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99 Cards in this Set
- Front
- Back
Substance Abuse: Overview
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-Umbrella Term
-Use and abuse of psychoactive substances -wide ranging physiological, psychological, and behavioral effects -Associated with impairment and significant costs |
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Levels of Involvement: Substance Abuse
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1st- Substance use
2nd- substance intoxication: reversible condition due to excessive use and significantly maladaptive 3rd- Substance Abuse: Repeted abuse leads to reiccuring problems -use in hazardous situations (drink and drive) -legal problems continued use despite social probs (fight, arguments, etc) 4th-Substance Dependence (most extreme): maladaptive pattern of use -tolerance (need to drink more than before to get same buzz) and withdrawal (maladaptive beh. when not taking drug) -use despite medical problems or social disapproval |
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5 main classes of substances
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1. Depressants: Behavioral Sedation (ex. alcohol)
2. Stimulants: increased alertness and elevate mood (ex. cocaine, nicotine) 3. Opiates: produce analgesia and euphoria (ex. heroin, morphine, codeine) 4. Hallucinogens: alter sensory perceptions (ex. weed, lsd) 5. Other drugs of abuse (ex. inhalants, anabolic steroids, medications) |
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Depressants: Disinhibition
-Blood Alcohol level -Lethal Dose -LD50 -Synergistic |
-disinhibit central nervous system especially GABA
-Disinhibition: substance inhibiting a natural brain inhibitory brain system -Blood Alcohol Level (0.08 BAL) -Lethal Dose: kills certain percentage of test animals -LD50 kills 50% of test animals (usually 0.40 BAL) -synergistic: multiplicative. multiples the intensity when drugs and alcohol are mixed. 3shots+3lines=9x effect |
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Alcohol (Depressants): Facts and Stats
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-Most adults condor themselves light drinkers = 92% will use in life
-Use is highest in Whites (56.5%) -Males use and abuse > females -23% of Americans binge drink -violence is associated with alcohol -Effects of Chronic alcohol use (Dementia, wernickes disease, fetal alcohol syndrome) |
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Alcohol: Chronic illness'
-Cirrhosis of liver -Korsakoff's Syndrome -Fetal alcohol Syndrome |
-Cirrhosis of liver: scar tissue replaces liver tissue leading to loss of function and possibly death
-Korsakoff's Syndrome: cognitive disorder (thiamine deficiency) involving confusion, memory loss, and coordination problems -Fetal alcohol Syndrome: facial abnormalities (flat nose, no ridge on lips, underdeveloped lips), slow physical growth, cognitive impairment, elementary school learning probs. |
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Depressants: Sedatives, Hypnotics, Anxiolytics
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-Calming (ex. Barbiturates), sleep inducing, and anxiety reducing (ex. benzodiazepines)
-similar to large doses of alcohol: combining with alc is synergistic -Depress Central Nervous System (through GABA) -Primary influence in GABA (inhibitory receptor) |
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Stimulants: Overview
-after effects |
-Stimulate Central nervous system (increase norepinephrine and dopamine)
-Most widely consumed drugs in the us -low doses/potency: Alertness, Reduce Fatigue, Elevated Mood -High doses/potency: Strong Euphoria, Increased sexual drive -After effects are the reverse of what the chemical is inducing= Fatigue and depression (similar to manic episode) |
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Stimulants: Nicotine
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-Smoking has declined since 1965 (use in kids has increased since 1992)
-25% of college students smoke regularly -Most (64%) were Women (maybe to suppress hunger) and White (81%) -Most smoked occasionally, not everyday -over half binge drank in the past 2 weeks -Consequences: high blood pressure, cancers, heart disease |
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Stimulants: Cocaine & Methamphetamine
-Speed Ball -Symptoms -Effects of use -Use in college students |
-Cocaine: mixed with other stuff called "speed ball"
-Severe Intoxication or overdose -Cocaine: euphoria, high energy, bizarre, paranoid beh. -Hallucinations, Intense Panic, Seizures, or coma -Methamphetamine: called CRANK and makes up 90% of all amphetamine abuse -Effects of use -Brain and Liver Damage -Skin infections, tooth decay (meth mouth) -Immune system complications and stroke -College Students: 9.5% tried and 6.6% past year |
