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527 Cards in this Set

  • Front
  • Back

Epimysium

Fibrous connective that covers all muscles
Tendon
Connective tissue that attaches muscle to bone
Bone Periosteum
Outer part of bone

Where muscle tendon attaches to the bone
Proximal

Closer to the Trunk

Distal
Farther from the Trunk
Superior
Closer to the head
Inferior
Closer to the feet
Origin
Proximal (towards the center) attachment of a muscle

Insertion

Distal (away from center) attachment of a muscle

Muscle Fibers

Muscle Cells

Muscle cells running the length of the entire muscle
- cylindrical cells
- multi nuclei

Fasciculi

Muscle fiber bundles or groups

Perimysium
Connective tissue surrounding fasciuli
Endomysium
Connective tissue surrounding each muscle fiber
Sarcolemma
Muscle fiber's cell membrane
Motor Neuron
Nerve Cell that innervates a muscle fiber
Neuromuscular Junction
Junction between motor neuron and muscle fiber
Motor Unit
A Motor Neuron and the Muscle Fibers it innervates
Sarcoplasm
The cytoplasm of a muscle fiber

Contains:
- Contractile Components (proteins, fats, enzymes, glycogen, mitochondria, SR)

Myofibrils

In sarcoplasm

Contain contraction apparatus
- Actin
- Myosin

Myofilaments
Myosin

Actin
Cross Bridges (Myosin)
Globular Heads of the myosin filaments the protrude to attach to Actin
Actin
The two strands with the double-helix shape
Sarcomere
Both myosin and actin filaments that are organized longitudinally

Smallest contractile unit in skeletal muscle
A-Band
The dark filament

Corresponds to the alignment of the Myosin Fillaments
I-Band
Corresponds with the areas in two adjacent sarcomeres

Contain only actin filaments
Z-Line
In the middle of I-Band

Appears as a thin, dark line

Runs Longitudinally
H-Zone
Area in the center of a sarcomere

Only myosin present
Sarcoplasmic Reticulum (SR)
Intricate tubules surrounding each myofibril

Terminates as vesicles in the vicinity of Z-lines

Stores Calcium + Ions (controls muscle contraction)
T-Tubules
Run perpendicular to the SR

Terminate in the vicinity of Z-lines between vesicles
Triad
The pattern of T-Tubules spaced between/perpendicular to 2 SR's
Action Potential
An Electrical nerve impulse
Action Potential w/ Muscle Contraction
Action Potential from MU
>
Releases Ca+ from SR to myofibril
>
Tension in Muscle
Sliding-Filament Theory
States that the actin filaments at each end of the sarcomere slide inward on myosin filaments, pulling the Z-lines toward the center of the sarcomere and thus shortening the muscle fiber
Troponin
A protein that is situated at regular intervals along the Actin filaments

Has high affinity for Ca+
Tropomyosin
Runs along the length of actin filament

In the grooves of the double-helix
Cross-Bridge/Force Pro Relationship
The number of Cross-Bridges that are attached to actin filaments dictate the Force Production of a muscle
Resting Phase (Muscle)
Little Ca+

Little cross-bridges
Excitation-Contraction Coupling Phase (Muscle)
Action Potential
>
Ca+ Release
>
Cross Bridging
>
Force Production
Contraction Phase (Muscle)
Ca+ and ATP are necessary for Myosin Cross-Bridging with Actin
Acetylcholine
Neurotransmitter

Diffuses across NM-Junction

Causes excitation of Sarcolemma
All-Or-Nothing Principle
Fibers all fire or they don't despite action potential strength/weakness
Twitch
The short period of activation of a muscle fiber within a MU after an AP reaches it
Tetanus
When twitches begin to merge and eventually completely fuse
Slow-Twitch
MU's develop force and relax slowly

Long twitch time
Fast-Twitch
MU's develop force and relaxes rapidly

Slow twitch time
Type I
Slow-Twitch

More efficient

Fatigue Resistant

High Capacity for Aerobic E+ supply

Limited potential for Rapid Force Development

Low ATPase activity and Low Anaerobic power
Type IIa/IIb
Fast-Twitch

Inefficient and fatigable

Low Aerobic Power

Rapid Force Development

High Actomyosin acitivites
Type IIx
Human muscle fibers
Motor Unit Recruitment
MU's are composed of Fibers with specific Morphological/physiological characteristics that determine their functional capacity
Recruitment
The number of MU's activated
Change in Force Production
Change in frequency of activation of individual motor units

Change in Number of Activated Motor Units
Preloading
Muscle fibers that are active early in the range of motion will not be fully activated unless the muscle is Loaded Prior to Muscle Action
Proprioceptors
Specialized sensory receptors

Located in:
- Joints
- Muscles
- Tendons

Sensitive to:
- Pressure
- Tension

Responsible for:
- Kinesthetic Sense
- Conscious Appreciation of Body Position
- Muscle tone
- Complex Coordinated Movements
Muscle Spindles
Proprioceptors in modified muscle fibers in the sheath of connective tissues

Provide Info On:
- Muscle Length
- Rate of Change in Length
Intrafusal Fibers
Modified Muscle Fiber with the Muscle Spindle attached to it

Run parallel to normal Fibers
Extrafusal Fibers
Normal Muscle Fibers
Golgi Tendon Organs (GTO)
Proprioceptors located in the Tendons of muscles

Attached end-to-end with Extrafusal muscle fibers

Sense tension in the Muscle
- If tension is in excess, the GTO kicks in and inhibits the muscle tension
Sarcopenia
Reduced muscle Size and Strength

Result of aging or inactivity
Heart
muscular organ comprised of two interconnected but separate pumps; the right side of the heart pumps blood through the lungs, and the left side pumps blood through the rest of the body.
Atrium
Left and Right

Deliver blood into the Right/Left Ventricles
Ventricles
Left and Right

Supply the main force of blood through:
- Pulmonary
- Peripheral
Circulations
Chambers of the Heart
Atria

Ventricles
Prevents Flow of Blood from the Ventricles back into the Atria During Systole
Tricuspid Valve

Mitral Valve (bicuspid)

Atrioventricular Valves (AV)
Systole
Ventricular Contraction
Prevents Backflow from the Aorta and Pulmonary Arteries into the Ventricles during Diastole
Aortic Valve

Pulmonary Valve (Semilunar Valves)
Diastole
Ventricular Relaxation
Sinoartrial Node (SA)
The intrinsic pacemaker-where rhythmic electrical impulses are initiated
Atrioventricular Node (AV)
Where impulses is delayed slightly before passing into ventricles
Atroventricular Bundle (AV)
Conducts the impulse to the Ventricles and L/R Bundle Branches
Purkinje Fibers
Conducts Impulses to all parts of the Ventricles
Myocardium
Heart Muscle
Parasympathetic

Sympathetic

Nervous Systems
Components of the Autonomic Nervous Systems

Takes control of rhythm from Medulla of the brain and transmits to heart
Sympathetic Nervous System (Heart)
Accelerates Depolarization

Causes the heart to beat faster
Parasympathetic Nervous System (Heart)
Slow SA node discharge

Causes the heart to beat slower
Bradycardia
Fewer than 60 bpm
Tachycardia
>100 bpm
Electrocardiogram (ECG)
Graphic Representation of electric activity of the heart recorded at the surface of the body
P-Wave
Generated by the Changes in Electrical Potential of Cardiac Muscle Cells that Depol that Atria and result in Atrial Contraction
Depolarization
The Reversal of the Membrane Electrical Potential
QRS Complex (ECG)
Generated by the Electrical Potential that Depols the Ventricles and results in Ventricular Contraction
T-Wave (ECG)
Caused by the Electrical Potential Generated as the Ventricles recover from the state of Depol
Repolarization
Occurs in the Ventricular muscle shortly after Depol
Arterial System
Carries blood away from the Heart
Venous System
Returns Blood toward the Heart
Arteries
Rapidly transport blood pumped from the Heart

