heart Disease
|
1% of pregnancies
- 40% inc cardiac output by 20-24 weeks - Pulse inc10+ bpm - dec cardiac reserve - dec capacity to handle pregnancy workload ¡E Congenital heart defects ¡E Rheumatic heart disease - mitral stenosis ¡E Mitral valve prolapse ¡E Peripartum cardiomyopathy - Multiparas, pre-eclampsia, twins, (inc edema) older, obesity |
|
Preggo Heart Disease Care
|
. Assess the stress of pregnancy
on the heart . Limit activity . Calm atmosphere . Head of bed up - Avoid supine hypotension . Monitor for signs of impending cardiac failure - Embolism - Vital signs . Fetal assessment . Family support and education |
|
Hyperthyroidism
|
0.15% pregnancies
1st trimester - Very high levels of hCG (similar to TSH) - multiple pregnancies, molar pregnancy Uncontrolled hyperthyroidism= - preeclampsia, depression - fetal tachycardia, morbidity, mortality, SGA, preterm Worsens again pp - Postpartum thyrotoxicosis, thyroiditis,psychosis Antithyroid drugs, cross the placenta Tx both |
|
Hypothyroidism
|
• Associated with 4% population
– maternal anemia, myopathy, chf, pre-eclampsia, aph, pph, and depression – Poorer neonatal outcomes • lbw • fetal brain development – cretinism • Care – Medication needs greatly increase in pregnancy – Thyroid hormones, levothyroxine and liothyronine (quick onset) – prenatal vitamins (iron, calcium) may inhibit absorption of L-T4A, take vit 4hr post med |
|
Epilepsy
|
Estrogen is epileptogenic
Seizures can put fetus and pregnancy at risk Anticonvulsant drugs (Valproate) associated with malformations, dec IQ 10 point Vitamin K supplement during the last trimester Control of nausea and management of vomiting Promote lifestyle factors that can dec risks for seizures |
|
Trauma During Pregnancy
|
• Types of trauma
– Blunt trauma – Penetrating injuries – Gunshot wounds – Motor vehicle accident – Uterine rupture is rare – Falls – Direct assaults • Impact – Traumatic separation of the placenta – High rate of fetal mortality – Premature birth – Early rupture of membranes – Maternal shock – Premature labor or spontaneous abortion – Placental abruption |
|
preggo trauma care
|
• Life-saving measures for woman
• Fetus near term and uterus damaged • Cesarean section • Fetus immature - Uterus can be repaired - Pregnancy continue to term • Minor injuries |
|
Substance use/abuse
|
18.9% socially high-risk pregnant women use recreational
drugs, 11% daily |
|
Cocaine and Crack
• Adverse maternal effects |
Adverse maternal effects
– Seizures and hallucinations – Pulmonary edema and cerebral hemorrhage – Respiratory failure and heart problems – Increased incidence of spontaneous abortion – Abruptio placentae, preterm birth, and stillbirth |
|
Cocaine and Crack Fetal neonatal effects
|
– Increased risk of intrauterine growth restriction (IUGR)
– Small head circumference – Cerebral infarctions – Altered brain development – Shorter body length – Malformations of the genitourinary tract – Lower Apgar scores – May have neurobehavioral disturbances – Marked irritability – An exaggerated startle reflex – Labile emotions |
|
Newborns exposed to cocaine in utero
|
– Increased risk of sudden infant death syndrome (SIDS)
–Cocaine crosses into breast milk –May cause symptoms in the breastfeeding infant – Extreme irritability and vomiting –Diarrhea, dilated pupils, and apnea –Cocaine use after childbirth • Prohibits breastfeeding |
|
Marijuana
|
• Impaired coordination, memory, and critical thinking ability
– Self-care, prenatal care • No strong evidence that marijuana is teratogenic • Risks are dose related • Increased risk of intrauterine growth restriction • Sudden infant death syndrome (SIDS) in infants • Impact of heavy marijuana use on pregnancy is difficult to evaluate – complex problems – postmaturity |
|
Ecstasy
|
• MDMA
(methylenedioxymethamphetamine) – produces euphoria and feelings of empathy for others • Deaths • Little is known about the effects on pregnancy • may be critical issue during fetal brain development |
