• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/104

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

104 Cards in this Set

  • Front
  • Back
diathesis
heritable tendency toward anxiety
trait neuroticism
relative frequently and ease with which negative emotions are generated- risk factor
autonomic restrictors
lower responses to physiological measures of heart rate and blood pressure but higher muscle tension

decreased activity in the right hemisphere, which is associated with low levels of image processing, and show increased activity in the left hemisphere, which is associated with increased verbal processing

images evoke higher levels of autonomic arousal and distress, and so by avoiding the processing of the image, their worrying is negatively reinforcing, don't actually face though, avoiding anxiety
behavioral inhibition system
-in septohippocampal system
-rich in serotonin and norepinephrine receptors: low levels (less activation) tend towards antisocial, high activation tend towards anxiety
-triggered by impending non-reward, punishment, novelty
-bottom-up and top-down
-activation: emotions of anxiety and frustration; behaviors of stop, slow down, freeze, observe, etc.
three “anomalies” in research regarding the role of cognitive change in effective cognitive behavioral
-no added value for cognitive intervention
-changes before changed cognitions
-no documentation for mediation effects for cognitive change
depressed thinking
-increased building on negative thinking
-stuck on negative thoughts
-deficits in cognitive control when processing negative information
empirically supported treatment
clearly specified psychological treatments shown to be effective in repeated experimental studies
depression and circadian rhythm
not regulated, too much or too little sleep
Interpersonal and Social Rhythm Therapy
-sleep and social rhythm interventions- get history, track events, stabilize rhythms and manage symptoms, find strategies for future success
-addition of the emphasis on social and biological rhythms
eating disorder NOS
-subthreshold of anorexia or bulimia
-combinations of different specifics
-binge eating
transdiagnostic treatment of eating disorder- overview
-the specific eating disorder is not of relevance – but its specific psychopathological features and the processes maintaining them
4 core features of eating disorders (maintaining mechanisms)
-mood intolerance
-interpersonal difficulties
-clinical perfectionism
-low self-esteem
anorexia/bulimia difference
"refusal" to maintain normal body weight (<85% expectation)- "success" in losing weight
anorexia death rate
20% (30% of that suicide)
social effects of starvation
-heightened preoccupation with eating
-social withdrawal
surprising about the finding that Interpersonal Psychotherapy is an effective treatment for bulimia nervosa
effective even though not specifically targeting disordered eating
why does a craving last for years?
-enduring cellular changes- sensitization of pathways to incentive salience through dopamine effects (craving)
-makes it easy to relapse
behavioral activation system
"pleasure pathway"
-mesolimbic system: amygdala, nucleus accumbens, prefrontal cortex- neurological circuit
-rich in dopamine receptors
-triggered by presence/possibility of reward
-activation triggers: emotions of hope and relief, approach behavior
which neurotransmitter plays a key role in activation in response to alcohol-related cues
glutamate
avoidant PD
avoid because scared of criticism/fear of rejection, want relationships
schizoid PD
doesn't want relationships
what is unique about personality disorders?
more pervasive/inflexible, less responsive to treatment, stable pattern of long duration
-Axis II
"borderline" origins
between neurotic and psychotic
biosocial theory of etiology (Linehan)- emotional vulnerability; biologically driven emotion dysregulation
-high sensitivity to emotional stimuli
-intense emotional responses
-slow return to emotional baseline
(each leads to next)
five components of emotional modulation in Linehan’s model of the etiology of borderline personality disorder
-inhibiting mood-dependent behaviors
-organizing behavior in the service of goals, independent of current mood (can't do something if inconsistent w/ mood)
-modulating physiological arousal as needed
-distracting attention from emotionally evocative stimuli
-experiencing emotion without immediate withdrawal or generation of extreme secondary emotions
three components of invalidating environments in Linehan’s model of the etiology of borderline personality disorder
-indiscriminate rejection of private experiences and self-generated behavior
-punish emotional displays and intermittently reinforce emotional escalation (when ppl pay attn)
-oversimplify the ease of problem solving and goal attainment
childhood sexual abuse and borderline
almost if not all- more (severe) abuse correlates with more symptoms
five behavioral skills addressed in the skill development component of borderline personality disorder
-distress tolerance
-emotion regulation
-interpersonal effectiveness
-self-management
-nonjudgemental awareness (mindfulness)
why borderline so challenging?
dissociation, missed/late to sessions, 3 am crisis calls, verbal attacks on the therapist
why dialectical behavior therapy "third wave"?
deals with trigger factors and context and experiential change as opposed to only treating symptoms
ADHD adverse outcomes
-risky behaviors- more accidents/health care
-comorbidity
-increased traffic offenses
-more early smoking
-reduced life expectancy
-more peer/sibling conflict
-increased HS dropout rate
problems associated with the DSM-IV approach to subtyping of ADHD
the disorder does not describe the condition- subtypes (ex. can have without inactivity); less symptoms as older but impairment can last
“spectrum” disorder
wide variations of symptoms and severity of symptoms
splinter skills
autism- great at something but no applicable skill (ex. birthdays)
spared skills
autism- undamaged
Yokohama study
proved no correlation by vaccines
“theory of mind”
separating your perspective from someone else's (ex. knowing that someone else doesn't know what you know)
mirror neurons and how are the implicated in the etiology of autism
less responsively (or fewer)
“expressed emotion” schizophrenia
-triggers relapse
-familial, expresses:
-criticism
-animosity
-emotional overinvolvement
Assertive Community Treatment
treatment for scizophrenia- go out into community and try to get people help in a non-hospital setting
prevalence rate of schizophrenia
1%
hypofrontality and how is it related to dopamine
underractivity to dopamine in frontal cortex, causes negative symptoms
positive symptoms of schizophrenia caused by
hyper-reactivity in the striatal system
overreactive to dopamine, positive symptoms; dopamine agonists increase symptoms (antagonists given as treatment, can cause Parkinson's-like symptoms)
panic attacks symptoms
abrupt experience of intense fear or acute discomfort, accompanied by physical symptoms that usually include heart palpitations, chest pain, shortness of breath, and possibly dizziness
three categories of panic attack
-Situationally bound-attack is cued by a particular setting or situation
-Unexpected-no clear cue or trigger for the attack
-Situationally predisposed-cues may, but don’t inevitably, cause an attack
treatment of panic disorder
benzos, SSRIs, exposure therapy (+interoceptive exposure)
generalized anxiety disorder
at least 6 months of excessive anxiety and worry that is ongoing for more days than not and is difficult to forget
GAD (symptoms) and panic attacks physiological?
GAD is characterized by muscle tension and mental agitation, fatigue, irritability, and difficulty sleeping
Panic attacks are associated with autonomic arousal
different types of phobia
-blood-injury-injection
-situational phobias
-natural environmental phobias (natural events eg. tornadoes etc.)
-animal phobias
-other
how phobias acquired
-classical conditioning
-may learn vicariously
-being warned repeatedly

