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43 Cards in this Set

  • Front
  • Back
Small, Weak Pulse
Characteristics
Diminished pulse pressure
Weak and small on palpation
Slow upstroke
Prolonged systolic peak
Small, Weak Pulse
Causes
Conditions causing a decreased stroke volume
heart failure
hypovolemia
severe aortic stenosis
conditions causing increased peripheral resistance
hypothermia
severe congestive heart failure
Large, Bounding Pulse
Characteristics
Increased pulse pressure
strong and bounding on palpation
rapid rise and fall with a brief systolic peak
Large, Bounding Pulse
Causes
Conditions causing an increased stroke volume or decreased peripheral resistance
fever
anemia
hyperthyroidism
aortic regurgitation
patent ductus arteriosus
conditions resulting in increased stroke volume due to decreased heart rate
bradycardia
complete heart block
conditions resulting in decreased compliance of the aortic walls
aging
artherosclerosis
Bisferiens Pulse
Characteristics
Double systolic peak
Bisferiens Pulse
Causes
Pure aortic regurgitation
Combined aortic stenosis and regurgitation
Hypertrophic cardiomyopathy
Pulsus Alternans
Characteristics
Regular rhythm
chagnes in amplitude (or strength) from beat to beat (you may need a sphygmomanometer to detect the difference)
Pulsus Alternans
Causes
left ventricular failure (usually accompanied by an S3 sound on the left)
Bigeminal Pulse
Characteristics
regular, irregular rhythm (one normal beat followed by a premature contraction)
alternates in amplitude (one strong pulse followed by a quick, weaker one)
Paradoxical Pulse
Characteristics
Palpable decrease in pulse amplitude on quiet inspiration
pulse becomes stronger with expiration
you may need a sphygmomanometer to detect the change (the systolic pressure will decrease by more than 10 mmHg during inspiration
Paradoxical Pulse
Causes
Pericardial tamponade
Constrictive pericarditis
Obstructive lung disease
Ventricular Impulses
Lift
diffuse lifting left during systole at left sternal border
associated with right ventricular hypertrophy caused by pulmonic valve disease, pulmonic hypertension, and chronic lung disease
may see retraction at apex from posterior rotation of left ventricle caused by oversized right ventricle
Ventricular Impulses
Thrill
palpated over 2nd and 3rd intercostal space
may indicated severe aortic stenosis and systemic hypertension
if palpated over 2nd and 3rd left intercostal spaces indicates pulmonic stenosis and pulmonic hypertension
Ventricular Impulses
Accentuated Apical Impulse
sign of pressure overload
increased force and duration
not usually displaced in left ventricular hypertrophy w/o dilation associated with aortic stenosis or systemic hypertension
Ventricular Impulses
Laterally Displaced Apical Impulse
sign of volume overload
found over a wider area
result of ventricular hypertrophy and dilatation associated with mitral regurg, aortic regurg, or left to right shunts
Abnormal Heart Rhythms
Premature Atrial or Junctional Contractions
beats occur earlier than the next expected beat and are followed by a pause
rhythm resumes with next beat
early beat has S1 of different intensity and a diminished S2
Abnormal Heart Rhythms
Premature Ventricular Contractions
beats occur earlier that the next expected beat and are followed by a pulse
rhythm resumes with next beat
early beat has an S1 of different intensity and a diminished S2
Abnormal Heart Rhythms
Sinus Arrhythmia
dysrhythmia
heart rate speeds up and slows down in a cycle
usually becomes faster with inhalation and slower with expiration
S1 and S2 sound are usually normal
S1 may vary with the heart rate
Abnormal Heart Rhythms
Afib and Atrial flutter w/ varying ventricular response
dysrhythmia
ventricular contraction occurs irregularly
short runs of the irregular rhythm may appear regularly
S1 varies in intensity
Extra Heart Sounds During Systole
high-frequency sounds heard just after S1 (ejection clicks)
produced by a functioning but diseased valve
can occur early or mid-to-late systole
best heard through diaphragm
