Incorporating physical activity is crucial for insulin therapy as it enhances cellular sensitivity to insulin and can escalate glucose intolerance. Exercise will also benefit cardiovascular function, weight control and overall increased health. Finding a cautious balance between moderate and vigorous exercise is important because increased exercise will cause hypoglycemia. Allowing time for consistent glycemic control before starting an exercise program is strongly encouraged. Blood sugar gets checked before and after exercise to oversee adequate blood sugar (Krzymien et al., 2015). The American diabetes association suggests 150 minutes of moderate-intensity aerobic exercise a week (Ignatavicius & Workman, 2013).
DKA
Diabetic ketoacidosis (DKA) is a critical …show more content…
DKA is the result of insufficient amounts of insulin production and the excess of glucagon production, distinguished by hyperglycemia, dehydration, acidosis, and electrolyte abnormalities. If left untreated the patient can slip into a coma. Without insulin the patient will return to a hyperglycemic state as she was prior to initial treatment. Blood glucose will rise as there is no insulin secreted to allow it into the cells. The blood becomes more acidic due to increased serum glucose concentration, shifting sodium and water out of the intracellular space to the extracellular space in efforts to lessen hyperosmolality. Experiencing polydipsia and polyuria returns with DKA (Sherry & Levitsky, 2008). Large amounts of water loss will result in dehydration leading to decreased blood volume, lowered blood pressure and increased heart rate attempting to compensate for inadequate tissue perfusion. Increased loss of electrolytes as a result of
DKA
Diabetic ketoacidosis (DKA) is a critical …show more content…
DKA is the result of insufficient amounts of insulin production and the excess of glucagon production, distinguished by hyperglycemia, dehydration, acidosis, and electrolyte abnormalities. If left untreated the patient can slip into a coma. Without insulin the patient will return to a hyperglycemic state as she was prior to initial treatment. Blood glucose will rise as there is no insulin secreted to allow it into the cells. The blood becomes more acidic due to increased serum glucose concentration, shifting sodium and water out of the intracellular space to the extracellular space in efforts to lessen hyperosmolality. Experiencing polydipsia and polyuria returns with DKA (Sherry & Levitsky, 2008). Large amounts of water loss will result in dehydration leading to decreased blood volume, lowered blood pressure and increased heart rate attempting to compensate for inadequate tissue perfusion. Increased loss of electrolytes as a result of