1. traumatic experience
2. fear develops more easily if "prepared" to fear (actually dangerous)
3. one must be susceptible to developing anxiety that the event will happen again
acute stress disorder vs. PTSD
acute stress disorder within 1st month
mania
characterized by exaggerated elation euphoria, little sleep, rapid speech, and grandiose plans
causes and treatments for seasonal affective disorder
winter, in cold environments
excess sleep, increased appetite, and weight gain
may produce too much melatonin, disrupted circadian cycles
relationship between depression and anxiety
almost all depressed patients are anxious, but not all anxious patients are depressed
-anhedonia is key to depression, the key to anxiety is the experience of autonomic activation, which can also occur in depressed people
role of neurotransmitters, brain-wave activity, and negative cognitions in the etiology and treatment of depression
norepinephrine and dopamine out of whack because low levels of serotonin
fight or flight system (FFS)
-panic circuit
-responds to unconditioned or innately aversive stimuli
-brain stem, hypothalamus, amygdala, central gray matter
-activation triggers emotion of fear, behaviors of alarm and escape
schizophrenia disorganized symptoms
erratic behaviors in many domains
disorganized speech (tangents, cognitive slippage, loose associations)
unusual behaviors (catatonia)
schizophreniform disorder
full range of symptoms, rapid onset, brief duration
-less than 6 month
schizoaffective disorder
mood disorder- hallucinations or delusions without mood symptoms
delusional disorder
only delusions, no other positive symptoms or negative symptoms
brief psychotic disorder
one or more positive symptoms for less than one month
anorexia subtypes
restricting type
binge-eating and purging type
bulimia
characterized by binge eating and compensatory behavior (vomiting, laxatives, etc.)
consequences of starvation (physiological)
amenorrhea (absence of menstruation), hair loss, lanugo, impaired temperature regulation, heightened sense of fullness
comorbidity in anorexia nervosa
33% depression
OCD overlap, often associated with substance abuse (with substance abuse increases suicide risk)
orthorexia
obsessive preoccupation with eating healthy food
bulimia nervosa treatment
CBT and IPT (50% effective), SSRIs (short-term effective)
anorexia nervosa treatment
family therapy (no strong evidence for effectiveness), hospitalization
substance abuse
impairment or distress over a 12 month period; dysfunction, more about how it effects your life than how it effects physiologically (failure to fulfill obligations, creation of physical hazards, continued use in spite of social issues, etc.)- dependence takes precedent, "step above"
substance dependence
-implies abuse criteria also met
-impairment or distress over a 12 month period
-dysfunction (more physiological; tolerance, withdrawal effects, higher doses or longer duration than intended, attempts to stop without success, etc.)
alcohol sensitivity
1st drink big effect, later drinks have less effect (early sensitivity, later insensitivity)- leads to drinking more to get same effect
why we like alcohol
-behavioral activation system ("pleasure pathway)
-dopamine- need to feel good, positive reinforcement
GABA and glutamate in alcoholism
GABA- inhibitory neurotransmitter, decreases sensitivity to other neurotransmitters (alcohol inhibits)