Have strong muscular walls, to withstand high pressure of blood from heart
Arterioles
Small Branches of Arteries

Act as control vessels through which Blood enter
Capillaries
Function is to exchange:
- Fluid
- Nutrients
- Electrolytes
- Hormones
- Other Substances
Between the blood and interstitial fluid in the various tissues in the body
Venules
Collect blood from the capillaries and gradually converge into Veins
Veins
Transport blood back to the heart
Cardiovascular System Function
Transport nutrients and removes waste products

Helps to maintain the Environment for all the body functions

Transports O2 from lungs to Tissues

Transports CO2 from tissues to Lungs
Hemoglobin
Part of the Blood that transports O2

Iron-Protein Molecule in RBC's

Acid Base Buffer
Red Blood Cells
Major component of Blood

Contain Carbonic Anhydrase (catalyzes CO2 and H2O to remove CO2)
Respiratory System (Function)
Basic exchange of Oxygen and Carbon Dioxide
Trachea
First-Generation Respiratory Passage
Bronchi
Left/Right

Second Generation Respiratory Passage
Brochiloes
All additional generations of Air Passage Alveoli
Alveoli
The location of gas exchange in Respiration
Pleural Pressure
Pressure in the narrow space between the Lung Pleura and Chest Wall
Pleura
Membranes enveloping the lungs and lining the chest walls
Alveolar Pressure
The pressure inside the Alveoli when the Glottis is open and no air is flowing into/out of Lungs
Diffusion
Random motion of molecules moving in Opposite Direction through the Alveolar Capillary Membrane

From High Concentration to Low Concentration
_____ are essentially a facility's rules and regulations; they reflect the goals and objectives of the program.
Policies
______ describe how policies are met or carried out.
Procedures
10 things that make up an effective mission statement:
(1)Short and focused (2)Clear and easily understood (3)Defines why we do what we do; why the organization exists (4)Does not prescribe means (5)Is sufficiently broad (6)Provides direction for doing the right things (7)Addresses our opportunities (8)Inspires our commitment (9)Matches our competence (10) Says what in the end we want to be remembered for
_____ _____ are the desired end products of a strength and conditioning program.
Program goals
_____ _____ are specific means of attaining program goals.
Program objectives
3 Hierarchical levels of strength and conditioning staff for a medium to large facility. In smaller ones you may have only 1–2.

"(1) Strength and Conditioning Director – is an administrator and practitioner

Speed

Skills and abilities needed to achieve high movement velocities
Agility
Skills and abilities needed to explosively change Movement Velocities or modes other than linear sprinting

Expression of athletes Coordinative Abilites

Basis of: Acceleration, Maximum–Velocity, Multidirectional Skills
Speed–Endurance
Ability to Maintain Maximal Movement Velocities

Repeatedly achieve maximal accelerations and velocities
Special Endurance
Ability to repeatedly perform Maximal/near Max or efforts in Competition–Specific exercise to rest ratio
Exercise:Relief (AKA. Work:Rest) Ratios
Metabolic power to execute specific techniques at targeted effort level

The metabolic capacity to do so
Force
Product of mass and acceleration
Impulse
Change in momentum resulting from a force measured as the product of Force and Time
Power
Rate of doing work

Product of force and velocity
Velocity Specificity
The final Movement velocity targeted when a mass is being Accelerated
Reactive Ability
Characteristic of Explosive strength exhibited in SSC action

Improved through reactive–explosive training
Reaction Time
Relatively Untrainable

Correlates poorly with movements action time/ sports performance
Ballistic Running
Flight phase

Single–leg support phase
Run Speed Interaction
Stride Frequency

Stride Length
Flight (Phases) Running
Recovery

Ground Preparation
Support (Phases) Running
Eccentric Breaking

Concentric Propulsion
Goal of Sprinting
Achieve high stride frequency and optimal stride length

w/ Explosive horizontal push–off

Minimal vertical impulse
Coordinative Abilities
Adaptive Ability

Balance

Combinatory

Differentiation

Orientation

Reactiveness

Rhythm
Practice Specificity
General Agility target development of basic coordinative abilities

Special tasks unify them in skill specific manner
Closed Agility Skills
Programmed assignments

Predictable/stable environments
Open Agility Skills
Non–programmed assignments

Unpredictable/unstable environments
Continuos Skills
No identifiable start or finish
Discrete Skills
Definite Start and finish
Serial Tasks
Discrete skill performed in sequence
Agility Characterized by Several Criteria
Initial speed and direction

Decrease or Increase in speed
redirection of movement

Final speed direction
Agility Needs Analysis
Two Fronts

Change velocity and Mode of Locomotion

vs.

Classifying motor skills according to basic schemes
Sprint Resistance
Gravity–Resisted Running or other means of achieving an overlaid effect

Provide resistance without arresting movement (>10% change detrimental)

Improve explosive strength
Sprint Assistance
Gravity assisted running (run down hill, high speed towing)

Achieve an Overspeed Effect

Improve Stride Rate
Tertiary Methods of SAQ
Mobility, Strength, Endurance
Dynamic Correspondence
Prioritizing strength training on rate/time force pro

Identify target activities
– Mechanics
– Metabolic
– Coordinative
Tactical Metabolic Training
Establish special endurance training criteria

According to competitive exercise–relief
Methods of SAQ Development
Primary:
Execution of Sound Movement Technique in a specific task

Secondary:
Methods – sprint resistance and sprint assistance training

Tertiary:
mobility, strength, speed–endurance
Periodization
Planned distribution or variation in training means and methods on a periodic or cyclic basis
Volume Load
Product of Work Volume and Intensity
Sequenced Training

Strategies based on the premise that Delayed effects of certain training stimuli can alter the response of others

Bioenergetics
Flow of energy in a Biological system

Concern Primarily the conversion of MacroNutrients
– CHO
– PRO
– FAT
Energy
Ability/Capacity to perform work
Catabolism
The breakdown of Large molecules into Smaller molecules

Associated with the Release of Energy
Anabolism
The Synthesis of Larger molecules from Smaller molecules from E+
Exergonic Reaction
Energy Releasing Reactions

Generally Catabolic
Endergonic Reaction
Require Energy

Include Anabolic Processes

Contraction of Muscles
Metabolism
The total of all the Catabolic (Exergonic) and Anabolic (Endergonic) Reactions in a biological system
Adenosine Triphosphate (ATP)
ATP allows the transfer of Energy from Exergon to Endergon Reactions
Hydrolysis
The breakdown of one Molecule of ATP to yield energy

B/C it requires one Molecule of H2O
Adenosine Triphosphatase (ATPase)
The enzyme that catalyzes ATP hydrolysis
Myosin ATPase
Specifically, the enzyme that catalyzes ATP hydrolysis for Cross–Bridge Recycling
Calcium ATPase
Enzyme for ATP Hydro

For Pumping Ca back into the SR
Sodium–Potassium ATPase
Enzyme for ATP Hydro

For Maintaining the Sarcolemmal concentration gradient Post–Depol
Adenosine Diphosphate (ADP)
Byproduct of Hydro of ATP

