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265 Cards in this Set
- Front
- Back
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What are the modes of infection of the CNS by Viruses?[3]
|
Hematogenous (most common)
Olfactory Nerve Intra-axonally |
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What are the outcomes of viral infection in the CNS?[4]
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Cell death
Cell transformation Persistent infection with altered cell function Persistent infection without altered cell function |
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What is the protein finding in a CSF sample with a viral infection?
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Increased protein
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What is the glucose finding in a CSF sample with viral infection?
|
Normal glucose
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What is the white cell profile in a CSF sample with a viral infection?
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Increased lyphocytes
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What would be the gram stain finding in a CSF sample of viral infection?
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No bacteria
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What are the common etiological agents of viral CNS infection?[3]
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ECHO
Coxsakie virus Poliomyelitis |
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What family is enterovirus?
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Picornaviridae
|
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Describe enterovirus?
|
ssRNA
Non-enveloped Icosohedral |
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What are the two classes of enterovirus?
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Poliovirus
Non-poliovirus |
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What viruses are included in the non-poliovirus enterovirus?[3]
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Coxsackie
Echovirus Unclassified |
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How many coxsackie A serotypes are identified?
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23
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How many coxsakie B serotypes are identified?
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6
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How many echovirus serotypes are identified?
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28
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At what point int he viral infection is there CNS involvement?[2]
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During major viremic phase
Later |
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What is the mode of spread of the enteroviruses?
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Feco-oral
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What season do the enteroviruses spread?
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Warm weather months
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What age group is the most susceptible to enterovirus CNS infection?[2]
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Infants
Children |
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What is the ultimate coarse of enterovirus CNS infection?
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It is self limiting
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What is the most causative agent of CNS viral infection?
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Enterovirus
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What are teh commonly encountered infections associated with enterovirus?[4]
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Hand foot and moth disease
Herpangia Myocarditis Pleurodynia |
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A viral infection (usually in children) marked by sore throat and fever and papules in the mouth and throat and headache and abdominal pain; usually subsides in a short time
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Herpangina
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Describe Herpangina
|
A viral infection (usually in children) marked by sore throat and fever and papules in the mouth and throat and headache and abdominal pain; usually subsides in a short time
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Pain in the pleural cavity
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Pleurodynia
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Pleurodynia
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Pain in the pleural cavity
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Affects infants and children, and is quite common. It is moderately contagious and is spread through direct contact with the mucus, saliva, or feces of an infected person. It typically occurs in small epidemics in nursery schools or kindergartens, usually during the summer and autumn months
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Hand foot and mouth disease
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Describe hand foot and mouth disease
|
Affects infants and children, and is quite common. It is moderately contagious and is spread through direct contact with the mucus, saliva, or feces of an infected person. It typically occurs in small epidemics in nursery schools or kindergartens, usually during the summer and autumn months
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What is the predominant presentation of enterovirus CNS infection?
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Asymptomatic or subclinical (60%)
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What type of CNS infection does enterovirus usually cause?
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Meningitis
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What is the result of Enterovirus mediated encephlitis?
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Permanent damage
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Describe coxsackie virus[3]
|
ssRNA
Non-enveloped Icosohedral |
|
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What family does coxsackie virus belong to?
|
Picornaviridae
|
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What is the route of transmission of coxsackie virus?[3]
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Feco-oral
Respiratory Fomites |
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What symptom does coxsackie A infection of the CNS cacuse?
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Flaccid paralysis
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What other diseases (of the skin) are caused by coxsackie A virus infection?[2]
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Herpangina
Hand Foot and Mouth disease |
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What is the ultimate course of coxasckie A virus infection (including time period)?
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Mild infection with recovery in 7 to 10 days
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What symptom does coxsackie B infection of the CNS cacuse?
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Spastic paralysis
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What other diseases are caused by coxsackie B virus infection?[4]
|
Pleurodynia
Myocarditis Pericarditis Hepatitis |
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What are symptoms seen with infection of coxsackie A and B?[4]
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Fever
Rash Upper Respiratory Tract Infection Aseptic Meningitis |
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What is the most common (>90%) cause for aseptic meningitis?[2]
|
Coxsackie virus
Echovirus |
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What are the symptoms of coxsackie aseptic meningitis?[8]
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Fever
Chills Nausea Vomiting Neck pain Photophobia Rash Upper Respiratory Symptoms |
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What is a complication which occurs with about 5-10% of coxsackie aseptic meningitis cases?
