• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/80

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

80 Cards in this Set

  • Front
  • Back

Major Depressive Episode

Depressed mood most of the day, every day


Has lasted 2 weeks or more


Cognitive symptoms: feelings of worthlessness and indecisiveness


Physical dysfunction: sleep, eating, motor agnition/ retardation


Anthedonia


Duration 4-9 months, unreated

Anhedonia

lack of interest in things you love and no longer enjoying them

Manic Episode

Exaggerated elation, joy, euphoria, energy, or irritability


Can cause overconfidence which can lead to impulsive decisions that greatly impact life


Last 1 week or less


Cognitive symptoms: flight of ideas, grandiosity


Physical dysfunction: Reduced sleep, hyperactive, pressured speech


Duration 3 to 4 months, untreated


Hypomanic

just below diagnosis threshold, can still function

Unipolar Disorders

Depression or mania alone


Typically depression

Bipolar Disorders

Depression and mania


Mixed episodes

Major Depressive Disorder

No mania/ hypomania


single episode possible but rare


usually recurrent


average 4 to 7 episodes (lifetime)


Average duration 4 to 5 months

Prevalence in Major Depressive Disorder

16% lifetime, 6% annual


Onset: 30


Female:male ration 2:1

Prevalence in Bipolar Disorder

1% lifetime, .8% annual


Fairly consistent across cultures

Persistent Depressive Disorder (Dsythymia)

Milder symptoms


2+ years to get diagnosis


chronic and persistent


Average onset: early 20s


Early onset: before 21= greater chronicity, poor prognosis, stronger family history


Median duration= 5 years--depends on comorbidity

Double Depression

Major depressive episodes and dysthymia


Dysthymia occurs first


Severe psychopathology--poor course and high recurrancy

Common Misdiagnosis in Children and Adolescence for Mania

ADHD and conduct disorder

Biological etiology for mood disorders

Heritability-- 20% for men, 40% for women, higher for bipolar disorders


Low levels of seretonin= depression


high levels of dopamine= mania


Endocrine system: elevated cortisol, supressed hippocampal neurogenesis

Pharmacological Tx for mood disorders

SSRIs- sexual side effects and possible suicide risk for children


Tricyclics- can be dangerous if not taken properly


MAOIs- fewer side effects; lots of interactions with food and drugs


Litium( for mania)- very significant weight gain, takes client off his or her high, increase blood toxicity levels

Electroconvulsive Therapy

Brief electrical current to brain that induces seizures


6 to 10 Tx; high efficiancy for severe depression


Few side effects-- short term memory loss


Relapse is common

Etiology Psychological

stress and negative life events can trigger depressive episodes


learned helplessness/ hopelessness


Negatively based attributional process


cognitive triad (aaron beck)


Automatic thoughts/ cognitive errors


negative schemas/ core beliefs

Cognitive Triad (Aaron Beck)

Negative thoughts about self, world, future

Cognitive Behavioral Therapy

4 Phases


1. Behavioral Activation--Greater suicide risk


2.chellenging automatic thoughts


3. identify negative thinking biases


4. Changing primary attitudes

Automatic thoughts

Beliefs about the moment occurences in life


Attributions

explanations for events

Assumptions

Beliefs about the nature of the world on a basic level- develops in childhood

Schemas

basic beliefs about nature or self

Cognitive Tx for Mood Disorders

Thought records


1. Situation


2. Emotions


3. Automatic thoughts that precede emotion


4. Rational Response


5. Outcome

Internal/External

Degree to which the event is caused by something within oneself or outside world

Global/Specific

Degree to which the cause will influence many events or few events

Stable/Unstable

Degree to which the course will be present in the future or for only a limited time

Combined Tx for Mood Disorders

Possible benefits above individual Tx


48% benefit from meds or CBT alone


73% benefit from combined

Abuse

Interference with life; could impact education, job, family, etc

Dependence

Symptoms: Tolerance and Withdrawl

Depressants

Alcohol


Initially slows inhibition the inhibits judgement, coordination, reflexs, etc.

Effects of chronic alcohol use

Intoxication, withdrawl (delirium tremors), dementia, medical and social problems

Alcohol Statistics

Males>Females, binge drinking=23%


Disorders flunctuations in involvement


course of dependence: progressive


course of abuse: variable



Stimulants

Most widely consumed drug (US)


Amphetamine, cocaine, nicotine, caffeine


Increase alertness and energy

Effects of Amphetamine

"Up"- elation, vigor, reduced fatigue


"Crash"- extreme fatigue, depression

Effects of Stimulants

Highly addictive, family and financial destruction, risk of stroke (cocaine), physiological destruction

Opiates

Heroin, morphine, codeine, opium


Effective analgesis- pain relief


Opiates Effects

Euphoria, Drowsiness, slow breathing--fatality risk due to OD

Opiate Withdrawl

Severe illness for a few days


Symptoms: excessive yawning, vomiting, nausea, chills, muscle aches, diarrhea, stomach aches, insomnia

Hallucinogens

Marijuana, Psilocybin Mushrooms, LSD


Alter sensory perceptions


Can produce delusions, paranoia, and hallucinations

Marijuana

Most frequently used illegal drug


Become tolerant


Withdrawl and dependence uncommon but can become psychologically addicted

Causes of Substance Abuse

Genetic Predisposition


Environment: Acceptability, attitudes, access


Psychological:


Positive reinforcement- adding really good and high feelings/ tendency to seek rewards


Negative Reinforcement- escape from unpleasntness / self-medication/ tension reduction/ coping mechanism for negative effect

