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148 Cards in this Set

  • Front
  • Back
DC-ML
discriminative touch, vibration, proprioception crosses at medulla
ALS/Spinothalamic Tract
pain, temperature and crude touch. Crosses at level of entry in spinal cord
Trigeminothalamic Tract
all sense from head. Pontine crossing
Spino/Cuneocerebellar Tracts
movement and postion sense from body. Crosses at spinal cord level of entry
Corticospinal Tract
voluntary movement of limbs. Pyramidal decussation
Corticobulbar Tract
motor control of head and neck. Crosses at brainstem level of exit
CN X→ Laryngeal/Pharyngeal
nucleus ambiguous
Taste from CN VII, IX, X
solitary nucleus
LMN Lesion
weak, hypotonia, hyporeflexia, atrophy
UMN Lesion
Corticospinal sign
UL flexors, LL extensors with distal weakness
Hyper-relfexia
UMN lesion, due to destruction of cortiocoreticulospinal inhibitory influence
Babinski
UMN lesion
Transient hypotonia/hyporeflexia
UMN lesion
Pure Corticospinal tract lesion
Ventral Root Lesion
LMN findings
Sympathetic deficits
C8-L2→ skin flushed, dry (no sweating), will not blanch. C8-T2 → Horners syndrome
Parasympathetic defecits (S2-S4)
erectile dysfunction, bowel, bladder dysfunction
Dorsal Root Lesion
hyptonia, hyporeflexia (sensory limb defect), anesthesia, atrophic skin changes.
Brown Sequard Syndrome
spinal hemisection. Ipsilateral paralysis of positon, vibration touch (DCML) and contralateral loss of pain and temperature one segment below lesion
Horners Syndrome
sympathetic lesions C8-T2. Ipsilateral ptosis, miosis, anhydrosis, flushing of skin and face.
Midbrain CN
II, III, IV
Pontine CN
V, VI, VII, VIII
Medullary CN
IX, X, XI, XII
Caudal Ventromedial Medulla
contralateral hemipareisis → corticospinal, contralateral loss of positioning and vibratory sense → DC-ML (medial leminicus)
Rostral Ventromedial Medulla
contralateral hemiparesis → corticospinal, Contralateral prop/vib→ DC ML*, ipsilateral tongue paralysis → CN XII rootlet
Caudal Ventrolateral Pons
contralateral hemiparesis → corticospinal tract, ipsilateral facial paralysis → facial nerve root
Rostral Ventrolateral Pons
contralateral hemiparesis → corticospinal tract, ipsilateral loss of jaw reflex and coreneal blink reflex and ipsilateral loss of sensation on face → trigeminal nerve root
Pontine Tegmentum, Rostral
contralateral loss of body position and vibratory sense → mediallemnisucs, Contralateral loss of pain and temperature frm body → spinothalamic tract, Ipsilateral loss of face pain and temperature, corenal blink reflex, and jaw jerk reflex → trigeminal nerve root
Pontine Tegmentum, Caudal
contralateral loss of body position and vibratory sense → medial leminiscus, contralateral loss of body poain and temperature → spinothalamic tract, Ipsilateral facial paralysis → Facial nerve root, Ipsilateral internal strabisumus and paralysis of eye abduction → abducens nerve root, ipsilateral loss of paoin and temperature → spinal trigeminal tract, nystagmus and vertigo → vestibular nuclei, horner’s syndrome → descending central sympathetics
Anterior internuclear opthalmoplegia, Multiple sclerosis
Paralsysis of eye adduction → medial longitudinal fasiculus, intact adduction on convergence → medial longitunidal fasiculus, nyastagimus of abducting eye on lateral gaze → medial longitudinal fasiculus
Ventral Pons/locked in syndrome
quadriplegia→ bilateral cortiospinal tracts, bilateral facial, laryngealm lingual paralysis → corticobulbar tracts. EYES are able to move. Basilar artery knock out
Central midbrain tegmentum