Only two Phosphate Groups
Adenosine Monophosphate (AMP)
Byproduct of Hydro of ADP
Anaerobic
Processes that do not require the presence of O2
Aerobic
Mechanisms that depend on O2
3 Systems of ATP Replenishment
Phosphagen

Glycolytic

Oxidative
Phosphagen System
Provides ATP primarily for:
– Short–Term
– High Intensity
Activities

Also, it's the reactive Start of ALL exercise regardless of intensity
Creatine Phosphate (CP) / Phosphocreatine (PCr)
High energy phosphate molecules used for E+ reproduction in the Phosphagen system
Creatine Kinase
The enzyme that catalyzes the synthesis of ATP from CP and ADP
Adenylate Kinase (aka Myokinase) Reaction
An important Single–Enzyme reaction that can rapidly replenish ATP
Type II Muscles and CP
Type II muscles have higher concentrations of CP
Law of Mass Action (aka Mass Action Effect)
Phosphagen system control

States:
– Concentrations of Reactants/Products (or both), in a solution, will Drive the Direction of the Reactions.
Near–Equilibrium Reactions
Slow Steady, equal Reaction

Proceed in a direction dictated by the concentrations of the Reactants due to the Law of Mass Action
Glycolysis
The breakdown of CHO

Either Glycogen stored in muscles
or
Glucose delivered in the Blood

To Resynthesize ATP
Pyruvate
The end result of Glycolysis

Can be converted to Lactate or Shuttled to Mitochondria
Anaerobic Glycolysis (Fast Glycolysis)
When Pyruvate is converted to lactate
– ATP Resynthesis occurs at a faster rate
– Limited duration
Aerobic Glycolysis (Slow Glycolysis)
When Pyruvate is shuttled into the Mitochondria for Krebs Cycle
– ATP Resynthesis rate is slower
– Occurs for Longer Duration during low intensity exercise
Lactate
Pyruvate is converted into lactate to be mobilized throughout the body
Creatine Kinase
The enzyme that catalyzes the synthesis of ATP from CP and ADP
Adenylate Kinase (aka Myokinase) Reaction
An important Single–Enzyme reaction that can rapidly replenish ATP
Type II Muscles and CP
Type II muscles have higher concentrations of CP
Law of Mass Action (aka Mass Action Effect)
Phosphagen system control

States:
– Concentrations of Reactants/Products (or both), in a solution, will Drive the Direction of the Reactions.
Near–Equilibrium Reactions
Slow Steady, equal Reaction

Proceed in a direction dictated by the concentrations of the Reactants due to the Law of Mass Action
Glycolysis
The breakdown of CHO

Either Glycogen stored in muscles
or
Glucose delivered in the Blood

To Resynthesize ATP
Pyruvate
The end result of Glycolysis

Can be converted to Lactate or Shuttled to Mitochondria
Anaerobic Glycolysis (Fast Glycolysis)
When Pyruvate is converted to lactate
– ATP Resynthesis occurs at a faster rate
– Limited duration
Aerobic Glycolysis (Slow Glycolysis)
When Pyruvate is shuttled into the Mitochondria for Krebs Cycle
– ATP Resynthesis rate is slower
– Occurs for Longer Duration during low intensity exercise
Lactate
Pyruvate is converted into lactate to be mobilized throughout the body
Metabolic Acidosis
The process of an Exercise–Induced Decrease in pH

Inhibits the enzymatic turnover rate of cell's E+ systems
Wet Muscle
Muscle that has not been Desiccated
Cori Cycle
Process of transporting Lactate in the blood to the liver
>
Then converted to Glucose
Mitochondria
Specialized cellular organelles where the reactions of aerobic metabolism occur
Reduced
Refers to the addition of Hydrogen
Phosphorylation
The process of adding an inorganic Phosphate (Pi) to another Molecule
Oxidative Phosphorylation
The resynthesis of ATP in the Electron Transport Chain
Substrate–Level Phosphorylation
The direct resynthesis of ATP from ADP during a single reaction in the Metabolic Pathways
Allosteric Inhibition
When an end Product Binds to the Regulation Enzyme
– Decreases turnover rate
– Slows production formation
Allosteric Activation
When an "Activator" binds with the enzyme and
– Increases its turnover rate
Rate–Limiting Step
The slowest step of a chemical reaction
– Limits and controls rate of reaction
Lactate Threshold (LT)
Intensity at which Blood Lactate
– Begins an Abrupt increase above baseline
– Marker of Anaerobic Threshold
Onset of Blood Lactate Accumulation (OBLA)
When the concentration of Blood Lactate reaches:
– 4 mmol/L
– During very Intense exercise
– Second increase of Lactate accumulation after LT
Oxidative System
The primary source of ATP at:
– Rest
– Low Intensity Exercise

Uses Primarily:
– CHO
– FAT
Krebs Cycle
A series of reactions that
– Continues to Oxidate the substrate from Glycolysis
– Produces two ATP
Electron Transport Chain (ETC)
The process of creating ATP from:
– ADP
– NADH
– FADH2
Cytochromes
Electron carriers in the Electron Transport Chain
Beta Oxidation
A series of reactions in which Free Fatty Acids are
– Broken Down
– Creates Acetyl–CoA and H–
Total ATP Yield from Oxidation of ONE Glucose Molecule
40
Gluconeogenesis
The process of converting Amino Acids into Glucose
Branched Chain Amino Acids
–Leucine
–Isoleucine
–Valine

Major amino acids that are oxidized in Skeletal Muscle
Total E+ Yield from Oxidation of ONE Triglyceride
463
Exercise Intensity
The Level of Muscular Activity that can be quantified in terms of Power Output
Power
Work performed per:
– Unit of Time
Relationship of Energy Systems
Inverse Relationship between:
– Energy System's max rate of ATP production
– Total amount of ATP production capable
E+ System for 0–6 seconds
(Intensity: Extremely High)
Phosphagen
E+ System for 6–30 seconds
(Intensity: Very High)
Phosphagen/Fast Glycolysis
E+ System for 30 secs – 2 mins
(Intensity: High)
Fast Glycolysis
E+ System for 2–3 minutes
(Intensity: Moderate)
Fast Glycolysis/Oxidative System
E+ System for >3 minutes
(Intensity: Low)
Oxidative System
Energy Substrates
Molecules that provide starting materials for:
– Bioenergetic Reactions
– Includes Phosphagens (ATP/CrP), Glucose, Glycogen, Lactate, Free Fatty Acids, Amino Acids
Time for Post–Ex Phosphagen Replenishment
3–5 mins
Glycogenolysis
Breakdown of Glycogen
Oxygen Uptake (O2 Consumption)
The measure of a person's ability to:
– Take in/Use Oxygen
Oxygen Deficit
The Anaerobic Contribution to the:
– Total E+ Cost of Exercise
Oxygen Debt
Post–Exercise Oxygen Uptake
Excess Postexercise Oxygen Consumption (EPOC)
The Oxygen Uptake:
– Above Resting Values
– Used to restore the body to pre exercise conditions

Factors:
– ATP REsynthesis
– O2 resaturation
– Repair Damage
– Increased body temperature
Metabolic Specificity of Training
The use of Appropriate:
– Ex. Intensities
– Rest Intervals
– Ex. Selection
based on the specific Energy Systems used during competition
Interval Training
A method of training that emphasis:
– Bioenergetic Adaptations
– For more efficient energy transfer
– Within Metabolic pathway
– Using Pre–Determined Intervals of Exercise and Rest Periods
Combination Training (Cross–Training)
Adding Endurance Training to Anaerobic Athletes training to:
– Enhance Recovery
– Due to recovery being Aerobic

