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43 Cards in this Set

  • Front
  • Back
Physical exam

What does Obesity/cyanosis suggest?
sleep apnea, RH failure
Physical exam

Tall w/ long arms and fingers
Marfan's syndrome
Phys exam

Cachexia
End stage heart failure
Phys exam

Slender, nervous
HyPERthryoidism
Phys exam

Slow, non-pitting edema
HyPOthryoidism
Phys exam

Febrile, ill, skin lesions
Endocarditis
What is pulsus paradoxus
systolic pressure fall >10mm Hg from cardiac tamponade (fluid fills pericardium)
- during INSPIRATION

(neg pressure --> RH^ --> septum pushed into left --> LH dimensions, stroke volume decrease --> drop in systolic BP
What are three positions to measure jugular venous waveform?
supine, 45 degrees, upright
What does jugular venous waveform tell us?
fluid status in right heart, cardiac rhythm
a-wave

abnormality
right atrial contraction

- giant = AV dissociation, ^RA volume, restriction to RV filling
c-wave
closure of tricuspid valve
x-descent

abnormality?
RA relaxation
RV contraction

- neg. slope = constriction (tamponade, pericarditis)
v-wave

abnormality?
RA filling

- ^ amplitude = tricuspid regurgitation
y-descent

abnormal?
tricuspid valve opening, RA filling

- decreased slope = impeded flow across tricuspid (stenosis, RV hypertrophy)

- increased slope = constriction (pericarditis, early disastole, limited RV filling, rapid atrial emptying)
Here is the PMI?

1. What does lateral displacement mean?
2. inferior displacement?
3. larger?
mid-clavicular, 5th intercostal

1. ventricular dilation (not hypertrophy)
2. obstructive lung disease
3. LV hypertrophy
How is the normal arterial pulse best assessed?
Bedside using the carotid artery
Instances of increased carotid upstroke?
1. AORTIC INSUFFICENCY
(LV SV^ = ^ systeolic volume = ^upstroke w/ peak amplitude
- aka "water-hammer pulse" (pulse pressure ^)

-reduced peripheral resistance
- sepsis
- anemia
- throtoxicosis
-peripheral AV malformation
-fever
What is Kussmail's sign?
paradoxic increase in jugular venous pressure during inspiration

- (inability of RH to accomodate increased flow, constrictive heart disease )
Classic example of increased carotid upstroke
aortic insufficiency
(water-hammer pulse)

^ LV volume = ^ systolic volume = ^upstroke
- reduced arterial resistance (compensatory ^ in CO)
What causes bisferiens pulse?

(spike and dome wave)
hypertrophy
obstructive cardiomyopathy
What causes pulsus Alternans?

(strong pulse followed by a weak pulse)
severe LV dysfunction
What causes pulsus parvus?

(diminished and delayed)
aortic stenosis
What are the four precordial sites for listening to heart sounds?
1. Aortic (2nd right interspace)
2. Pulmonic (2nd left interspace)
3. Tricuspid (4th left lateral sternal interspace)
4. Mitral (cardiac apex)
What generates S1?

When does it occur?

Which component occurs first?
close of AV valves

- prior to upstroke of carotid pulse

- mitral
What generates S2?

When does it occur?

Which component occurs first?
close of aortic/pulmonary valves

- after carotid pulse

- aortic (LV activated first + higher systemic resistance)
What happens to A2 and P2 during inspiration?

S2 splitting?
get further apart (RV filling^ = prolonged RV contraction = delays pulmonic valve closure)
What affects intensity of S2?

Factors affecting...
-leaflet mobility
- pumonary hypertension
- systemic hypertension

-afterload
-systole duration
-sequence of elec activation
What is a fixed split S2 pathognomonic of?
atrial septal defect

balance b/w systemic venous return & shunted blood results in preserved RA filling & RV stroke volume

A2 & P2 timing remain constant
When is S3 found?

What does it indicate?

causes? (2)
ventricular filling during DIASTOLE

congestive heart failure

1. decreased vent compliance (dialted cardiomyopathy)
2. ^ flow across AV valve (mitral regurg, aortic insufficiency, ventricular septal defect)
When is S4 heard?

What does it indicate?

cause
prior to ventricular activation, secondary to atrial contraction

ventricular hypertrophy

decreased ventricular compliance (arterial hypertension, aortic stenosis, hypertrophy)
When are ejection sounds heard?
early in ventricular systole secondary to blood flow across aortic or pulmonic valve
What does an aortic ejection sound indicate?
- biscupid aortic valve
- root disease
- coarctation
What does a pulmonic ejection sound indicate?
- valvular pulmonic stenosis
- pulm hypertension
- dialted pulm artery
How do you classify cardiac murmurs?

(Levine and Harvey scale)
1. timing
2. location
3. quality
4. intensity
5. pitch
Determination of the valve and condition requires what info? (5)
1. clinical scenario/other exam findings
2. timing of murmur (systole/diastole)
3. location of murmur
4. quality of murmur
5. response to maneuvers (right-handed get louder w/ inspiration)
What is a murmur caused by aortic stenosis characterized by?

How is timing related to severity?
harsh and diamond-shaped (crescendo-decrescendo)

- more severe, later the peak
What does a single S2 or paradoxical split S2 indicate?

What is is accompanied by?
severe aortic stenosis

pulsus parvus
What is a paradoxical split of S2?
P2 before A2
What characterizes mitral regurgitation?

Begins with? Ends with?

What is is frequently accompanied by?
-soft
-blowing
-high-pitched
-holosystolic murmur (lasting an entire systole)

- S1, A2

- S3
Where does murmur direct when the cause is:

1) anterior leafleft
2) posterior leaflet
1. axilla, thoracic spine

2. base of the heart
What characterizes mitral valve prolapse?

When is it more audible?

How is it affected by body position?
mid-systolic ejection click and late systolic murmur

when there is LESS blood in ventricle (more size-leaflet mismatch)

extended when upright
What characterizes hypertropic cardiomyopathy?

What increases its intensity?
systolic ejection murmur w/ increased intensity of S1 and preservation of S2

- maneuvers that ^ outflow obstruction
What characterizes aortic value regurgitation?

How is it best heard?
- diastolic
- high pitched
- blowing decrescendo murmur
- follow normal S2

- patient sitting, leaning forward, at end of expiration