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109 Cards in this Set

  • Front
  • Back
leading cause of disability in the USA
arthritis
functions of the system
-protection
-framework
-mobility
-produces heat
-blood return via vascular massage
-stores immature RBCs
-Ca, phos, Mag, Fluoride storage

*most of body's Ca is in BONE
how many bones in our body
206
4 classifications of bones (by shape)
-long
-short
-flat
-irregular

*the shape depends on F and forces it must support
types of bone tissue
cancellous (trabecular)
-lacunae layered in irregular lattice work
-like most epiphyses

cortical (compact)
-like in most diaphysis
epiphyseal plate
allows for longitudinal growth
-calcified in adults
articular cartilage
tough, elastic, nonvascular tissue
long bones
-made for weight bearing/movement

cancellous (trabecular)
-like most epiphyses

cortical (compact)
-like in most diaphysis
short bones
cancellous bone covered by a layer of compact bone
Flat bones
cancellous layer between compact
osteoblasts
BUILD
bone formation by secreting matrix
matrix
collagen and ground substance that make a framework where minerals (phosphorus and calcium) are deposited
osteocyte
mature bone cells
involved in bone maintenance
located in lacunae(bone matrix unit)
osteoclasts
dissolve and resorb bone

located in Howship's lacunae
(sm pits in the bone)
periosteum
dense,fibrous membrane covering bone
-nourishes bone/facilitates growth
-attachment of tendons/ligaments
endosteum
thin vascular membrane lining the marrow cavity of long bones and spaces in cancelous bone
Red bone marrow
located in long bones b4 puberty

sternum/ ilium/ vertebrae/ ribs
after puberty

site of RBC production AKA hematopoiesis
osteogenesis
bone formation
ossification
process by which bone matrix is formed and hard mineral crystals (STRENGTH) are bound to collagen fibers (RESILIENCE)
bone is a dynamic tissue -explain
constant state of turnover
modeling
bone growth during childhood continues until early 20's
remodeling
simultaneous bone resorption and osteogenesis that maintains structure and function

starts in early 20's
factors that influence bone formation and resorption
-physical activity
-calcitriol (activated vit D)
-PTH
-calcitonin
-thyriod hormone
-growth hormone
-estrogen
-testosterone
-blood supply
calcitriol
-activated vitamin D
-increases amount of Ca absorbed by GI
-helps mineralization of osteoid tissue
-absorbed in duodenum (bypass)
-fortified milk is the best source
osteon
microscopic functional bone unit
-center is Haversian canal (capillary)
-encircled by lamellae (matrix)
paresthesia
abnormal sensation
(like burning,numbness,tingling)
isometric contraction
-increased muscle tension w/out change in length or joint movement

-almost all of energy is released as heat (like shivering)
isotonic contraction
muscle tension unchanged, muscle shortened, joint moved

-some energy is used for movement
major hormonal regulators of Ca homeostasis
PTH and calcitonin
calcitonin
secreted by the thyriod gland when serum Ca is HIGH
-inhibits bone resorption
-increases Ca deposits in bone
-tells kidneys not to keep Ca???
-decreases w/age

Miacalcin
-nasal spray/ IM/ SQ
-not given if SEAFOOD ALLERGY
PTH
-released when serum Ca is LOW
-causes bone deminieralization
-tells kidneys to keep Ca ???
-increases w/age
thyriod hormone
-increased bone resorption and decreased bone formation

-CORTISOL has the same effect

-and since cortisol is a steriod then corticosteriods will also have the same effect
growth hormone
-stimulates LIVER and bone to make insulinlike growth factor-1
AKA IGF-1

-accelerated bone modeling in kids/adolescents

-also stimulates skeletal muscle growth in this age group
estrogen
-stimulates osteoblasts and inhibits osteoclasts= increased bone formation and reduced resorption
testosterone
-skeletal growth in adolescents
-continued effect on skeletal muscle growth

Remember increased muscle mass = greater weight bearing stress on bones=increased bone formation