contralateral intention tremor → red nucleus, Paralysis of vertical and medial movments of ipsilateral eye, pupillary dilation, loss of light rflex, ptosis, external strabismus → oculomotor nucleus, contralateral loss of body and vibratory sense → medial leminiscus
Ventromedial Midbrain (webbers)
ipsilateral oculomotor nerve signs → oculomotor nerve rootlets. Contralateral hemiparesis → corticospinal tract, Contralateral lower facial muscle paresis, contralateral tongue paresis → corticobulbuar tracts
Diencephalon Lesion
contralateral hemihypoalgesaia and intense pain → ventral posterolateral thalamic nucleus, Contralateral hemiparesis, Babinski, hyperreflexia→ corticospinal fibers in posterior limb of internal capsule, homonymous hemianopsia → lateral geniculate nucleus
Lateral Medulla/Wallenberg’s Syndrome/PICA
cont loss pain/temp → lateral spinothalamic tract. Ipsi loss of pain/temp → spinal tract and CN V, dysphagia/dysphonia → nucleus ambiguous, horner’s syndrome → descending cntral sympathetics, ipsi atazia of limbs → inferior cerebellar peduncle, ipsi dearness and tinnitus → cochlear nuclei, nystagumus, nausea, vomiting, vertigo → vestibular nuclei, ipsi loss of taste → solitary nucleus
DC-ML
discriminative touch, vibration, proprioception crosses at medulla
ALS/Spinothalamic Tract
pain, temperature and crude touch. Crosses at level of entry in spinal cord
Trigeminothalamic Tract
all sense from head. Pontine crossing
Spino/Cuneocerebellar Tracts
movement and postion sense from body. Crosses at spinal cord level of entry
Corticospinal Tract
voluntary movement of limbs. Pyramidal decussation
Corticobulbar Tract
motor control of head and neck. Crosses at brainstem level of exit
CN X→ Laryngeal/Pharyngeal
nucleus ambiguous
Taste from CN VII, IX, X
solitary nucleus
LMN Lesion
weak, hypotonia, hyporeflexia, atrophy
UMN Lesion
Corticospinal sign
UL flexors, LL extensors with distal weakness
Hyper-relfexia
UMN lesion, due to destruction of cortiocoreticulospinal inhibitory influence
Babinski
UMN lesion
Transient hypotonia/hyporeflexia
UMN lesion
Pure Corticospinal tract lesion
Ventral Root Lesion
LMN findings
Sympathetic deficits
C8-L2→ skin flushed, dry (no sweating), will not blanch. C8-T2 → Horners syndrome
Parasympathetic defecits (S2-S4)
erectile dysfunction, bowel, bladder dysfunction
Dorsal Root Lesion
hyptonia, hyporeflexia (sensory limb defect), anesthesia, atrophic skin changes.
Brown Sequard Syndrome
spinal hemisection. Ipsilateral paralysis of positon, vibration touch (DCML) and contralateral loss of pain and temperature one segment below lesion
Horners Syndrome
sympathetic lesions C8-T2. Ipsilateral ptosis, miosis, anhydrosis, flushing of skin and face.
Midbrain CN
II, III, IV
Pontine CN
V, VI, VII, VIII
Medullary CN
IX, X, XI, XII
Caudal Ventromedial Medulla
contralateral hemipareisis → corticospinal, contralateral loss of positioning and vibratory sense → DC-ML (medial leminicus)
Rostral Ventromedial Medulla
contralateral hemiparesis → corticospinal, Contralateral prop/vib→ DC ML*, ipsilateral tongue paralysis → CN XII rootlet
Caudal Ventrolateral Pons
contralateral hemiparesis → corticospinal tract, ipsilateral facial paralysis → facial nerve root
Rostral Ventrolateral Pons
contralateral hemiparesis → corticospinal tract, ipsilateral loss of jaw reflex and coreneal blink reflex and ipsilateral loss of sensation on face → trigeminal nerve root
Pontine Tegmentum, Rostral
contralateral loss of body position and vibratory sense → mediallemnisucs, Contralateral loss of pain and temperature frm body → spinothalamic tract, Ipsilateral loss of face pain and temperature, corenal blink reflex, and jaw jerk reflex → trigeminal nerve root
Pontine Tegmentum, Caudal