*Aerobic Endurance training WILL REDUCE ANAEROBIC TRAINING*
* Especially:
– High Strength
– High Power
Exercise–to–Rest Intervals

Phosphagen:
5–10 secs (1:12 – 1:20)

Fast Glycol:
15–30 secs (1:3 – 1:5)

Fast Glycol/Oxid
1–3 mins (1:3 – 1:4)

Oxidative
>3 mins (1:1 – 1:3)

3 Principles of Resistance, Plyometrics, and Speed
1. Specificity

2. Overload

3. Progression
Specificity
Most basic concepts to incorporate

Method where an athlete is trained in a Specific manner

To produce Specific adaptation or training outcome

S.A.I.D. Specific Adaptation to Imposed Demands
S.A.I.D. Principle
Specific Adaptations to Imposed Demands

The type of demand placed on the body dictates the type of adaptation that will occur

The more similar the training activity is to the actual sport movement, the greater the positive transfer
Overload
Assigning a workout or training regime of Greater intensity than the athlete is accustomed to
Progression
To attain higher levels of performance

Intensity of the truing must Progressively become greater
Program Design
Complex process of designing a resistance program with recognition and manipulation of the 7 Program Design Variable
7 Variables of Program Design
1. Needs Analysis

2. Exercise Selection

3. Training Frequency

4. Exercise Order

5. Training Load and Repetitions

6. Volume

7. Rest Periods
Needs Analysis
Two–Stage process

Includes evaluation of the Requirements and Characteristics of the sport

Assessment of the athlete
Evaluation of Sport (Needs Analysis)
Movement Analysis

Physiological Analysis

Injury Analysis
Movement Analysis
Body/Limb movement patterns and muscular involvement
Physiological Analysis
Strength, Power, Hypertrophy, and Muscular Endurance priorities of a sport
Injury Analysis
Common sites for Joint and Muscle injury and causative factors
Profile (Assessment of the Athlete)
Profile athletes Needs and Goals

Evaluate training/injury status

Conducting maximum strength testing
Training Status
An athletes current condition or level of preparedness to being new/revised program
Training Background/ Exercise History
Training that occurred before he/she began a new/revised program
– type of training
– length of program
– level intensity
– Degree of exercise technique
Exercise Technique Expérience
Knowledge and Skill to perform resistance training exercises properly
Primary Goal of Resistance Training
Improve Strength, Power, Hypertrophy, muscular endurance

Pick one to focus on per season
Exercise Selection
Choosing exercises for a resistance training program
Core Exercises
Recruit one or more large muscle areas

Involve two or more primary joints

And receive priority when selecting exercises due to direct application to sport
Multijoint Exercises
Involves two ore more primary joints
Assistance Exercises
Usually recruit smaller muscle areas/groups

Involve only one primary joint

Less important to improving sport performance
Single–Joint Exercises
Involve only one primary joint
Structural Exercise
Core exercise that emphasizes loading of the spine directly or indirectly
Power Exercise
Structural exercises that are performed very quickly or explosively
Agonist
Muscle/group actively causing the movement
Antagonist
Sometimes passive (not concentric) muscle/group that is on the opposite side if the limb from the agonist
Muscle Balance
Not always equal strength

Proper Ratio of Strength/Power/Muscle Endurance
Training Frequency
Number of training sessions completed in a given time period
Split Routine
Different muscle groups are trained on different days
Exercise Order
The Sequence of resistance exercises performed during one training session
Resistance Training Frequency Based on Training Status
Beginner 2–3/week

Intermediate 3–4/week

Advanced 4–7/week
Resistance Training Frequency Based on Sport Season (Trained)
Off–Season 4–6/wk

Preseason 3–4/wk

In–season 1–3/wk

Postseason (active rest) 0–3/wk
Preexhaustion
Reverse Exercise arrangement
– purposely fatigue single–joint prior to multijoint for greater exhaustion of larger groups
Circuit Training
Exercises performed with minimal rest periods (20–30 secs)
Superset
Two sequentially performed exercise that stress two Opposing muscle/groups
Compound Set
Sequentially performing two Different exercises for the Same Muscle Group
Load
Amount of weight assigned to an exercise

Often most critical aspect of a resistance training program
Mechanical Work
The product of Force and Displacement (distance)
Load–Volume (AKA. Volume–Load)
Reps x Weight

Highly related to mechanical work

Total amount of work performed

Associated Metabolic energy demands and physiological
Load Equations
Reps x Weight
Intensity
Quality of Work Performed

Sets and reps
Repetitions
Number of times an exercise can be performed
1–Repetition Maximum (1RM)
Greatest amount of weight

Lifted with proper technique

For only one repetition
Repetition Maximum (RM)
Most weight lifted for a specific number of repetitions
Goal Repetitions
The number of repetitions to perform in a given exercise
2–for–2 Rule
Conservative Method to increase athletes training loads

If athlete can perform 2+ reps over his RM in the last set of 2 workouts weight should be added to the exercise in the next session
Volume
Total amount of weight lifted in a training session
Set
Group of repetitions sequentially performed before the athlete stops to rest
Rest Period (AKA. Interset Rest)
Time dedicated to recovery between sets/exercises
Repetition Volume

Total Number of Reps

Resistance Training on Endocrine System
Can be manipulated naturally by Resistance Training

Can enhance/develop of Target Tissues

*Improves Performance
Hormones
Chemical Messengers that are:
– Synthesized
– Stored
– Released in blood

by Endocrine Glands
Endocrine Glands
Body Structures specialized for:
– Secretions

*maybe other cells
Neuroendocrinology
The Study of the Interactions between:
– Nervous System
– Endocrine System
Nucleus Receptor for Hormones are in these hormones
Steroid

Thyroid
Target Tissue
The tissue for which hormones are created for
Myosin Heavy–Chain Proteins (MHC)
Can go through a change in their molecular structure
– From IIx to IIa
Anabolic Hormones
Hormones promoting:
– Tissue Building
– Block catabolic hormones
Catabolic Hormones
Attempt to degrade cell proteins to support:
– Glucose synthesis
Lock–and–Key Theory
The Receptor = Lock

The Hormone = Key
Cross–Reactivity
A given receptor partially:
– Interacts with Hormones that not specifically designed for it
Allosteric Binding Sites
When a substance Other than Hormones can:
– Enhance/Reduce cellular response to the Primary Hormone
Downregulation
Inability of a hormone to interact with a receptor
Hormone–Receptor Complex
(H–RC)
A binded Hormone/Receptor
– Shifts the receptor
– Activates Receptor
– Opens cells Nucleus
Polypeptide Hormones
Hormones made up of Amino Acids

i.e. HGH, Insulin
Secondary Messengers (STAT)
Messengers that get polypeptide hormones messages across cellular membranes
Heavy Resistance Exercise and Hormonal Increases
Specific Force Produced in Activated fibers:
– Stimulates: receptor and membrane Sensitivities
– To anabolic factors:
– Includes: hormones
– Leads to muscle growth and strength training
Hormone link to Resistance Training
Hormone responses are tightly linked to:
– Characteristics of the Resistance Exercise Protocol
Diurnal Variations
Normal fluctuations in Hormone Levels throughout the day
Large Muscle Group Training and Hormone Response
Large Muscle Group Exercise results in:
– Acute increased Serum Total Testosterone concentrations in men
Growth Hormone (Reactions)
– Normal development of children

– Plays a vital role to Adapting to stress of Resistance Training

Secondary Effects of HGH Injections:
– Change in muscle size/strength
– Hypertrophy
Proteolytic Enzymes
Enzymes that break down proteins
Cortisol Hormone and Resistance Training
Resistance Training:
– High Volume
– Large Muscle Groups
– Short Rest Periods