-also it converts to estrogen in adipose tissue
RANKL
receptor for activated nuclear factor-kappa B ligand =binds to RANK on osteoclast precursurs causing them to mature into ostoeclasts

1) produced by osteoblasts

2)produced by T cells during inflammatory response
OPG
osteoprogerin =blocks the effects on RANKL, turning off bone resorption

-also made by osteoblasts
synarthrosis jionts
immovable -like the skull after maturity
amphiarthrosis joints
-allow limited motion,joined by cartilage

like the vertebra, symphysis pubis
diarthrosis joints
freely movable

include:
-ball and socket (full freedom)
-hinge (bending in 1 direction)
-saddle aka biavial (movement in two planes at right angles to each other as in the thumb)
-pivot (permit rotation) as in radius and ulna
-gliding (limited movement in all directions) as in the carpal bones
joint capsule
-surrounds articulating bones
-lined w/synovium=synovial fluid

so bones DO NOT touch each other
tendons
attach muscle to bone
ligaments
attach bone to bone
bursa
fluid filled sac of sinovial fluid that cushions movement of tendons,ligaments, and bones at point of friction

elbow,shoulder,hip,knee
stages of fracture healing
1) REACTIVE PHASE
-bleeding and hematoma formation
-cytokines released cause fibroblasts to come
-fobroblasts cause angiogenesis
-granulation tissue forms from clot

2) REPARATIVE PHASE
-granulation tissue replaced w/procallus
-fibroblasts invade procallus forming a denser callus of fibrocartilage
-this is replaced by bony callus in 3-4 weeks
-lamellar bone forms as it calcifies

3) REMODELING
-new bone reformed to former structure
-can take months or years
skeletal muscle contraction
-each muscle cell/fiber is made of sarcomeres
-sarcomeres are the contractile unit of skeletal muscle
-sarcomeres contain thick myosin and thin actin filaments
-contract in response to electrical stimulation =cell depolarization and action potential
-action potential=release of Ca ions that are stored in sarcoplamic reticula
-Ca ions cause actin and myosin to cross each other
-the Ca ions are then reabsorbed into the sarcoplasmic reticulum

Ca needed for muscle contaction
energy for muscle contraction
primary source is ATP

1)low levels of activity
-ATP made from oxidation of glucose to H2O and CO2

2) strenuous activity
-glucose metabolized into lactic acid and stored glycogen
-this is not as effective

muscle fatigue d/t depletion of glycogen and accumulation of lactic acid
myoglobin
hemoglobin like protein pigment found in striated (skeletal) muscle that transports O2

RED muscles-have lots of this
-contract slow but w/force
-like respiratory/postural muscles

WHITE muscle-have less of this
-contract quickly -like EOM

most muscle have both
muscle tone
relaxed muscles in a state of readiness to respond
AKA tonus
-produced by some muscle fibers remaining in a contracted state
-reduced in sleep/increased w/anxiety
-muscle spindles =are sense organs that monitor tone
flaccid
limp without tone
spastic
greater than normal tone
atonic
denervated muscle that will atrophy
prime mover

synergists

antagonists
-muscle that causes the movement

-assist the prime mover

-must relax to allow prime mover to contract
hypertrophy
increase in muscle fiber SIZE not number

atrophy is the opposite

isometric exercise helps if immobile
pain assessment
bone=dull,deep ache, "boring"

muscle=sore,ache, cramping

fracture=sharp relieved w/immobilization

increases w/activity= sprain, strain, compartment

steady increase= progression of infetious process/tumor/etc
kyphosis
increased forward curve of thoracic spine
-ab muscles relax
-breathing may be impaired

-often seen in elderly associated w/OP
lordosis
AKA swayback
increased curve of lumbar spine

