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60 Cards in this Set
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measuremetn of measure, flow and oxygenation arterial pressure includes: CVP, PAWP, CO/CI, SV, SaO2 SvO2
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Hemodynamics
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volume pumped by heart in 1 minute
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CO
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adjusted for body size
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CI
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volume pumped with eat heartbeat
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SV
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resistance to flow by the vessels
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SVR, PVR
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pressuer generated by volume in a a cardiac centricle chamber at end of diastole,
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preload
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LV end diastolic preload
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PAWP
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rt ventricle preload
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CVP
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volume ejected from vent wtih each hreat beat (40-60%)
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EF
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force opposing vent. ejection SVR + atrial pressures = LV afterload
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afterload
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increased workload of heart, vasoconstriction outflow obstr (aortic stenosis)
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increased afterload
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strength of contraction; positive inotropes will increase contractility -depa, dobut, isoproterenol, primecor
negative inotropes will decrease contractility, alcohol, pronestyl, CCB, barbituates |
contractility
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4 IC at sternum, mid axillary line to level invasive lines
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phlebostatic axis
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b/p and MAP, radial, femoral, brachial, complications: hemorrhage, infection, thrombus, neruovasc. impairment, flush (heparinized saline), meassure bag inflated-1-3 mL per hour
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Art Line
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PAD + PAW-indicate fluid volume + cardiac function increased with overload and heart failure, decrease volume deficit
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PA Cath
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monitor pressures, mixed venous samples
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PA port
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for CVP, project CO, draw labs
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RA port
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PAS peak, PAD lowest PA mean-average, end expiration
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PAP
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inflation; risk rupture of PA
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PAW
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take mean pressure, waveform similart o PA waveform, if increased RV failure or volume overload, if decreased=hypovolemia
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CVP
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2.2-4L/min/m2; decreased wtih hypovolemia, cardiogenic shock, heart failure, increased: hyperdynamic, fever,sepsis, thermodilution is continuous-uses heat filamnet, injectate 3X take average
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CI
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measured when CO measured
increases: vasoconstriction-shock, LV failure, epi decreased: vasodilation-sepsis, septic shock, neurogenic shock, |
SVR
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determines adequacy of tissue oxygenation, oxygenation, tissue perfusion and tissue O2 consumpt. 60-80% normal at rest, decreased O@, low CO, low Hgb., increased O2 consumpt. increased improvement sepsis, paralysis
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SVO2
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infection/sepsis, air embolus, pulm. infarction, PA rupture, vent arrhythmias
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PA cath complications
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decreased afterload, confirm placement X-ray, systole= balloon decrease, early diastole= blood to coronaries,
late diastole-asssited systole |
balloon pump
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dislodged plaques, aortic dissection, compromised distal circulation, smashes platelets-thrombocytopenia, leak-helium gas embolus
immoble-HOB decrease 45 degrees no flexing of legs ratios: 1:1, 1:2 |
balloon pump complications
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chemical reset, cardioversion, use for sinus tach, uncontrolled rate, will get asystole for 6 sec. then should get rhythm back
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adenosine
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like a low shock, syncs rhythm with QRS
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sync cardioversion
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uncontrolled A fib, a flutter
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cardiazem
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OLD drug for multiple PVCs and other ventricular arrhythmias,
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Lidocaine
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NEW drug for multiple PVCs and otehr ventriuclar arrhythmias
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amiodarone
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treats tachy if pt. has good B/P (olols)-metopropolol
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beta blockers
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treats asystole, V tach, V fib
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epinephrine
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NEW treatment for asystole, V tach, and V fib. Can give vasopressin after epi
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vasopressin
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3rd degree heart block
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pacemaker
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given when patient is in V tach or V fib
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shock then epi or vasopressin
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fraction of inspired oxygen. range 21% to 100%. in most cases the ____ should be at the lowest setting that will maintain the desired blood oxygen levels
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FIO2
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set volume of gas that is delivered when the ventilator gives a breath
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Tidal Volume
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ventilator breath rate control. determines the minimum number of breaths that will be delivered to the patient (usually around 650)
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Rate (respiratory rate)
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rather than returning to zero at teh end of exhalation a set amount of pressure is maintained. helps to reverse underlying pathophysiologic changes (such as small airway and alveolar collapse) by increasing teh functional residual capacity of splinting the lung in a position of function
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PEEP
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a ventilator mode in which pressure is applied to a breathing circuit when there is no ventilator delivered tidal volumes being provided
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CPAP
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ventilator mode in which teh set tidal volume is delivered in resonse to all inspiratory efforts (assist). Or, breaths are delivered at intervals determined by the rate setting if teh patient is apneic (control)
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Assist control
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mode in which the ventilator delivers a predetermined number of mandatory breaths, adn teh patient is allowd to take spontaneous breaths in between. the mandiotry breathes are "synchronized" with inspiratory efforts
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syncrhonized intermittent manditory ventilation (SIMV)
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mode of ventilation in which a breath is delivered up to a set amt of pressure rather than a set volume. the time of inspiration, inspiratory flow settings, and the compliance of the lungs affect the amount of volume given.
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Pressure control (PC)
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an adjunct to venitlation inw hich flow is added to the circuit to decrease the word of breathing during spontaneous breathing. enough flow is provided to raise and maintain the inspiratory pressure of teh PS level until the patient begins to exhale
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Pressure Support
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the volume or amount of air the machine delivers wtih each ventilator initiated breath also refers to the amount of air we breathe with each spontaneous breath
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Tidal Volume
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typically set at 10-15 breaths/minute. respiratory rate is adjusted according to teh pCO2 ( and pH) which is the index of ventialtion
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respiratory rate
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ability of the heart to initiate an impulse spontaneously without an external stimulus
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automaticity
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the ability of cardiac cells to reach a threshold and resond to a stimulus.
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excitability
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ability of muscle cells to shorten in response to an electrical stimulus
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contractility
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when sodium and potassium move otu and calcium moves in. electral excitation of teh cell mbembrane resulting from the flow of ions across the membrane; wave of excitation spreads from cell to cell through the conductionn system
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depolarization
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recovery stage-return of cell membrane to it's resting state; cells must repolarize before they can depolarize again
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repolarization
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recovery period after stimulation
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refractory period
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heart cannot respond to another stimulus regardless of stimulus strength
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absolute refractory period
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follows absolute refractory period, heart can respond to very strong stimulus, but response will be abnormal . can respond to stimulus if stimulus is really strong
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relative refractory period
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represents AV conduction time, or the time it takes the impulse to travel through teh atria,through teh AV node, and down to where the ventricles begin to depolarize
measure from beginning of P wave to beginning of QRS normal:.12-20 seconds |
PR interval
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ventricular depolarization as the impulse travels through both ventricles. Normal: .04-.10
follows the P wave |
QRS complex
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ventricular repolarization as ventricles return to resting electrical state; relative refractory period (vulnerable period)
normal: smooth, rounded, upright in most leads |
T wave
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represents early repolarization phase and extends from end of QRS to beginning of T wave; should be at isoelectice line; myocardium still in absolute refractory period; eleveation or depression represnets mycoardial injury or ischemia
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ST segment
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time from ventricular depolarization to ventricular repolarization: measrued from beginning of QRS to end of T wave; QT interval varies with heart rate
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QT interval
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