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14 Cards in this Set
- Front
- Back
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Route of SVC blood and IVC blood |
Svc-ra-rv-ductus arteriosus-desc aorta Ivc-foramen o vale- la- lv-ascending aorta-brain |
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Ducts arteriosus how is it kept open |
Mediated by hypoxia so pilm arteria vasoconstrict Also maintained by PGE2 |
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Reason for DA closure |
First breath, increase in BP and decrease in pulm prrsuure Closure due to increase in 02 content from breathibg |
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2,3 DPG |
Intermediate in glucose metabolism Changes haemoglobin from tense to weak stage Weakens haemoglobin affinity for 02 In chronic hypoxia used to advertise to increase DPG levels |
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Sickle cell |
Valine for glutamate in position At Low 02 concerned distortion is worse |
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Haemoglobin breakdown |
Broken down into haem and globin Heam- iron and bilirubin Excessive breakdown/impaired excretion-jaundice |
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Labour prostaglandins |
PGF2a-Indirectly through oxytocin promotes contrax Short lived and local hormone From arachiodonic acid PGE2- Relaxes cervix |
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Contrax |
Compresses útero placental vessels so reduced flow Stretch receptors activate reflex to abd muscl contact in unison |
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Mill ejection reflex |
From suckling baby- hypothalamus stimulates release of oxy from pp- myoepithelial contrax Prolactin secretion also stimulated by suckling |
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Surfactant |
Secreted by type 2 alveolar cells Stim by **cortisol**, prolactin and thyroxine Defic treated by continuous airway (CPAP) |
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Placenta issues |
Previa- painless, bleeding Abruption- painful, concealed/leaking blood, lifethreatening Prolapsed cord- membrane rupture Preeclampsia |
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Preeclampsia |
Blood pressure higher than 120/140? Protein in urine Placenta perfusion inadequate Seen as shock Mat: hyperten, fluid retention,convulsions Fet: coagulopathy, renal damage, liver damage |
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Anti d |
Routinely given at 28/34 weeks Within 72 hrs postpart if baby is rh+ (igG) ABO antibodies too big to cross placenta (igm) |
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Fetal cloack |
Phosphocholine |