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Opiates (narcotics):
-Heroin -33 year addict study -Painkillers |
-Relieve pain and induce sleep (ex. Heroin, morphine, opium)
-Influence natural opioid system (i.e. endorphins, enkephalins) -Heroin (derived from morphine): -college students: 0.9% tried and 0.4% past year -Risk associated with injecting heorin -33 year follow-up data on addicts: 22% died (1/2 overdosed) and of the survivors- 80% not using and 20% being treated -Painkillers one of the fastest growing forms of sub. disorders (2.5% of college kids used oxycotin in last year) |
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Hallucinogens:
-Symptoms (esp. LSD&MDMA) -low dose/high dose -tolerance/withdrawal |
-drugs that cause symptoms of psychosis
LSD-spurs dopamine into the brain (like schizophrenia) Ecstacy (MDMA)- is both stimulant and hallucinogens -After sensory perception or create sensory experiences (ex. marijuana, LSD, mescaline, ecstacy) -low doses/potency: relaxation, shifts in attention, impaired memory -High doses: synthesia (exp. senses with other senses- eating color, hearing color), hallucinations, depersonalization, derealization -tolerance develops and dissipates quickly -withdrawal symptoms are uncommon |
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Hallucinogen: Marijuana (cannabis sativa)
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- stimulates canabinoid receptors
-Most common illicit drug -49% in college tried and 33% in last year -Adverse Effects: -longterm: impaired cognitive functioning -contributes to psych probs later -impares lung shape/function -Theraputic effects -reduces pain signaling -reduces side effects of chemo and treats glaucoma |
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Other drugs: Inhalants
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-EX. glue, gas
-significantlly impairing -volatile liquids that are breathed directly into the lungs -rapidly absorbed with effects like alcohol intoxication -tolerance and prolonged symptoms of withdrawal common -9% college kids tried |
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other drugs: Designer Drugs & Steroids
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-Rave type drugs
-produced by pharmacies for treatment of diseases -Ecstasy, MDEA (eve), Ketamine (Special K) all artificial drugs -heigtened auditory and visual perceptions, decreased inhibition, disorientation (roofies) -increased popularity in nightclubs -produce tolerance and dependence Steroids: synthetic substances to enhance muscles growth and secondary sexual characteristics to gain competitive edge |
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STUDY- Research on Addictiveness: Hastings
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-Experts rated "addictiveness" of commonly used drugs
-how easy is it to get hooked on a substance? -how hard is it to stop using? -Rankings reflect only addictive potential -all vary in levels of addictiveness -LEGAL: nicotine has highest addictive potential -ALCOHOL is in top 50% -Stimulants also in top percent |
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Substance Abuse: Biological Risk factors
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-high heritability for alcoholism (40-60%) but not clear cut b/c of envio. risk factors (may be overestimate)
- genetics for Alcohol may effect metabolism of alc. and sensitivity, and cravings. -high heritability for cocaine, opiates, and weed -8x more likely if relative has disorder -interact significantly w/ fam. and envio. factors -use highly likely determined by envio. factors -abuse/dependence determined by genes |
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Substance Abuse: Brain Regions
-Priming -Drug Cues -Cravings -Mesolimbic Pathway -THEORY: Reward deficiency syndrome |
-Priming: single drug dose leads to uncontrollable binging
-Drug cues: stimuli associated w/ drug leads to further drug use -Cravings: obsessive drive for drug use -Mesolimbic system:Tons od dopamine w/in mesolimbic pathway or "pleasure pathway" (lower d2 receptors) -goes from prefrontal cortex thru nucleus accumbus to ventricle tagmental area -Reward deficiency syndrome: fewer specialized dopamine receptors (d2) -everyday events aren't rewarding - seek reward/ pleasure thru substances |
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Substance Abuse: Environmental Risk Factors
-Stress induced relapse -Cognitive factors -Learning -emphysema |
-Stress induced relapse: an activation of certain brain related stress, such as cortisol, that help us cope but also increase dopamine activity in the mesolimbic pathway.
