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54 Cards in this Set

  • Front
  • Back

Atherosclerosis

plaque buildup affecting moderate-large arteries


thickening and narrowing of blood vessel wall

Coronary Artery Disease (CAD)

spectrum of clinical entities ranging from angina to infarction to death




symptoms present when lumen is at least 70% occluded

Angina Pectoris

chest pain or pressure due to ischemia




may be accompanied by Levines sign (fist over sternum)




inbalance in myocardial oxygen supply and demand brought on by increased demand on heart, exertion, stress, smoking, extreme temps, overeating, vasospasm

Stable angina

classic exertional angina




occurs at a predictable rate pressure product, relieved with rest and/or nitorglycerin



Unstable angina

coronary insufficiency at any time without any precipitating factors or exertion




chest pain increases in severity, frequency, and duration; refractory to treatment

Variant angina

caused by vasospasm of coronary arteries in the absence of occlusive disease




responds well to nitroglycerin or calcium channel blocker

Angina pain in women

sensations of discomfort, crushing, pressing, and bad ache

Symptoms of angina

SOB ,fatigue, diaphoresis, weakness

Myocardial infarction

prolonged ischemia, injury, and death of an area of the myocardium caused by occlusion of 1 or more coronary arteries

Transmural MI

full thickness of myocardium


ST elevated MI or Q wave MI




STEMI

Non-transmural MI

subendocardial, subepicardial, intramural




non-ST elevated MI---NSTEMI




non-Q wave MI

MI impaired ventricular function

decreased SV, CO, and ejection fraction




increased end diastolic ventricular pressures

Heart failure

heart in unable to maintain adequate circulation of the blood to meet the metabolic needs of the body

Left sided CHF

pulmonary congestion, edema, and low CO due to back up of blood from the left ventricle to the left atrium and lungs




occurs with insult to the left ventricle from myocardial disease, excessive workload of heart, arrhythmias, valvular disease

Right sided CHF

characterized by increased pressure load on the right ventricle with higher pulmonary vascular pressures




mitral valve disease, chronic lung disease,




produces hallmark signs of jugular vein distension and peripheral edema

Biventricular failure

severe LV pathology producing backup onto the lungs




increased PA pressure and RV signs of HF

Compensated HF

heart returns to functional status with reduced CO and exercise tolerance




control is achieved through physiological compensatory mechanisms

What physiological compensatory mechanisms are used in compensated HF

SNS stimulation, LV hypertrophy, anaerobic metabolism, cardiac dilatation, arterial vasoconstriction

Pulmonary signs and symptoms of Left CHF

dyspnea, dry cough,


orthopnea


paroxysmal nocturnal dyspnea


pulmonary rales, wheezing

pulmonary signs and symptoms of Right CHF

dependent edema


weight gain


ascites


liver engorgement

Signs and symptoms of low CO in Left CHF

hypotension


tachycardia


lightheaded, dizzy


fatigue, weakness


poor exercise tolerance


enlarged heart on x-ray


S3 heart sound


murmurs

signs and symptoms of low CO in Right CHF

anorexia, nausea, bloating


cyanosis


right upper quadrant pain


jugular vein distension


right sided S3 heart sound


murmurs of pulmonary or tricuspid

PAD

chronic, occlusive arterial disease




diminished blood supply to affected extremities with decreased or absent pulses

Associated conditions with PAD

HTN, HDL, CAD, cerebrovascular disease, diabetes, metabolic syndrome, hx smoking

Early stages of PAD

intermittent cladication




burning, searing, aching, tightness, or cramping




occurs regularly and predictably with walking, and relieved with rest

Late stages of PAD

pain at rest, muscle atrophy, trophic changes

Thromboangiitis obliterans (Buergers disease)

chronic, inflammatory vascular occlusive disease of small arteries and also veins




occurs commonly in young adults, males, who smoke




begins distally and progresses proximally in both UE and LE

Signs and symptoms of Buergers disease

paresthesias or pain, cyanotic cold extremity, diminished temperature sensatio, fatigue, risk of ulceration, gangrene

Diabetic angiopathy

an inappropriate elevation of blood glucose levels and accelerated atherosclerosis




Neuropathy--major complication




neurotrophic ulcers may lead to gangrene and amputation

Raynauds Phenomenon

episodic spasm of small arteries and arterioles




abnormal vasoconstrictor reflex exacerbated by exposure to cold temperature or emotional stress




tips of fingers develop pallor, cyanosis, numbness, and tingling




largely affects females

Superficial vein thrombophlebitis

clot formation and acute inflammation




localized pain usually in saphenous vein

DVT

clot formation and acute inflammation in a deep vein




associated with venous stasis--bed rest, hyperactivity of blood coagulation, vascular trauma




early ambulation is prophylactic

signs ans symptoms of DVT

TTP, dull ache, tightness, or pain in calf


swelling, warmth, skin discoloration and venous distention




DO NOT USE HOMANS SIGN TO EVALUATE

DVT to PE

presents abruptly with chest pain and dyspnea




diaphoresis, cough, apprehension,




requires EMERGENCY TREATMENT-life threatening

Medical management of DVT

anticoagulation therapy--heparin


thrombolytic agents--streptokinase


ambulation and mobility encouraged after 1 dose of low weight heparin




compression stocking 30-40 mmHg

Arterial Insufficiency skin changes

pale, shiny, dry


loss of hair


nail changes


coolness

venous insufficiency skin changes

hemosiderin--dark, cyanotic brown skin


fibrosis

arteial insufficiency ulceration

may develo in toes, feet, or areas of trauma




pale or yellow to black eschar




regular in shape--punched ouy

venous insufficiency ulceration

may develop at medial side of ankle along course of vein




painful, shallow, exudative, granulation tissue at base




irregular borders

chronic venous stasis/incompetence

venous valvular insufficiency, venous dilation




grade I-mild aching, minimal edema




grade II- increased edema, dilated veins, changes in skin pigmentation




grade III- venous claudication, severe edema, ulceration

Primary lymphedema

congenital condition with abnormal lymph node or vessel formation

Secondary lymphedema

acquired, due to injury of 1 or more parts if the lymphatic system




surgery, tumors, trauma, infection, radiation therapy, chronic venous insufficiency,

Acute lymphangitis

acute bacterial infection spreading throughout lymph system




usually strep

Aneurysm

localized abnormal dilation of a blood vessel

Aortic aneurysm

usually asymptomatic




may have generalized abdominal or low back pain




may cause pulsations near the navel

Cerebral aneurysm

can cause a sudden and severe headache, nausea, vomiting, stiff neck, seizure, LOC, double vision

Cardiomyopathy

a group of conditions that affect the myocardium




impairing the ability of the heart to contact and relax

3 types of cardiomyopathy

dilated


hypertrophic


restrictive

Cor pulmonale

pulmonary heart disease




hypertrophy of the right ventricle caused by altered structure or function of the lungs

Signs and symptoms of cor pulmonale

cardinal sign--progressive SOB, esp with exertion




fatigue, atypical chest pain swelling of LE, dizziness, syncope

Endocarditis

inflammation of the endothelium that lines the heart and cardiac valves




if left untreated, can cause damage or destroy heart valves and become life-threatening!!!!




bacterial infection

Myocarditis

inflammation and weakness of the myocardium




can cause myocardium to become thick and swollen, which can lead to symptoms of heart failure




viral or bacterial

Pericarditis

inflammation of the pericardium of the heart




pericardium has inner and outer layer with fluid in the middle which can cause pericardial effusion (increased fluid)

Signs and symptoms of pericarditis

chest pain, SOB, dry cough, anxiety, fatigue, fever