|
Heroin
|
• CNS depressant
– narcotic • Alters perception and produces euphoria • An addictive drug, generally IV administered • Associated with malnutrition, poor, late or no prenatal care |
|
Fetus of heroin-addicted woman
|
• Increased risk for IUGR and meconium aspiration
• Hypoxia • Restlessness and shrill, high-pitched cry • Irritability and fist sucking • Vomiting and seizures • Signs of withdrawal usually appear within 72 hours – May last for several days |
|
Methadone
|
• Blocks withdrawal symptoms
– Reduces or eliminates the craving for narcotics – Crosses the placenta • Associated with pregnancy complications and abnormal fetal presentation • Prenatal exposure – Reduced head circumference and lower birth weight – Withdrawal symptoms |
|
Alcohol
|
• Central nervous system (CNS) depressant
• Potent teratogen – FASD • Maternal effects – Malnutrition – Bone marrow suppression – Increased incidence of infections – Liver disease – Withdrawal seizures • Pregnancy – May inhibit labour |
|
Care
|
Mother
– Watch for signs of withdrawal – Use • Leave unit, leave baby unattended, high Baby – Watch for signs of withdrawal – Active use • NICU for observation and treatment |
|
Common complications of pre-term births
|
• Prematurity
• Thermoregulation • Respiratory Distress • Hyperbilirubinemia • Hypoglycemia • Sepsis • Neonatal Abstinence Syndrome |
|
Preterm Birth Survival
|
Gestation Survival %
23 weeks 15-30 28 weeks 85-90 |
|
The Pre-term Infant
|
Respiratory:
- Lack of surfactant - respiratory distress syndrome (RDS) - apnea - bronchopulmonary dysplasia (BPD) Cardiovascular: - Patent ductus arteriosis (PDA) - inc respiratory effort - CO2 retention Thermoregulation: - dec brown fat, dec brown fat & subcutaneous fat, poor muscular development, less flexed tone, inability to shiver, thin skin, inc BSA, inc exposure during resuscitation Gastrointestinal: - Small stomach, immature feeding reflexes - Necrotizing Enterocoitis (NEC) Renal: - dec ability to concentrate urine - dec ability to excrete drugs/medications Hepatic / Hematologic: • Immature liver = dec conjugate bilirubin = inc jaundice • At risk for hypoglycemia • Limited iron stores = anemia • Blood volume is less Neurological: • Intraventricular hemorrhage (IVH) |
|
Preterm Temperature Control
|
Less able to produce heat - higher ratio of body surface to body weight
Lack of brown fat Thin skin - insensible water loss Lack of flexion increases heat loss Resuscitation efforts Birthing room or OR ambient temperature Maintain room temperature ~ 25° - 26°C put them in a baggy and keep them moist |
|
Late Preterm Infant
34 – 36 completed weeks |
Often overlooked
• do not appear dramatically sick Immature • < 4 – 6 wks of the 3rd trimester Brain size at 34 – 35 wks • 60% of that of infant @ term Largest proportion of preterm births • Respiratory distress syndrome (RDS) • Transient Tachypnea of the Newborn • Pulmonary infection • Recurrent apnea • Temperature instability • Jaundice (discharge delay) • Re-hospitalization for various Dx., • Feeding difficulties • Long-term behavioural difficulties • Mortality |
|
Neonatal Respiratory Distress
|
Signs
- tachypnea - apnea -cyanosis - grunting - nasal flaring - retractions - poor feeding - increased effort to breathe - hyper capnia - dec BP and shock Common Causes - respiratory distress syndrome (RDS) - meconium aspiration (MSAF, MAS) - transient tachypnea of the newborn (TTN) |
|
Risk Factors for RDS
|
-20% of neonatal deaths (0-28d)
-Lack of surfactant -Prematurity ** • Maternal diabetes (type 1 & 2) -Stress a protector • Acidemia • Multiples, 2nd • Hypovolemia • Male infant • Genetic predisposition • C/S without labour • Caucasian • APH • Hydrops fetalis |
|
RDS Management
|
Management
• Antenatal Corticosteroids • Exogenous Surfactant Endotracheal • Continuous positive airway pressure (CPAP) and positive end-expiratory pressure (PEEP) • Fluids and other supportive care Nursing assessment • Respirations ? • Colour ? • Edema |
