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33 Cards in this Set
- Front
- Back
DKA reflects amplification of the same physiologic mechanism as ______
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ketosis
p1286 |
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As the body switches to fat-based metabolism, the blood level of ketones ______
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rises
p1286 |
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In DKA when the ph balance is from 7.4 to ____ the pt is hours from death if left untreated
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6.9
p1286 |
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The onset of clinically obvious DKA is slow usually lasting from ___ to ___ hours
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12-24 hours
p1286 |
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Signs and Symptoms of DKA
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Diuresis, increased urine production, dry warm skin and mucous membranes, general malaise, tachycardia, physical weakness
p1286 |
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Name a major compensatory mechanism as ketoacidosis appears, it helps expel cardon dioxide
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Kussmaul's respirations
(rapid deep breathing pattern) p1285 |
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A hallmark sign of DKA is ?
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A sweet fruity odor on the pt's breath
p1286 |
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Hyperglycemia, acidic pH, electrolyte abnormalities, low bicarb levels, hypokalemia all occur in
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DKA
p1287 |
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True or False
A fever is characteristic of ketoacidosis |
False
a fever is a signal of infection p1287 |
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It is not uncommon for pts w/ketoacidosis to have BGL in excess of
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300mg/dl
p1287 |
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Field management for DKA:
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Maintenance of ABC's and fluid resuscitation to counteract dehydration
p1287 |
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HHNK coma develops when 2 conditions occur:
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1-sustained hyperglycemia
2-dialysis, high-osmolarity feeding supplements, infection, drugs can also be associated p1287 |
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HHNK's level of hyperglycemia is often much higher than that of DKA usually around
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1000 mg/dl
p1287 |
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The mortality rate for HHNK ranges between ___ to ___%
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40-70%
p1287 |
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HHNK primarily affects the ____
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elderly
p1287 |
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Onset of HHNK is slower than DKA, onset development occurring ____
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over several days
p1288 |
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Signs and Symptoms of HHNK
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Increased urication and thirst, orthostatic hypotension, dry skin and mucous membranes, tachycardia
p1288 |
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True or False
Kussmaul's respirations are often seen in pts with HHNK |
False
Kussmaul's is rarely seen because of the lack of acidosis p1288 |
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Management of HHNK
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Same as DKA, ABC's & fluid resuscitation, expedited transport
p1288 |
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As hypoglycemia lengthens, risk rises that brain cells will be permanetly damaged due to lack of
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glucose
p1288 |
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Glucagon may be ineffective in raising BGL because of
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high insulin levels
p1288 |
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Almost half of glucose normally produced is of ____ origin and the activity is stimulated by ______
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Renal origin
epinephrine p1288 |
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The most important sign/symptom of hypogycemia
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Altered Mental Status
p1288 |
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Signs and symptoms of hypoglycemia
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bizarre behavior, diaphoresis, tachycardia, Altered mental status, may have a hypoglycemic seizure
p1288 |
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Management of hypoglycemia
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BGL<60: IV on NS, admin 50-100ml of D50 IV, if pt is conscious and can swallow use dextrose paste
If BGL cannot be obtained: IV of NS, admin 50-100ml of D50, if alcoholism is suspected admin 100mg of thiamine If IV cannot be started: admin 0.5-1.0 mg of glucagon IM p1288 |
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You are more likely to see thyroid disfunction as part of the _____ than as an emergency
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Medical history
p1289 |
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Name the 4 most common thyroid disorders
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1-Hyperthyroidism
2-Thyrotoxicosis 3-Hyptothyroidism 4-Myxedema p1289 |
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More than 95% of thyrotoxicosis are due to _____
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Graves' disease
p1289 |
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Graves' disease has a strong hereditary role and is about ___ times more common in women than men
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6
p1289 |
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Signs and symptoms of Graves' disease
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Agitation, emotional changeabilty, insomnia, poor heat tolerance, weight loss, weakness, dyspnea, tachycardia, new-onset of A-Fib,
p1289 |
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Nervous symptoms tend to be more common in _____, where cardiovascular symptoms tend to be common in ____
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young adults
older individuals p1289 |
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Management of Graves' disease
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Reducing cardiac stress and dealing with cardiac dysfunction
p1289 |
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____ is probably the most likely context w/in an emergency call may arise in Graves' disease
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Cardiac dysfunction
p1289 |