-often seen during pregnancy
scoliosis
lateral curve to spine

-may be idiopathic or d/t muscle damage
effusion
excessive fluid within capsule
-suspect if joint is swollen and normal bony landmarks are obscured

may use balloon sign or ballottement of knee
fasiculation
involuntary twitching of muscle fiber groups
when measuring muscle size
-measure at greatest width at rest
-document distance from bony landmark
-change greater than 1cm is significant
arthrography
-used to ID tears in capsule,ligaments
-dye or air injected into joint
-passive ROM performed
-X-rays taken
-compression bandage and rest for 12h after
-clicking or crackling normal for next 2-3 days
bone densitometry
-tests for bone density
-uses X-rays or ultrasound

-DXA (dual energy X-ray absorptiometry) is best for predicting hip fracture risk r/t OP so it is the most commonly used
-uses T-scores or standard deviation
bone scan
-radioisotope given IV 2-3h before
-isotopes taken up my metabolicaly active bone tissue
-encourage fluids to clear isotopes
-empty bladder if scanning pelvis
arthroscopy
-direct visualization of joint
-compression wrap, ice, extended and elevated joint
arthrocentesis
-joint aspiration
electromyography
-needle electrodes used to stimulate muscle to differentiate muscle issues from nerve issues

-may use warm compress after
bone GLA protein
AKA serum osteocalcin
-indicates the rate of bone turnover
lab values for increased osteoclast activity
-urinary N-telopeptide of type 1 collagen (N-Tx)
-urinary deoxypyridinoline (Dpd)
lab values for increased osteoblast activity
-serum bone-specific alkaline phosphatase (ALP)
-osteocalcin
-intact N-terminal propeptide of type 1 collagen (P1NP)
casts
1)plaster
-used more in adults
-get cool water for DR
-longer dry time
-wet is dull, sounds "thud"
-dry is shiny,sounds hallow
-CANNOT GET WET

2)fiberglass
-used more in kids
-can get wet but must dry
-get warm water for DR

BOTH
-handle with palms
-leave open to air, avoiding plastic
-will feel warm while drying
-burning over bony prominence may be d/t to pressure ulcer
-exercise any joint not immobilized hourly when awake
-compartment syn,ulcers,disuse
-bivalving=or splitting of cast
neurovascular assessment
Pain
Pallor
Pulse
Paresthesia
Paralysis

q2-4h w/any musculoskelatal injury
nursing pt w/immobilized upper extremity
-when lying down position arm so wrist is higher then elbow, is higher than shoulder

-Volkmann's contracture = type of compartment syndrome will see contracture of the fingers and wrists, unable to/painful to extend fingers, loss of circulation
nursing pt w/immobilized lower extremity
-elevated to heart level and ice packs for 1-2 days
-then elevated when sitting
-should assume a recumbent position several times a day

-Peroneal nerve injury can cause footdrop= can't keep foot in flexed position
nursing pt w/immobilized body or spica cast
-impaired mobility is huge
-turn to uninjured side q2h
-use at LEAST 3 people to turn
-may require a trapeze
-turn to prone position x2/d to provide postural drainage (a sm pillow under belly may help)
-line w/plastic sheets during elimination if not Gore-Tex
-monitor bowel sounds and for cast syndrome q4-8h

-cast syndrome=
psychologically-claustrophobic
physically- GI dysfunction
External Fixator
-used when there is soft tissue damage
-may be replaced by cast after healing
-disuse/immobility less of an issue
-pt may be freaked out by appearance
-PIN CARE is very important
-encourage mobility AS ORDERED
-DO NOT ADJUST THE PINS

Ilizarov=used to lengthen bones and correct other deformities it IS ADJUSTED daily as ORDERED
Traction
-pulling force to a body part
-body/part is countertraction

-straight/running= pulling force in straight line of part like Buck's
-balanced suspension=part is elevated off of bed
-skin traction=no pins
-skeletal traction=PINS
-manual traction=hands
Skin traction
-muscle spasms and immobility b4 sx
-max 4.5-8lbs an extremity
-assess circulation in 10-15min then q1-2h
-encourage exercise q1h WA
Buck's traction
-straight/running skin traction
-leg pulled straight out
-do not turn side to side
-use manual traction to remove boot x3/day to inspect skin
-palpate areas of traction tape
-provide back care q2h
Skeletal traction

pins
-allows more mobility
-uses more wt (15-25 lb)
-inspect weight and pulley system
-NEVER REMOVE WEIGHTS
-DVT is huge risk do calf pump 10xh
-REPORT/ASSESS ANY PAIN ASAP