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Encephalitis
|
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What demographic has the highest incidence of aseptic meningitis?
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Infants < 1 year old
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What are the long term deficits appear with coxsackie aseptic meningitis?
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The are no long term deficits with coxsackie aseptic meningitis
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What are the symptoms of coxsackie encephalitis?[6]
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Altered mental status
Personality changes Neurologic deficits Speech impairments Motor impairments Sensory impairments |
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What history is often present with a patient who has coxsackie encephalitis?
|
Rash
Preceded by Upper Respiratory tract infection |
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Patient has altered mental status, speech and motor impairments. Presented 8 days earlier with upper respiratory infection and at that time you noted the presence of a rash. What is the most likely diagnosis?
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Coxsackie encephalitis
|
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Vesicular lesions that appear on the tongue, palmar surfaces and the buttocks.
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Hand foot and mouth disease
|
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Vesicular exanthma of the oropharynx with fever sore throat odynophagia and dysphagia.
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Herpangina
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Disease of the muscles with fever, paroysmal spasmotic pain in chest and upper abdomen.
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Pelurodynia
|
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How would you diagnose for enterovirus CNS infection?[3]
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CSF sample PCR for enterovirus
PCR in Non-CNS site lesions(throat) EEG |
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What are some Non-CNS site for infection by enterovirus, from which detection can be made?[2]
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Throat
Stool |
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In what regions is Polio virus still endemic?[4]
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Afghanistan
Nigeria Pakistan India |
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What are the manifestations of polio virus infection?[5]
|
Unapparent infection
Abortive polio Nonparalytic polio Paralytic polio Polioencephalitis |
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Polio virus resides in the Reticular-epithelium system and is asymptomatic.
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Unapparent infection
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Polio infection which is similar(symptomatically) to other enterovirus causes of aseptic meningitis.
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Nonparalytic polio
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Nonparalytic polio
|
Polio infection which is similar(symptomatically) to other enterovirus causes of aseptic meningitis.
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"Minor illness" polio infection which resembles other viral illnesses
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Abortive polio
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Abortive polio
|
"Minor illness" polio infection which resembles other viral illnesses
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What are the types of paralytic polio?[2]
|
Spinal
Bulbar |
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Spinal paralytic polio/Bulbar paralytic polio
Flaccid paralysis |
Spinal paralytic polio
|
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Spinal paralytic polio/Bulbar paralytic polio
Paralysis of CN. IX & X |
Bulbar paralytic polio
|
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Spinal paralytic polio/Bulbar paralytic polio
Lysis of the anterior horns |
Spinal paralytic polio
|
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Spinal paralytic polio/Bulbar paralytic polio
Encroachment on respiratory centers |
Bulbar paralytic polio
|
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What cranial nerves are affected in parlytic polio?[2]
|
CN IX
CN X |
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Why is there flaccid paralysis in spinal paralytic polio?
|
Degeneration of the anterior horn cells
|
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Spinal paralytic polio/Bulbar paralytic polio
Needed iron lung for treatment in the past |
Bulbar paralytic polio
|
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What is the route of transmission of polio?
|
Feco-oral
|
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Progressive weakness developing in a patient several decades after a polio infection
|
Post polio syndrome
|
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Post polio syndrome
|
Progressive weakness developing in a patient several decades after a polio infection
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T/F Post polio syndrome is a recurrence of polio virus infection
|
False
|
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T/F Post polio syndrome results in degenerated motor neurons
|
True
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Motor neurons which regenerate after a polio infection are (weaker/stronger/same)?
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Weaker
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Weaker motor neuron after regeneration after polio virus infection can lead to ...
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Post polio syndrome
|
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How long after polio infection does post polio infection occur?
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Decades
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What family does Lymphocytic Choriomeningitis Virus (LCMV) belong?
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Arenaviridae
|
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How does a patient acquire Lymphocytic Choriomeningitis Virus (LCMV)?
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Contact with rodent excreta
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Contact with rodent excreta puts a person at risk for acquiring what CNS virus?
|
Lympocytic Choriomeningitis Virus
|
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What percentage of meningitis are due to herpesviruses?
