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103 Cards in this Set

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SC - in the cardiac cath lab

1. C arm gantry may make chest compressions difficult and complicate airway control.


2. Table less stable.


3. Interventionalist usually heavily task loaded.



1. Spontaneous VT/VF due to myocardial irritability and provoked by coronary contrast dye - often involves RCA: rapid defib alone.


2. VT/VF secondary to coronary ischaemia and injury: early defib and treatment of ischaemic substrate.


3. Arriving to lab after prolonged arrest: revascularisation last resort.



-consider tamponade or ventricular rupture: ECHO +/- percardiocentesis

3 + 3

SC - arrest in the operating theatre

-5-7/20,000


-survival to discharge 30% (with 2/3 having good neuro outcomes)


-PEA or asystole


-4H and 4T plus think: CO2 embolus, cement embolus, LA toxicity, malignant hyperthermia



1. Interruptions in compressions may be necessary to achieve surgical control of cause.


2. Manual hyperventilation common in stressed environment.


3. Leadership issues with task overloading seniors

SC - following cardiac surgery

1. Peri-OP MI most common i.e graft occlusion.


2. Post-OP: MI, shock (haemorrhage or hypovolaemic), tamponade, pacing system disconnect, tension pneumothorax, electrolyte imbalance (potassium)



1. Tamponade, haemorrhage and graft occlusion require re-opening and therefore internal cardiac massage


2. Reopen chest if; no output with compressions, shockable rhythm refractory to defib, asystole unresponsive to internal pacing


3. Adrenaline and resulting hypertension may cause failure of graft anastomosis


4. Consider emergency cardiopulmonary bypass


Peri-op: 1


Post-op: 6


4

SC - on intubation of critically ill

-complications in 78% of Emergency intubation


-3% of critically ill inpatients die within 30 mins of intubation


-15% if hypotensive at time <90


-PEA (cardiovascular collapse) usually

4

SC - Anaphylaxis

1. Remove source


2. Adrenaline


3. Standard ALS protocol in arrest


4. May need large vol fluids


5. Prolonged resus may be necessary

5

SC - Asthma

Near fatal: raised arterial CO2 and/or requires high pressure ventilation.


Life-threatening: PEFR <33%, SpO2 <92, normal CO2, silent chest, cyanosis, feeble resp effort, bradycardia, arrhythmias, hypotension, exhaustion, confusion, coma



1. Critical care area


2. IV access


3. Senior help early


4. O2 to maintain sats >92%


5. Salbutamol nebs


6. Hydrocortisone 200mg IV


7. Neb ipratropium


8. Magnesium sulphate 2g IV over 20 mins


9. IV salbutamol 250-500 micrograms IV slowly then infusion 3-20mic/min


10. Fluids


11. Arterial line for repeat ABGs



Other: aminophylline 5mg/kg loading over 20-30min followed by infusion 500-700 micrograms/kg/hr. Heliox. Ketamine. NIV.



Causes:


1. Severe bronchospasm and mucous plugging - hypoxia


2. Gas trapping and hyperinflation


3. Tension pneumothorax


4. Arrhythmias from hypoxia/drugs/electrolyte disturbances



SC


1. Intubate early


2. Gas trapping - apnoea or external decompression


3. Early needle decompression and chest drain (may need bilateral) if necessary

11


Causes: 4


SC: 3

SC - drowning

The process of experiencing respiratory impairment from submersion/immersion in liquid. (Drowing or non-fatal drowning)



If found within 60 mins of submersion start CPR.


No difference between salt and fresh water.



RESCUE BREATHS in water.


High oxygen concentration.


LMA may fail due to high airway pressures or decreased lung compliance.



No evidence to suggest drowing in cold water has better survival outcome.



Delayed lung complications.

SC - pregnancy

Cardiac arrest incidence 1:20,000 - poor outcomes.



Post-mortem caesarean: if rosc not achieved within 4 mins. Survival can occur up to 15 mins from arrest. Higher sternum compressions to adjust for elevated diaphragm.



