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40 Cards in this Set

  • Front
  • Back

SI joint movements during


-standing


-sitting


-supine


-SL standing


-trunk extension


-trunk flexion


-Respiratory motion


- nutation


- nutation


- counternutation


- almost maximal nutated position on WB leg


- nutation


- counternutation


- inhale=counternutation; exhale=nutation

Describe Force vs. Form closure of the SI joint

Force: muscular contraction at the joint to hold the joint in place


-ex: Glenohumeral joint; rotator cuff muscles



Form: Shape of the joint and bony structure give support


-ex: hip joint; deep ball and socket



*SI joint relies on both form and force closure

Describe common characteristics of SI joint pain

- SIJ pain will never occur above the PSIS


- SIJ pain most commonly presents at the PSIS, inner buttock pain, buttock pain, buttock and posterior thigh pain


- Typical upper, inner buttock (S1,S2 dermatomes)


- SIJ pain never produces LBP


- SIJ pain is never midline; always


caudal(below) to the PSIS or at the PSIS


*should never have findings of parasthesia, weakness, or numbness



Describe some some characteristics of SIJ affliction or instability

- more common in women vs. men (males more likely to have SIJ pain due to macrotrauma)


- 18-35 years old = increased incidence of instability


* 35 and over = incidence decrease in males


- Birth control increases risk for SIJ instability


- Commonly seen during pregnancy due to hormonal changes and relaxation of the ligaments


- chronic injury can be unilateral or bilateral; acute is typical unilateral

What is the capsular pattern for a hip injury differentiated from a LB injury

- Equal loss in IR, ABD, and Flex > loss in Ext. and ER > loss in ADD of the hip

How do you differentiate Hip pathology from a Lumbar pathology

- Patient has a limp (not commonly seen with LB injuries)


- Groin pain


- Limited hip med. rotation


- follow capsular patterns of the hip


- Positive sign of buttock


- Major lesion = infection, tumor, fx


- Minor lesion = bursitis, tendonopathy, arthritis


- Restricted hip movement (may be related to LBP)

What are the 2 types of hip impingement and how do they differ?

Cam impingement: "pistol grip deformity" bony deformity or abnormality that changes how the neck of the femur is shaped


- neck of the femur is flatter in angle and


therefore contacts with the acetabulum


w/ hip flex. and IR


Pincer Impingement: Acetabulum contacts the head/neck at end ROM, causing the femoral head to jam into the acetabulum w/ hip flex. and IR



*most common cause of OA in young men

What are some commonalities with Hip Labral Tears?

- 86% occur anterior*


- almost 1/2 of labral tears are accompanied by a chondral defect


- Rare to occur w/out a bony abnormality


- most w/ chondral defect


- Athletic hip injury (acute) w/ C/O intra-articular pain usually means a labral tear*

What are some common S&S for a hip labral tear?

- buckling, catching, clicking, and loss of ROM


- pain in the groin exacerbated by activity*, standing up, or going down stairs


- deep buttock pain



*clinically, more often diagnosis are hip pointers groin strain, deep muscle strain, or contusion


** if pain persists or worsens re-evaluate

Describe the Hip Flexion Mechanical Deficits (W,SD,C,AP)

W:


Iliopsoas weakness


SD:


TFL


Rectus femoris


ADD longus (become the hip flexors)


C:


Anterior glide of the femur


lumbopelvic instability (anter. tilt)


AP:


Anter Lab. tear


Rectus femoris and ADD strains


IT band issues


L-spine pathology

Describe the Hip Extension Mechanical Deficits (W,SD,C,AP)

W:


Glute max


quads


SD:


Hamstrings


ADD overuse (magnus and longus)


C:


Anterior glide of the femur


lumbopelvic instability (anter./ex tilt)


Iliopsoas shortness/tightness


AP:


Hamstring strain


labral tear

Describe the Femoral ADD/IR Mechanical Deficits (W,SD,C,AP)

W:


Glute med


SD:


TFL


C:


TFL/IT band stiff/short


Deep ER weakness


ADD stiff/short


medial hamstring stiff/short


QL stiff/short


Femoral retroversion


compensatory Lumbar rotation


AP:


IT band syndrome

Describe the Femoral ER Mechanical Deficits (W,SD,C,AP)

W:


Glute med.


SD:


biceps femoris


C:


Anterior femoral glide


anteversion


compensatory lumbar rotation


AP:


kyphosis


L-spine hyperextension


T-spine to dysfxn side


Lateral shear of the pelvis leading to


hyperpronation

Describe a sacral torsion, and the different types of possible torsions

- a rotation about an oblique axis along with dysfunction of L5 on S1



Forward (pain with backward walking)


- L on L: left rotation on the left oblique axis


- R on R: right rotation on the right oblique axis



Backward (pain with forward walking, overall more discomfort)


- L on R: left rotation on the right axis


- R on L: right rotation on the left axis

Describe the static and dynamic positions of a backward R on L sacral torsion

Static


- R superior sulcus is shallow


- L ILA deeper


- Lumbar curve is convex to R


Dynamic


- Seated flexion test on R (+)


- Positive lumbosacral spring test


- Positive backward bending test


- L5 is flexed or extended, sidebent L and rotated L (or SB and rotated toward concavity)


Describe the static and dynamic positions of a backward L on R sacral torsion

Static


- L superior sulcus is shallow


- R ILA deeper


- Lumbar curve is convex to L


Dynamic


- Seated flexion test on L (+)


- Positive lumbosacral spring test


- Positive backward bending test


- L5 is flexed or extended, sidebent R and rotated R (or SB and rotated toward concavity)