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<3%
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What is the course of CNS infection with Lymphocytic Choriomeningitis Virus (LCMV)
|
Recovery
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What herpes virus in most common in CNS infections?
|
HSV-2
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Most common cause for fatal sporadic encephalitis?
|
Herpes simplex encephalitis
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What virus causes Herpes simplex encephalitis in adults?
|
HSV-1
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What causes Herpes simplex encephalitis in children?
|
HSV-2
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Focal damage of the anterior lobe of the cerebellum and the orbitofrontal lobe of the cerebrum.
|
Herpes simplex encephalitis
|
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What are eh symptoms of herpes simplex encephalitis?[5]
|
Personality changes
Bizarre behavior Hallucinations Seizures Hemiparesis |
|
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What is the incidence of herpes simplex encephalitis?
|
2-4 per million per year
|
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What CSF findings would be consistent with herpes simplex encephalitis?[3]
|
RBC
WBC + HSV PCR |
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T/F Image studies such as MRI and CT can be used int he diagnosis of herpes simplex encephlopathy?
|
True
|
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What is the treatment for herpes simplex encephalopathy?
|
Acylovir IV over 10 days
|
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How does Acylcovir work?
|
Inactivates viral DNA synthesis by inactivating thymadine kinase
|
|
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What is an alternative to Acyclovir?
|
Famcyclovir
|
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Describe Herpes viruses.
|
ds DNA
Enveloped Icosadeltahedral capsid |
|
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What is the incubation period for Herpes varicellae?
|
10 to 23 days
|
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T/F Herpes varicellae can survive outside of the body.
|
True
|
|
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What are some complications from Herpes vericellae infection in the pregnant female early in gestation?[3]
|
Microcephaly
Motor disability Cataracts |
|
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Massive and fused necrosis in the cerebrum of a patient with HIV.
|
VZV encephalitis
|
|
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What is the route of tranmission of cytomegalovirus?[5]
|
Respiratory droplet
Blood Urine Feces Through mucous membranes |
|
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What herpes virus causes CM-5
|
HHV-5
|
|
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T/F CMV infection can cause CNS disturbances in the immunocompetent
|
False
|
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What are teh complications of in utero infection of an infection with CMV?[2]
|
Hearing loss
Brain damage |
|
|
What are teh high risk population for complications concerning CMV?[2]
|
HIV+/AIDS
Organ transplant recipients on immunosuppresive medication |
|
|
What are the symptoms of CMV infection of the CNS?[3]
|
Mild flu-like symptoms
Lethargy Fever |
|
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Where is CMV shed?[3]
|
Blood
Sputum Urine |
|
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When observing the neurons of a patient with CMV encephalitis what is seen?
|
Intranuclear inclusion bodies
|
|
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What type of inclusion bodies are seen in CMV and what are they known as?
|
Intranuclear inclusion bodies
Owl's eye inclusion body |
|
|
Describe CMV[3]
|
ds DNA
Enveloped Icosadelthedral |
|
|
Describe VZV[3]
|
ds DNA
Enveloped Icosadeltahedral |
|
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Where is CNS CMV infection generally seen?
|
Periventricular parenchyma
|
|
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What is the treatment for CMV infection?
|
Gancyclovir
|
|
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What percentage of adults between 35 and 40 are infected with EBV?
|
95%
|
|
|
What herpes virus is EBV?
|
HHV-4
|
|
|
Describe EBV[3]
|
ds DNA
Enveloped Icosadeltaherdal |
|
|
What common disease is seen with adolescent infection with EBV?
|
Mononucleosis
|
|
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What virus causes mononucleosis
|
EBV (HHV-4)
|
|
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What neurological disease can be caused by EBV?[2]
|
Aseptic meningitis
Encephalitis |
|
|
How common is CNS infection with EBV?
|
7% of hospitalized patients with EBV may develope neurological complications
|
|
|
What is the most common symptom for neurological involvement of EBV?
|
Headache(only symptom in 50% of cases)
|
|
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What virus causes roseola in children?
|
HHV-6
|
|
|
Exanthema subitum
|
Roseola
|
|
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What cells are infected be HHV-6?[2]
|
T-cells
NK cells |
|
|
What patients are susceptible to HHV-6 infection of the CNS?
|
Bone marrow transplant patients
|
|
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What diseases has HHV-6 infection been associated with?[2]
|
MS
Chronic fatiguesyndrome |
|
|
What virus is commonly found in simian species who present with no symptoms?
|
Monkey Pox Simian B virus
|
|
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What percentage of patients infected with Monkey pox simian B virus will die if untreated?
|
80%
|
|
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What sequelae is seen with survivors of monkey pox simian B virus?
|
Severe neurological deficits
|
|
|
How long until death occurs with infection of monkey pox simian B virus?
