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50 Cards in this Set
- Front
- Back
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BLOOD VOLUME PERCENTAGE
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45% FORMED ELEMENTS
55% PLASMA |
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AVERAGE BLOOD FOR ADULTS
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MALE-5-6 LITERS
FEMALE- 4-5 LITERS |
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HEMATOCRIT
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% OF TOTAL BLOOD VOLUME COMPOSE OF RED BLOOD CELLS AS DETERMINED BY CENTIFUGE
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MAIN FUNCTION OF BLOOD
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TRANSPORTATION- AND ASSIST IN MAINTAINING BODY TEMPATURE-IMMUNITY-CLOT FORMATION
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PLASMA
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LIQUID PART OF BLOOD
91% WATER 9% PROTEINS,IONS,NUTRIENTS,WASTE PRODUCTS,HORMONES |
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SERUM
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PLASMA WITHOUT CLOTTING FACTORS
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FORMED ELEMENTS
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ERYTHROCYTES(rbc)95%
LEUKOCYTES(wbc)AND THROMBOCYTES(PLATLETS) 5% |
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HEMATOPOIESIS
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PRODUCTION OF ALL TYPES OF BLOOD CELLS
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DIFFERENCE IN MARROW BETWEEN CHILDREN AND ADULTS
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NEARLY ALL OF CHILDREN IS RED BONE MARROW- WHILE ADULTS RED IS FOUND IN RIBS,STERNUM,VERTEBRAE,PELVIS,FEMUR& HUMERUS OTHERS ARE YELLOW(ADIPOSE)MARROW
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RED BLOOD CELLS
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MOST NUMEROUS FORMED ELEMENT IN BLOOD-25 TRILLION. CANNOT MOVE ON THEIR OWN MOVE PASSIVLY BY CIRCULATION
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BIOCONCAVE STRUCTURE OF RBC
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INCREASES SURFACE AREA ALLOWS CELL TO BEND & TRAVEL THROUGH SMALL BLOOD VESSELS
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HEMOGLOBIN
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PIGMENTED PROTEIN, COMPOSES 1/3 OF TOTAL CELL VOLUME- ALLOWS RBC TO TRANSPORT 1 BILLION 02 MOLECULES
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HEMOLYSIS
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DESTRUCTION OF RBC IS A PATHOLOGY- RBC-RUPTURES
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REPLACEMENT OF RBC
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2.5 MILLION AND ARE REPLACED A DAY
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RBC LIVE AND CIRCULATE
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4 MONTHS
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WBC- LEUKOCYTES
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LACK HEMOGLOBIN- DO HAVE NUCLEUS- PROTECT AGAINIST MICROBES
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DIAPEDESIS
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ABILITY TO THIN, ELONGATE TO SLIP BETWEEN OR THROUGH CELLS OF VESSELS WALLS
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PUS
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ACCUMULATION OF DEAD WBC & BACTERIA WITH FLUID & CELL DEBRIS
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NEUTROPHILS
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60-70% OF WBC
MOST PROMIENT |
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LYMPHOCYTES
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20-25% OF WBC
SMALLEST WBC HELPS WITH IMMUNITY AND LYMPH SYSTEM |
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EOSINOPHILS
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2-4% WBC
STAIN RED WORK AGAINIST PATHOGENS |
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BASOPHILS
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1/2 TO 1 %WBC
ALLERIGES MAST CELLS |
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MONOCYTES
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3-8% WBC
BODY JANITORS LARGEST WBC TRANSFORM INTO MACROPHAGES |
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PLATLETS-THROMBOCYTES
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MINUTE, DISC SHAPED CELL FRAGMENTS WITH A STICKY SURFACE. PROMOTE WOUND CLOTS
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HEMOSTASIS
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STOPPAGE OF BLEEDING
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VASCULAR SPASM
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IMMEDIATE BUT TEMPORARY VESSEL CONSTRICTION
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PLATELET PLUG
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ACCUMULATION OF PLATLETS TO SEAL SMALL BREAK IN VESSEL
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4 COMPONETS OF PLATLET PLUG
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1- PLATLET ADHESION
2- PLATLET RELEASE REACTION 3- PLATLET AGGREGATION 4- PLATLET EXPRESS PHOSPHOLIPIDS AND COAGULATION CHEMICALS |
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COAGULATION
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BLOOD CLOOTING WHICH STOPS LARGE TEARS & CUTS
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BLOOD CLOT
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A " NET" OF FIBRIN THAT TRAPS BLOOD CELLS& PLATLETS
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PROTHROMBINASE
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CAUSES PRODUCTION OF THROMBIN
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THROMBIN
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CONVERTS FIBRINOGEN INTO FIBRIN
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ANTICOAGULANTS
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CHEMICALS IN THE BODY TO TURN OFF POSITIVE FEEDBACK OF CLOTTING. PREVENTS COAGULATION FROM SPREADING .
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ANTIGENS
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MOLECULES ON THE SURFACE OF RBC
ABLOOD HAS A ANTIGEN B BLOOD HAS B ANTIGEN |
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ANTIBODIES
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MOLECULES PRESENT IN PLASMA; TARGET SPECFIC ANTIGENS
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AGGLUTINOGENS
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SELF ANTIGENS- ANTIGENS YOU DO HAVE
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AGGLUTININS
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SELF- ANTIBODIES- ANTIGENS YOU DON'T HAVE AND ATTACK THEM
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AGGLUTINATION
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CLUMBING OF RBC
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TYPE A
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A ANTIGENS AND ANTI- B ANTIBODIES
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TYPE B
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B ANTIGENS AND ANTI- A ANTIGENS
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TYPE AB
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A&B ANTIGENS AND NEITHER ANTIBODIES
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TYPE O
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NEITHER A OR B ANTIGENS BOTH A & B ANTIBODIES
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RH- POSITIVE
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CERTAIN RH ANTIGEN IS PRESENT ON SURFACE OF RBC
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RH- NEGATIVE
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RH ANTIGEN IS NOT PRESENT
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ERYTHROBLASTOSIS FETALIS
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AGGLUTINATION& HEMOLYSIS OF FETAL BLOOD CELLS
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CBC
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ANALYSIS OF BLOOD.
RBC,HEMOGLOBIN,HEMATOCRIT MEASURED, WBC COUNT |
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ANEMIA
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LOWER THAN NORMAL RBC
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LEUKOCYTOSIS
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ABNORMALLY HIGH WBC COUNT
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LEUKEMIA
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CANCER OF RED BONE MARROW
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THROMBOCYTOPENIA
HEMOPHELIA |
LOW PLATLET COUNT, FAILURE TO CLOT& CHRONIC BLEEDING
ABNORMAL OR ABSENT CLOTTING MECHANISM |