|
Meconium Stained Amniotic Fluid
(MSAF) |
Why does MSAF occur in ~ 12% of live
births ● Fetal stress/hypoxia ● Breech ● Post Term Fluid aspirated (thin, thick, particulate) ●Amount and consistency varies ●During first few breaths taken by newborn (?) ●Does suctioning at perineum help? 4-5% of MSAF result in MAS |
|
Complications of Meconium
Aspiration Syndrome (MAS) |
• RDS
• Pneumothorax • Pneumonia • Irritation • PPHN |
|
Care with Meconium Aspiration
|
Prevent
• avoid post maturity • amniotic infusion • endotracheal suction by trained individual (?) Assisted ventilation Surfaxin, Steroids Close observation, supportive, nutrition • Respiratory distress • crackles, rhonci |
|
Transient Tachypnea of Newborn
(TTNB) |
"wet lung syndrome¨
Excess fluid in the lungs or delayed reabsorption of fetal lung fluid ~ 1% of all deliveries Normal @ birth -develops respiratory distress symptoms in 4-6 hours May be related to aspiration of amniotic fluid, excess secretions or tracheal fluid - C-section - Preterm Resembles RDS Nursing care involves - Oxygen - Intravenous fluids for fluid and electrolyte requirements - Restrict oral feedings until respiratory status improves Usually resolves within 12-72 hrs |
|
HYPERBILIRUBINEMIA
|
Hyperbilirubinemia
• excessive concentration of bilirubin in the blood Jaundice - bile pigment deposited in the skin, mucous membranes and sclera = - Cephalocaudal (head to toe) progression - Well lit area, preferably natural daylight - Gentle pressure to blanch skin to reveal underlying color of skin/subcutaneous tissue |
|
Physiological Jaundice
|
Most common cause of jaundice - 50-60% newborns:
Primary mechanism: - inc RBC volume, short RBC life span = inc RBC hemolysis after birth = inc bilirubin load Additional mechanisms include: - Decreased clearance of bilirubin from plasma - liver immature, dec ability to excrete inc conjugated bilirubin - immaturity of bilibrubin conjugation in the liver at birth Usually peaks at 3 - 5 days Typically resolved by day 8 |
|
Pathological Jaundice –within 1st
24hr |
Excessive erythrocyte destruction
inc Extravascular blood Polycythemia Other |
|
Breastfeeding Jaundice
|
~ 0.5% to 2.4% of all newborns
mild jaundice that occurs or persists past one week of life in an otherwise healthy and thriving breast-fed infant ? factors in the breast milk block certain proteins in the liver that break down bilirubin ? runs in families |
|
Severe Hyperbilirubinemia
|
Risk factors include:
Č Visible jaundice Č Previous sibling with jaundice Č Visible bruising / cephalhematoma Č Exclusive / partial Breastfeeding Č Certain ethnic backgrounds Č Dehydration / weight loss after birth Č Infant of Diabetic Mother (IDM) |
|
Bilirubin Encephalopathy- kernicterus
|
Neurological effects of unconjugated bilirubin in the brain may be reversible or
permanent deep-yellow staining of neurons andneuronal necrosis of the basal ganglia and brainstem nuclei |
|
Guidelines for Testing (CPS, 2007)
|
TSB - Total Serum Bilirubin
- Severe >340 μmol/L at any time during 1st 28 days - Critical >425 μmol/L during 1st 28 days only 50% of babies with a TSB concentration greater than 128 μmol/L appear jaundiced (TcB) transcutaneous bilirubin measured in infants 24 h -72 h • results plotted to risk of progression to severe hyperbilirubinemia |
|
Phototherapy
|
Eye patches
• remove q shift Temperature • Isolette and baby Expose • maximum skin surface Serum bilirubin levels • Turn lights off while blood is drawn & protect sample from light Change position q2h • Prevent and assess for pressure sore |
|
Other Treatment (jaundice)
|
Early and frequent feedings
• Promote breastfeeding • increase excretion through feces & urine Prevention of dehydration • supplemental fluids when phototherapy Exchange transfusion for severe cases |
|
Hypoglycemia
|
IDMs, SGA infants, Preterm AGA, LGA