|
10 days
|
|
|
Who is at greatest risk for infection of monkey pox simian B virus?[2]
|
Veterinarians
Lab workers with contact to monkey and monkey tissue |
|
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Veterinarians and Lab workers in contact with monkeys and monkey tissue are at greater rick for contracting what virus?
|
Monkey Pox Simian B Virus
|
|
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What treatment might be useful for infection with monkey pox simian B virus?
|
Acyclovir
|
|
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Mumps virus is part of what viral family?
|
Paramyxovirdae
|
|
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What CNS conditions are seen with infection with the mumps virus?[2]
|
Meningitis
Encephalitis |
|
|
What season does mumps infection generally occur?[2]
|
Winter
Spring |
|
|
What percentage of patients infected with mumps virus develop CNS symptoms?
|
10% to 30%
|
|
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What percentage of patients who develop CNS manifestations of mumps infection do not develop parotitis?
|
40% to 50%
|
|
|
What are the typical symptoms of mumps?[3]
|
Fever
Swollen Parotid Gland Malaise |
|
|
What is the course of measles encephalitis?
|
Self limiting
|
|
|
What is SSPE?
|
Subacute sclerosing panencephalitis
|
|
|
What is the course of SSPE?
|
Slow developing syndrome with progressive neurological degeneration which is ultimately fatal
|
|
|
Slow developing syndrome with progressive neurological degeneration which is ultimately fatal. Happen about 10 years post measles infection.
|
Subacute Sclerosing Panencephalitis (SSPE)
|
|
|
What viral family does measles belong to?
|
Paramyxoviridae
|
|
|
What is the length of survival for the fulminant course of SSPE?
|
1 to 3 years
|
|
|
What is the length of the survival of the chronic course of SSPE?
|
4 to 10 years
|
|
|
What viral family does rubella belong to?
|
Togaviridae
|
|
|
What is PRP?
|
Progressive Rubella Panencephalitis
|
|
|
What demographic is generally affected by rubella virus?
|
Males ages 8 to 19
|
|
|
What are the symptoms of PRP?[7]
|
Behavior problems
Ataxia Dementia Spastic quadraparesis Koplik's spots Lymphadenopathy Maculopapular rash |
|
|
Small red spots on the mucous membrane of the cheek, with a minute bluish-white speck in the center, regarded as symptomatic of measles
|
Koplik's spots
|
|
|
Koplik's spots
|
Small red spots on the mucous membrane of the cheek, with a minute bluish-white speck in the center, regarded as symptomatic of measles
|
|
|
Describe paramyxoviruses[3]
|
(-)ssRNA
Enveloped Helical capsid |
|
|
Describe Rhabdoviruses[3]
|
(-)ssRNA
Enveloped Helical capsid |
|
|
What family does the rabies virus belong to?
|
Rhadoviridae
|
|
|
How is rabies virus transmitted?[4]
|
Scratches
Abrasions Open wounds Contaminated saliva |
|
|
What is the incubation period of rabies?
|
60 to 365 days
|
|
|
How does the rabies virus enter the cell?
|
Absorption
|
|
|
What are the symptoms of the early infection with rabies virus?[3]
|
Malaise
Fever Headache |
|
|
What are the symptoms of the prosgressive infection with rabies virus?[6]
|
Insomnia
Confusion Slight or partial paralysis Agitation Hypersalivation Dysphagia |
|
|
What is the coarse of rabies infection?
|
Death
|
|
|
How long until Death occurs after onset of symptoms of a rabies infection?
|
Within days
|
|
|
What is the tissue tropism for the rabies virus?[2]
|
Neurons
Salivary glands |
|
|
How can rabies be diagnosed from saliva?
|
RT-PCR
|
|
|
How can rabies be diagnosed by serum or CSF?
|
Detection of rabies virus antibodies
|
|
|
What is the name of the inclusion bodies seen in rabies infected neurons?
|
Negri bodies
|
|
|
What histopathological findings will be seen in the brain tissue of an animal infected with rabies virus?[3]
|
Negri bodies
Lymphocytic foci Mononuclear infiltration |
|
|
What is the treatment for a rabies infection?
|
Post exposure prophylaxis
|
|
|
What is the post exposure prophylaxis for rabies infection?
|
1 dose Immunoglobulin
5 doses rabies vaccine -for 28 days |
|
|
What is the time period in which post exposure prophylaxis should be administered?
|
Over 28 days
|
|
|
What microbes are responsible for recurrent benign lymphocytic meningitis?[2]
|
HSV-2(95%)
HSV-1(5%) |
|
|
What is the course of recurrent benign lymphocytic meningitis?