contralateral loss of body position and vibratory sense → medial leminiscus, contralateral loss of body poain and temperature → spinothalamic tract, Ipsilateral facial paralysis → Facial nerve root, Ipsilateral internal strabisumus and paralysis of eye abduction → abducens nerve root, ipsilateral loss of paoin and temperature → spinal trigeminal tract, nystagmus and vertigo → vestibular nuclei, horner’s syndrome → descending central sympathetics
Anterior internuclear opthalmoplegia, Multiple sclerosis
Paralsysis of eye adduction → medial longitudinal fasiculus, intact adduction on convergence → medial longitunidal fasiculus, nyastagimus of abducting eye on lateral gaze → medial longitudinal fasiculus
Ventral Pons/locked in syndrome
quadriplegia→ bilateral cortiospinal tracts, bilateral facial, laryngealm lingual paralysis → corticobulbar tracts. EYES are able to move. Basilar artery knock out
Central midbrain tegmentum
contralateral intention tremor → red nucleus, Paralysis of vertical and medial movments of ipsilateral eye, pupillary dilation, loss of light rflex, ptosis, external strabismus → oculomotor nucleus, contralateral loss of body and vibratory sense → medial leminiscus
Ventromedial Midbrain (webbers)
ipsilateral oculomotor nerve signs → oculomotor nerve rootlets. Contralateral hemiparesis → corticospinal tract, Contralateral lower facial muscle paresis, contralateral tongue paresis → corticobulbuar tracts
Diencephalon Lesion
contralateral hemihypoalgesaia and intense pain → ventral posterolateral thalamic nucleus, Contralateral hemiparesis, Babinski, hyperreflexia→ corticospinal fibers in posterior limb of internal capsule, homonymous hemianopsia → lateral geniculate nucleus
Lateral Medulla/Wallenberg’s Syndrome/PICA
cont loss pain/temp → lateral spinothalamic tract. Ipsi loss of pain/temp → spinal tract and CN V, dysphagia/dysphonia → nucleus ambiguous, horner’s syndrome → descending cntral sympathetics, ipsi atazia of limbs → inferior cerebellar peduncle, ipsi dearness and tinnitus → cochlear nuclei, nystagumus, nausea, vomiting, vertigo → vestibular nuclei, ipsi loss of taste → solitary nucleus
DC-ML
discriminative touch, vibration, proprioception crosses at medulla
ALS/Spinothalamic Tract
pain, temperature and crude touch. Crosses at level of entry in spinal cord
Trigeminothalamic Tract
all sense from head. Pontine crossing
Spino/Cuneocerebellar Tracts
movement and postion sense from body. Crosses at spinal cord level of entry
Corticospinal Tract
voluntary movement of limbs. Pyramidal decussation
Corticobulbar Tract
motor control of head and neck. Crosses at brainstem level of exit
CN X→ Laryngeal/Pharyngeal
nucleus ambiguous
Taste from CN VII, IX, X
solitary nucleus
LMN Lesion
weak, hypotonia, hyporeflexia, atrophy
UMN Lesion
Corticospinal sign
UL flexors, LL extensors with distal weakness
Hyper-relfexia
UMN lesion, due to destruction of cortiocoreticulospinal inhibitory influence
Babinski
UMN lesion
Transient hypotonia/hyporeflexia
UMN lesion
Pure Corticospinal tract lesion
Ventral Root Lesion
LMN findings
Sympathetic deficits
C8-L2→ skin flushed, dry (no sweating), will not blanch. C8-T2 → Horners syndrome
Parasympathetic defecits (S2-S4)
erectile dysfunction, bowel, bladder dysfunction
Dorsal Root Lesion
hyptonia, hyporeflexia (sensory limb defect), anesthesia, atrophic skin changes.
Brown Sequard Syndrome
spinal hemisection. Ipsilateral paralysis of positon, vibration touch (DCML) and contralateral loss of pain and temperature one segment below lesion
Horners Syndrome
sympathetic lesions C8-T2. Ipsilateral ptosis, miosis, anhydrosis, flushing of skin and face.
