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94 Cards in this Set

  • Front
  • Back
5 frontal lobe syndromes
orbitofrontal
frontal convexity
medial frontal
massive lesion
Broca's aphasia
orbitofrontal syndrome p/w
disinhibition
impulsivity
frontal convexity syndrome p/w (2)
apathy
aggressive behavior
medial frontal syndrome p/w (3)
mutism
gait disturbance
urinary incontinence
massive frontal lesion p/w (2)
akinesis
abulia
3 parietal lobe syndromes
Gerstmann
alexia with agraphia
Balint
Gerstmann syndrome is caused by lesion in ___
dominant angular or supramarginal gyrus
Gerstmann syndrome p/w (4)
agraphia
acalculia
finger agnosia
L-R confusion
alexia with agraphia is caused by lesion in ___
dominant angular gyrus
Balint syndrome is caused by lesion in ___
this happens in context of ___
b/l occipito-parietal cortex
watershed infarct
Balint syndrome p/w (4)
optic ataxia
ocular apraxia
visual inattention
simultagnosia
3 Temporal lobe syndromes
Prosopagnosia
Klüver-Bucy
Wernicke's aphasia
prosopagnosia is caused by lesion in ___
nondominant or b/l temporo-occipital cortex
Klüver-Bucy syndrome is caused by lesion in ___
b/l medial temporal lobes
3 occipital lobe syndromes
anton syndrome
palinopsia
alexia without agraphia
anton syndrome is caused by ___ lesion and pw ___ (2)
b/l parieto-occipital
anosagnosia
preserved pupillary reflexes
palinopsia is caused by ___ (2) and pw ___
occipitotemporal lesion
recovery from cortical blindness
persistence of prior retinal image while gazing at another
alexia without agraphia is caused by ___ (2)
dominant occpital lobe lesion
splenium of corpus callosum
2 deficits associated with alexia without agraphia
R homonymous hemianopia
color anomia
3 syndromes of optic chiasm
anterior chiasm syndrome
body of chiasm syndrome
posterior chiasm syndrome
anterior chiasm syndrome pw ___ (2)
it is aka ___
ipsilateral monocular vision loss
contralateral superior temporal field cut
junctional scotoma
superior temporal field cut in anterior chiasm syndrome is caused by compression of ___ aka ___
contralateral inferior nasal fibers
Wilbrand knee
body of chiasm syndrome pw ___
bitemporal hemianopia
posterior chiasm syndrome pw ___ or ___
bitemporal scotoma with intact peripheral field
contralateral homonymous hemianopia
posterior chiasm lesion causes scotoma, i.e. ___, because ___
defect in macular vision
macular fibers decussate posteriorly
3 non-movement-related deficits from BG lesion
impaired learning
abulia
poor executive function
5 midbrain syndromes
Claude's
Weber's
Benedikt's
top of basilar
Parinaud's
Claude syndrome has ___ (2)
CN3 palsy
contralateral ataxia
ataxia in Claude's syndrome is due to ___ lesion
rubrospinal tract
Weber's syndrome has ___ (2)
CN3 palsy
contralateral hemiparesis
hemiparesis in Weber's syndrome is due to ___ lesion
corticospinal
Benedikt's syndrome has ___ (3)
CN3 palsy
contralateral ataxia
contralateral hemiparesis
Parinaud's syndrome has ___ (6)
upgaze palsy
impaired convergence
convergence retraction nystagmus
light-near dissociation
lid retraction
skew deviation
lid retraction in Parinaud is aka ___
Collier's sign
8 pontine syndromes
basis pontis
Millard-Gubler
Foville
Raymond
Raymond-Cestani-Chenais
Marie-Foix
AICA
locked-in
3 basis pontis syndromes
ataxic hemiparesis
dysarthria-clumsy hand
pure motor hemiparesis
Millard-Gubler syndrome results from lesion in ___
structures affected are ___ (3)
lateral ventral pons
fascicles of CN6
fascicles of CN7
CS tract
Millard-Gubler syndrome presents with ___ (3)
ipsilateral peripheral CN6 palsy (no conjugate gaze palsy)
ipsilateral CN7 palsy
contralateral hemiparesis
Foville syndrome results from a ___ lesion in ___
structures affected are ___ (4)
laterally extensive
medial ventral pons
fascicles of CN6
fascicles of CN7
CS tract
PPRF
Foville syndrome presents with ___ (3)
ipsilateral CN6 palsy (with conjugate gaze palsy)
ipsilateral CN7 palsy
contralateral hemiparesis
Raymond syndrome results from a ___ lesion in ___
structures affected are ___ (3)
non-laterally-extensive
medial ventral pons
fascicles of CN6
CS tract
Raymond syndrome presents with ___ (3)
ipsilateral abducens palsy
contralateral central facial palsy
contralateral hemiparesis
Raymond-Cestan-Chenais syndrome results from a lesion in ___
structures affected are ___ (3)
rostral pons
medial lemniscus
superior cerebellar peduncle
supranuclear oculomotor tract
Raymond-Cestan-Chenais syndrome presents with ___ (2)
ataxia
contralateral hypesthesia
ipsilateral gaze palsy
Marie Foix syndrome results from a lesion in ___
structures affected are ___ (3)
lateral pons
middle cerebellar peduncle
CS tract (basis pontis?)