Stressed, infection Asymptomatic - Feeding interventions - inc frequency of Breastfeeding - supplement-BM, BM fortifier, formula - Evaluate for response in 1 hr Symptomatic or < 2mmol/L - IV infusion of glucose - Target 2.6 mmol/l or „´ (CPS) Blood glucose - Q 1-4h in high risk - Varies with institution - At or below 2.5/2.6 mmol/L |
|
Symptoms of Hypoglycemia
|
1. Jitteriness or tremors
2. Apathy 3. Episodes of cyanosis 4. Convulsions 5. Intermittent apneic spells 6. Tachypnea 7. Weak or high-pitched cry 8. Limpness or lethargy 9. Difficulty in feeding 10. Eye rolling 11. sweating, sudden pallor, hypothermia, and cardiac arrest and failure also occur |
|
Newborn Sepsis
|
Immature immune system
Exposure to Bacteria (GBS) PPROM Maternal fever Chorioamnionitis Early onset - within 24-48 hrs - usually respiratory (pneumonia) Late onset - after 48 hrs - usually meningitis and bacteremia Subtle behavior changes - not doing well - Lethargic or irritabilitable - Feeding intolerance Temperature INSTABILITY Tachycardia Poor Peripheral Circulation ¡E Pallor ¡´Duskiness ¡´Cyanosis Respiratory Distress Hyperbilirubinemia |
|
Parents of Newborn with
Complications |
Sick or preterm baby may have
• Delayed or lack of periods of reactivity due to poor condition of newborn • More disorganized in sleep-wake cycle Assess • knowledge of infant's condition or anomaly Communication • Keep informed about infant condition Promote care • Flexible visits • Skin to skin care • Breastfeeding/pumping |
|
NAS “Withdrawal” from opiates
|
Withdrawal
Irritability Tremors, tight tone, trouble with sleep &temp Hyperactive, high pitched cry, hypotonia Diarrhea, diaphoresis, dehydration, disorganized suck Respiratory distress, rhinorrheoa, Apneic attacks Weight loss Alkalosis – respiratory Lacrimation |
|
Heroin, other opiates (Oxycontin,
Percocet), including methadone |
- Significant withdrawal in the baby
- NICU care to stabilize and observe - Withdrawal can start at 2 hrs, depending on last dose by mom - Lasts up to 6 months - *Administer drug to wean baby off drug over 1-2 months - Avoid naloxone, can induce seizures - Seizures ¡V phenobarbital - Polydrug use etc. - Increased need to suck, frantic movement, diarrhea, sleep deprivation |
|
Cocaine and Methamphetamine effect on newborn
|
Different mechanism for addiction than heroin and different effects on newborn
Amphetamines (crystal meth, crank) - inc HR and BP, vasoconstriction dec blood flow to fetus - Low birthweight, IUGR, prematurity, intracranial hemorrhage - No withdrawal symptoms like optiates but rather poor suck and feed, lethargy, ¡õ tone Cocaine - Similar to amphetamines but also - Abruptio - Anemia, fetal death, Neonatal SIDS Long term-language delays until 2+ years & behavioural problems unless add support @ home (i.e. mom clean) |
|
Marijuana
|
• Postdates, SGA
• dec prolactin affects breastfeeding • Inattentiveness to baby afterwards |
|
Alcohol
|
- FASD/FASE
- withdrawal symptoms may start at 2 hours (depending on last drink) and last up to 18 months |
|
Nicotine
|
• SGA
• SIDS, respiratory, behavioural problems, headaches |
|
Care for drug baby
|
Manage newborn complications
Serologic tests for • syphilis, HIV, hepatitis B Meconium, cord, or urine drug screen Social service referral Nutritional support Reduce withdrawal symptoms Promote adequate respiration, temperature, and nutrition, second-hand smoke/drugs |
|
Therapeutic handling
|
1. Reduce stimulation
• Calm subdued environment (↓ light, ↓ noise) • Avoid eye contact • Soother • Take cues from baby 2. Swaddling to help control body and tremors 3. “C” position to feed and cuddle • Hold baby and move up & down carefully with baby held away from person swaddled in c position |
|
Treating Withdrawal
|
Opium
Morphine Methadone Phenobarbital ***Wean Slowly!!!*** Naloxone (Narcan) - Contraindicated in addicted baby - rapid withdrawal & seizures |
|
Parents of drug babies
|
High levels of stress/anxiety
• More in 1st week • More for mother - Guilt, own recovery, grief • Financial – esp if out of region PTSD • delivery NICU • Stressful environment • Buddy • Social work • Discharge nurse • LC |