Lateral displacement of uterus to left in over 20 weeks. Can improve CO 20-30%. 2 or 1 handed technique.



Rapid securement of airway. May need smaller tracheal tube. Cricoid pressure recommended.



Causes of collapse:


1. Amniotic fluid embolism


2. PE


3. Haemorrhage


4. Pre-eclampsia/eclampsia


5. Cardiac disease (peri-partum cardiomyopathy)


6. Sepsis


7. Toxicity

TCA - causes

Most common - hypoxia from airway obstruction and blood loss


Others - tension pneumothorax, tamponade, hypothermia

5

TCA - usual rhythms

PEA and asystole

TCA - considerations

1. Jaw thrust preferred to avoid hyperextension of neck


2. Potential for tension pneumothorax, do bilateral decompression with fibger/needle thoracostomy followed by bilateral chest drains


3. Control catastrophic external haemorrhage


4. Use blood early to correct hypovolaemia


5. If pericardial tamponade, thoracotomy and pericardiotomy may be indicated outside theatre if the right expertise is present

5

Trauma care sequence

1. Primary survey


2. Resus


3. Adjuncts to primary survey


4. Secondary survey


5. Definitive care

5

Trauma primary survey SC - A and c-spine

A


- jaw thrust over head tilt


- oropharyngeal over NP if risk of basal skull fracture



C-spine


- assume all neck pain, neuro deficit, and/or impaired consciousness to have c-spine injury unless proven otherwise


- semi-rigid collars no longer recommended pre-hospital, risk; moving head while sizing, discomfort and pain, airway compromise if vomits, pressure on neck veins causing raised ICP, hiding other injuries.


-in-line bimanual stabilisation, head blocks or Philadelphia collar ok


-intubate with someone providing manual stabilisation from side or below.

2, 4

Trauma primary survey SC - B

- insert chest drain for significant haemothorax, if >1500ml drained immediately or ongoing >200ml/hr then rapid transfer to theatre


-open pneumothorax treatment is assisted with bag to mask device then chest drain. 3-way occlusive dressing no longer initial management.

Trauma primary survey SC - C

- earliest sign of hypovolaemia = progressively narrowing pulse pressure


- systolic hypotension occurs late after 30-40% blood loss


- shock with a high JVP suggests tamponade, tension pneumothorax or pump failure


- permissive hypotension higher in head injury (110) vs non head injury (90)

3

Trauma primary survey SC - D

- AVPU (GCS secondary survey)


- severe hypotension ie from blood loss can cause severe brain dysfunction and fixed dilated pupils - don't assume brain injury until other causes ruled out and managed.

Trauma primary survey adjuncts

ECG - dysrhythmias, tachycardia, AF, ventricular ectopic and ST change may indicate blunt cardiac trauma. Bradycardia, aberrant conduction and premature beats may mean hypoxia and hypoperfusion. PEA may indicate tamponade, tension pneumothorax or profound hypovolaemia.



Ventilatory rate and waveform capnography



Pulse oximetry



BP



Xrays - only CXR and pelvic xray useful in initial management



USS



Cathaterisation to monitor UO



PR exam only consider if; incubated, perineal trauma, penetrating groin injury, spinal cord injury



Orogastric over nasogastric tube in incubated pt: Size 14 or 16FG preferable

Secondary survey sequence

1. Head


2. Face


3. Neck


4. Chest and shoulder girdle


5. Abdomen


6. Pelvis


7. Perineum and rectum


8. Lower limbs


9. Upper limbs


10. CNS incl GCS


11. Back


12. History


13. Tubes


14. Investigations

Best chance of neuro intact survival

1. Witnessed collapse


2. CPR started immediately


3. Rhythm is VF or pVT


4. Defib performed early

What is targeted temperature management

Maintaining constant temp 32-36 with strict avoidance of hyperthermia for those who have arrested and remain unresponsive during ROSC. Rapid infusion of ice-cold IV fluids up to 30ml/kg or ice packs are safe to lower temp by up to 1.5 degrees C. Not recommended pre-hospital.

