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27 Cards in this Set

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Indicators of Severe Asthma Attack
Normal PCO2 levels (should be depressed from hyperventilation)
Speech diffiuclty, Diarphoresis, AMS, Cyanosis & SIlent Lungs
Restrictive Lung Dzs: Mechanical vs Ts:
Increased Functional Reserve Capacity in Mechanical
Mechanical --> Ankylosing Spondylitis
Why is excess O2 bad for COPD:
blunted hypoxic drive
oxygenated hemoglobin has higher CO2 affinity (Haldante effect --really?)
causes pulmonary vasodilation --> increased dead space ventilation --> worsens V/P mismatch
O2 Therapy & COPD
Oxygen improves COPD lifespan
INdications
PaO2 <55, SaO2 <88, Hematocrit >55%
even more beneficial than vaccines apparently
pt with cirrhosis has pleual effusion
think hepatic hydrothorax = loss of albumin
salt restriction & diruetics --> TIPS
best way to follow pt with Guillian Barre
is through serial measurements of vital capacity (as much as you can suck in to as much as you can blow out)
Tx: IVIG & Plasmapheresis
CSF reveals elevated protein without elevated cellularity or change in glucose
progressive dyspnea, dry cough & "Velcro-like" inspiratory crackles-->
ideiopathic pulm fibrosis
Tx for ARDS:
Low Tidal Volumes ~6ml/kg (so normally ~400mL) (prevents barotrauma) +
& PEEP up to 15 mmHg to keep alveoli from collapsing
Diagnostic Criteria: ARDs
Acute onset, patchy bilateral arispace dz (CXR), PCWP <18, or no clinical evidence of LVEDP, PaO2/FiO2 <200
inspiration vs expiration in those pulmonary flow graphs
inspiration is on top expiration is on bottom
differentiating COPD from asthma
bronchodilator challenge increases FEV1 in asthma but not COPD
Differentiating types of restrictive diseases
Diffusion of CO:
Normal: chest wall weakness (neuromuscular)
decreased: interstitial lung disease
bradycardia, AV block hypotension, diffuse wheezing
--> beta blocker OD
Tx: glucagon
glucose in pleural exudate
30-50: lots of thigns incl. malignancy, SLE TB, eosphageal rupture
<30: empyema or rhematic effusion with active WBC's
Hemothorax vs Pneumothorax
Hemo: neck veins collapsed
Pneumo: neck veins engorged
Signs of consolidation:
E-->A, Prominent expiration, dullness to percussion, often crackles
Aspirin Exacerbated Respiratory Dz:
asthma, chronic rhinosinusitis c nasal polyps, bronchospasm or nasal congestion following aspirin or NSAIDs; surgery is palliative, but avoid aspirin and NSAIDs
increase in PCO2 + decrease in PO2
= alveolar hypoventilation
NB most other things which cause a decrease in PO2 would cause an increase in PCo2 from hyperventilation
PAO2 =
(FiO2 x (Patm - PH2O)) - (PaCO2/R)
Cancer inside the Nose
Nasopharyngeal Carcinoma
Extremely linked to EBV
usually metastatic at presentation
recurrent URI, persistent nasal discharge, & bilateral nasal polyps
--> CF
Pt has subcutaneous emphysema
-->knee jerk CXR for Pneumothorax
Pt with COPD treated for pneumonia now has Headaches, insomnia, nausea, vomiting and arrhtymia
Erythromycin or Ciprofloxacin --> altered theophylline metzm --> Theophyllin toxicity
Theophyllin Toxicity: CNS, GI, Cardiac toxicities
Pulmonary Renal Sros:
Goodpasture - circulating anti-glomerular BM abs Tx with Plasmapheresis
Wegeners'
Polyarteritis Nodosum
Idiopathic rapidly proegressive glomerulonephritis
SLE
COPD + infeciosu exacerbation
Even receives corticosteroids as a 2 week taper.
list the light criteria for exudate vs transudate
fluid protein/serum protein ratio >0.5, fluid LDH/serum LDH >0.6, pleural fluid LHD greater than 2/3 ULN of the serum LDH

if exudate: complivated vs uncomplciated
complicated: positve gram stain, positive cx, pH <7,2, glucose <60, & requires chest tube drainage, more likely to progress to empyema/be unresponvie to abx

always drain if pH <7.2 OR glucose <60 = diagnostic for empyema.
high mixed venous o2 saturation with shock
spetic shock (hyperdynamic circulation, inability of
ts to access o2, inproper distribuption of Cardiac Output)