-Stress triggers dual responses in the brain 1) coping and 2) desire for reward -cognitive distortions- hard to break cycle that creates dependency on substance for anxiety relief -Learning: subtances dependence learned thru classic conditioning (enviornmantal cues) and substances create a positive reinforcement while the withdrawal creates neg. reinforcement. (operant conditioning) -Neg. effects often far off and not considered like EMPHYSEMA: lung disease marked by damage to air sacs and hard to breath. |
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Substance Abuse: Environmental Risk Factors
-Modeling -Personality -Family Factors |
-Modeling: ppl immitate their friends drug use especially between the ages of 13-16 yrs (esp. tobacco, marijuana, and alcohol)
-"addictive personality" are people high in impulsivity (risk taking, lack of planning, chaotic lifestyle, immediate gratification, and explosiveness) - impulsivity associated with dramatic PD's and especially psychopathy (ppl use drugs to cope with stress and "perceived threats") -Family F |
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• 5 factor model of personality:
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• Dimensional Perspective: personality traits differ in degree
• 5 factor model of personality: o Extroversion: High= talkative, assertive, active LOW= silent, passive, reserved o Agreeable: High= kind, trusting, warm LOW= hostile, selfish, mistrust o Conscientiousness: High= organized, thorough, reliable LOW= careless, unreliable o Neuroticism: High= nervous, moody, temperamental LOW= even temp., stable emotions o Openness to new experience: High= curious, imagine, creative LOW= practical, routine, focused |
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• Personality Disorders: Overview
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• Two reasons they differ from other MHD’s
• Distinct type of MHD- Axis II • Enduring stable thru life • Pattern across 4 domains/multi areas: o Cognitions: perceiving/interpreting self, others and events o Affect: range, intensity, ability, appropriateness o Interpersonal functioning o Impulse control o Like traditional disorders = maladaptive, causing distress &/or impairment |
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• Personality Disorders: Clusters (A,B,C)
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o Cluster A: Odd or eccentric (Prev = 2.1%)
• Paranoid • Schizoid • Schizotypal o Cluster B: Dramatic, emotional, erratic (Prev= 5.5%) • Antisocial • Borderline • Histrionic • Narcissistic o Cluster C: fearful or anxious (prev= 2.3%) • Avoidant • Dependent • Obsessive compulsive |
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• Cluster A: Paranoid PD
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o Pervasive and unjustified mistrust and suspicion
o Ppl “out to get them” o Disloyal and untrustworthy (blame other for their misfortunes) o Misinterpret events as threats to themselves o Hold grudges o Rigid, controlling, critical, blaming, jealous |
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• Cluster A: Paranoid PD Causes and treatments
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o Biological/ psychological factors= unclear
o Early learning that ppl/world =dangerous and untrustworthy • Cluster A: Paranoid PD treatments o Few seek treatments o Focus on developing trust o Cognitive therapy to counter neg. thoughts o Lack good outcome studies o Pervasive disorder but treat= lower impairment |
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• Cluster A: Schizoid PD
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o Pattern of detachment from social relationships
• Almost always choose solitary activity • No interest in sex/pleasurable activities/relationships • No close friends or fam. o Very limited range of emotions • Not effected by praise/criticism • Emotional coldness/lack of expression • Do not show paranoia or suspicion |
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• Cluster A: Schizoid PD Causes & treatment
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o Unclear
o Childhood shyness/ preference for social isolation like autism o Treatments: few seek treatment o Focus on value of interpersonal relationships o Building empathy and social skills o Lack of outcome studies |
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• Cluster A: Schizotypal PD
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o Think events have personal relevance (ideas of reference)
o Social anxiety/ paranoia- does not go away o Odd/ eccentric in beh. Or appearance o Inappropriate or constricted affect o Few friends outside immediate fam. o Perceptual and cognitive disturbances o Odd thinking and speech patterns o Differ from other PD’s: more odd behavior and more cog. Disturbances |
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• Cluster A: Schizotypal PD treatment/cause
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o Show brief transient psychotic episodes in response to stress (relatively short)
o Most likely to seek treatment for anxiety or mood disorders |
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• Cluster B: Antisocial PD
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o Failure to comply w/ social norms
o Disregard for /violation of others rights o Irresponsible, impulsive, deceitful o Lack of remorse (savvy and smooth talking) o Unlikely to maintain steady employment o Must be preceded by Conduct disorder |
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• Conduct disorder (Antisocial PD)
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o Aggression towards ppl/animals
o property destruction o deceitfulness or theft o serious violations of laws/rules o before the age of 15 to be relevant to ASPD |
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• Cluster B: Psychopathy
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o Related to but not synonymous w/ antisocial PD
o More interpersonal and affective traits that ASPD o Not in DSM b/c it takes more time clinically to assess and is not clear cut (behaviors) o Affect: callous, shallow emotion, say the “right” things but don’t have emotion behind them o Interpersonal: charming, manipulative, grandiosity, deceitful o Severe variant of ASPD o Goal directed to money, sex, and status o 1/3 of ppl with ASPD |
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• Cluster B: Antisocial PD Biological Risk
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o Sig. genetic contributions
o Under-arousal hypothesis: abnormally low levels of cortical arousal (limbic system) • Low heart rate, skin response, etc. o Fearlessness Hypothesis: abnormally low amygdala reactivity |
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• Cluster B: Antisocial PD psychological risk
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o Early history of behavioral probs.