|
As baby recovers…
|
Ensure parents understand
• routine well-baby care • special procedures • expected growth and development of infant Referral • infant screening procedures • follow-up care - Team approach • special equipment required at home |
|
Cancer
|
! Cancer is diagnosed in an average of 1300 children every year in ages ranging from 0-19 years of age
! Leukemia accounted for 25% of new cancer cases and is the most common childhood cancer. ! Since the 1950’s, mortality rates for childhood cancer have declined more then 50%. ! Approximately 85% of Canadian children and adolescents with acute ALL are alive 5 years after diagnosis. |
|
Assessment
cancer |
! Unusual mass or swelling
! Unexplained paleness and loss of energy ! Spontaneous bruising ! Prolonged, unexplained fever ! Headaches in morning ! Sudden eye or vision changes ! Excessive – rapid weight loss |
|
Cancer Dx
|
! X-ray, Skeletal survey
! CT scan ! Ultrasound ! MRI ! Bone marrow aspiration ! CBC with absolute neutrophil count ! Urinalysis ! Lumbar puncture ! Urine catecholamines |
|
Cancer Treatment for children
|
! The improvement in childhood survival rates reflects biological differences in adults compared to children, and differences in treatment approaches.
! The success of clinical trials in identifying new agents and treatment approaches has been significant. ! A shift towards multidisciplinary approach has improved overall outcomes and decreased mortality Determined by: ! Type of cancer ! Location ! Extent of disease |
|
Osteosarcoma
|
! Also called osteosarcoma, osteogenic sarcoma is one of
the most common types of bone cancer in children. ! The disease usually occurs in the long bones, such as the arms (humerus), legs (femur/tibia), and pelvis. ! It rarely occurs in the jaw and fingers, but often occurs at the ends of these bones near growth plates. ! Peak incidence is during rapid growth years. ! Bone tissue produced by osteosarcoma never matures into compact bone. ! At the time of diagnosis, most children hav emetastases. |
|
Osteosarcoma
Symptoms |
! Pain
! Swelling ! Pulmonary metastasis occurs in 20% of cases. When lung metastasis is the only site, lung resection may be successful for treatment. ! Disseminated metastases and bone lesions have poorer prognosis. |
|
Leukemia
|
! In leukemia, the bone marrow, for an unknown reason, begins to make white blood cells that do not mature correctly, but continue to reproduce themselves.
! With leukemia, these cells do not respond to the signals to stop and reproduce, regardless of space available. ! Leukemia can occur at any age, although it is most commonly seen in children between 2 and 6 years of age. ! The disease occurs slightly more frequently in males than in females. |
|
Leukemia
ALL |
! Acute lymphocytic leukemia (ALL), also called
lymphoblastic or lymphoid, accounts for about 75 to 80 percent of the childhood leukemias. ! The low death rate in ALL reflect the major advances in treating the cancer. ! In this form of the disease, the lymphoid cells are affected. |
|
Leukemia
AML |
! Acute myelogenous leukemia (AML), also called
granulocytic, myelocytic, myeloblastic, or myeloid. ! accounts for about 20 percent of the childhood leukemias. ! Acute myelogenous leukemia is a cancer of the blood in which too many granulocytes are produced in the marrow. |
|
Leukemia
Symptoms may include: |
! Anemia
! Bleeding and/or bruising ! Recurrent infections ! Bone and joint pain ! Abdominal distress ! Swollen lymph nodes |
|
Nursing Diagnosis
|
! Potential for Pain
! Potential for nutrition deficit. ! Impaired physical mobility ! Disturbed body image ! Knowledge Deficit |
|
Nursing Interventions
|
! Support
! Systems assessment- Monitoring pain, mouth sores, hydration ! Ensure adequate fluid intake. ! Monitor output ! Pain and antiemetic medications ! Monitor blood work |
|
Chemotherapy
|
! Chemotherapy is a medication that can help fight cancer.