=

Increased Serum Cortisol Values (if acute, maybe a sign of muscle tissue remodeling)
General Concepts (Resistance Training and Endocrine Response)
– More Muscle fibers recruited = more muscle remodeling

– Only muscle fibers activated by Resistance Training are Subject to Adaptation
To Increase Serum Testosterone Concentrations
– Large Muscle Group Exercise

– Heavy Resistance Training (85%–95% of 1RM)

– Moderate–High Volume of Exercise

– Short Rest Intervals (30–60 secs)
To Increase Growth Hormone Levels
– Use workouts with High Lactate Concentrations

– High Intensity (10RM/Heavy Resistance)

– High Total Work (3 sets)

– Short Rest Periods (1 min)

– Supplement Diet with Carbohydrate and Protein Before/afterwards workout
To Optimize Responses of Adrenal Hormones
Use:
– High volume
– Large Muscle Groups
– Short Rest Periods

But:
– Vary training protocol/rest period length/volume

Prevents:
– Adrenal gland chronic catabolic response of Cortisol
Growth Hormone (Function)
Stimulates:
– IGF–1
– Protein Synthesis
– Growth
– Metabolism
Thyroid–Stimulating Hormone (Function)
Stimulates:
– Thyroid Hormone Synthesis
– Secretion
Luteinizing Hormone (Function)
Stimulates:
– Ovulation
– Secretion of Sex Hormones in Ovaries/Testes
Insulin Hormone (Function)
Stores:
– Glycogen

Promotes:
– Glucose Entry into Cells

Involved In:
– Protein Synthesis
Glucocorticoids (Function) (Cortisol, Cortisone, etc)
Inhibits:
– Amino Acid Incorporation into Proteins

Stimulates:
– Conversion of Amino Acids into: CHO

Maintains:
– Normal Blood Sugar Levels

Conserves:
– Glucose

Promotes:
– Fat use
Insulin Like Growth Factor –1 (Function)
Increase Protein Synthesis in Cells
Epinephrine (Function)
Increases:
– Cardiac Output
– Blood Sugar
– Glycogen Breakdown
– Fat Metabolism

Norepinephrine (Function)

Same as Epinephrine

Also:
– Constricts Blood Vessels

Testosterone (Function)

Stimulates:
– Growth
– Protein Anabolism
– Development/Maintenance of Male Sex Characteristics

Anatomy
The Study of the components that make up the "Musculoskeletal" machine
Biomechanics
The Mechanism through which the Musculoskeletal system interact to create Movement
Axial Skeleton
Consists of:
– Skull (cranium)
– Vertebral Column (C1–Coccyx)
– Ribs
– Sternum
Appendicular Skeleton
Consists of:

Shoulder Girdle
– L/R Scapula
– Clavicle
Bones of the Arm:
– Humerus
– Radius
– Ulna
– Carpals
– Metacarpals
– Phalanges

Pelvic Girdle:
– L/R Coxal Bones

Bones of Legs/Ankles/Feet
– Femur
– Patella
–Tibia
– Fibula
– Tarsals
– Metatarsals
– Phalanges
Joints
Junction of Bones
Fibrous Joints
Allow Virtually no Movement

i.e. Sutures of the Skull
Cartilaginous Joints
Allow Limited Movement

i.e. Intervertebral Disks
Synovial Joints
Allow considerable movement

i.e. Knee and Shoulder Movement
Hyaline Cartilage
The smooth cartilage on the end of Bones at the joint
Synovial Fluid
Liquid in the Joint Capsule that allows for lubrication and nutrient diffusion
Uniaxial Joints
Operates as Hinges

Rotates on One Axis

i.e. Elbow
Biaxial Joints
Movement around:
– Two Perpendicular Axes

i.e. Ankle/Wrist
Multiaxial Joints
Ball and Socket Joints
– Allow movement in all 3 Perpendicular axes

i.e. Hip and Joints
Vertebral Column
Made up of:
– Several Vertebral Bones
– Flexible Disks
Cervical Vertebrae
7

In Neck
Thoracic Vertebrae
12

In middle upper back
Lumbar Vertebrae
5

Make Up lower back
Coccygeal Vertebrae
3–5

Inner tail of pelvis
Origin (Muscle)
The muscles:
– Proximal Attachement
Proximal
Toward the Center of the Body
Insertion (Muscle)
The muscles:
– Distal Attachement
Distal
Away from the Center of the Body
Fleshy Attachment
Found at the Proximal End of a Muscle

Muscle Fibers:
– Directly affixed to the Bone
– Usually over a Wide Area for Force Distribution
Fibrous Attachment
i.e. Tendons

Blend into/are:
– Continuous with both Muscle Sheaths and Connective Tissue surrounding the bone
Agonist
Prime Mover

The muscle most Directly Involved in Bringing about a movement
Antagonist
The muscle that can slow down/stop movement
Synergist
A muscle that assists
– Indirectly in a movement
Lever
A Rigid/Semirigid Body that:
– When Subjected to force (when actions does not pass through pivot point)
– Exerts Force on any Object impending its Tendency to Rotate
Fulcrum
The Pivot Point of a Lever
Moment Arm (Force Arm/Lever Arm/Torque Arm)
The Perpendicular Distance from the:
– Line of Actions
– Infinitely Long Line passing point of application of force
– Oriented in the Direction in which the Force is Exerted
Torque (Moment)
The Degree to which a Force tends to:
– Rotate an object about a Specified Fulcrum
Muscle Force
Force Generated by:
– Biomechanical activity
– Stretching of Noncontractile Tissue

Tend to draw the opposite ends of the muscle together
Resistive Force
Force generate by:
– Sources External to the Body (e.g. gravity, inertia, friction)
– Acts contrary to Muscle Force
Mechanical Advantage
The Ratio of the Moment Arm
– Through Which an applied Force act to that
– Through which the Resistive Force Acts

Mechanical Advantage >1.0 allows:
– Muscle force to be less that resistive force to produce an equal torque
– Visa Versa On <1
First–Class Lever
A lever for which the muscle force and resistive force are on Opposite side of the Fulcrum

Force
>
Axis
>
Resistance

"See–Saw"
Second–Class Lever
A lever for which the Muscle Force and Resistance Force act on the:
– Same Side of the Fulcrum
– Muscle force acting through the moment arm

Muscle Force
>
Resistance
>
Axis of Rotation

"Wheel Barrel"
Third–Class Lever
A lever for which the:
– Muscle force and Resistance Force act on the same side
– Muscle force works through shorter Moment Arm

Axis
>
Muscle Force
>
Resistance Movement

"Bicep Curl"
Patella Function
Keep mechanical advantage at knee joint
– Keep Quad tendon perpendicular to knee axis
Most Muscles Operate on what Type of Advantage
Disadvantage
– Cause a lot of injury because of exaggerated forces
Anatomical Position
Body Erect

