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60 Cards in this Set

  • Front
  • Back
measuremetn of measure, flow and oxygenation arterial pressure includes: CVP, PAWP, CO/CI, SV, SaO2 SvO2
Hemodynamics
volume pumped by heart in 1 minute
CO
adjusted for body size
CI
volume pumped with eat heartbeat
SV
resistance to flow by the vessels
SVR, PVR
pressuer generated by volume in a a cardiac centricle chamber at end of diastole,
preload
LV end diastolic preload
PAWP
rt ventricle preload
CVP
volume ejected from vent wtih each hreat beat (40-60%)
EF
force opposing vent. ejection SVR + atrial pressures = LV afterload
afterload
increased workload of heart, vasoconstriction outflow obstr (aortic stenosis)
increased afterload
strength of contraction; positive inotropes will increase contractility -depa, dobut, isoproterenol, primecor
negative inotropes will decrease contractility, alcohol, pronestyl, CCB, barbituates
contractility
4 IC at sternum, mid axillary line to level invasive lines
phlebostatic axis
b/p and MAP, radial, femoral, brachial, complications: hemorrhage, infection, thrombus, neruovasc. impairment, flush (heparinized saline), meassure bag inflated-1-3 mL per hour
Art Line
PAD + PAW-indicate fluid volume + cardiac function increased with overload and heart failure, decrease volume deficit
PA Cath
monitor pressures, mixed venous samples
PA port
for CVP, project CO, draw labs
RA port
PAS peak, PAD lowest PA mean-average, end expiration
PAP
inflation; risk rupture of PA
PAW
take mean pressure, waveform similart o PA waveform, if increased RV failure or volume overload, if decreased=hypovolemia
CVP
2.2-4L/min/m2; decreased wtih hypovolemia, cardiogenic shock, heart failure, increased: hyperdynamic, fever,sepsis, thermodilution is continuous-uses heat filamnet, injectate 3X take average
CI
measured when CO measured
increases: vasoconstriction-shock, LV failure, epi
decreased: vasodilation-sepsis, septic shock, neurogenic shock,
SVR
determines adequacy of tissue oxygenation, oxygenation, tissue perfusion and tissue O2 consumpt. 60-80% normal at rest, decreased O@, low CO, low Hgb., increased O2 consumpt. increased improvement sepsis, paralysis
SVO2
infection/sepsis, air embolus, pulm. infarction, PA rupture, vent arrhythmias
PA cath complications
decreased afterload, confirm placement X-ray, systole= balloon decrease, early diastole= blood to coronaries,
late diastole-asssited systole
balloon pump
dislodged plaques, aortic dissection, compromised distal circulation, smashes platelets-thrombocytopenia, leak-helium gas embolus
immoble-HOB decrease 45 degrees
no flexing of legs
ratios: 1:1, 1:2
balloon pump complications
chemical reset, cardioversion, use for sinus tach, uncontrolled rate, will get asystole for 6 sec. then should get rhythm back
adenosine
like a low shock, syncs rhythm with QRS
sync cardioversion
uncontrolled A fib, a flutter
cardiazem
OLD drug for multiple PVCs and other ventricular arrhythmias,
Lidocaine
NEW drug for multiple PVCs and otehr ventriuclar arrhythmias
amiodarone
treats tachy if pt. has good B/P (olols)-metopropolol
beta blockers
treats asystole, V tach, V fib
epinephrine
NEW treatment for asystole, V tach, and V fib. Can give vasopressin after epi
vasopressin
3rd degree heart block
pacemaker
given when patient is in V tach or V fib
shock then epi or vasopressin
fraction of inspired oxygen. range 21% to 100%. in most cases the ____ should be at the lowest setting that will maintain the desired blood oxygen levels
FIO2
set volume of gas that is delivered when the ventilator gives a breath
Tidal Volume
ventilator breath rate control. determines the minimum number of breaths that will be delivered to the patient (usually around 650)
Rate (respiratory rate)
rather than returning to zero at teh end of exhalation a set amount of pressure is maintained. helps to reverse underlying pathophysiologic changes (such as small airway and alveolar collapse) by increasing teh functional residual capacity of splinting the lung in a position of function
PEEP
a ventilator mode in which pressure is applied to a breathing circuit when there is no ventilator delivered tidal volumes being provided
CPAP
ventilator mode in which teh set tidal volume is delivered in resonse to all inspiratory efforts (assist). Or, breaths are delivered at intervals determined by the rate setting if teh patient is apneic (control)
Assist control
mode in which the ventilator delivers a predetermined number of mandatory breaths, adn teh patient is allowd to take spontaneous breaths in between. the mandiotry breathes are "synchronized" with inspiratory efforts
syncrhonized intermittent manditory ventilation (SIMV)
mode of ventilation in which a breath is delivered up to a set amt of pressure rather than a set volume. the time of inspiration, inspiratory flow settings, and the compliance of the lungs affect the amount of volume given.
Pressure control (PC)
an adjunct to venitlation inw hich flow is added to the circuit to decrease the word of breathing during spontaneous breathing. enough flow is provided to raise and maintain the inspiratory pressure of teh PS level until the patient begins to exhale
Pressure Support
the volume or amount of air the machine delivers wtih each ventilator initiated breath also refers to the amount of air we breathe with each spontaneous breath
Tidal Volume
typically set at 10-15 breaths/minute. respiratory rate is adjusted according to teh pCO2 ( and pH) which is the index of ventialtion
respiratory rate
ability of the heart to initiate an impulse spontaneously without an external stimulus
automaticity
the ability of cardiac cells to reach a threshold and resond to a stimulus.
excitability
ability of muscle cells to shorten in response to an electrical stimulus
contractility
when sodium and potassium move otu and calcium moves in. electral excitation of teh cell mbembrane resulting from the flow of ions across the membrane; wave of excitation spreads from cell to cell through the conductionn system
depolarization
recovery stage-return of cell membrane to it's resting state; cells must repolarize before they can depolarize again
repolarization
recovery period after stimulation
refractory period
heart cannot respond to another stimulus regardless of stimulus strength
absolute refractory period
follows absolute refractory period, heart can respond to very strong stimulus, but response will be abnormal . can respond to stimulus if stimulus is really strong
relative refractory period
represents AV conduction time, or the time it takes the impulse to travel through teh atria,through teh AV node, and down to where the ventricles begin to depolarize
measure from beginning of P wave to beginning of QRS
normal:.12-20 seconds
PR interval
ventricular depolarization as the impulse travels through both ventricles. Normal: .04-.10
follows the P wave
QRS complex
ventricular repolarization as ventricles return to resting electrical state; relative refractory period (vulnerable period)
normal: smooth, rounded, upright in most leads
T wave
represents early repolarization phase and extends from end of QRS to beginning of T wave; should be at isoelectice line; myocardium still in absolute refractory period; eleveation or depression represnets mycoardial injury or ischemia
ST segment
time from ventricular depolarization to ventricular repolarization: measrued from beginning of QRS to end of T wave; QT interval varies with heart rate
QT interval