• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/110

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

110 Cards in this Set

  • Front
  • Back
Neurological changes, optic nerve
myelination is not complete at birth
Neurological changes, LGN
layers are visible, but not complete at birth.
Parvo pathways develops at
1 year
Magno pathway develops at
2 years
tracking and neurological development
good tracking early showed good neurological development. poor tracking early was a indicator of neurological development problems
visual cliff
babies 6-9 months old had developed depth perception
PEVP
pattern evoked visual potential, electrodes on the occipital lobe. Amplitude was adult like by the 6th month
OKN
optokinetic nygstagmus. directionally appropriate for newborns, better in nasal/downward direction
FLP.
birth
1 month
6 months
1 year
3-5 years
forced choice preferential looking. measured in octaves.
birth = 20/600 to 20/1000
1 month = 20/400 to 20/600
6 months = 20/200
1 year = 20/50
3-5 years = 20/20
Why the difference between vep versus fpl and okn?
FPL and OKN require motor responses and good behavior, while VEP doesn't
Color vision at birth
prefers red, least sensitive on the tritan axis.
Rod dominance theory
rods better developed at birth, this dominance causes a decreased response to color since little to no cones.
Uniform loss theory
cones are just not well developed at the rods, which makes them less sensitive.
color vision development at 1 month, 2 months and 3 months.
1 month, scotopic is much more functional.
2 months, dichromat
3 months, trichromat
contrast sensitivity shifts
curve shifts vertically and horizontally in early years. will shift up and to the right as we age, finally down to the left in elderly.
CFF
critical flicker frequency, measured temporal resolution, poor at birth but actually very similar to adults. Adult like at 4 years of age.
Accommodation
18.5 D at birth, amplitude reduces 1 d every 3 years. reasonably accurate at 75 cm.
Verneir acuity
infants verneir acuity worse than grating acuity before 11-12 weeks.
stereopsis
eyes should be aligned by 6 months. stereo emerges at 3.5-6 months, and adult like at 24 months.
Interactionist
Nature versus nurture, interactionist believes both play a role
zygote
first 2 weeks after conception, fertilization to implantation of embryo, not susceptible to outside influences
embryo
2-8 weeks after conception, development of major organ systems, especially vision in the 4-5 week.
fetus
8 weeks after conception to birth
apgar score
take 1 and 5 minutes after birth, scores are 0-2.
A = appearance
P = pulse
G = grimance
A = activity level
R = respiratory effort
reflexes present at birth
1. grasping
2. rooting
3. sucking
4. motor/startle
5. babinski
6. stepping
cepahlocaudal
growth from heal to tail
proximodistance
development from the inside out
sensitive period of attachment
attachment to primary caregiver within the 1st year
separation anxiey
end of the first year, through second year, secure, insecure - ambivalent and insecure - avoidant
Piaget's stages
1. sensorimotor 0-2
2. pre-operational 2-7
3. concrete operations 7-11
4. formal operations 11 and up
Sensorimotor 0-2
child is an active scientist, object permanence is established at the end of this period.
Pre-operational 2-7
will make logic errors, conversation develops during this time period.
Concrete operations 7-11
child uses logic, can't yet deal with abstract subjects
Formal Operations 11 and up
child can now deal with abstract thought.
Schema
mental represenation of an idea or concept
adapatation
adapt to a new environment or stimulus
assimilation
force new stimulus into existing schema
accomodation
create new schema for new stimulus
LGN structure
6 major layers
4 dorsal layers - parvo layers
2 ventral layers - magno layers
Monocular cortical neurons
OD monocular neuron fires only when OD is stimulated
Binocular cortical neurons
responds when either eye is stimulated or when both stimulated (facilitation)
layer 4c only has
monocular cells, so only has cells belonging to 1 and 7. It is the first point of contact for information to the lgn
each vertical column contains cells
having the same degree/extend of binocularity. Binocularity of successive columns change gradually. There is a shift gradually from 1 column to another, spanning 7 columns.
each vertical column has cells of similar
orientation specificity.
hypercolumn
small chunk of cortex containing columns of cells spanning the entire ocular dominance column distribution and slabs of orientation distribution.
competition
right and left eye lgn fibers compete to innervate as many cortical cells as possible
inhibition
suppressive force actively prevents 1 eye's fibers from innervating
Monocular deprivation
constant unilateral strab.
affected eye has:
reduced luminance sensitivity
reduced contrast sensitivity
lower CFF