Arm's down at the side

Palm's facing forward
Sagittal Plane
Split:
– Left/Right
Frontal Plane
Split:
– Front Back
Transverse Plane
Split:
– Upper/Lower
Acceleration
Change in Velocity Per Unit Time
Force =
Force = Mass x Acceleration
Strength
The Maximal Force that a Muscle/Muscle Group can Generate at a Specified Velocity
Power
The Time Rate of Doing Work
Work
The Product of the Force Exerted on an Object

and

The Distance the Object moves in the Direction in which the Force is Exerted
Work = (Equation)
W = Force x Distance
Power = (Equation)
P = Work/Time
Weight
Mass kg2 x 9.8 m/s2
Angular Displacement
The Angle through which an Object Rotates
Angular Velocity
An Objects Rotational Speed
– Measured in Radians/sec
Rotational Work Equation
W = Torque x Displacement
Rotational/Linear Power Equation
Power = 19,600 J / Secs
Recruitment
Which and How Many Motor Units are involved in a Muscle Contraction
Rate Coding
Rate at which the Motor Units are Fired
Pennate Muscle
Fibers that Align:
– Obliquely w/ tendon
– Featherlike arrangement
Angle of Pennation
The Angle between the Muscle Fibers and an Imaginary line Between:
– the Muscles Origin
– and Insertion
Concentric Muscle Action
A Muscle Action in which the:
– Muscle Shortens
– Muscle Force > Resistance Force
Eccentric Muscle Action
A Muscle Action in which the:
– Muscle Lengthens
– Muscle Force < Resistance Force
Isometric Muscle Action
A Muscle Action in which the:
– Muscle Length Does Not Change
– Muscle Force (=) Resistance Force
Classic Formula
Loaded Lifted / BW2/3

Used to figure Relative Wieght Lifted
Gravity Formula
Gravity = Mass x Local Acceleration
Inertial Force
an imaginary force which an accelerated observer postulates so that he can use the equations appropriate to an inertial observer
– To describe Inertia, in a non–intertia base
Bracketing Technique
The Athletes performs the:
– Sport Movement
– With Less than Normal
– and Greater than Normal
– Resistance

Form of Acceleration Training
i.e. Shot–Putter with Extra–Heavy Shot–Putt
Force/Weight/Acceleration Relationship
When a Weight is Held in a
– Static Position/Constant Velocity

It Exerts:
– Constant Resistance
– Only in the Downward Direction

However:
– Upward/Lateral Acceleration of the Weight
– Requires Additional Forces
Friction (Definition)
The Resistive Force Encountered when
– One Attempts to move an Object
– Pressed against another Object
Friction (Equation)
Resistance Force = Coefficient of Friction (for both objects) x Normal Force
Fluid Resistance
The Resistance Force Encountered By:
– an Object Moving through Fluid (Liquid or Gas)

or

Fluid Moving Past or Around
– an Object
– or Through an Orifice
Suface Drag
Result from the Friction of a Fluid:
– Passing Along the Surface of an Object
Form Drag
Results from the way in which a Fluid:
– Presses Against
– the Front/Rear of an Object
– Passing Through It
Lordotic
Slightly Arched
– Better Advantage in Back
– Avoid Back
Kyphotic
Slightly Rounded
Ventral
Towards the Anterior
Dorsal
Towards the Posterior
Valsalva
Glottis Closed (prevents air escaping lungs)

Muscles of the Abdomen and Rib Cage Contract

– Creates Rigid Compartments of –– Liquid in Lower Torso
–– Air in the Upper Torso

– Increases Rigidity of Entire Torso
– Easy to Support Heavier Loads
Specificity

Training is Most Effective When:
– Exercises are Similar to the Sport Activity, which improvement is needed (target activity)

– Major consideration during Program Design

– Observe the Sport Movement :
–– Analyze Qualitatively/Quantitively to determine – Joint Movement

Anaerobic Training
Consists of:
– High Intensity
– Intermittent Bouts
– of Exercise

i.e.:
– Weight Training
– Plyometric Drills
– Speed/Agility
– Internal Training
Anaerobic Training Adaptations
Improvements in:
– Muscular Strength
– Power
– Hypertrophy
– Muscular Endurance
– Motor Skill Performance
Size Principle
Governs:
– De/recruitment of MU's in an orderly manner

Relationship between:
– MU Twitch Force
– Recruitment Threshold

MU's are recruited in Order According to their:
– Thresholds
– Firing Rates
Adaptation to Resistance Training (Muscle Fibers)
With Heavy Resistance Training:
– All fibers grow larger

Experienced Lifters:
– CNS adaptations allow greater MU activation of Larger MU's first
Selective Recruitment
Exception to Size Principle

Fast–Twitch MU"s may occur under Circumstances that allow the Athlete to:
– Inhibit Lower–Threshold MU"s
– Instead will Activate Higher MU's Thresholds to produce force
Neuromuscular Junction (NMJ)
Interface between the:
– Nerve
– Skeletal Muscle
– Potential Site for Neuro–Adaptations

All from Anaerobic Training
Electomyography (EMG)
Common Research Tool

Used to examine:
– Magnitude of Neural Activation following training
Cross–Education
Training only One Limb
– Can Result in an Increase in Strength in the Untrained Limb!
Bilateral Deficit
Untrained Individuals

The Force Produced when Both Limbs are Contracting
– is Less than the Sum of
– The Forces when produced Unilaterally
Hypertrophy
Muscular Enlargement
– from Training
– Increase in Cross–Sectional Area
Structural Proteins
Titin

Nebulin

Part of Hypertrophy Adaptation
Myogenesis
Muscle Regeneration
Proteins Increased in Hypertrophy
Actin

Myosin

Myofibrils
Hyperplasia
Increase in the actual
– Number of Muscle Fibers
– Via Longitudinal Fiber Splitting

Response to H.I.T. (Only in Animals, not so much Humans)
Mechanical Loading
Forces from Exercise that:
– Cause Deformation of Specific Regions of the Skeleton
– Created by Muscular Actions
– On Tendinous Insertion into Bone
– Bending, Compressive, Torsional
Osteoblast
Cells that:
– Manufacture
– Secrete Proteins (Collagen)
– Placed in–between bone cells
– Increase bone strength

Migrates to Bone's Surface:
– Begin Bone Remodeling
Bone Matrix
Space Between Bone Cells
Hydroxyapatite
Calcium Phosphate Crystals
– Mineralized Collagen
Periosteum
Outer Surface of the Bone
Trabecular Bone
Spongy Bone
Cortical Bone
Compact Bone
– Dense
– Compact outer shell of bone
Minimal Essential Strain
The Threshold of Stimulus
– Initiates new Bone Formation
– From Enhanced Mechanical Strain
Bone Mineral Density
The Quantity of Mineral Deposited in a Given Area of Bone
Specificity of Loading
Using exercises that:
– Directly Load a Particular Region of the Skeleton
Osteoporosis
A Disease in which:
– BMD
– Bone Mass
– Reduced to Critical Levels
Osteogenic Stimuli
Factors that Stimulate New Bone Formation
Structural Exercises
Exercises that Involve:
– Multiple Joints
– Direct Force Vectors Through
– – The Spine and the Hip
Progressive Overload
Progressively Placing:
– Greater than Normal Demands
– On the Exercising Musculature
– Training that increases Bone Mass
Stress Fractures
Micro–fractures in the Bone Due to:
– Structural Fatigue
Peak Bone Mass
Maximum Bone Mass Achieved during:
– Early Adulthood
Component of Mechanical Load for Bone Growth
Magnitude Load
– Intensity

Rate of Loading:
– Speed

Direction of Forces

Volume of Loading
– Number of Repetitions
How Do Athletes Stimulate Bone?
– Exercise for Direct Load (Specificity)

– Structural Exercises

– Progressively Overload

– Vary Exercise Selection

– Weight Bearing
Collagen
The primary structural component of
– All Connective Tissue
– Type I for Bone/Tendon/Ligaments
– Type II for Cartilage
Procollagen
The Parent Protein to Collagen