Describe the static and dynamic positions of a bilateral sacral flexion

Static


- R and L superior deep sulci


- Increased lumbar curve


- R and L ILAs shallow


Dynamic


- Negative lumbosacral spring test


- Restricted springing on ILAs B


- False negative on seated flexion and standing flexion



*Both SIs restricted

Describe the static and dynamic positions of a bilateral sacral extension

Static


- R and L superior shallow sulci


- decreased lumbar curve


- R and L ILAs are deep


Dynamic


- Positive lumbosacral spring test


- Restricted springing on superior sulci B


- False negative on seated flexion and standing flexion


*Both SIs restricted


Describe the static and dynamic positions for a unilateral sacral flexion on the R

* sacral base is shifted anterior on the R


Static:


- R superior sulcus is deep


- R ILA is inferior (slight) and shallow


Dynamic:


- Seated flexion (+) on the R


- restricted spring test on R ILA



** flipped for sacral flexion on the L

Describe the static and dynamic positions for a unilateral sacral extension on the R

*sacral base is shifted posterior on the R


Static:


- L superior sulcus is deep


- L ILA superior (slight) and shallow


Dynamic:


- Seated flexion (+) on the R


- restricted spring test on R superior pole


- (+) lumbosacral spring test


- (+) backward bending test

Diagnosis for SIJ should be based upon...

- (+) history


- BFE for lumbar spine is negative


- (+) pain provocation tests

Possible causes of SIJ pain

- locking


- instability

Describe the kinematics of the PFJ in the frontal plane

- slight lateral displacement is normal




- slight medial displacement from 45 to 15 deg. knee flexion, followed by slight lateral displacement at end range (15-0 deg.)




- Med to Lat shift is explained by the screw home mechanism




- patellar motion is highly related to tibial rotation

Describe kinematics of the PFJ in the transverse plane

- knee ext of 45 - 0 deg. patella tilts medial 5-7 deg. instead of 10 deg. due to groove structure




*lateral placement or lateral tilt = pathology is result of degree rather than motion

Describe kinematics of the PFJ in the saggital plane

- flexion: inf pole moves posterior




- extension: inf pole moves anterior




*patellar flexion is 20 deg less than knee flexion; Same with knee ext.

Describe the reaction force mechanics of the PFJ

- 1x body weight while walking


- 3.8 x while walking up/down stairs


- 11 x while running

How does contact area effect the kinematics of the PFJ

-3x greater during flexion even at 60 deg


- greater contact area distributes force for less stress to the PFJ


- less contact equals higher stress during malalignment



characteristics of excessive lateral pressure syndrome (ELPS)

describe the 3 patellar shapes and designate the most common

- type 1: both facets are concave and symmetrical; med. and lat. are equal in size




- type 2: med. facet is smaller than lat.; lat. is flat and med. is concave *




- type 3: med. facet is slightly convex and smaller; marked by lat. facet predominance




*most common type

what are factors that increase Q angle for the PFJ

- excessive femoral anteroversion




- lateral tibial torsion




- lateral displacement of the tibial tuberosity




- genu valgum

describe the possible abnormal kinematics at the tibia and femur

Tibial rotation


- lateral rotation = > Q angle


- medial rotation = < Q angle




Femoral rotation


- > medial rotation > Q Angle, affects patellar alignment


- > lateral rotation < Q angle (alignment with the ASIS)


- knee valgus

What are some differential Dx for PFP and how do they present?

- Patellar tendonitis/tendonosis: pain will be sup. and inf. with tendon not retropatellar or med/lat. with PFP




- ITB: pain on the lateral epicondyle and localized




-meniscal/ligamentous




-referred pain: L3-L4 nerve roots

who qualifies for surgery to the PFJ

- persistant patellar tilt


- pathological plica


- infrapatellar plica


- severe post-traumatic chondrosis/arthrosis


- postoperative neuroma


- scar pain


- recurrent dislocation

What are the preferred diagnostic testing for the PFJ

- x-ray: while w/b


- CT: midpatellar transverse through the post condyles w/ flex of 0,15,30, and 45 deg


- KMRI is more effective than MRI or CT


- bone scan: recognizes intraosseous dysfxn

4 main reasons for lower leg overuse injuries

-environmental factors: training surface; uneven soft, hard, same side




- training parameters: intensity, frequency, duration




- footwear




- mechanics

describe the complications of an ankle sprain

- limited dorsiflex: need 10 deg to walk; 20-30 to run


- failure to train eccentric response


- failure to complete rehabilitation


- syndesmotic sprain fails to heal: at least 7 days immobilization ot heal properly


* min of 4 weeks for any sprain


*completed rehab reduces reinjuy by 50%



characteristics of the plantar fasciitis

- Fxn: stabilize and lock the foot into supination prior to toe-off


- strained by the "windlass mechanism": hyperext. of the MTP and hyperpron. of the foot


- found in rigid (pes cavus) and hypermobile feet


- caused also by biomechanical abnormalities



characteristics of a midfoot sprain

-Lisfranc injury


-needs stabilization of the foot


-intrinsic mm strengthening


-emphasize normal and correct gait


*can be either: hypermobile that needs to stabilize; hypomobile that is forced out of place into injury

acute injuries to the lower leg to be aware of

- DVT (usually due to travel or prolonged sitting)


- Fracture


- Strain


- Hematoma


- Sprain


- Acute Compartment Synd.



chronic injuries to the lower leg to be aware of

- periostium injury: MTSS; stess fracture


- Vascular: Popliteal A. entrapment vs. intermittent claudication


- referred pain: N. entrapment (peripheral or spinal); knee or hip abnormality


- Muscular/Tendon: exertional comp. syndrome; strain; tendinopathy