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58 Cards in this Set
- Front
- Back
Why care about testing CAPD?
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Understanding of behavior, remove blame, develop remediation, educational placement, differential diagnosis, site of lesion testing.
Determining if the deficit is fixed or plastic: shat conditions improve performance and can that be maintained? If fixed, what skills/behaviors can be improved? |
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"Language"
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Employs pitch, duration and rhythm: tonal and temporal characteristics to convey meaning.
Language performance depends on: verbal working memory, phonological awareness (perception). These skills can be taught leading to improved receptive and expressive language. |
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Phonological Working Memory
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Ability to process and hold verbal information in immediate attention.
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Phonological Awareness
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Ability to reflect and manipulate sound components of language.
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What is auditory processing?
List auditory abilities that belong to AP |
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Ability to attend, discriminate, recognize, remember and comprehend. Depends on accurate timing of neural events. Involves attention, language, motivation and decision processes. Listener actively controls processing and constructs message. |
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ASHA definition of auditory processing
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The efficiency and effectiveness by which the CNS utilizes auditory info.
Perceptual processing of auditory info in the CNS & the neurobiologic activity that underlies that processing and gives rise to electrophysiologic auditory potentials. |
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Bellis definition of auditory processing
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Extraction of information at different stages of processing.
Complex interactions between sensory and higher-level cognitive/linguistic operations occurring simultaneously (parallel) and sequentially (distributed) throughout the system. |
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Behaviors of auditory processing: temporal aspects of hearing
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Resolution (important for e.g. voice onset time)
Integration Ordering (sequencing) (melody perception) -“Boots” vs. “boost” ISI of only 2ms needed to perceive 2 signals, but 17 ms needed to perceive order of signals. |
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Behaviors of auditory processing: performance with competing messages
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Binaural separation (attend to signal in one ear only)
Binaural integration (keep signals from each ear separate) |
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Behaviors of auditory processing: binaural interaction
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Binaural interaction: how the ears work together. (brainstem very important)
–Localization (direction of source) –Lateralization (place in head) –Binaural release from masking (MLD test) –Detection of signals in noise –Binaural fusion |
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Behaviors of auditory processing: pattern recognition
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“He saw the snow drift by the window.” vs. “He saw the snowdrift by the window.”
Project vs. Project |
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Behaviors of auditory processing: auditory closure
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Ability to "fill in the gaps"
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Auditory coherence/streaming
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How sounds occurring simultaneously will be perceived as grouped together (one source) or separate (two sources).
Ability to stream depends on most basically: audibility, normal temporal resolution, normal frequency resolution. |
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Bottom-up processing
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Sensory and data driven.
Encoding of auditory signals. Depends on good peripheral hearing function and good signal. **Bottleneck and subtlety principles** |
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Top-down processing
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Central resources and concept driven.
Higher order cognitive, linguistic and other influences on processing. |
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Phase locking
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A nerve will only respond to a particular phase of a signal.
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Bottleneck principle
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All information must pass through the cochlea and 8th nerve. Problems will show up very clearly.
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Subtlety principle
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The redundancy of internal pathways makes finding problems more difficult because system can more easily compensate when the problem is more central.
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Intrinsic or internal redundancy
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Built into CNS…multiple representations. Impacted by disorder.
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Extrinsic or external redundancy
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Built into signal (syntax, morphology, semantics) which enhance comprehension of the signal.
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Information extraction
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VIIIN: signal analysis re: intensity, frequency and time.
CN: contrast enhancement of intensity. Convergence/divergence. Differential cell responses. SOC: codes binaural cues via con/divergence from both sides. ID: modulation enhancement, binaural cue enhancement. Divides pathway into primary and diffuse systems. MGB: primary connection between BS and cortex. Codes slowly changing stim (vowel/syllable duration contrasts, binaural coding, feature extraction, complex signal processing, integration with other modalities. Primary Auditory Cortex: coding of rapid acoustic events for fine discrim. e.g. consonants, auditory space developed for localization. Auditory Association Cortex: recognition of linguistic stimuli, comprehension of spoken lang, lang formation. |
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Corpus Callosum
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4 parts: rostrum, genu, body, splenium.
Anterior commissure a separate structure. Complex rich network, connections to most areas of the cortex, allowing for multimodality integration. Auditory segment mainly in the posterior segment. |
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What is APD?
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A deficit in the perceptual processing of auditory stimuli and the neurobiological activity underlying that processing.
Abnormal or misrepresentation of sounds in the auditory system which leads to difficulty listening and comprehending (especially in background noise). |
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Signs of APD
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Normal peripheral hearing common
Inconsistent auditory responses, often misunderstands Poor localization, discrimination Poor AFG skills: understanding speech in noise/reverb Poor auditory memory Poor listening skills, trouble paying attention, concentrating, easily distracted Trouble following instructions Difficulty with rapid speech, degraded speech Frequently says “huh?” or “what?”, asking for repetition Relies on visual cues Hx chronic OM Difficulty following instructions Gives slow/delayed response to verbal stim Poor reading/spelling and academic achievement Has language deficits, deficient phonemic skills, memory deficits, auditory memory, phonologic awareness Lower self esteem and behavior problems |
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APD vs LD vs ADHA vs OM
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APD: difficulty with perception or deriving meaning from incoming sounds.
