Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
23 Cards in this Set
- Front
- Back
Name the 2 kinds of strokes and describe the pathology.
|
ischemic stroke (85% of strokes) caused by thrombotic stroke, precursors are TIAs. Embolic Stroke under this category too. Other kind is Hemorrhagic stroke--dont treat with thrombolytics.
|
|
TIA--patho
|
temporary loss of neurological function, at rest or sleep when BP lowered and cant perfuse past plaque, resolves within 24H without any deficits.
|
|
TIA S/S
|
weakness
slurred speech (carotid artery involvement feeding the middle cerebral artery in Broca's area) loss of words (temp. aphasia) visual changes |
|
What are the most common areas to get a thrombotic stroke?
|
on top of artherosclerotic plaque: carotids (5 x more common than other arteries), middle cerebral artery, vertebral artery. Internal carotid is most devestating.
|
|
Thrombotic Stroke: Vertebral Artery Signs and Symptoms
|
dizziness, ataxia, dysphasia, syncope, numbness, vertigo, visual field disturbances
|
|
Thrombotic Stroke: Middle cerebral artery and carotids signs and symptoms
|
blindness in one eye
hemiparesis or hemiplegia hemianesthesis speech disturbances confusion symptoms progress as cerebral edema develops |
|
Embolic Stroke--patho
|
Frequently cardiogenic in origin.
A-Fib clots and subsequent clot formation is the most common cause. occur when pt awake and active most frequent route is thru the middle cerebral artery (straightest route from heart) Symptoms appear rapidly, seconds to minutes Emboli begin to break up in 12H and smaller emboli may contribut to further neurological deficits. |
|
CVA--history data to get
|
when was the pt last neurologically intact? any recent trauma or surgery? are you on anticoagulants antihypertensives, anticonvulsants, are you diabetic?clock starts when you start asking questions, you have to give thrombolytics in <3H or may have significant neurologic deficit.
|
|
CVA--embolic stroke--evaluation
|
CT scan to r/o hemorrhagic BUT ischemic strokes dont show up for a while. MRI may be better to assess for an ischemic injury. Check PT, is it < 15? more than 14 days since major surgery, more than 3 months since previous stroke or head trauma. SBP < 185 or DBP<110. Must have SIGNFICANT neurologic deficits because its so risky to give a thrombolytic. is it worth it?
|
|
CVA evaluation
|
rt-PA given within 90-180 min. reduces the extent of the neurological damage (if less than 1/3 of a hemisphere--if more wont respond). They are increased risk for intra-cerebral hemorrhage.
|
|
CVA TESTS to assess ischemic stroke
|
Require a baseline CT to determine if ischemic or hemorrhagic
If embolic, recommend anticoagulation (ASA). 80% will secondary event. Recommend ASA 325mg within 48 H unless hypertensive or have a large infarct--then wait 10-14 days. If a-fib, start ASA during the acute phase (as soon as documented that it is non-hemorrhagic) |
|
CVA--ischemic-- Clinical Mgmt
|
hyperglycemia increases neuronal damage and extends the infarct!! it may be a marker of severity of brain injury. Monitor carefully!!
Treat fevers: fevers may worsen the prognosis and look for the cause. |
|
CVA--ischemic--clinical mgmt 2
|
HT may be protective. Ischemic penumbra has lost autoregulation. it may require higher pressures to perfuse it. Avoid anti-Htn meds unless SBP > 220 or DBP >140.
Dont lower the BP more than 10-15% in 24H Recommend: Labetalol (10-20mg) IV or Nicardipine (5mg/hr). Prophylactic seizure control NOT recommended for ischemic stroke. |
|
CVA--ischemia--other things to watch
|
naubtaub euvolumia, BR w/ HOB 30-35%, laxatives, DVT prophylaxis, mild sedation, pulmonary care, speech and swallowing assessment (40% will have abnormalities), nutrition--may use dobhoff for swallowing difficulties but usually resolves in 7-10 days.
Treat fevers--look for the source. ASA if anticoagulation is desired. |
|
CVA--Hemorrhagic Stroke--patho
|
2 kinds Subarachnoid 25% and Intracerebral (75%). Subarachnoid is from a ruptured aneurysm, or its a bleed from an AV malformation. Intracerebral, more common, bleeding into the parenchema. Most commonly assoc. w/ HTN. see in elderly doing valsalva. s/s quick and progress over hours. Hematoma enlarges and increase ICP leads to herniation.
|
|
CVA--Hemorrhagic stroke--Right Hemisphere
|
L sided hemiplegia
L sided Hemoparesis spatial-perceptual deficits denial of affected side ***** impulsive behavior poor judgement deficits in L visual fields |
|
CVA--Hemorrhagic stroke--Left Hemisphere
|
Right sided hemiplegia
Right sided hemiparesis Expressive, receptive or global aphasia Decreased performance on cognitive tests (math) shows cautious behavior deficits in R visual field difficulty distinguishing R from L |
|
CVA--hemorrhagic stroke--vertebral artery
|
pain in face, nose, eye
ipsilateral numbness and weakness of face dizziness staggering gait nystagmus clumsiness dysphagia |
|
CVA--Hemorrhagic--Diagnosis
|
CT without contrast. get tox screen on young ppl
|
|
CVA goals of care
|
restore circulation, stop ischemic process, reduce ICP. Might have reflexive HTN to protect cerebral perfusion. decline starts in 24H, gradually normalizes in 7-10 days. Hematoma impairs autoregulation in the surrounding area and can extend injury. Recommendation to gradually lower BP if SBP>200 or DBP > 110
Start Rehab early!!! |
|
Hemorrhagic stroke--treatment
|
rest, treat HTN, d/c anticoagulation drugs for 1-2weeks. watch for seizure activity. can put in an ICP monitor, or do surgical decompression of hematoma >3cm if s/s. start rehab on admission!!
|
|
CVA--complications
|
DVT
decubitus ulcers Gastric ulcers (curlings ulcer) aspiration PNA hydrocephalus (more likely with hemorrhagic stroke) |
|
Ranchos Scale
|
used for stroke patients to guide for determining treatment modalities. the highert the number the better the function. goes from level 1 to VIII
|