Agonist substitution

safer drug introduced

Antagonist drugs

block or counteract the effects of psychoactive drugs

Aversive Tx

perscribed drugs that make ingesting the abused substance extremely unpleasant

Psychological Tx for Substance Abuse

Impatient facilities


12 step program put on in AA and its variations


Contingency Managment


CBT helps prevent relapse

Dissociative Disorders

all have to do with serious loss in memory that is not organically caused

Amnesia

Loss of memory, usually traumatic events

Fugue

sudden travel with new identity or confusion

Tx for Fugue

None usually necesarry, onset and remission are both typically rapid

Dissociative Trance

Not in the DSM bc it doesnt occur in western cultures


Symptoms: sudden shift in personality, sterotypic behavior of being possesed, distressing to individual, not culturally accepted


Etiology: Life stress or Trauma


NO TX

Depersonalization and Derealization Disorder (DDD)

Feelings of unreality and detachment from self or surrounding


Severe/ frightening


Significant impairment

Etiology of DDD

People who have developed this disorder tend to have cognitive defects ( attention, short-term memory, spatial reasoning, easily distracted) and decreased emotional response

Statistics of DDD

.8-2.8% / female: male ratio 1:1


High comorbidies with anxiety and mood disorders


Tx has not been studied--Prozac not effective

Dissociative Identity Disorder

Dissociation of personality


adopt several new identities or alters--average 15


Unique characterisitics--no limitations


May be aware of each other or not

Etiology of DID

Severe abuse and trauma history- links with PTSD


High Suggestibility


Latrogenic

Latrogenic

may be inadvertly caused in Tx by well intentioned therapist asking leading questions

Statistics of DID

1.5% year/ Female: male Ratio= 9:1/ onset= childhood (typically before the age of 9)/ lifelong chronic course

DID Tx

CBT


reintegration of identities


identify and neutralize cues and trigger


visualization


coping skills


Antidepressants may help in some cases

Bulimia and Anorexia

Intense fear of obesity


Driven to be thin


Preocupied with food, weight, and appearance


High need for perfection--Struggle with anxiety and depression


Distorted body image


often begin after a period of dieting

Bulimia Nervosa

Binge eating followed by a compensatory behavior


Belief that popularity and self-esteem are determined by weight and body shape


Most are within 10% of normal weight

Medical Consequences of Bulimia Nervosa

Salivary Gland enlargment


Erosion of dental enamel


Electrolyte imbalance (potentially fatal)


Intestinal problems and permanent colon damage

Bulimia Nervosa Statistics

Female: Mostly caucasian females of middle to upper class, onset is teenage years, lifetime pervalence is 1.5% and is more prevalent among college women


Chronic if untreated


Male: Mostly caucasian middle to upper class, bisexual or gay, athletes with weight restricitons, onset is earlier

Anorexia Nervosa

Overly successful weight loss-- 15% below expected weight


Intense fear of gaining weight and losing control of eating


Relentless pursuit of thinness


Often begins with dieting


body image disturbance


pride in diet and control


rarely seek Tx

Anorexia Nervosa Medical Consequences

Amenorrhea, dry skin and brittle hair and nails, sensitivity to cold, lanugo, cardiovascular problems--low blood pressure and heart rate


Electrolyte imbalance


Severe malnutrition

Anorexia Nervosa Statistics

Mostly caucasian, middle to upper class


Onset=15 years


Chronic, resistant to Tx


Comorbid with anxiety, mood, and substance abuse disorders


Suicide is very prevalent with anorexia

Eating Disorders Etiology Social and Cultural

Cultural imperatives (Thinness= success)


Ideal body size standards-change rapidly and culturally bound


Media Standards


Social and Gender standards-femal perceptions of ideal weight is lower than males actual prefrences

Eating Disorders Etiology Psychological

Diminished sense of personal control in life


Low confidence and self-esteem


High standards of perfectionism


Preoccupation with how others view self


Isolation-both a cause and result


Mood tolerance- leads to binging and burging as an effect to regulate mood

Dyssomnias

problems with quality, quanity, and sleep onset


Insomnia, sleep apnea, and narcolepsy

Parasomnia

Abnormal behavior, physiological events: sleep terrors and nightmares


Type of sleep disorder assesment

Polysomnographic (PSG) evaluation

Electroencephalogram

(EEG) brain waves

Electrooculogram

(EOG) eye movement

Electromyogram

(EMG) muscle movement

Electrocardiogram

(EKG) heart activity

Actigraph

watch-like device that meausres wrist movement

Insomnia

Disrupted sleep or nonrestorative sleep


Prevalence 33% (year)


Female:Male= 2:1


Frequently associated with anxiety, deprsession, substance use (alcohol)


NUMEROUS CASES- biological, psychological, environmental...

Narcolepsy

Daytime sleeiness plus cataplexic attacks-- direct transition into REM sleep--sudden loss of muscle tone--triggered by strong emotion


Sleep paralysis


Hypnagogic hallcinations


Prevalence .03%-.16%


Female:Male 1:1


Onset= adolescense


Cataplexy, sleep paralysis and hallucinations improve over time, daytime sleepiness persisit

Hypnagogic Hallucinations

very vivid experience that can include all senses, occur at sleep onset (sensation of falling)

Sleep Apnea

10-20% lifetime prevalence, mostly male


Disrupted sleep at night and daytime sleepiness--restricted airflow, brief cessations of breathing, can be fatal but rare


Associated with Obesity


Tx: CPAP machine

Psychological Tx for Sleep-Wake disorders

Combined medication/ behavioral Tx


Stimulus control-improved sleep hyqiene


Modify unrealistic expectations about sleep


Reduce stress and anxiety


Relaxation techiques- Diaphragmatic breathing, progressive muscle relaxation