Extra Heart Sounds During Systole
Aortic Ejection Click
heard during early systole at the 2nd right intercostal space and apex
click occurs with opening of aortic valve and doesn't change with respiration
Pulmonic Ejection Click
Best heard at the 2nd left intercostal space during early systole
click often becomes softer with inspiration
Midsystolic Click
heard in middle or late systole
heard over mitral or apical area
result of mitral valve leaflet prolapse during left ventricular emptying
late systolic murmur typicall follows indicating mild mitral regurg
Extra Heart Sounds During Diastole
Opening Snap
early diastole
heard with opening of stenotic or stiff mitral valve
does not vary w/ respirations
often mistaken for split S2 or S3
occurs earlier in diastole than S3 and has a higher pitch
Extra Heart Sounds During Diastole
S3
Ventricular Gallop
low frequency
heard best with bell at apical area or lower right ventricular area w/ pt. in left lateral position
accentuated during inspiration
rhythm of "Ken-tuc-ky"
result of vibrations caused by blood hitting ventricular wall during rapid filling
normally found in young children, ppl w/ high CO, and in 3rd trimester
rarely normal for ppl over 40
associated with decreased myocardial contractility, myocardial failure, congestive heart failure, and volume overload of ventricl from vascular disease
Extra Heart Sounds During Diastole
S4
Artial Gallop
low-frequency occurring at end of diastole when atria contract
caused by vibrations from blood flowing rapidly into the ventricles after atrial contraction
"Ten-nes-see"
may increased with inspiration
best heard with bell over apical area w/ pt. in supine or left lateral position
never heard in absence of atrial contraction
can be normal for athletes and some older pts.
associated with coronary artery disease, hypotension, aortic and pulmonic stenosis, and acute myocardial infarction
Extra Heart Sounds During Diastole
Summation Gallop
simultaneous occurrence of S3 and S4
caused by rapid heart rates in which diastolic filling is shortened
associated with severe congestive heart disease
Extra Heart Sounds in Both Systole and Diastole
Pericardial Friction Rub
heart best in left 3rd intercostal space
caused by inflammation of pericaridal sac
high pitched, scratchy, scaping sound
rub may increased with exhalation and when pt. leans forward
use diaphragm, w/pt. leaning forward, exhale and hold breath
usually heard 1st week after MI
Extra Heart Sounds in Both Systole and Diastole
Patent Ductus Arteriosus
PDA
congenital anomaly
leaves open channel between aorta and pulmonary artery
found over 2nd left intercostal space and may radiate to left clavicle
continuous murmur
medium pitch
harsh machinery sound
loudest late in systole
Extra Heart Sound in Both Systole
Venous Hum
Common in children
benign sound cause by turbulent blood in jugular vein
heard about medial 3rd of clavicles (right)
may radiate to 1st and 2nd intercostal spaces
low pitch
humming or roaring continuous murmur
loudest in diastole
obliterated with pressure on jugular vein
Midsystolic murmurs
most common type
occur during ventricular ejection
can be innocent, physiologic, or pathologic
crescendo-decrescendo shape
peak near midsystole and stope b4 S2
Midsystolic murmurs
Innocent Murmur
no physical abnormality
occur when ejection of blood in aorta is turbulent
very common in children and young adults
older ppl w/ CVD
pt may have another murmur as well
2nd to 4th left ICS between LSB and apex
little radiation
grade 1 or 2
medium pitch
variable quality
usually disappear when pt sits
Midsystolic Murmurs
Physiologic Murmur
caused by temporary increase in blood flow
occur with anemia, pregnancy, fever, and hyperthyroidsim
2nd to 4th ICS between LSB and apex
little radiation
grade 1 or 2
medium pitch
harsh quality
Midsystolic Murmurs
Pulmonic stenosis
pathologic
occurs from impeded blood flow across pulmonic valve and increased RV afterload
commonly found in children
atrial septal defect may mimic
if severe, S2 is widely split and P2 is diminished