cortical cells are abnormal, primary site affected by MD is visual cortex. Layers recieving input from deprived eye are shrunken. ocular dominance stripes occupied by MD eye are narrower. Parvo layers are more effected.
Alternating monocular deprivation
most cortical cells belong to 1 or 7. mainly monocular cells, very few binocular cells.
for normal binocular vision to develop it's not good enough to just have the 2 eyes open
the 2 eyes must be given simultaneous, not alternating, visual experiences
4 types of deprivation
1. MD 1 bump at 1 or 7
2. Alternating deprivation 2 bumps at 1 and 7.
3. Binocular deprivation
4. Pattern deprivation
ageism
any situation in which individuals are discriminated against based on age
85+
fastest growing age group in the US, 1 in 5 americans will be over 65 by 2030.
biological theories of aging
2 categories:
1. genetically controlled aging
2. non genetic cellular aging
a. stress theory
b. deprivation theory
c. free radical theory
genetically controlled aging
controlled by our genetic blueprint DNA. DNA code determines sequence of physiological growth and decline.
Hayflick limit
number of times a cell is able to replicate before ceasing to function is limited
non genetic cellular aging
views aging as cumulative effect or errors occuring in sequence of transfer of information at a cellular level. Increase in aging results in errors in transferring in genetic code, thus deterioration of code.
wear and tear theory
human cells wear out over time
stress theory
accumulation of stressful life events over lifetime impacts on body's ability to adapt.
deprivation theory
inadequacy of essential nutrients and oxygen to cells linked to arteriosclerosis
free radical theory
unstable free radicals combine with other cellular molecules, causing damage - impacts on the body's ability to repair itself.
process of aging Senescence
begins at age 30
secondary aging
factors that influence the natural process of primary aging, treatable diseases, disorders, or social problems.
taste peaks between
30-60
loss of brain cells after age of 30
between 20,000 and 100,000 per day.
secondary effects
1. increased dependance on others.
2. challenges in ability to accept assistance
3. willingness and availability of family to help
4. financial constraints.
integrity
comes from the ability to emotionally accept one's life and its limitations with awareness of its shortness and finality
despair
occurs when one believes life is too short, no further chance of feeling a sense of fulfillment.
peck's theory
continued psychological growth depends on the outcome of 3 major developmental tasks:
1. occupational retirement: finding personal satisfaction beyond work.
2. physical decline: degree to which one's happiness and well being is dependant on physical dimensions
3. Human mortality: the way in which one faces the realization of impending death
cattell-horn theory of fluid and crystallized intelligence
general intelligence conglomeration of up to 100 abilities working together in various ways in different individuals resulting in different levels of intelligence.
fluid intelligence
abilities enabling one to perceive relationships, draw inferences, interpret abstract information and comprehend implications.
Dementia
general term for collection of chronic multiple acquired neuro cognitive deficits.
Dementia most common causes
1. alzheimer's disease
2. multi infarct vascular disease
3. depression
4. frontal lobe disease
alzheimer's disease
rare before 50. most common form od dementia. progressive and irreversible deterioration of brain tissue primarily in the cerebral cortex.
4 phases of alzheimer's
1. forgetting names, misplacing objects.
2. short term memory loss, disorientation
3. partial dependency on others for basic needs
4. total dependence.
Depression
most common treatable psychiatric disorder among adults.
Marriage
less likely to experience loneliness, lower incidence of mental illness, increased longevity, lower vulnerability to outside stressors.
Widowhood
greatest emotional and social loss in the normal course of the lifespan. loss of spouse impacts men more significantly than women.
Institutionalization
only 5% of older adults live in institutions. 33% die within a year of entering facility. 85-90% die in the facility.
critical period
interval during which it is possible to disrupt or re-establish function that were lost during deprivation period
critical period for direction and ocular dominance
4-7 weeks
development periods in humans
1. prestereoptic, 0-4 months
2. onset of stereopsis 4-6 months
3. post stereoptic 6 months - 2 years
critical period in regards to magno versus parvo
critical period ends earlier for magno than parvo
GAP-43
growth associated protein, helps axons find their way to target neurons.
NMDA receptors
activation of NMDA receptors leads to activation of 2nd messengers, enzymes and protein syn. without NMDA receptors free plastic changes are reduced.
Total number of NMDA receptor sites is at their highest during
the peak of the critical period for ocular dominance.
Hebb's synapse hypothesis
if pre and post synapse fire together the synapse is strengthened.
Activation of NMDA receptors has more
specific effect on plasticity
norepi modulates the state of
depolarization in the brain, this is less specific.
Spherical refractive error, premature
correlation between birthweight and myopia
spherical refractive error, newborns
99% are between -6.00 and +10.00
Spherical refractive error, early childhood
disppearance of extremes during first 2 years of life
Spherical refractive error, school age
more hyperopia than myopia, +1.25- -0.25, after age 10 it skews towards myopia.
5-6 years
> or = +1.50 D
+0.50 to +1.25
plano to +0.49
any minus
13-14
hyperopic
emmetropic
myopic
more myopic
ATR/WTR/Oblique
ATR and WTR decreases, obliques will remain stable.
with aging there is a loss in the visual field
greater loss in the upper field
aging on stereo
marked decline in stereo after 60
tonic vergences changes with aging
increase with age
positive fusional vergence changes with aging
decrease
negative fusional vergence changes with aging
no change
with age exophoria or esophoria?
exophoria at near with age.
9 stages of language development
1. phonetic expression, babbling
2. phonetic contraction
3. one word sentences
4. two word sentences
5. telegraphic speech
6. denotation of the word, definition of word.
7. connotation of word, how word is used in specific culture.
8. semantics
9. syntax
Stage 1
0-2 trust versus mistrust
Stage 2
2-4 autonomy versus shame
Stage 3
4-7 initiative versus guilt
Stage 4
7-11 industrious versus inferiority
Stage 5
11-19 identity versus identity confusion
Stage 6
20-30s intimacy versus isolation
Stage 7
40-50s generativity versus stagnation
Stage 8
old age integrity versus dispair.