o Harsh and inconsistent parental discipline o Conduct probs. Before 15 (to be able to diagnose) o Fam. w/ criminal and violent beh. |
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• Cluster B: Antisocial PD treatments
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o Few seek treatment on own
o Poor prognosis (esp. if early onset) o Emphasis on prevention and rehab o Often incarceration=only viable option o PREVENTION! Reduces prevalence |
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• Cluster B: Histrionic PD
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o Discomfort in situations where they are not the center of attention
o Emotions that shift rapidly and do not seem “real” o Suggestible/easily influenced o Inappropriately provocative or sexually seductive interactions o Phys. Appearance used to draw attention o Very dramatic beh. And expression of emotions o Relationships thought to be more intimate and close than they really are o Intense need to be loved, desired, and intimate w/ others o More in women |
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• Cluster B: borderline PD
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o On “border” of psychosis and neurosis
o Unstable mood and relationships (affect deregulation) o Impulsivity and FEAR of abandonment o Very poor self image o Self-mutilation and suicidal gestures o Chronic feeling of emptiness o Dissociations or paranoid thoughts in response to stress |
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• Cluster B: Borderline PD risk factors and comorbidity
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o Comorbid with: major depressive disorder
• Bipolar disorder • Sub. Abuse and bulimia o Suicide rates are high (10%) o Sig. genetic predisposition o Early trauma/abuse o Most frequently diagnosed PD in inpatient treatment o More in women o Agression/impulsivity= low seratonin |
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• Cluster B: Borderline PD treatments
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o Antidepressants
o Dialectical beh. Therapy =most promising |
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• Dialectical Behavior Therapy
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o Treatment focuses on dialectics
o Acceptance and change o Zen philosophy and mindfulness (being in the moment) o 3 simultaneous treatment methods • 4th treatment deals directly w/ therapist o 1. Individual sessions→ eliminate beh. That harm self, therapy, and life o 2. Skill training (group therapy): • mindfulness: not attaching to things • distress tolerance: dealing w/ intense neg. emotions • emotion regulation: identify, evaluate, control • interpersonal effectiveness: improve rel. while keeping self-respect o 3. Skills generalization (phone calls) |
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• Dialectical Behavior Therapy: Outcomes
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• Dialectical Behavior Therapy: Outcomes
o Reduces: • Self injury/suicide • Emergency/impatient treatments • Sub. Abuse o Improves: • Depression/aggression • Impulse control • Social adjustment |
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• Cluster B: Narcissistic PD
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o Exaggerated and unreasonable sense of self-importance
o Preoccupation w/ receiving attention o Lack sensitivity and compassion- “serial friendships” o Highly sensitive to criticism o Envious and arrogant o Marked grandiosity |
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• Cluster B: Narcissistic PD risk and treatments
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o Failure to learn empathy
o Treatments target grandiosity, building empathy, and lower unrealistic thoughts o Little evidence that treatment works o More in men |
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• Cluster C: Avoidant PD
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o Extremely sensitive to others opinions
o Highly avoidant of interpersonal Rel. o Interpersonally anxious and fear rejection o See self as inept, unappealing, and inferior o Want social interaction but are scared |
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• Cluster C: Avoidant PD causes and treatments
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o Difficult childhood temp. and early rejection
o Treat: similar to social phobia • Seek to improve social skills and lower anxiety (cog.beh. therapy) o More in women |
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• Cluster C: Dependent PD
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o Difficulty making simple decisions w/out advice/reassurance
o Fear of loss of support or approval = do not disagree w/ others o Desperate for support and nurturance o Giving up responsibility for major life areas o Extreme difficulty doing things alone b/c lack of self esteem o Helplessness and fear of being left alone o More in women |
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• Cluster C: Obsessive Compulsive PD
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o Excessive and rigid fixation on doing things the right way
o Preoccupied w/ details, rules, lists, etc o Highly perfectionistic → interferes w, completing tasks and deadlines o Miserly, stubborn, rigid o OBSESSIONS/CUMPULSTIONS =RARE |
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• Cluster C: Obsessive Compulsive PD risk and treatments
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o Unknown causes, weak genetic link
o More in women o Limited data on treatment o Address fears related to needs for orderliness o Rumination, procrastination, feelings of inadequacy |
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• Summary of PD’s
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o Long standing patterns of beh. (begin early and are chronic)
o Dimensional approach→ traits are apparent in everyone to some degree o DSM has 10 PD’s- 3 clusters o Causes start in childhood but are difficult to specify o Treatment= difficult and prognosis= poor |