Midbrain CN
II, III, IV
Pontine CN
V, VI, VII, VIII
Medullary CN
IX, X, XI, XII
Caudal Ventromedial Medulla
contralateral hemipareisis → corticospinal, contralateral loss of positioning and vibratory sense → DC-ML (medial leminicus)
Rostral Ventromedial Medulla
contralateral hemiparesis → corticospinal, Contralateral prop/vib→ DC ML*, ipsilateral tongue paralysis → CN XII rootlet
Caudal Ventrolateral Pons
contralateral hemiparesis → corticospinal tract, ipsilateral facial paralysis → facial nerve root
Rostral Ventrolateral Pons
contralateral hemiparesis → corticospinal tract, ipsilateral loss of jaw reflex and coreneal blink reflex and ipsilateral loss of sensation on face → trigeminal nerve root
Pontine Tegmentum, Rostral
contralateral loss of body position and vibratory sense → mediallemnisucs, Contralateral loss of pain and temperature frm body → spinothalamic tract, Ipsilateral loss of face pain and temperature, corenal blink reflex, and jaw jerk reflex → trigeminal nerve root
Pontine Tegmentum, Caudal
contralateral loss of body position and vibratory sense → medial leminiscus, contralateral loss of body poain and temperature → spinothalamic tract, Ipsilateral facial paralysis → Facial nerve root, Ipsilateral internal strabisumus and paralysis of eye abduction → abducens nerve root, ipsilateral loss of paoin and temperature → spinal trigeminal tract, nystagmus and vertigo → vestibular nuclei, horner’s syndrome → descending central sympathetics
Anterior internuclear opthalmoplegia, Multiple sclerosis
Paralsysis of eye adduction → medial longitudinal fasiculus, intact adduction on convergence → medial longitunidal fasiculus, nyastagimus of abducting eye on lateral gaze → medial longitudinal fasiculus
Ventral Pons/locked in syndrome
quadriplegia→ bilateral cortiospinal tracts, bilateral facial, laryngealm lingual paralysis → corticobulbar tracts. EYES are able to move. Basilar artery knock out
Central midbrain tegmentum
contralateral intention tremor → red nucleus, Paralysis of vertical and medial movments of ipsilateral eye, pupillary dilation, loss of light rflex, ptosis, external strabismus → oculomotor nucleus, contralateral loss of body and vibratory sense → medial leminiscus
Ventromedial Midbrain (webbers)
ipsilateral oculomotor nerve signs → oculomotor nerve rootlets. Contralateral hemiparesis → corticospinal tract, Contralateral lower facial muscle paresis, contralateral tongue paresis → corticobulbuar tracts
Diencephalon Lesion
contralateral hemihypoalgesaia and intense pain → ventral posterolateral thalamic nucleus, Contralateral hemiparesis, Babinski, hyperreflexia→ corticospinal fibers in posterior limb of internal capsule, homonymous hemianopsia → lateral geniculate nucleus
Lateral Medulla/Wallenberg’s Syndrome/PICA
cont loss pain/temp → lateral spinothalamic tract. Ipsi loss of pain/temp → spinal tract and CN V, dysphagia/dysphonia → nucleus ambiguous, horner’s syndrome → descending cntral sympathetics, ipsi atazia of limbs → inferior cerebellar peduncle, ipsi dearness and tinnitus → cochlear nuclei, nystagumus, nausea, vomiting, vertigo → vestibular nuclei, ipsi loss of taste → solitary nucleus
DC-ML
discriminative touch, vibration, proprioception crosses at medulla
ALS/Spinothalamic Tract
pain, temperature and crude touch. Crosses at level of entry in spinal cord
Trigeminothalamic Tract
all sense from head. Pontine crossing
Spino/Cuneocerebellar Tracts
movement and postion sense from body. Crosses at spinal cord level of entry
Corticospinal Tract
voluntary movement of limbs. Pyramidal decussation
Corticobulbar Tract
motor control of head and neck. Crosses at brainstem level of exit
CN X→ Laryngeal/Pharyngeal
nucleus ambiguous
Efferent motor fibers of CN IX
nucleus ambiguous
Taste from CN VII, IX, X
solitary nucleus
LMN Lesion
weak, hypotonia, hyporeflexia, atrophy
UMN Lesion
Corticospinal sign
UL flexors, LL extensors with distal weakness
Hyper-relfexia
UMN lesion, due to destruction of cortiocoreticulospinal inhibitory influence
Babinski
UMN lesion
Transient hypotonia/hyporeflexia
UMN lesion
Pure Corticospinal tract lesion
Ventral Root Lesion
LMN findings
Sympathetic deficits
C8-L2→ skin flushed, dry (no sweating), will not blanch. C8-T2 → Horners syndrome
Parasympathetic defecits (S2-S4)
erectile dysfunction, bowel, bladder dysfunction
Dorsal Root Lesion
hyptonia, hyporeflexia (sensory limb defect), anesthesia, atrophic skin changes.