Synthesized and Secreted by:
– Fibroblasts

3 Protein Strands Twisted Around Each Other (Triple Helix)
Microfibril
The Parallel Arrangement of Collagen Filaments
Cross–Linking
Strong Chemical Bonds of Collagen

Collagen True Strength

Chemical Bonds Forms Between Adjacent Collagen Molecules throughout collagen bundles
Elastin
Elastic Fibers in Ligaments
Sites Where Connective Tissue Can Increase: Strength/Load Bearing
At Junctions Between the:
– Tendon/Ligament
– Bone Surface

Within Body of the
– Tendon/Ligament

In the Network of:
– Fascia within Skeletal Muscle
Increase of Strength in a Tendon Are From What Adaptations
Increase in Collagen Fibril Diameter

Greater Number of Covalent Cross–Linking in Hypertrophied Fiber

Increase in the Number of Collagen Fibrils

Increase in the Packing Density of Collagen Fibrils
Tendon Stiffness
Force Transmission:
– Per Unit of Strain (Tendon Elongation)
Main Function of Cartilage
– Provide Smooth Joint Articulating Surfaces

– Act as a Shock Absorber for Forces Directed Through the Joint

– Aid in the Attachment of Connective Tissue to the Skeleton
Hyaline Cartilage (Articular Cartilage)
Found on the:
– Articulating Surface of Bones
Fibrous Cartilage
Very Tough form of Cartilage

Found in:
– Intervertebral Disks of Spine
– At Junctions where Tendons Attach to Bone
Athletes Training for Connective Tissue Adaptations (Tendons, Ligaments, Fascia)
High–Intensity Exercise
Athletes Training for Connective Tissue Adaptations (Cartilage)
Weight–Bearing Forces

Complete Movements (Full ROM)

Moderate Aerobic Exercise
Acute Anabolic Hormonal Response to Anaerobic Exercise
Critical for Exercise Performance/Training Adaptations

– Upregulation of Anabolic Hormone Receptors is Important for:
– – Mediating the Hormonal Effects
Acute Anaerobic Exercise Results in:
Increased:
– Cardiac Output
– Stroke Volume
– Heart Rate
– Oxygen Uptake
– Systolic BP
– Blood Flow to active Muscles
Reactive Hyperemia
When Contractions >20% max voluntary contraction
– Impedes Blood Flow

BUT…
– Blood Flow Increases During Rest Periods (Reactive Hyperemia)
Rate Pressure Product
RPP = Resting Heart Rate x Systolic Blod Pressure

A Measure of Myocardial Work
Ventilation Equivalent
The Ration of:
– Air Ventilated to
– Oxygen Used by the Tissues
Possible Decrease of Power/Strength Output From Aerobic Training
Adverse Neural Changes

Alterations of Muscle Proteins n Muscle Fibers
Overtraining
Excessive:
– Frequency
– Volume
– Intensity

Of Training that Results in:
– Extreme Fatigue
– Illness
– or Injury

Due to Lack of:
– Sufficient Rest
– Recovery
– m/b Nutrient Intake
Overreaching
Excessive training on a Short–Term Basis
Overtraining Syndrome
The Condition resulting from
– Overtraining

Happens when Overreaching continues beyond a Reasonable Period of Time

aka. Staleness, burnout, chronic overwork, etc.
Sympathetic Overtraining Syndrome
Increased:
– Sympathetic Activity at Rest
Psychological Markers of Anaerobic Overtraining
Decreased Desire to Train

Decreased Joy from Training
Hormonal Markers of Anaerobic Overtraining
Acute:
– Epinephrine/Norepinephrine
– Increased beyond normal Exercise–Induced Levels
– aka. Sympathetic Overtraining Syndrome
Performance Markers of Anaerobic Overtraining
Performance Decrements
Detraining

The Cessation of:
– Anaerobic Training

Or Substantial Reduction in:
– Frequency
– Volume
– Intensity
– or a combo of all 3

Results in:
– Decrements in Performance
– Loss of Physiological Adaptations

Happens ~2 weeks of no training
– Maybe more for Well Trained Athletes

Primary Function of CV System During Aerobic Exercise
Deliver O2 and Nut to muscles
Cardiac Output
The amount of Blood Pumped by the Hear in
– Liters/Min

Q = Stroke Volume x Heart Rate
Stroke Volume
Quantity of Blood Ejected with Each Beat
Heart Rate
Hearts Rate of Pumping
Max Heart Rate Estimation
220–Age
Fick Equation
Q (Cardiac Output) = VO2 (Oxygen Consumption) / (Ca – Cv) (Venous Return)
Rate Pressure Product (Equation)
HR+BP = Rate Pressure Produce = Double Product

HR + BP = the Work of the Heart
Oxygen Uptake (Equation)
Figured by:
– Fick Equation

Expresses Relationship Between:
– Cardiac Output
– Oxygen Uptake
– Arteriovenous Oxygen Difference
Maximal Oxygen Uptake
The Greatest Amount of Oxygen that:
– Can be used at the Cellular Level for the Entire Body

– Correlation is accepted as Measurement of Cardiorespiratory Fitness
Diastolic Blood Pressure
Used to estimate the:
– Pressure exerted against the arterial walls when
– No Blood is being forcefully ejected through the walls
– AKA Diastole
Systolic Blood Pressure
Estimates the Pressure Exerted against the:
– Arterial Walls as Blood is Forcefully Ejected during the Ventricular Contraction
– aka Systole
Mean Arterial Pressure (Definition/Equation)
The average blood pressure throughout the Cardiac Cycle

Mean Art. BP = (SBP–DBP/3) + DBP
Total Peripheral Resistance
The resistance of the Entire Systematic Circulation

ion = Increased Resistance

vasodilation = Decreased resistance
Adaptation to Acute Aerobic Exercises
Increased:
– Cardiac Output
– Stroke Volume
– HR
– Vo2
– SBP
– Blood Flow to active muscles
– Decrease in DBP
Minute Ventilation
The Volume of Air Breathed in a Minute
Tidal Volume
The Amount of Air Inhaled and Exhaled with each breath
Ventilatory Equivalent
The ratio of:
– Minute Ventilation to
– Oxygen Uptake

Ranges between:
20–25L of Air/liters of O2 consumed
Physiological Dead Space
The Alveoli in which Poor:
– Blood Flow
– Ventilation
– Other problems with Alveolar Surface

Impair Gas Exchange
Aerobic Gas Exchange Process
Large Amounts of O2 Diffuse from:
– Capillaries to Tissues

Increased levels of CO2 move from:
– Blood to Alveoli

Minute Ventilation Increases to Maintain Appropriate Alveolar Concentrations of these Gases
Diffusion
The Movement of O2 and CO2 Across
– Cell Membrane

Is a function of the Concentration of Each Gas

Molecular motion is determined by Partial Pressure
Aerobic Training Adaptions
Increased:
– Max Cardiac Output
– Increased Stroke Volume

Reduced:
– Heart Rate at Rest/submax exercise
Vasoconstriction
Narrowing of Blood Vessels as a result of:
– Contraction of the Muscular wall of the vessel
Vasodilation
Widening of Blood Vessels as a Result of:
– Relaxation of the Muscular wall of the Vessel
Venous Return
The amount of Blood Returning to the Heart
Ventilatory Equivalent
The Ration of:
– Minute Ventilation to
– Oxygen Uptake
Alveoli
The functional unit of the Pulmonary System

Where gas exchange occurs
Anatomical Dead Space
During Inspiration:
– Air also Occupies Areas of Respiration:
– Nose
– Mouth
– Trachea
– Bronchi
– Bronchioles