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• Normal Sexual behaviors
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o Consensual activity
o Non-distressing to you or partner o Culturally/socially appropriate o Does not interfere w/ interpersonal rel., work, finances o Legal and private endeavor |
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• Sexual Dysfunction: overview
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o Disturbance in sexual response cycle
• Desire→ arousal→ orgasm o Or pain during sex o In both men and women equally o Effect 43% of women and 31% of men o Typically axis I |
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• Sexual Dysfunction: classifications
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o Lifelong vs. acquired (previous period of normal orgasm)
o Generalized vs. situational o Psychological factors only o Psych factors and Med. Condition |
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• Hypoactive Sexual Desire Disorder
• Sexual Aversion Disorder |
o 1st stage of SRC disrupted (desire)
o Low or no interest in sex o Masturbation, sexual fantasy, and intercourse =rare o 50% of all complaints @ sexuality o Little interest in sex o Extreme fear, panic, or disgust related to phys. Or sex contact |
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• Male Erectile Disorder
• Female Sexual Arousal Disorder |
o 2nd stage of SRC disrupted (arousal)
o impotence o difficulty achieving/ maintaining an erection o main reason men seek help o Inadequate lubrication o Problem =AROUSAL not desire |
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• Inhibited orgasm disorder
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o Female and male orgasmic disorder
o No orgasm despite adequate sexual desire and arousal o Rare in men common in women o Female orgasmic disorder: same except an orgasm may occur but is was less intense than usual |
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• Premature Ejaculation
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o Ejaculating too soon
o Not a problem during masturbation o Common in younger and inexperienced men o Most common disorder in men (21%) |
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• Dyspareunia
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o Extreme pain during sex
o Adequate sex. Desire o Ability to attain arousal and orgasm o Must rule out Medical reason |
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• Vaginismus
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o Perineal muscles at outer 1/3 of vagina spasm painfully
o Feeling of ripping, burning, or tearing |
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• Sexual Dysfunction: Biological and psychological Risk
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o Physical disease, prescription medication
o Use or abuse of alc./other drugs o Anti-hypertensive meds o Anxiety: worry during sex of pleasing others/self o Distraction→ Spectator Role: evaluating performance rather than enjoying it o Sexual dysfunctions increase w/ age |
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• Sexual Dysfunction: sociocultural risk and psych/physiological interaction
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o Erotophobia: learned neg. attitudes about sex
o Neg. or traumatic sexual experience o Deterioration of rel. and lack of communication o Blacks= lower desire/pleasure during sex o Whites= more pain during sex o “balancing scale”→ presence of risk factors tilts sexual performance towards successful or dysfunctional |
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• Sexual Dysfunction: assessment
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o Comprehensive interview: detailed history of sexual beh. Lifestyle, and associated factors
o Medical Exam: rule out sole med. Cause o Psychophysiological Evaluation: exposure to erotic material and determine extent/pattern of sexual arousal • Male penile plethysmograph • Female vaginal photoplethysmograph |
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• Sexual dysfunction: Biological treatments
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o Medications→ sildenafil= increases blood flow to penis to form erection
o Implants→ inflatable, semi-rigid, rigid o Vacuum system: penis is placed in tube to help draw blood to it o Antidepressants are used for premature ejaculation and compulsive sexual beh. o Vaginal lubricants or hormone replacement therapy for women w/ painful intercourse o Low sexual desire= testosterone hormone therapy (men) or estrogen/androgen hormone treatment (women) |
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• Sexual dysfunction: Psychological treatments
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o Sex therapy:
o Stop-start procedure: for premature ejaculation→ stimulate penis until ejaculation is about to occur then pinch top of penis to suppress stimulation and block ejaculation= longer erection o Sensate focus: low sexual desire→ ban sexual contact and rebuild couples sexual repertoire (and introducing fantasy training). Touching nonsexual, touching sexually, foreplay, etc. o Masturbation Training: orgasmic disorders/painful sex→ clitoral stimulation and prohibiting intercourse, gradually inserting dilators to increase vaginal size, relaxation tech. and kegal exercises o Sex schedule and increasing sex knowledge |
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• Paraphilia
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o Misplaced sexual attraction and arousal
o Focused on inappropriate ppl/objects o High comorbidity (anxiety, mood, sub abuse) o Several may be present @ once o Sometimes associated w/ violent beh. And/or rape |
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• Exhibitionist & voyeurism
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o Exhibitionist: exposure of genitals to unsuspecting strangers
• Elements of thrill and rick necessary for sexual arousal o Telephone scatalogia: sexual arousal via obscene phone calls o Voyeurism: observing an unsuspecting individual undressing and/or naked • Risk associated w/ peeping= sexual arousal o Abnormal b/c ppl = UNCONSENTING |
