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45 Cards in this Set

  • Front
  • Back
5 Types of Arrhythmias
Irregular Rhythms
Escape
Premature Beats
Tachy-arrythmias
3 Types of Irregular Rhythms
Wandering Pacemaker
Multifocal Atrial Tachycardia
Atrial Fibrilation
Wandering Pacemaker
Paced by SA Node and other atrial automaticity foci
Irregularly Irregular
Different shaped P waves
Normal Rate
Multifocal Atrial Tachycardia
Same as wandering pacemaker but tachy-arrhythmia because atrial foci have entrance block (not overdrive suppressed).
COPD, digitalis toxicity
Atrial Fibrilation
Rapid firing multiple atrial automaticity foci
No impulse depolarizes atria completely (no P waves).
Irregularly Irregular
May be fast or slow ventricular rate
Escape Rhythms / Beats
SA node stops pacing for a beat or all together and another foci starts pacing
Atrial Escape Rhythm / Beat
Junctional Escape Rhythm / Beat
Ventricular Escape Rhythm / Beat
Atrial Escape Rhythm
Rate: 60-80
P waves not identical to SA P waves
May see an arrest of SA pacing followed by escape rhythm
Junctional Escape Rhythm
Rate: 40-60 (may become accelerated)
Failure of SA node and atrial foci OR complete AV conduction block
No P waves but may get retrograde depolarization leading to inverted P waves before, after or within QRS complex.
Ventricular Escape Rhythm
Rate: 20-40 (may become accelerated)
Complete AV conduction block or downward displacement of pacemaker
Leads to Stokes-Adams Syndrome
Ventricular escape beat may occur with burst of parasympathetic activity as blocks all other foci
Premature Beats
Premature Atrial Beat
Premature Junctional Beat
Premature Ventricular Beat
Cause of Atrial and Junctional Premature Beats
Adrenaline
Increased sympathetic stimulation
Caffeine, amphetamines, cocaine, Beta 1 receptor agonists
Digitalis, ethanol
Hyperthyroidism
Premature Atrial Beat
Early P prime wave that resets SA node so next cycle is at regular interval.
Abberant condution: early depolarization down one bundle branch causing widened QRS for that cycle.
Non-Conducted PAB: AV node still repolarizing so no QRS but resets SA node for next cycle. Looks like a block because missing QRS.
Atrial Bigeminy/Trigeminy
PAB coupled to the end of a normal cycle leading to two successive QRS depolarizations with a stretch in between.
Trigeminy: PAB after every second QRS.
Premature Junctional Beat
Premature irritable focus from AV junction.
Aberrant Ventricular Conduction: widened QRS if one BBB depolarizes before the other
Retrograde Atrial Depolarization: inverted P wave before, during or after. This will reset SA node pacing.
(AV) Junctional Bigeminy / Trigeminy
Premature junctional beat coupled with normal cycle or after every second normal cycle. May see retrograde P waves.
Causes of Irritable Ventricular Focus
Hypoxia: airway obstruction, air with poor O2, minimal blood oxygenation (PE, pneumothorax), reduced CO, poor coronary blood supply
Hyokalemia
Pathology: mitral valve prolapse, myocarditis
Premature Ventricular Contraction
Produces a Premature Ventricular Complex (PVC)
QRS: wide, large amplitude, inverse
Pause afterwards caused by repolarization of ventricles, not resetting of SA node
>6/min = pathological
>2 in a row = V tach, if >30seconds = sustained V tach
Ventricular Bigeminy / Trigeminy
Quickly exceeds 6 PVCs/min so hypoxia likely
Ventricular Parasystole
Ventricular automaticity focus with entrance block.
Paces at its own rate regardless of SA node pacing.