|
Recurrent meningitis with spontaneous recovery
|
|
|
What percentage of patients have transient neurological symptoms after recurrent benign lymphocytic meningitis?
|
50%
|
|
|
What is the sequelae of recurrent benign lymphocytic meningitis?
|
Transient neurological symptoms
|
|
|
What virus is bullet shaped?
|
Rabies virus
|
|
|
What family does the "bullet" shaped virus belong to?
|
Rhabdoviridae
|
|
|
What acute CNS condition can HIV cause?
|
Meningitis
|
|
|
What condition can be caused by HIV in AIDS?
|
Subacute encephalitis
|
|
|
What are the morphological features of the brain in subacute encephalitis caused by HIV in AIDS?[2]
|
Brain shrinks
Enlarged ventricles |
|
|
What condition is difficult to differentiate from HIV-related dementia?
|
Multifocal leukoencephalopathy caused be the JC virus
|
|
|
What condition is difficult to differentiate from multifocal leukoencephalopathy caused by JC virus?
|
HIV-related dementia
|
|
|
What are the symptoms of subacute encephalitis caused by HIV in AIDS?
|
Pre-senile dementia
|
|
|
What is PML?
|
Progressive multifocal leukoencephalopathy
|
|
|
What microbial agent causes PML?
|
JC virus
|
|
|
What viral family does JC virus belong to?
|
Polyomaviridae
|
|
|
Describe Polyoma viruses[3]
|
ds circular DNA
Nonenveloped Icoshedral capsid |
|
|
What are the symptoms of PML?[7]
|
Hemiparesis
Visual loss Aphasia Seizures Dementia Personality changes Gait problems |
|
|
What is the course of PML?
|
Death
|
|
|
What is the characteristic finding of neuroimaging in a patient with PML?
|
White matter lesion in the area of the occipital region
|
|
|
How long is the survival period of patients with PML?
|
Weeks to months
|
|
|
What are the most common agents of community acquired meningitis?[3]
|
H. influenzae
S. pneumoniae N. meningiditis |
|
|
In what type of patients does PML affect?
|
Immunosuppressed
|
|
|
What is the mechanism of action of the JC virus in PML?
|
Demyelination
|
|
|
What are the Artropod-borne viral encephalitides?[7]
|
Eastern Equine Encephalitis
Western Equine Encephalitis Venezuelan Equine Encephalitis St.Louis Virus Japanese Encephalitis Virus West Nile Virus La Crosse Virus |
|
|
What are the arthropods that usually transmit viruses that affect the CNS?[2]
|
Ticks
Mosquitoes |
|
|
What family of viruses causes EEE
|
Togaviridae
|
|
|
What is the vector for transmission of the Togaviridae?
|
Culiseta mosquito
Aedes mosquito |
|
|
What is the geographical area of EEE?
|
North America
|
|
|
What is the mortality rate for EEE?
|
High mortality rate
|
|
|
Is the CNS involvement EEE infection focal or diffuse?
|
Diffuse CNS involvement
|
|
|
What are the symptoms of prodrome EEE?[6]
|
Fever
Chills Weakness Headache Myalgia Nausea/vomiting |
|
|
What are the symptoms of rapid progression of EEE?[7]
|
Headache
Nausea/vomiting Confusion Somnolence Siezures(50%) Coma |
|
|
A state of drowsiness; sleepiness
|
Somnolence
|
|
|
Somnolence
|
A state of drowsiness; sleepiness
|
|
|
What causes the prodromal symptoms of EEE?
|
Viral replication in non-neural tissue
|
|
|
What causes the rpid progression symptoms in EEE?
|
Spread through the microvascular permeability of the brain
|
|
|
How does cell to cell infection of togaviruses in EEE occur?
|
Through the cell to cell interactions of dendrites and axons
|
|
|
What viral family causes WEE?
|
Alphaviridae
|
|
|
What is the vector for the alphviridae?
|
Culex mosquito
|
|
|
What is the geographic area of WEE?
|
Rural US
|
|
|
What is the seasonality of WEE?
|
Summer
|
|
|
What is the fatality rate of WEE?
|
3-4%
|
|
|
What is the rate of CNS sequlae?
|
30%
|
|
|
What are the prodromal symptoms of WEE?[4]
|
Fever
Headache Chills Nausea/vomiting |
|
|
what is the duration of prodromal symptoms in WEE?