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• Fetishism and Transvestic fetishism
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o Fetishism: sexual attraction to nonliving objects (inanimate and/or tactile) like rubber, hair, feet , shoes
• Held, seen, smelled to achieve sexual arousal o Transvestic Fetishism: sexual arousal with the act of cross dressing • Helps person engage in the fantasy of being the opposite sex • Can be secret or obvious • Imagine themselves as female (autogynephilia |
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• Frotteurism
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o Physical contact w/ someone who has not consented
o Ex. Rubbing against someone in subway o Contact ranges form light in the form of “an accident” to very intrusive (grabbing boobs) o Fantasy of having long term relationship w/ victims |
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• Sexual Masochism and Sadism
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o Masochism: suffer pain or humiliation to attain sexual gratification
o Ex. Bondage, treated like a baby, verbal abuse, oxygen deprivation o Sadism: inflicting pain or humiliation to attain sexual gratification o Often about controlling the other person during sex |
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• Pedophilia
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o Sexual attraction to young children
o Victims= male/female kids of very young adolescents o Acts range from observation→penetration o Associated features: • Mostly men • Most rationalize their beh. (eg. Love or education) • Not distressed by actions o @least 16yrs old and victim 5 year age difference |
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• Pedophilia: Child Molestation
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o Someone who molests children may not be sexual attracted to them but may have sexual contact b/c they:
• lack partner own age • wish to hurt childs parents • intoxicated • cognitive/intellectual deficits |
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• Paraphilia: Risk factors
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o Biological: not well known but genetics may influence related factors (ex. Violence)
o Associated w/ sexual and social probs. • Patterns of inappropriate arousal and deficits o High sex drive coupled w/ suppression on urges o More common in men o Typical onset is early 20’s or adolescence |
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• Paraphilia Risk factor Taxonomy
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o 1) Early inappropriate sexual associations or experience (accidental or vicarious)
o 2) possible inadequate development of consensual adult arousal pattern OR o 2)possible inadequate development of appropriate social skills for relating to adults o 3) inappropriate sexual fantasies repeatedly associated w/ masturbatory activities and strongly reinforced o 4) repeated attempts to inhibit undesired arousal and beh. Resulting in (paradoxical) increase in paraliphic thoughts, fantasies, and behaviors o 5)paraphilia |
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• Paraphilia: assessment
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o Psychophysiological assessment: deviant pattern of sexual arousal
o Desired sexual arousal to adult consent o Social skills o Nature of adult rel. |
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• Paraphilia: Biological Treatments
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o Medications: equivalent to chemical castration
• Anti-androgens, low testosterone, sexual urges and fantasies (used for dangerous sex offenders) o Anti-depressants –SSRI’s- reduce sexual urges, diminish performance, or stop depression o Good efficacy: low sexual desire, fantasy, and arousal o Relapse is high if meds. =discontinued |
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• Paraphilia: psychological treatments
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o Most= behavioral : target deviant and inappropriate sexual associations
o Aversion treatment: paraphilia stimuli→ masturbate→punishment o Covert sensitization: Imagining aversive cons o Orgasmic Reconditioning: masturbate + appropriate stimuli o Masturbatory satiation: after orgasm, keep masturbating to paraliphic stimuli= irritating and boring (mild punishment) o Family/marital therapy: address interpersonal probs o Coping and relapse prevention: self control and risk management o Efficacy: 75%-95% show improvement but poorest outcome in rapists/multi paraphilics o Chronic course w/ high relapse rate |
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• Gender identity disorder
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o NOT a paraphilia
o Desire/arousal to be opposite sex (not for sex related reasons) o Biologically one sex and psychologically the opposite |
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• Gender identity disorder: treatments
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o Hormone therapy
o Gender reassignment prerequisites→ 1) live 1-2 yrs as opposite sex 2) hormone therapy 3) psychological, financial, and social stability o Up to 97% are stratified w/ surgery |
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• Schizophrenia: History
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o Emil Kraeplin- “Dementia Precox”
o Symptoms of catatonia, hebephrinia, and paranoia o Focus on early onset and “loss of mind” o Eugen Bleuler- “schizophrenia” o “splitting of the mind” b/w thoughts an beh. o Lead to common yet inaccurate confusion w/ multi personality dis. (DID) o Many of Kraeplins and Bleulers ideas are still practiced o Importance of understanding onset and course |
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• Schizophrenia: overview
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o Disturbances in thinking (delusions), perceptions (hallucinations), speech, emotions, and beh.