early pulmonic ejection is common
right-sided S4 present
RV impulse often strongere and prolonged
2nd to 3rd ICS
radiates toward left shoulder and neck
soft to loud intensity
medium pitch
harsh quality
loudest during inspiration
Midsystolic Murmurs
Aortic Stenosis
impedes blood flow across valve and increase LV afterload
result from rheumatic disease or a degenerative process
mimics include aortic sclerosis, bicuspid aortic valve, dilated aorta, aortic regurg
if severe, A2 may be delayed resulting in an unsplit S2 or paradoxical split S2
S4 may occur due to decreased left ventricular compliance
aortic ejection suggests congenital cause
right 2nd ICS
radiates from neck down to LSB to apex
loud with thrill
medium pitch
harsh but musical at apex
heard best with pt sitting and leaning forward
loudest during expiration
Midsystolic Murmur
Hypertrophic Cardiomyopathy
cause by rapid ejection of blood from LV during systole
massive hypertrophy of ventricle muscle
coexisting obstruction to flow
mital regurg may result
pt may have S3 and S4
3rd and 4th left ICS
decreases w/ squatting
increases w/ straining down
variable intensity
medium pitch
harsh quality
Pansystolic Murmurs
occurs when blood flows from a chamber w/ high pressure to a chamber of low pressure through a orfice normally closed
pathologic
begin with S1 and continue through systole to S2
Pansystolic Murmurs
Mitral Regurg
occurs when mitral valve fails to close fully in systole
result of blood flowing from LV back into LA
volume overload occurs in LV causing dilation and hypertrophy
S1 sound is decreased
apical impulse is stronger and prolonged
LV volume overload suspected if S3 heard
Apex
radiates to left axilla less often LSB
soft to loud w/ apical thrill assoc. w/ loud
medium to high pitch
blowing quality
heard best w/ pt in LL decubitus position
doesn't become louder w/ inspiration
Pansystolic Murmurs
Tricuspic Regurg
blood flowing from RV back to RA over a tricuspid valve not fully closed
RV failure is most common cause resulting from pulmonary hypertension or LV failure
RV impulse is stronger and prolonged
may be S3 along LLSB and jugular venous pressure is often elevated with visible v waves
LLSB
radiates to R of sternum, to xiphoid area, and to MCL, no radiation to axilla
Variable intensity
medium to high pitch
blowing quality
may increase slightly with inspiration
Pansystolic Murmurs
Ventricular Septal Defect
congenital abnormality
blood flows from LV into RV through hole in septum
causes loud murmur that obscures A2 sound
3rd, 4th, and 5th ICS
wide radiation
very loud intensity with thrill
high pitch
harsh quality
increases with exercise
Diastolic murmurs
indicative of heart disease
two types:
early decrescendo (flow through incompetent SL valve (aortic is common)
rumbling indicate valve stenosis (usually mitral)
Diastolic murmurs
Aortic Regurg
occurs when leaflets of aortic valve fail to close completely
result of blood flowing from aorta back into LV
results in LV volume overload
ejection sound may be present
severe regurg suspected if S3 and S4 present
apical impulse becomes displaced downward and laterally with a widened diameter and increased duration
pulse pressure increases, arterial pulses large and bounding
2nd to 4th ICS
radiate to apex or LSB
Grade 1 to 3
high pitch
blowing, sometimes mistaken for breath sounds
heard best with pt. sitting, leaning frwd
have pt exhale then hold breath
Diastolic murmurs
Mitral Stenosis
result of blood flow across disaeased mitral valve
thickened, stiff distorted leaflets result of rheumatic fever
loud during mid-diastole as ventricle rapidly fills, then grows quiet, and become loud again immediately b4 systole as atria contract
pt w/ Afib the second sound is absent because no atrial contraction
Loud S1 palpable at apex
often OS after S2
P2 becomes loud and the RV impulse becomes palpable if pulmonary hypertension develops
apex
little or no radiation
grade 1 to 4
low pitch
rumbling
best heard with bell exactly on apex w/ pt in LLP
mild exercise and listening during exhalation makes easier to hear