! Chemotherapy stops the growth of cancer cells, either by killing the cells or by stopping the cells from dividing. ! Often a combination of chemotherapy drugs are used to fight a specific cancer ( PO, IV or intrathecal). |
|
Chemo Precautions
|
! Gloves
! Special storage and disposal of body fluids and Chemo products. ! Pregnant women should not handle Chemo meds. |
|
Side effects of Chemotherapy
|
! Bone marrow suppression
! Mouth sores ! Nausea ! vomiting and diarrhea ! Hair loss, also called "alopecia“ |
|
Goals of Chemotherapy include:
|
a) Cure
b) Control c) Palliation d) Prophylaxis |
|
Radiation Therapy
|
! The use of ionizing radiation to break apart bonds
within a cell causing cell damage and death. ! External beam therapy accounts for the majority of radiation treatments in children. ! Problems: radiation beams cannot distinguish between malignant cells and healthy cells. |
|
Side effects of radiation
|
! Hair loss, also called "alopecia"
! Bone growth ! Skin changes ! Diarrhea, nausea, and vomiting |
|
Delivery of Medications and
Fluids |
Portacatheters
! Two decades ago, CVAD were introduced as an integral part of the pediatric oncology patient’s treatment plan. ! Used to deliver chemotherapy, blood components, antibiotics, fluids, TPN, medications and blood sampling. |
|
Portacatheter
|
! Single/double lumen
! Sometimes difficult to access ! Decrease risk of infection ! Totally implanted: cannot be pulled out by client and easy to bathe client. ! Requires an incision to remove |
|
Growth and Development cancer kids
|
! Promote normal G & D
! Allow decision making ! Establish daily routines ! Play therapy ! Friends ! School attendance or tutor |
|
Caring for Families
|
! Trusting relationship with nurse
! Open communication ! Daily contact with oncology team ! Support groups Patient / family education ! Begins at time of diagnosis ! Continues through treatment phases ! Maintained in post-survival years ! Support if death of child |
|
Stages of Grief
|
The stages Kubler-Ross identified are:
! Denial (this isn't happening to me!) ! Anger (why is this happening to me?) ! Bargaining (I promise I'll be a better person if...) ! Depression (I don't care anymore) ! Acceptance (I'm ready for whatever comes) |
|
Potential Feelings of children &
Parents diagnosed with cancer |
! Anger
! Guilt ! Depression ! Loneliness ! Fear of death |
|
Nursing interventions to assist families to cope with a loss of a child or pending death.
|
! Take time to spend with the family
! Allow family time to Cry. Allow family time alone. ! Try to assist parents to retain healthy behaviors.: ie enough sleep. Eat a healthy diet. Going outside in the sunshine for exercise or a mild walk. |
|
Nursing Interventions
|
! Encourage family to seek help from others. Reassure that Now is not the time to try to do everything by yourself.
! Seek out grief counseling ! Remember that grief is individual. Not everyone’s grief is identical. ! Families can share some similarities with others, but grieving is a very personal and very individual process. |
|
Wilms
|
! Wilm's tumor, also called nephroblastoma, is a
malignant (cancerous) tumor originating in the cells of the kidney. ! It is the most common type of renal (kidney) cancer and accounts for about 6 percent of all childhood cancers. |
|
Rhabdomyosarcoma
|
! Rhabdomyosarcoma is a cancerous tumor that
originates in the soft tissues of the body, including the muscles, tendons and connective tissues. ! The most common sites for this tumor to be found include the head, neck, bladder, vagina, arms, legs and trunk. |
|
Ewing Sarcoma
|
! Ewing sarcoma is a cancer that occurs primarily in the
bone or soft tissue. ! Ewing sarcoma can occur in any bone, but is most often found in the extremities and can involve muscle and the soft tissues around the tumor site. |
|
Neuroblastoma
|
! Neuroblastoma is a cancerous tumor that begins in nerve tissue of infants and very young children. The abnormal cells are often found in the nerve
tissue that is present in the unborn baby and later develops into a detectable tumor. ! The tumor usually begins in the tissues of the adrenal gland found in the abdomen, but may also begin in nerve tissue in the neck, chest or pelvis. |
|
Lymphoma
|
! Lymphoma is the second most common group of cancer in children.
! Sixty percent of lymphomas are non-Hodgkin's lymphomas. ! Approximately 17% of new cases of cancer. |