Causes of arrest in infants (0-1)

- hypoxia


- resp failure


- SUDI


- airway obstruction


- sepsis


- drowning


- poisoning


- neuro disease

8

Causes of arrest in older children

- trauma

At what age does adult protocol 30:2 take over child 15:2?

9

Continuous ventilation no longer recommended

Simultaneous ventilation and compression reduces survival

What rhythm might you suspect gas trapping

PEA

Drug doses in children

-Adrenaline 10microgram/kg (0.1ml/kg of 1:10000)


-Amiodarone 5mg/kg


-Atropine (bradycardia caused by vagal stimulation or cholinergic drug toxicity) 20 micrograms/kg (bradycardia caused by hypoxia treat with vent and o2, severe Brady or with hypotension treat with adrenaline- not atropine)


-Glucose (hypoglycemia) 0.25g/kg by IV/IO I.e 0.5ml/kg of 50% or 2.5ml/kg of 10%


-Glucose (maintainence) in infants 5-8mg/kg/min

2

Shock joules in children

4J/kg

Low CO2 (<10mmHg) in expired breath from a child during CPR may indicate

. A treatable condition: pneumothorax, hypovolaemia, tamponade


. Inadequate compression


. Excessive ventilation

3

High CO2 (>60mmHg) in expired breath implies

Inadequate ventilation

IO placement in children

18-gauge needle or IO cannula inserted perpendicular into anterior surface of tibia 1-3cm below tibial tuberosity. All drugs followed by 5-10ml flush

Alternative to Amiodarone if not available shock-resistent arrest

Lignocaine - 1mg/kg IV/IO

How to avoid dosing errors in children

1. Rounding weight to nearest 5kg


2. Use resus chart with printed drug doses


3. Rounding doses to nearest whole milligram


4. Double check drugs

4

Correction of hypovolaemia in children

-0.9% saline 20ml/kg bolus (25% of childs blood vol)


-blood products are required if 40-60ml/kg of saline failed to reverse hemorrhagic shock

2

PEA or VT

Don't need to waste time feeling for a pulse - if unresponsive and not breathing normally treat as arrest

Describe the types of VF

Coarse- usually earlier and more responsive to defib


Fine - may be indistinguishable from asystole - if unsure continue CPR as it may increase the amplitude



Decreasing amplitude assoc with 5-10% decrease in likelihood of successful defib with each minute of arrest



Recurrent VF/VT with ROSC inbetween indication for earlier amiodarone

4

Defib energy in adults

200J or max energy

Rhythms that may resemble VF

. Polymorphic VT


. AF with WPW



However if either of these rhythm present and pt is in arrest then defib indicated. Very fast SVT may also cause shockable arrest.

When to do a pre cordial thump

. Monitored


. Defib not immediately available


. Onset of VT is witnessed



NOT for VF, if defib immediately available or VT without arrest.

3

Precordial thump - steps

. Clenched fist held approx 25-30cm above sternum


. Fist brought down sharply so ulnar side hits mid-sternum


. Immediately afterwards begin chest compressions (don't feel for pulse)

3

Tachycardia

>120/min

Bradycardia

<60/min

Evidence of low CO

. Pallor


. Sweating


. Cold extremities


. Impaired consciousness or agitation


. Hypotension


. Chest pain

6

Evidence of Heart Failure

. Pulmonary oedema


. Raised JVP


. Peripheral oedema

3

Extreme tachycardia

Narrow complex >200


Broad complex >150

2

Excessive bradycardia

<40

OPA size

Ear lobe to corner of mouth


3 and 4 for average adults

Fitting an LMA

. Size 4 whores and 5 guys


. Fully deflate cuff with 30ml syringe


. Lubricate the back


. Hold like a pen with opening to chin


. Insert along hard palate


. Press down and backwards until will go no further (may need assistant to apply jaw thrust)