Areas of no Gas Exchange
Arteriovenous Oxygen Difference
The Difference in:
– Oxygen Content

Between:
– Arterial and Venous Blood
Blood Doping
The Practice of Artificially Increasing:
– Red Blood Cell Mass
Bradycardia
Fewer than 60 bpm
Ejection Fraction
The Fraction of the:
– End Diastolic Volume Ejected from the Heart
End–Diastolic Volume
The Volume of Blood Available to be Pumped by the:
– Left Ventricle
– At the the End of the Diastole (filling phase)
Frank–Starling Mechanism
The Force of the Contraction is a:
– Function of the Length of the Fibers of the Muscle Wall
Hyperoxic Breathing
Breathing Oxygen–Enriched Gas Mixtures
Hyperventilation
Increase in Pulmonary Ventilation
Metabolic Equivalent of Tasks
3.5 ml of O2/KG/BW

The ability of the Heart/Circulatory to Transport Oxygen, and the Body tissues t use it
Myoglobin
The protein that transports Oxygen within the Cell
Detraining
Succeeds Aerobic Inactivity

Most sensitive detraining happens in:
– The Aerobic Enzyme Activity
– Revers to Normal, Untrained State
Overtraining (Aerobic)
Extreme levels of:
– Frequency
– Volume
– Intensity
– Combo of above

Rest, to recover
– Even longer period with Aerobic Athletes
Overtraining Syndrome (Aerobic)
Performance Decrements

Low Body Weight

Low Body Fat
Overreaching (Aerobic)

Same as Overtraining,
– But Symptoms only last a few days

Resistance Exercise
Specialized Method of Conditioning that Involves:
– The Progressive use of Resistance to
– Increase one's Ability to Exert Force
Preadolescence
The Period of Life Before:
– The Development of Secondary Sex Characteristics
Adolescence
Refers to the Period:
– Between Childhood and Adulthood
Growth
Increase in Body Size or a Particular Body Part
Development
The Natural Progression from:
– Prenatal Life to Adulthood
Maturation
The Process of Becoming:
– Mature
– Fully Functional
Puberty
Period of Time in Which:
– Secondary Sex Characteristics Develop and a Child is Transformed
– Into a Young Adult
Chronological Age
Development by Age in:
– Months or
– Years
Biological Age
Development of:
– Maturation
– Pubertal Development

Measured by:
– Skeletal Age
– Somatic (physique) Maturity
– Sexual Maturity
Menarche
The onset of Menstruation in Girls
Training Age
Length of Time the:
– Child has been
– Resistance Training
Peak Height Velocity
Pubertal Growth Spurt
– May increase the Risk of Injury in Adolescents
– S&C Pro: Focus on balance and correction
Diaphysis
Central Shaft of a Long Bone
– Where Bone Formation Occurs
Growth Cartilage
Located at Three Sites in a Child
– Epiphyseal (Growth) Plate
– Joint Surface
– Apophyseal Insertions of Muscle–Tendon Units

Damage to These May Impair Growth and Development in Affected Bone
Important Factor of Strength Expression in Children
Most comes from the:
– Development of their Nervous System
– Myelination of Nerve Fibers
Experts on Child Resistance Training
Safe and Effective Method of Conditioning in Children
– Under a Qualified and Competent S&C Pro
– Children aren't Miniature Adults
Benefits of Resistance Training in Children
May Favorably Alter:
– Selected Anatomic/Psychosocial Parameters
– Reduce Injuries in Sport/Rec
– Improve Motor Skills
– Improve Sports Performance
Osteoporosis
Clinical Condition:
– Characterized by Low Bone Mass and
– Increased Susceptibility to Fractures
Absolute vs. Relative Strength (Men vs. Women)
Absolute: Women have about 2/3 the Strength of Men

Relative: Equal in Lower Body, Slightly Less in Upper Body
Women Risk Factors in Knee Joint (Other Joint) Injuries
Possible that:
– Joint Laxity
– Limb Alignment
– Notch Dimensions
– Ligament Size
– Body Movement
– Shoe–Surface Interaction
– Skill Level
– Hormonal Changes
– Training Deficiencies

Reason for the Difference in Knee Injuries
Most ACL Injuries Happen in Women at:
Non–Contact Mechanisms:
– Deceleration
– Lateral Pivoting
– Landing
Osteopenia
Bone Mineral Density Between:
–1 and –2.5 from normal Young Adult

>–2.5 is Osteoporosis
Sarcopenia

Loss of Muscle Mass from:
– Inactivity
– Disease
– Loss in Type II Muscle Fibers

*Focus on Power with Older People

Ideal Performance State
Ultimate Goal of an Athlete

Marked by:
– Psychological
– Physiological
Efficiency

Efficiency = Only employing the amount of:
– Psychic and Physical energy required to perform a Task
Athlete
Someone who Engages in a Social Comparison (Competition) involving:
– Psychomotor Skill
– Physical Prowess
– Or Both

In an Institutionalized Setting

Typically under:
– Public Scrutiny
– or Evaluation
Sport Psychology
Subdiscipline of Exercise Science that seeks to Understand the:
– Influence of Behavioral Processes on
– Skilled Movement
Three Major Goals of Sports Psychology
1. Measuring Psychological Phenomena

2. Investigating the Relationships between:
– Psychological Variables
– Performance

3. Applying Theoretical Knowledge to:
– Improve Athletic Performance
Anxiety (State Anxiety)
A Subjective Experience of:
– Apprehension and Uncertainty

Accompanied by:
– Elevated Autonomic
– Voluntary
– Neural Outflow
– Increased Endocrine Activity

Experience
Trait Anxiety
A Personality Variable or Disposition Relating to:
– The Probability that One will Perceive an Environment as Threatening

Characteristic
Arousal
Simply the Intensity Dimension of:
– Behavior Physiology
"Psyched–Up" Athlete
Psychological Arousal

Athlete May Experience:
– Tremendous Mental Activation

Characterized by:
– Positive Thoughts
– Strong Sense of Control
– i.e. Psychic Energy
Anxiety and Athletic Performance
Causes doubt by:
– High Degree of Ego Involvement
– Athlete May Perceive a Threat to Self–Esteem

– Perceived Discrepancy between Ones:
– Ability vs. Demands for Athletic Success

– Fear of the:
– Consequences of Failure (i.e. Loss of Approval from: teammates, coach, family, or peers)
Cognitive Anxiety
Relates to
– Psychological Processes
– Worrisome Thoughts
Somatic Anxiety
Relates to such Physical Symptoms as:
– Tense Muscles
– Tachycardia
– Butterflies
Stress
Considered any Disruption from:
– Homeostasis or
– Mental/Physical Calm
Stressor
Environmental and/or Cognitive that:
– Precipitates Stress (i.e. Stress Response)
Types of Stress
Distress (Negative) = Comprises Cognitive and Somatic Anxiety

Eustress (Positive) = Comprises Psychic Energy and Physiological Arousal
Attention
The Processing of Both:
– Environmental and
– Internal Cues that
– Come to Awareness
Selective Attention
The Ability to Inhibit Awareness of some Stimuli in order to Process Others

Suppresses:
– Task–Irrelvant Cues (i.e. people on sidelines)
– In order to Process Task–Relevant Cues

Athlete's Focus
Preparatory Routine
To Deal with Anxiety and Attentional Challenge by:
– Adopting a Ritual or Mental Checklist

Consciously Directs thought to:
– Task–Relevant and Controllable Concerns
Cue Utilization

Theory Explains the Effect of Stress or Increased Levels of Physiological Arousal on:
– Attentional Processing of Information