Is The Human Form Of Mad Cow Disease Case Study
a. Migraine episode
b. Seizure episode
c. Hallucination episode
d. All of the above
e. A and B only
2. ____________ is the human form of mad cow disease which is caused by the consumption of contaminated beef from cattle.
a. Familial Creutzfeldt-Jakob Disease
b. Iatrogenic Creutzfeldt-Jakob Disease
c. Variant Creutzfeldt-Jakob Disease
d. Sporadic Creutzfeldt-Jakob Disease
3. A 25-year-old female patient was referred to a neurology clinic due to her complaints of vision changes, numbness and muscle weakness. An MRI scan was performed at that time and revealed a white matter disease with increased T cell …show more content…
b. Guilliane Barre syndrome
c. Myasthenia Gravis
e. Both A and B
4. In Alzheimer’s disease, the protein _______ changes to form neurofibrillary tangles _______ the nerve cell. When this happens, ______ disintegrates and destroys the structure of the cell's cytoskeleton, which collapses the neuron's transport system.
a. Amyloid, inside, microfilaments
b. Tau, outside, microfilaments
c. Tau, inside, microtubules
d. Amyloid, outside, microtubules
5. The ____________ normally exert a constant inhibitory influence on a wide range of motor systems, preventing them from becoming active at inappropriate times. When a decision is made to perform a particular action, inhibition is ___________ for the required motor system thereby releasing it for activation.
a. Basal ganglia, reduced
b. Thalamus, reduced
c. Basal ganglia, increased
d. Thalamus, increased
6. Which of the following changes can be observed in neurological diseases but not in psychiatric disorders?
a. Changes in imaging (e.g. CT scan)
b. Changes in behavior
c. Changes in neurotransmitter levels
d. None of the above
7. _____________ was the first drug made for epilepsy that is useful for the prevention of tonic-clonic seizures and partial seizures, but not absence …show more content…
In Alzheimer’s disease, excess glutamate can be released from damaged cells, leading to chronic overexposure to calcium that can speed up cell damage. __________ helps to prevent this destructive chain of events by partially blocking the NMDA receptor
d. None of the above
23. _______________ is a disease that expresses severe muscle degeneration and death of motor neurons. Typical life expectancy for most people with this disease is 2-4 years after diagnosis.
a. Amyotrophic lateral sclerosis (ALS)
b. Myesthania gravis
c. Multiple sclerosis
24. ___________ was a disease caused by consuming human brains in which cerebellum is damaged and displays symptoms such as tremors and loss of coordination.
a. Gerstmann-Sträussler-Scheinker disease (GSS)
c. Fatal familial insomnia
d. Creutzfeldt-Jakob Disease
25. Tremors seen in Parkinson’s disease are called:
a. Essential tremor
b. Action tremor
c. Postural tremor
d. Resting tremor
26. A 25-year-old woman presents with recurrent slurring of speech, trouble swallowing, droopy eyelids and double vision. Her symptoms have progressively deteriorated over the past 6 months. She has intermittent weakness in her legs and arms. After testing, it was revealed that her acetylcholine receptors are being attacked. Her most probable diagnosis would