-pins will be covered 1st 48 hrs
-some redness drainage WNL 1st 72h
-assess q8h
Immobilty complications
-anxiety
-self care
-atelectasis/pneumonia
-constipation/anorexia
-urinary stasis/infection
-DVT
Hip precautions (sx)
-maintain abduction, neutral rotation, and flexion LESS than 90
-no crossing the legs
-hips higher than knee when sitting
-fracture pan
-keep leg extended and pivot w/ transfer
-avoid sitting more than 45min
-followed for about 4 months
continuous passive motion device
basically moves a part for you
-used w/knee replacements/etc
s/s DVT
-unilateral calf swelling
-tenderness/warmth/redness
s/s hypovolemic shock
-increased pulse (100+)
-low B/P (90/60)
-increased pulse pressure (20+)
-decreased Hct/Hgb
-decreased urine output (30/h)
-change in LOC/thirst
Low back pain

herniated nucleus pulposus
-most often L4-L5 and L5-S1
-most will end in 4 weeks
-acute=less than 3 months
-take it easy
-assess pt prone d/t relaxation of paraspinal muscles
-head up w/knees flexed or side w/pillows
-avoid sleeping prone
-avoid high heels,bad posture,lifting
-loose weight

AKA herniated disc =pressure on nerve root
-radiculopathy/sciatica=radiating pain
-postive straight leg test=pain w/raise
-depressed/absent Achilles reflex
carpal tunnel syndrome
-entrapment neuropathy syndrome
-median nerve compressed
-most often d/t repeated movement
-pain,numbness,paresthesia, weakness along median nerve (thumb, index, and middle fingers)
-Tinel's sign=tap on inner aspect of wrist to illicit s/s
-splints
cane usage
COAL

C-ane
O-pposite
A-ffected
L-eg

bad leg and cane move together
cane/crutches and stairs
"Up with the good (foot), Down with the bad"
Ostoeporosis
-primary
-secondary d/t dz or meds
-osteoclasts more than osteoblasts
-bone becomes porous/brittle
-1st s/s may be fracture
-compression fracture most common
-age r/t loss begins after peak(40)
-sm white women at highest risk
-may not see in X-ray until advanced
-DX w/ DXA
daily Ca intake

daily Vit D intake
1000-1200
-split doses in 2

800-1000

NEITHER taken w/bisphosphonates
Osteomalacia
-metabolic bone dz
-bone is not being mineralized
-soft weak bones=fracture/deformity
-may see waddling or limping gait
-primary deficit is lack of calcitriol
-but also renal/liver/vit D/etc issue
-X-ray shows demineralization
-biopsy of osteoid AKA prebone
-handle pt gently support w/pillows
Paget's Disease
AKA osteitis deformans
-localized rapid bone turnover
-skull/long bones/pelvis/spine most
-primary proliferation of osteoclasts
-compensatory increase in osteoblasts
-classic mosiac pattern of growth
-areas of reduced and overgrowth
-insidious
-skull may thicken=hats/loss of hearing/ sm triangular face
-legs bow=waddling gait
-spine bent forward with chin to chest
-thorax compressed=breathing hard
-arms bent out and forward=look long
-pain,tenderness,warmth over bone
-pain with weight bearing
-vascular lesions=cardiac failure
-normal serum Ca levels
-fractures and arthritis common
Osteomyelitis
-infection of bone
-causes inflammation,necrosis,bone growth
-post sx w/in 30 days or 1y after implant
-bone avascular =hard to tx
-50% caused by staph.aureus
-bone abscess forms if not tx
-sequestrum=the abscess
-involucrum=bone around abscess
-s/s of sepsis if blood born
-constant, pulsating pain, worse w/move
-chronic=non healing ulcer that drains