Brown Sequard Syndrome
spinal hemisection. Ipsilateral paralysis of positon, vibration touch (DCML) and contralateral loss of pain and temperature one segment below lesion
Horners Syndrome
sympathetic lesions C8-T2. Ipsilateral ptosis, miosis, anhydrosis, flushing of skin and face.
Midbrain CN
II, III, IV
Pontine CN
V, VI, VII, VIII
Medullary CN
IX, X, XI, XII
Caudal Ventromedial Medulla
contralateral hemipareisis → corticospinal, contralateral loss of positioning and vibratory sense → DC-ML (medial leminicus)
Rostral Ventromedial Medulla
contralateral hemiparesis → corticospinal, Contralateral prop/vib→ DC ML*, ipsilateral tongue paralysis → CN XII rootlet
Caudal Ventrolateral Pons
contralateral hemiparesis → corticospinal tract, ipsilateral facial paralysis → facial nerve root
Caudal Ventromedial Pons
Contralateral hemiparesis → corticalspinal tract, Ipsilateral internal strabismus and paralysis of eye abduction → abducens nerve root
Rostral Ventrolateral Pons
contralateral hemiparesis → corticospinal tract, ipsilateral loss of jaw reflex and coreneal blink reflex and ipsilateral loss of sensation on face → trigeminal nerve root
Pontine Tegmentum, Rostral
contralateral loss of body position and vibratory sense → mediallemnisucs, Contralateral loss of pain and temperature frm body → spinothalamic tract, Ipsilateral loss of face pain and temperature, corenal blink reflex, and jaw jerk reflex → trigeminal nerve root
Horner’s syndrome
descending central sympathetics
Pontine Tegmentum, Caudal
contralateral loss of body position and vibratory sense → medial leminiscus, contralateral loss of body poain and temperature → spinothalamic tract, Ipsilateral facial paralysis → Facial nerve root, Ipsilateral internal strabisumus and paralysis of eye abduction → abducens nerve root, ipsilateral loss of paoin and temperature → spinal trigeminal tract, nystagmus and vertigo → vestibular nuclei, horner’s syndrome → descending central sympathetics
Anterior internuclear opthalmoplegia, Multiple sclerosis
Paralsysis of eye adduction → medial longitudinal fasiculus, intact adduction on convergence → medial longitunidal fasiculus, nyastagimus of abducting eye on lateral gaze → medial longitudinal fasiculus
Ventral Pons/locked in syndrome
quadriplegia→ bilateral cortiospinal tracts, bilateral facial, laryngealm lingual paralysis → corticobulbar tracts. EYES are able to move. Basilar artery knock out
Central midbrain tegmentum
contralateral intention tremor → red nucleus, Paralysis of vertical and medial movments of ipsilateral eye, pupillary dilation, loss of light rflex, ptosis, external strabismus → oculomotor nucleus, contralateral loss of body and vibratory sense → medial leminiscus
Ventromedial Midbrain (webbers)
ipsilateral oculomotor nerve signs → oculomotor nerve rootlets. Contralateral hemiparesis → corticospinal tract, Contralateral lower facial muscle paresis, contralateral tongue paresis → corticobulbuar tracts
Diencephalon Lesion
contralateral hemihypoalgesaia and intense pain → ventral posterolateral thalamic nucleus, Contralateral hemiparesis, Babinski, hyperreflexia→ corticospinal fibers in posterior limb of internal capsule, homonymous hemianopsia → lateral geniculate nucleus
Caudal Pons
CN → CN VI, CN VII
Lateral Medulla/Wallenberg’s Syndrome/PICA
cont loss pain/temp → lateral spinothalamic tract. Ipsi loss of pain/temp → spinal tract and CN V, dysphagia/dysphonia → nucleus ambiguous, horner’s syndrome → descending cntral sympathetics, ipsi atazia of limbs → inferior cerebellar peduncle, ipsi dearness and tinnitus → cochlear nuclei, nystagumus, nausea, vomiting, vertigo → vestibular nuclei, ipsi loss of taste → solitary nucleus