Reading...
Play button
Play button
Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
11 Cards in this Set
- Front
- Back
|
Venous circulation of lower extermities
|
2 components:
Superficial veins- used for temp. control Deep venous channels 95% of blood Perforation or commmunication veins connect these 2 Venous valves prevent the backwards flow of blood |
|
|
Varicose veins
|
Dilated or tortuous veins that result from a sustained increase in pressure that causes the venous vavles to become incompetent, allowing for reflux of blood and vein engorgment.
Primary - orginate in syphanous vein Secondary- impaired flow |
|
|
Tx. for varicose veins
|
Elastic support stockings
Sclerotherapy(injection of sclerotic agent into collapsed vein to produce fibrosis) Surgical Tx. consists of removing the varicosities and the incompetent perforating veins |
|
|
Chronic Venous Insufficiency
|
Incompetent valves, venous tension
|
|
|
Clinical manifestations of CVI
|
Tissue congestion
Edema Skin atrophy Brown pigmentation of the skin Impaired tissue nutrition causes stasis dermatitis and the development of stasis or venous ulcers. |
|
|
Venous thrombosis
|
Presence of a thrombosis in a vein and the accompanying inflammatory response within the vessel wall.
Can develop in superficial and deep veins DVT most common in the lower extremities Can occur in upper extremities |
|
|
Risk factors for venous thrombosis
|
Venous stasis: Bed rest, immobility, Acute MI, CHF, Venous obstruction
Hyperreactivity of blood coagulation: Pregnancy, childbirth, oral contraceptive use, DEHYDRATION, cancer Vascular trauma: Surgery, Massive trauma or infection, fractured hip, orthopedic surgery |
|
|
Tx for Venous thrombosis
|
Elevation of legs
Heat bedrest anticoagulation therapy Prevent extension or mobilization of thrombi |
|
|
Clinical manifestation of VT
|
50% asymptomatic
inflammation deep muscle tenderness malaise increased ESR pain fever |
|
|
Difference between: CVI and PAD
CVI |
Pulses are normal but hard to locate
Ulcers medial, around ankle Edema, pitting edema(Pallymaliorlar, around ankle) Pain, dull/heavy discomfort(relieved with walking, compression stockings) Skin changes Purple, reddish, Brownish pigmentation. Skin is thin |
|
|
PAD
|
Decreased or absent pulse past the point of occulusion. Tibial
Wounds form- toes, foot, distal heel Limited/absent edema Complains of cramping pain when walking or exercising. Relates to where the occulusion is.(intermediate claudecasion) Pain r/t lactic acid build up. By product of metabolism(r/t anarobic metabolism). Pain subsides when at rest. Nocturnal pain relieved by hanging feet off the bed. Skin will be pale, cool to touch, loss of hair follicles |