3 types:
-hematogenous=from blood
-contiguos-focus=contamination
-w/vascular insufficiency (DM)
strain

sprain
-pulled muscle or tendon

-pulled ligament/tendon around joint

BOTH
-graded in stages
1) stretching
2) some tear
3)major or complete tear

-avulsion fracture possible
contusion
-soft tissue injury d/t blunt force
-causes rupture of vessels
-takes 1-2 weeks to heal
tx for sprains, strains,contusions
RICE
then heat after 1st 24-48 hrs
dislocation

subluxation
-articular surfaces no linger aligned

-partial dislocation

BOTH
-considered an ER if traumatic
-may result in avascular necrosis
rotator cuff tears

Impingement syndrome= general term to describe ALL lesions that involve the rotator cuff
-rotator cuff is made of 4 muscle and there tendons and stabilizes the humeral head

-supraspinatous
-infraspinatous
-teres minor
-subscapularis

s/s
-night pain unable to sleep on side
-cannot do over-the-head stuff
epicondylitis
-chronic painful condition
-caused by moving forearms too much
-results in tendonitis

-lateral epicondylitis AKA tennis elbow
-medial epicondylitis AKA golfers or pitchers elbow=d/t repeated wrist flexion
lateral and medial collateral ligament injury
foot planted on floor..........
-lateral ligament if struck medially
-medial ligament if struck laterally
ACL and PCL injury
foot planted on floor............

ACL -anterior cruciate ligament if struck forward

PCL-posterior cruciate is struck from behind

s/s
-may feel/hear a "pop"
-suspect if severe swelling in 2h
meniscal injury
may cause leg to "give out"
or "lock up"
Achilles tendon rupture
-attaches soleus and gastrocnemius muscles to the os calcis (the heel)

-suddden contraction of calf muscle w/foot planted may cause it
-will not be able to plantar flex foot
types of fractures (basics)

S/S
-complete=across entire bone
-incomplete=does not go all the way
-comminuted=several fragments
-closed/simple=does not break skin
-open/compund/complex=breaks skin
-intraarticualr=ends of bones and hard to see w/X-ray

-pain=will get worse
-muscle spasms=possible
-loss of function
-deformity
-shortening=esp w/long bones
-crepitus=assessing may cause injury so keep it minimal
-edema and ecchymosis =hrs after
Dupuytren's disease
-slow progression of contracture of palmar fascia
-results in flexion of 4th/5th and sometimes 3rd finger
-looks like making a GUN w/hand
-inherited autosomal dominant
-also arthritis,cigs, alcoholism,DM
Benign bone tumors
-more common than malignant
-symetric controlled growth pattern
-primary tumors=bone destruction

-osteochondroma most common=
starts as projection on end of long bone
-echondroma=tumor of hyaline cartilage
-giant cell tumors(osteoclastoma)= may become malignant
Malignant bone tumors
-primary are RARE
-metastatic is more common=NO TX

-osteosarcoma is most common and most fatal
-chondrosarcomas=from hyaline
complications of bone fractures
EARLY
-shock (often hypovolemic)=

-fat embolism syndrome (FES)=
hypoxia, tachypnea, tachycardia, dyspnea, crackles, wheezes, thick WHITE SPUTUM, edema, PaO2 below 60, x-ray shows SNOW STORM infiltrate, change in LOC, petechia, fever over 103, fat in urine

-compartment syndrome=
pain that increases w/passive ROM not relieved by narcotics or seems way to extreme for injury

-DVT
---------------------------------------
DELAYED
-delayed union=takes longer to heal
-nonunion=ends do not unite
-malunion=do not unite right
-avascular necrosis of bone= loses blood supply and dies
-reaction to internal devices
-complex regional pain syndrome= sympathetic nervous system goes nuts and skin temp and look changes frequently
-hetorotopic ossification= bone forms where it should not including soft tissue/muscle/etc