. Inflate cuff to (size-1) x 10 (or whatever is on tube)


. Connect self-inflating bag and ventilate gently (<20cm water pressure and slow)


. Place waveform capnography between LMA and the bag to ensure ventilation

9

Intubation assistance

Prepare equipment and drugs


Be ready to inflate cuff


Apply capnography


Keep track of time to avoid prolonged attempts

4

Common mistakes managing an airway

. Removing the pillow


. Inadequate jaw thrust


. Using an OPA the wrong size


. Inadequate seal with the mask


. Attaching the oxygen to the wrong place


. Not enough oxygen


. Focusing on airway at expense of other aspects of care


. Incubating oesophagus


. Failure to assess airway patency with waveform capnography


. Hyperventilation

10

Suction flow rate and pressure

Flow >40L/min


Pressure 300mmHg when tubing occluded

NPA size

6.5 - 8mm

Capnography - normal waveform and ETCO2

Intubation

Oesophageal intubation

What will happen to ETCO2 when CO is reduced?

Will decrease as heart not delivering CO2 to the lungs

ETCO2 of what level indicates ineffective resuscitation

<10mmHg -> push harder and faster!

Loss of CO

Primary action of adrenaline in shockable arrest

Peripheral vasoconstriction to increase coronary perfusion pressure (also may improve cerebral blood flow)

Adrenaline use in non-shockable arrest

Improving coronary and cerebral blood flow through peripheral vasoconstriction.



Overcome cholinergic induced profound bradycardia or asystole.



If in PEA, may improve cardiac contractility.

Adrenaline

1mg IV every 4 mins.



Kids - 10microgram/kg (0.1ml/kg 1:10000)



Shockable - after 2nd shock and every other loop



Non-shockable - immediately after rhythm recognition and every other loop.



Follow each dose by flush to reach central circulation.

5

Amiodarone

Shockable rhythm - after 3rd shock



Earlier if shock appears to be transiently successful.



300mg IV.


Second dose of 150mg may be considered.



5mg/kg in kids.



Lignocaine 1mg/kg as alternative

5

Causes of arterial hypoxia

1. Low inspired oxygen


2. Diffusion abnormalities


3. Ventilation/perfusion mismatch


4. Shunt

4

Such PEEP be used to overcome hypoxia?

100% oxygen should be given to overcome hypoxia.



If someone arrests from a disease responsive to PEEP use with extreme caution as increasing intra-thoracic pressure impedes venous return and therefore CO

What rhythms might hypovolaemia cause?

PEA with progressively more abnormal complexes until asystole.



Occasionally may be VF/VT due to myocardial ischaemia, especially if underlying IHD.

How to rewarm at 30-32 degrees

Passive using blankets.


Active using hot air blankets, warm baths, warm packs on truncal areas.

2

Rhythms in hypothermia

Sinus Brady 》 AF 》 VF 》 asystole

Rewarding techniques in arrest due to hypothrmia

. Cardiopulmonary bypass ideal


. Warmed saline lavage of peritoneal and thoracic cavities


. Warmed IV/IO fluids


3

What is malignant hyperthermia?

A life-threatening generic generic sensitivity of skeletal muscles to volatile anesthetics and depolarisaing neuromuscular blocking drugs (suxamethonium) occurring during or very soon after anaesthesia

How to manage malignant hyperthermia

1. Stop triggering agents


2. Call for help (senior anaesthetist)


3. Dantrolene 2-5mg/kg repeated as necessary


4. Correct accompanying acidaemia and electrolyte imbalance

Causes of hyperkalaemia

. Excessive production - oral intake in renal failure


. Tissue breakdown - rhabdomyolysis, tissue necrosis, burns, tumour loss syndrome


. Ineffective elimination - aldosterone deficiency


. Impair excretion by kidneys

4

ECG findings in hyperkalaemia

Mild-mod - reduced size of p waves and peaked t waves


Severe - widening of QRS complex

9

Management of hyperkalaemia

. Reduce myocardial contractility: calcium chloride 10% (x3 calcium than calcium gluconate), onset less than 5 mins, duration 30-60 mins. Repeat if ECG doesn't normalise in 3-5 mins



. Lower K temporarily:


- insulin 10 units IV insulin with 50ml of 50% dextrose.