o 6+ Months o Schizophrenia Vs. psychosis o Psychosis: broad diagnostic category characterized by hallucinations and loss of contact w/ reality o Schizophrenia: type of psychosis o Schizophrenia and psychosis= heterogeneous o Symptoms aren’t necessarily shared w/ all sufferers o 2 clusters of symptoms: neg and pos. |
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• Schizophrenia: Positive Symptoms
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o Active manifestations of abnormal thoughts, feelings, beh.
o Sig. distortions of normal beh., thoughts, etc o Includes: • Delusions • Hallucinations • Disorganized speech/beh. |
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• Schizophrenia: Positive Symptom Delusions
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o Irrational beliefs that are gross misinterpretations of reality
o Highly fixed and resistant o Persecutory Delusions: irrational beliefs that one id being harmed/harassed in some way o Control Delusions: • Thought insertion: putting thought into their mind • Thought broadcasting: transmitting thoughts to everyone around you • Thought withdrawal: stealing thoughts and creating memory loss o Delusions of reference: everyday events have something to do with them o Grandiose Delusions: belief that one is especially powerful /important o Somatic delusions: ones physical body is neg. affected by external source |
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• Schizophrenia: Positive Symptom Hallucinations
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o Sensory exp. w/ no external input
o Can involve all senses o Auditory: voices o Tactile: bizarre skin sensations o Visual: seeing images or invisible visions o Olfactory: smelling unusual odors |
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• Schizophrenia: Positive Symptom Disorganized symptoms
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o Fragmented, illogical, incoherent
o Neologism: make up words o Clanging: saying words b/c they rhyme or repeating them o Tangentality: rapidly and randomly switching topics w/out warning o Echolalia: repeating others words o Echopraxia: repeating others actions o Alogia: not speaking @ all o Disorganized beh: inappropriate effect o Catatonia: unusual motor symptoms • Being unresponsive to envoi. Factors or excited agitation • Catalepsy/waxy flexibility: One body part staying in unusual position for long periods |
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• Schizophrenia: Negative Symptom
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o Absence/deficits I normal beh
o Avolition (or apathy): lack of initiation and persistence o Anhedonia: lack of pleasure from once pleasurable things o Affect flattening: unemotional o Lack of insight: poor awareness of MHD (think everyone else is “sick”) |
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• 4 Phases of schizophrenia
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o 1) Prodomal- peculiar beh. Such as minor disturbances in speech /thoughts, odd/withdrawn social interactions, perceptual distortions, anxiety or depression
• mostly neg. symptoms and may resemble depression o 2) Psychotic Prephase- first full-blown pos. symptom (such as hallucinations) for less than 2 months. Positive sym. Get stronger and person admitted to treatment o 3) Active phase: 6+ months (1 month of acute symptoms) of full blown positive and negative symptoms. Usually hospitalized. o 4) Residual Phase: following treatment but resembles prodromal phase (most people remain here most of their life) |
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• Schizophrenia Subtypes
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o Paranoid: persecutory or grandiose delusions and auditory hallucinations but no probs putting thoughts together and communicating (best prognosis for subtypes)
o Disorganized: fragmented, incoherent, odd and/or inappropriate speech, beh, emotions o Catatonic: abnormal motor movements- immobility, odd movements, excessive activity o Undifferentiated: mix of symptoms o Residual: after being treated for pos symptoms. Neg symptoms still in effect. Oddities in speech and ongoing perceptual differences o 3 dimensions: psychotic, disorganized, negative |
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• Video Case Study- Schizophrenia
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o Positive symptoms:
o Persecutory delusions: • Thought mom was gonna shoot him • Ppl are keeping his kids away o Disorganized beh. • Inappropriate affect- laughed at fiancé when she talked about his phases and smiled when calling her a liar o Neg Symptoms (harder to recover from neg. sym.): • Flat affect • Moderate lack of insight |
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• Schizophreniform Disorder
• schizoaffective Disorder |
o Only a few months (less than 6)
o Daily functioning not impaired o Resume normal lives o 2/3 become schizophrenic o symptoms of schizophrenia and mood disorder (are independent form each other)- depressive or bipolar subtype o prognosis like schizophrenia |
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• Delusional Disorder