R on T
PVC falls on T wave which can cause dangerous rhythm
Tachyarrhythia Rate
150-250: Paroxysmal Tachycardia
250-350: Flutter
350-450: Fibrillation
Paroxysmal Tachycardia
Types: Atrial, Junctional, Ventricular
Irritable focus suddenly paces rapidly 150-250
Paroxysmal Atrial Tachycardia with AV block
150-250
Spiked P prime waves
2:1 P:QRS
Digitalis Toxicity (excites atrial foci but causes depression of AV node)
Paroxysmal Junctional Tachycardia
150-250
May see retrograde P prime waves
May have aberrant ventricular conduction wtih widened QRS
AVNRT - AV nodal re-entry
Ventricular Tachycardia
150-250
SA still paces atria but hidden by huge QRS
Looks like consecutive PVCs
Usually from hypoxia
SVT with aberancy vs. V tach
V tach: coronary artery disease, QRS > .14s, AV dissociation, extreme R axis deviation
Torsades de Pointes
From long QT segment
1. hypokalemia
2. Long QT syndrome
Atrial Flutter
250-350
Saw tooth pattern
2:1 or 3:1 P:QRS
Vagal maneuvers slow AV node so possible to see difference between P and QRS
Ventricular Flutter
250-350 - smooth sine waves
Usually decompensates into v fib which requires defib
No good CO
Fibrillation
Multiple irritable foci.
Parasystolic: no overdrive suppression
Atrial: irregular ventricular rhythm; QRS rate depends on AV refractoriness
Ventricular: type of cardiac arrest; defibrillation required
Wolff-Parkinson-White Syndrome
Bundle of Kent accessory pathway conducts before normal delay through AV node producing pre-excitation and delta waves. Shortened PR interval.
May get paroxysmal tachycardia in 3 ways
1. rapid conduction: supraventricular tachycardia conducted 1:1 causing high ventricular rate
2. may contain automaticity foci that initiate tachycardia
3. Re-entry: ventricular depolarization may restimulate atria retrograde causing re-entry loop.
Lown-Ganong-Levine Syndrome
AV node bypassed by extension of Anterior Internodal Tract (James tract).
Conducts tachy atrial rate 1:1 to ventricles
P adjacent to QRS (no PR interval)
Sinus Block
SA node does not pace for one cycle. May start next cycle or may be an escape beat in between.
Sick Sinus Syndrome
SA node dysfunction with unresponsive atrial and junctional automaticity foci so no escape beats.
Sinus bradycardia
Bradycardia-Tachycardia Syndrome: Sinus brady with intermittent SVT
1st Degree AV Block
Prolonged PR interval > .2s (1 large square) - measured from beginning of P wave to start of QRS.
Seen in every cycle the same amount.
Second Degree AV Block
Wenkebach (Type I): progressive PR interval lenghtening until 1 P wave is not transmitted. Consistent P:QRS ratio (3:2, 4:3, 5:4). Increased parasympathetic activity (not harmful). Originates in AV node so vagal maneuvers increase P:QRS ratio.
Mobitz (type II): several P waves that don't conduct followed by normal cycle with consistent P:QRS ratio (3:1, 4:1). Pathological. May see widened QRS (originates in His Bundle).
Third Degree Heart Block
Independent atrial and ventricular rates (AV dissociation)
Block high in AV node - junctional escape rhythm (40-60).
Block lower - Ventricular escape rhythm (20-40).
May lead to Stokes Adams Syndrome (not enough CO).
Bundle Branch Block
1. wide QRS >.12s (3 small boxess)
2. Check V1/V2 (RBBB), V4/V5 (LBB) for R/R prime
Axis (normal and abnormal causes)
0 to 90
1. Body habitus: R = obese, L = slim
2. Hypertrophy: toward
3. Infarction: away
Lateral Leads
Inferior Leads
1. I, AVL
2. II, III, AVF
Axis
Left Axis Deviation in Degrees
Left Axis Deviation in Degrees
Right Axis Deviation in Degrees
+ / - QRS in chest leads
1. negative
2. negative - through AV node and over anterior and posterior LV wall
3. isoelectric
4. slightly positive
5/6. positive
Heart rotates in horizontal plane toward hypertrophy and away from infarct.