LD: disorder in using language (spoken/written) decreasing ability to listen, think, speak, read, write, spell, math. ADHA: academic or emotional difficulties from inattention, impulsivity, and hyperactivity. OM: persistency of fluid in ME. |
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Why the controversy of APD, ADHA and LD?
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1. Highly complex parallel (simultaneous) and sequential (distributed) processing.
2. Involves at lease 4 cortical levels including 15 or more areas in the brain. 3. Interconnected with other sensory modalities. |
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Gestational age
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Time between the first day of the last menstrual period and the date of birth.
2 weeks longer than conceptional age; they are not synonymous terms. |
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Corrected age
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Subtracting the number of weeks born before 40 weeks of gestation from the chronological age
Important if born premature…can’t expect a premie to be as developed as the full term infant |
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When do we first hear in the womb?
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6-7 months GA. All auditory mechanisms seem ready somewhere between 20-30 weeks (premies born at 25 weeks react to sound).
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What is the final event that allows us to hear?
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Simultaneous mechanical, chemical, neural events.
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Sensory cell development
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Inner hair cells: mature at 14 wks GA, base before apex, neural connections before OHC, afferent before efferent.
Outer hair cells: maturing into the 3rd trimester, >24 wks Innervation: afferent mature by 24 wks, develops up & down, efferent present at 22 wks but not hooked up. |
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Neuromaturation
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Neural development prior to adult like responses.
Structural vs. functional development. IE structure by end of 5 mo GA (20 wks): 26 wk GA earliest response. BS structure functional by 30 weeks GA: 30 wks earliest response |
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What do we hear in the womb?
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Average 85 dB SPL, low pass filtered (20-200 Hz).
Nearby voices may reach fetus. Mostly mothers vocalizations. |
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Postnatal hearing
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Acoustic reflex fully developed at birth with adult like thresholds.
ABR: 17 dB higher thresholds than adults. (But, what does an ABR measure???????) Behavioral: some behavioral response to high levels, subtle responses at moderate levels: 55-60 dB at 500 and 4000 Hz at 2-5 wk using OPP, 15 -20 dB SPL at 5mo+ using VRA. Infant-adult gap varies in development by frequency up to 10 years. |
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Recording ABR after birth
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PCA (post conceptional age) of 35 wks required for reliable ABR for wave V id. Bipolar clicks with threhsold detection around 40 dB nHL.
ABR can be recorded as early as 27-28 weeks CA (30 GA). Wave I more prominent and prolonged (by .3-1.0ms) latency decreases after birth. Later waves are also prolonged in neonates. Interwave I-V is easily 5.0 ms (4.0 ms in adults). Infants more sensitive to changes in click rate. Middle latency response: more affected by sleep in infants Late evoked potentials: age 17 |
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OAE possible for PCA (post-conceptional age) of ______
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28 weeks
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Neuroplasticity
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Reorganize, adapt in response to internal and external changes: throughout life
–Stroke –Auditory deprivation studies |
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Cochlear structure and size is mature by
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22-26 weeks GA
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Cochlear function is mature by
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Term birth. Subtle aspects of cochlear function or efferent function mature by 33 weeks.
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Auditory nerve maturation
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Myelination by 1 year. Synaptic transmission by one year or more.
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Central auditory system maturation
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ABR at 30 weeks GA (BS structurally complete). New and more efficient synaptic connections and myelination continues into adulthood. Arborization (branching of dendrites). Myelination caudal to rostral, cortex continues into early adulthood. CC myelination starts 5-6 mo of age and complete in mid 20s. Last to develop and first to degenerate.
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Waves I and V of the ABR are adult-like by
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????
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Goal for APD testing
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ID presents of APD or confirm site of lesion?
Goal dictates approach Always include: objective/physiological tests, linguistically loaded tests (e.g. SSW, comp sent), less loaded tests (e.g. dichotic digits, pitch pattern), tests with known sensitivity and specificity. |
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Dichotic
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Different signal to each ear at same time
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Diotic
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Identical signal to each ear
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Monotic
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One or more signals to one ear only
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Monaural
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Using or involving one ear
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Binaural
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Using or involving both ears
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Interaural
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Between the ears
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Drawbacks to APD test battery
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Time: client fatigue, attention problems, busy clinic.
Complexity: many tests to choose from, need to test across different tasks. |
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Patient factors affecting APD testing
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Hearing loss, WRS in quiet, cognition, medications/drugs
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Possible causes of CAPD
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Neurodegeneration, hypoxemia, toxicity, maldevelopment, etc.
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Comprehensive test battery
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Functional tests, questionnaire, pure tone & speech audiometry, immittance measures, evoked potentials, OAEs. For CAPD you want a subset of this betters that is sensitive, specific and efficient.
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Aims of APD assessment
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?
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CAPD subtypes
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Decoding, integration, prosodic, output-organization, associative
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CAPD management
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Top down: compensatory strategies
Bottom up: environmental changes, therapies |
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Therapies for adults
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Formal auditory training (e.g. LACE)
Playing a musical instrument Note taking Audiobooks |
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APD assessment can have 4 basic goals. What are they?
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1) Judge site of lesion
2) Screening prior to medical referral 3) Identifying communication disorders 4) Monitor disease progression |