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o Delusions contrary to reality
o Lack of positive or negative symptoms o Extremely rare better prognosis than schizophrenia o Delusions are NOT as bizarre o Erotomanic delusions: person loves them form afar o Grandiose delusions: special or powerful o Jealous delusions: spouse is having affair o Persecutory delusions: someone trying to hurt them o Somantic delusions: serious medical disease |
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• Brief Psychotic Disorder
• Shared Psychotic Disorder (folia due) |
o One or more pos. symptoms of schizophrenia
o Usually brought on by extreme stress/trauma o Lasts less than 1 month o Remits on its own o Postpartum psychosis o Delusions form one person manifests in another person o Little is know |
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• Schizophrenia: Epidemiology
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o Onset and prevalence WORLD WIDE
o About 0.33% → 0.72% o Age of onset= 22yrs old o Somewhat more frequent/severe among men o Generally chronic o Moderate to severe impairment o Life expectancy slightly lower than avg. o Most often victims of violence b/c of stigma= live on edge of society |
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• Schizophrenia: Biological risk factors
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o Strong genetic inheritance from twin and fam. Studies
o Inherit a tendency for schizophrenia (not subtypes) o Removal of subtypes in DSM V o Risk higher w/ genetic relatedness o Kids of ppl with schiz. = 12x more likely o -50% concordanance rate w/ identical twins o adoptions studies: risk for schiz. Still high o genes- polygenic model (lots of genes working together) |
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• Schizophrenia: Biological risks neurotransmitters
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o Excessive dopamine hypothesis
o Drugs that are high in dopamine (agonist) result in schiz. Beh. o Drugs that decreased dopamine (antagonists) reduce schiz. Beh. o Alladopa- side effects are parkinsins like symptoms o Dopamine receptors in striatal region of brain: D1 and D2 may be denser o Dopamine hypothesis=overly simplistic and problematic o Current Theories- emphasize many transmitters (glutamate) |
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• Schizophrenia: Brain Structure
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o Enlarged fluid filled ventricles and reduced TISSUE volume in cortical areas
o Hypofrontality- low active frontal lobes (organizes thoughts) o Viral infections during early prenatal development o Findings = not conclusive |
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• Schizophrenia: Psychological Risk Factors
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o Stress: may active underlying vulnerability and increase relapse risk
o Family intercations: ineffective communication patterns and high expressed emotion= associated w/ relapse (open criticism/pessimism and highly emotionally over involved) o Psychological Factorsexert only a minimal effect on schiz. |
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• Schizophrenia: Culture, Labeling, and stigma
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o More common in developing countries and among immigrants/migrant workers b/c of Social isolation and low social support
o More sensitive dopamine systems (found through animal models) o Labeling and stigma: Rosenhan (1973) “psuedopatients” STUDY o Acted as tho they had a psych disorder to be hospitalized→ started acting normally in hospital and DR’s interpreted the normal beh and “psychotic” o Neg. effects of labeling/stigma are strong and difficult to challenge |
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• Schizophrenia: Medical Treatments
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o Antipsychoti (neuroleptic) Meds: often the first line of treatment
o Most reduce/eliminate pos. symptoms o Acute and permanent side effects =common o Extrapyrmidial parkinsin side effects o Tardive dyskinesia- involuntary tics of the face, mouth, tounge and upper body (Repetative beh.) and potentially irreversible o Compliance w/ meds=problem |
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• Schizophrenia: Historical Psychological Treatment
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o Historicaly viewed as having poor prognosis
o Goals were to reduce neg. outcomes o Psychosocial approach involves: o Token economy o Social/living training o Cog. Vocational rehab (memory skills, work skills, etc) o Beh. Family therapy o Community care programs |
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• Schizophrenia: New Psychological Treatments
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o Stigma, trauma, and neurocognitive impairments create neg. selp-experience
o Treatment needs to help reconstruct self-experiences that are richer and more positive o Personal narratives: writing about self in detail helps to show them that they are more than their stereotypes o Metacognition-thinking about ones own thinking |