- bicarbonate 50ml (50mmol) over 5 mins


- salbutamol neb 10-20mg



. Increase elimination: haemodialysis/haemofiltration. Furosemide.

6

ECG finding hypokalemia

U waves


T wave flattening


Arrhythmias (ventricular)

Management of hypermagnesaemia

Calcium chloride 10% 5-10ml


Calcium gluconate 10% 15-30ml

2

Management hypomagnesaemia

1-2g MgSO4 as IV bolus



Far more common than hypermagnesaemia.



Associated with pVT incl torsades de pointes

3

Needle thoracostomy steps

. 14-16G cannula into 2nd space mid clavicular line


. Advance needle until air aspirated into syringe attached to needle


. Leave cannula in


. Follow with chest drain

4

Finger thoracostomy steps

. Identify 4th or 5th ICS mid axillary line


. Clean skin


. LA in conscious patient


. Scalpel incision along upper border of rib


. Blunt dissection into pleural cavity


. Finger inserted into pleural cavity


. Follow with chest drain

7

Causes of tamponade

. Infection (TB)


. Trauma


. Tumour


. Aortic dissection


. Post cardiac surgery

5

What is Beck's Triad?

Clinical signs of tamponade


. Hypotension


. JVP distension


. Absent or distant HS

Management of tamponade

Pericardiocentesis

Antidote to beta blockers

Glucagon


Milrinone


Insulin


Calcium


Bypass

5

Antidote to benzos

Airway and vent support


Flumazanil may precipitate seizures so should generally be avoided

2

Antidote to calcium channel blockers

Calcium


Glucagon


Milrinone


Bypass

4

Antidote to carbon monoxide

100% oxygen

1

Antidote to cyanide

Oxygen


Amyl nitrate


Sodium thiosulphate


Sodium nitrate


Hydroxycobalamin

5

Antidote to digoxin

Correct hypomagnesaemia/hypokalaemia


Digoxin Fab fragments


Cardiac pacing

3

Antidote to ethylene glycol

Alcohol


Hemodialysis

2

Antidote to iron

Whole bowel irrigation


Desferrioxamine

2

Antidote to lithium

Hemodialysis


Whole bowel irrigation

2

Antidote to LA

Intralipid

1

Antidote to methanol

Alcohol


Dialysis

2

Antidote to opiates

Ventilation


Naloxone

2

Antidote to organophosphates

Atropine


Pralidoxime


Benzos

3

Antidote to paracetamol

N-acetyl cysteine

1

Antidote to salicylates

Haemodialysis

1

Antidote to TCA

NaHCO3


Intralipid

2

Where should the tip of the endotracheal tube sit?

At least 2cm above the carina

How to alter oxygenation in the incubated person

1. Altered inspired oxygen


2. Alter the PEEP up to 15cmH20 if normovolaemic. Cautious in cardiac arrest or hypovolaemia as increasing intra thoracic pressure may impede venous return and thus CO.



3. Recruitment manoeuvres


4. Inhaled pulmonary vasodilators ie nitric oxide


5. Prone positioning


6. Alternate ventilation strategies ie inverse ratio ventilation

6

Normocapnia

35-40 mmHg

Hypocapnia may cause

Cerebral vasoconstriction



(Hypercapnia may cause cerebral vasodilatation)

How is ventilation altered?

1. Alter RR


2. Alter volume of each breath



Ventilation = tidal vol x RR


Ventilation is inversely proportional to PaCO2 (doubling ventilation halves PaCO2 and vice versa)

4

Ventilation rate

5-6ml/kg at rate of 15 breaths/min

How different is ETCO2 to arterial CO2?

Lower by 5-6mmHg in healthy individuals (more following arrest)