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45 Cards in this Set
- Front
- Back
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reflex arc
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sensory meets motor
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posterior horn acute
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hyperesthesia
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posterior horn chronic
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anesthesia
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anterior horn acute
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hypertonicity/spasm
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anterior horn chronic
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atonia/flaccidity
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lateral horn acute
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vaso/bronchodilation
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lateral horn chronic
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vaso/bronchodilation
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five components of vertebral subluxation complex
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1. Neuropathophysiology
2. Kinesiopathophysiology 3. Myopathology 4. Histopathology 5. Pathophysiology |
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Neuropathophysiology
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Biomechanical insult to nerve tissue is proposed to case neural dysfunction in 3 forms
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Neuropathophysiology 3 forms
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1. Irritation
2. Compression 3. Decreased axoplasmic transport |
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Kinesiopathology
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hypomobility, diminished or absent joint play, or segmental hypermobility due to compensation
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Myopathology
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spasm or hypertonicity of muscles as a result of compensation, facilitation, hilton's law.
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Histopathology
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damage on the cellular level via subluxation/inflammation.
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5 signs of Inflammation
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Redness (rubor), Heat (calor), swelling (tumor), Pain (dolor), loss of function (functio laesa)
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Histopathology substances released:
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histamines, prostaglnadins, kinins
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Pathophysiology
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abnormal function of the spine and the body via subluxation
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Nerve Compression
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DD Palmer.
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Spinal nerve root compression would expect to do what to nerve fiber function
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decrease
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Nerve roots are more or less mechanically predisposed to irritation or compression than are peripheral nerves.
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More
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Axoplasmic Transport
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how nerve is provided nutirion through the flow of proteins and other materials.
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Anterograde
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forward moving. Proximal to distal. faster
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Retrograde
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Back-ward. Nerve terminals to cell bodies. Slower
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Cord compression AKA?
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Compressive Myelopathy
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Cord Compression
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B.J Palmer
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Grisel's syndrome
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infectious process results in ligamentous laxity and atlantoaxial subluxation in which patients complain of unrelenting throat and neck pain. compressive myelopathy
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What BEST describes the nature of the bertebral subluxation?
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Theory of spinal dysfunction
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alar Ligament
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"check" ligament
sides of dens to occipital condyles limits rotation |
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dentate ligaments
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connect pia to dura
21 ligaments |
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Stages of Adaptation
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Alarm, Resist, Adapt, Exhaust
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Brain Hybernation, Cerebral Dysfunction syndrome, diachisis
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theory: reduction of cerebral blood flow can occur and cause dizziness, fatigue, slurred speech, glare distress
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Kinesiopathology
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hypomobility, diminished or absent joint play, or segmental hypermobility due to compensation
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Myopathology
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spasm or hypertonicity of muscles as a result of compensation, facilitation, hilton's law.
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Histopathology
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damage on the cellular level via subluxation/inflammation.
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5 signs of Inflammation
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Redness (rubor), Heat (calor), swelling (tumor), Pain (dolor), loss of function (functio laesa)
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Histopathology substances released:
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histamines, prostaglnadins, kinins
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MOST accurate statement to describe a reduction of pain after a spinal adjustment
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Chiropractic encounter can often result in an analgesic response
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neurodystrophic hypothesis
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lowered tissue resistance is the cause of disease
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Leach defines segmental dysfunction
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lessened mobility, altered pressure threshold to pain, and signs of neuromuscular dysfunction
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Subluxation Syndrome
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signs and symptoms that relate to pathophysiology or dysfunction of spinal and pelvic motion segments or to the peripheral joints
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3 phases of Unified model for vertebral subluxation
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phase of segmental dysfunction, instability, and stabilization.
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Neopspinothalamic tract
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lateral spinothalamic system
primary pathway in the spine for nociceptive sensation |
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What amount of pressure could decrease conduction up to 60% in 30 minutes
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10mm Hg
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A nerve that includes preganglionic and postganglionic fibers
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Visceral efferent
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Spinal Learning
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alterations from spinal fixations are due to changes in spinal reflex circuits and peripheral inputs
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Most abundant afferent fibers that innervate joints and muscles
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group IV (C-fibers)
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