|
1 to 4 days
|
|
|
What are the rapidly diffuse symptoms of WEE?[3]
|
Confusion
Somnolence Coma |
|
|
How is CNS damage mediated in WEE?
|
Immunologically active cells that enter the brain
|
|
|
What are the agents of CNS destruction in WEE?[2]
|
Neutrophils
Macropahges |
|
|
What is the type of cell death that occurs in the CNS with WEE
|
Apoptosis
|
|
|
What cells die by apoptosis in WEE?[2]
|
Glial cells
Inflammatory cells |
|
|
What virus causes VEE?
|
Togavirdae alphavirus
|
|
|
What is the importance of Togaviridae alphvirus?
|
Potential as a biological weapon
|
|
|
What is the vector for Togaviridae alphavirus?[2]
|
Culex mosquito
Aedes mosquito |
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What family if St Louis Virus a part of?
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Flaviviridae
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What is the vector of St. Louis virus?
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Culex mosquito
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What are the factors that determine CNS infection by St. Louis Virus?[3]
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Efficacy of viral replication at extraneural sites
Degree of viremia Age of host |
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How dies the St. Louis virus enter the brain parenchyma?[2]
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Through cerebral capillary endothelial cell
Across fenestrated endothelium |
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T/F St. Louis virus can cross the BBB.
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True
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What is the mortality rate for St. Louis virus infection?
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2% to 20%
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T/F St. Louis virus is more dangerous in the young.
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False (St. Louis virus affects elderly more)
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What are the prodromal symptoms for St. Louis virus?[2]
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Malaise
Fever |
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What percentage of people with St. Louis virus develop sequelae?
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20%
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What is the sequelae for St. Louis infection?[6]
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Headache
Nausea/vomiting Irritability Memory loss Movement disorders Motor deficits |
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T/F Seizures and coma are common in patients with St. Louis virus
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True
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T/F There is a chronic course with St. Louis Virus infection.
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False (there is no chronic infection)
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What is a chronic development of St. Louis Virus infection?
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Deep cranial nerve palsy
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What viral family does JEV belong to?
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Flaviviridae
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What is an Arbovirus?
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An arthropod borne virus
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Describe Flaviviruses.[3]
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(+)ssRNA
Enveloped No visible capsid |
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What is the vector for JEV?
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Culex mosquito
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What is the geographic region of JEV?
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Rural Asia
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What is the incubation period for JEV?
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4 to 14 days
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What are the prodromal symptoms of JEV?
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Fever by 2nd week
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What are the symptoms of JEV encephalitis?
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Tremors
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T/F There are seizures associated with JEV?
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False
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What sign in JEV is a risk for death?
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Low IgG/IgM ratio
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What percentage of patients relapse from recovery of JEV in 1 year?
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50%
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What viral family does Wet Nile Virus belong to?
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Flavivirdae
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What is the reservoir for West Nile Virus?
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Wild birds
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What is the vector of West Nile Virus?
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Mosquitos
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T/F Person to person transmission of West Nile Virus is not possible.
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False (Person to person transmission is RARE but there have been a few confirmed cases)
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What is the physician duty in the case of West Nile Virus?
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Physician must report the disease
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What percentage of West Nile virus Cases are fatal?
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3% to 15 %
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What are the prodromal symptoms of West Nile Virus?
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Maculopapular rash of trunk and extremities
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What are the symptoms of severe infection with West Nile Virus?[8]
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Headache
HIGH fever Nuchal rigidity Stupor Loss of consciousness Tremors Seizures Paralysis |
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What are the CSF findings in a patient with West Nile Virus?[3]
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Negative bacterial stain
Pleocytosis(WBC between 5 and 1500cells) Elevated protein(>40mg) |
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What family does La Crosse Virus belong to?
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Bunyaviridae
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Describe Bunyaviridae[3]
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3 (+)ssRNA segments (L,M,S)
Enveloped Nucleocapsid |
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What is the geographic location of La Cross Virus infection
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Forrested areas in Upper Midwest
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What are the symptoms of severe infection with La Crosse infection?[3]
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Seizures
Coma Paralysis |
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What is the usual course of La Crosse Virus infection?
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Spontaneous resolution
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What is the fatality rate with La Crosse Virus infection?
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<1%
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What is EEE?
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Eastern Equine Encephalitis
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What is WEE?
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Western Equine Encephalitis
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What is JEV?
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Japanese Encephalitis Virus
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What is SLEV?
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St. Louis Virus
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What is VEE?
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Venezuelan Equine Encephalitis
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T/F WEE presents with a Rash
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False
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