glutamate- excitatory neurotransmitter, shift from dopaminergic regulation to glutamate regulation in frontal cortex (move from pleasure to craving)
personality disorder- enduring pattern of atypical behavior in two or more areas
-cognition
-affectivity
-interpersonal functioning
-impulse control
controversies of personality disorders
-comorbidity rule rather than exception
-poor empirical basis
-challenges categorical approach of DSM-IV
-extremes of personality; dimensional phenomena
3 clusters of personality disorders
-eccentric group
-dramatic-emotional group
-anxious-fearful group
eccentric group of personality disorders
-paranoid
-schizoid
-schizotypal
dramatic-emotional group of personality disorders
-antisocial
-borderline
-histrionic
-narcissistic
anxious-fearful group of personality disorders
-avoidant
-dependent
-obsessive-compulsive
borderline symptom picture
-extreme efforts to avoid real or imagined abandonment
-unstable interpersonal relationships
-identity disturbance
-impulsivity
-suicidal and parasuicidal behavior (self-injuring)
-affective instability
-anger
-dissociation
etc.
borderline personality disorder suicide rate
6%
biosocial theory of borderline personality disorder (Linehan)
most with vulnerability to emotion dysregulation do not develop BPD
-invalidating environments
-high incidence of childhood sexual abuse
emotional vulnerability for borderline personality disorder- the experienced process
-freezing or dissociating in the face of intense emotions
-rage
-intense dispair
dialectical behavioral therapy
3rd wave empirically supported behavioral treatment for borderline personality disorder
1. attaining basic capacities
2. posttraumatic stress reduction
3. resolving problems in living and increasing respect for self
4. attaining the capacity for freedom and sustained contentment
-ultimate goal: "having a life worth living"
Broca's area in schizophrenia
involved in speech production, activated- like "talking to self"
alogia
not talking
avolition
apathy
negative symptoms in schizophrenia
avolition
alogia
anhedonia
affective flattening
delusion
gross misrepresentations of reality, disorder of thought content
hallucinations
sensory experience in absence of environmental stimuli or input
diagnosis of schizophrenia
2 or more symptoms for at least one month
onset of schizophrenia- gender differences
men- earlier onset and likelihood of onset diminishes with age
women- onset later and likelihood of onset diminishes with age
ADHD subtypes
-inattentive subtype
-hyperactive-impulsive type
-combined type
-NOS
ADHD must have clinically significant impairment in which settings?
social, academic, or occupational (two or more)
ADHD and behavioral interventions?
-no empirical support; only experimental
-can help to treat comorbidities
core symptoms of autism
-social impairment
-communication impairment
-repetitive (stereotypes) behaviors and interests
explanations for possible increase of autism rates
-changes in diagnostic criteria
-finding more individuals with autism
-actual increase- pseudoscience, toxic exposure (potentially during a specific critical point in development)
-past- refrigerator mothers
4 types of medications for ADHD
amphetamines
methylphenidate
atomoxetines
guanfacine
antisocial personality disorder
irresponsible, impulsive, deceitful behavior that violates social and legal norms
lack conscience and empathy
paranoid-type schizophrenia
characterized by delusions and hallucinations that have a theme, typically of grandeur or persecution
do not show disorganized speech or flat affect
better prognosis
catatonic-type schizophrenia
unusual motor responses, including remaining in fixed positions and engaging in excessive activity
odd facial and physical mannerisms, may also mimic speech
undifferentiated-type schizophrenia
includes people who have the major symptoms of schizophrenia but do not meet the criteria for the other types
residual-type schizophrenia
people who have had at least one episode of schizophrenia but who no longer manifest major symptoms of the disease
schizophrenia prenatal cause
fetal exposure to viral infection, pregnancy complications, and delivery complications have all been implicated in schizophrenia