spinothalamic tract
Marier Foix syndrome presents with ___ (2)
ipsilateral ataxia
contralateral hemiparesis
contralateral ST hypesthesia
locked-in syndrome results from a lesion in ___
structures affected are ___ (3)
bilateral ventral pons
CS tract
corticobulbar tract
CN 6 fascicles
locked-in syndrome presents with ___ (5)
quadriplegia
b/l facial palsy
b/l abducens palsy
dysarthria
aphonia
2 medullary syndromes
lateral
medial
LMS is aka ___
Wallenberg
8 structures affected in LMS are ___ (3-1-3-1, medial to lateral, dorsal to ventral)
dorsal motor nucleus of 10
NTS
CN8
descending autonomic fibers
N Ambiguus
spinal nucleus of 5
inferior cerebellar penduncle
lateral spinothalamic tract
LMS syndrome presents with ___ (6)
dysphagia
vertigo
ataxia
Horner's
ipsi trigeminothalamic hypesthesia
contra spinothalamic hypesthesia
3 structures affected in LMS are ___
CN12 nucleus
pyramidal tract
medial lemniscus
MMS presents with ___ (4)
ipsilateral tongue deviation
contralateral hemiparesis
contralateral ST hypesthesia
contralateral DC hyepsthesia
crossed motor hemiparesis is aka ___ and results from a lesion in ___
hemiplegia cruciata
inferior medulla
hemiplegia cruciata presents with ___
ipsilateral arm weakness
contralateral leg weakness
hydroxyamphetamine distinguishes ___ from ___ order Horner's syndrome
1st or 2nd
3rd
tilting head ipsi/contra to weak side worsens diplopia in CN4 palsy
this is called ___
ipsi
Bielschowsky's sign
modalities in CN7
GSA
GVE
SVA
SVE
nervus intermedius contains ___ (3) fibers and joins with ___ fibers at ___
GSA
GVE
SVA
SVE
geniculate ganglion
8 branches of CN7 from proximal to distal
greater petrosal
n. to stapedius
n. to auricular branch of X
chorda tympani
posterior auricular branch
n. to digastric
n. to stylohyoid
facial n. proper
CN7 deficits a/w hyperacusis localize to ___
the part proximal to the nerve to stapedius
Gradenigo syndrome is ___ (3)
it is caused by ___ which may result from ___ (3)
CN6 palsy
CN7 palsy
retro-orbital facial pain
lesion at petrous apex
OM
mastoiditis
nasopharyngeal Ca
2 kinds of aberrant regeneration after Bell's palsy
crocodile tears
gustatory sweating
crocodile tears is aka ___ (2)
gustatolacrimal reflex
Bogorad syndrome
9 parameters to distinguish central from peripheral vertigo
intensity
duration
position-dependency
n/v
direction of nystagmus
nystagmus modulation by gaze
fatiguability
latency
associated neuro sx
central/peripheral vertigo is more intense
peripheral
central/peripheral vertigo is continuous
central
central/peripheral vertigo is position-dependent
peripheral
central/peripheral vertigo has worse n/v
peripheral
direction of vertigo in peripheral vertigo
horizontal, rotational
2 features specific for central nystagmus
can be vertical
can change direction
central/peripheral vertigo has nystagmus affected by fixation
peripheral
nystagmus in peripheral vertigo does ___ with gaze fixation
dampens
central/peripheral vertigo has delay before nystagmus onset on provokation
peripheral
4 spinal cord syndromes
transverse myelopathy
Brown-Sequard
anterior cord
central cord
3 categories of abnormalities for SC syndromes
motor
sensory
reflexes
motor abnormalities for transverse myelopathy
LMN weakness at level of lesion, UMN below
sensory abnormalities for transverse myelopathy
hyperesthesia/dysesthesia at level of lesion,
loss of all modalities 2-3 segments below
reflex abnormalities for transverse myelopathy
hyporeflexia at level of lesion, hyperreflexia below
motor abnormalities for Brown-Sequard syndrome
LMN weakness at level of lesion
UMN weakness below
sensory abnormalities for Brown-Sequard syndrome
ipsilateral hyperesthesia at level of lesion
ipsilateral loss of proprioception/fine touch/vibration below lesion
contralateral loss of pain/temperature 2-3 segments below lesion
reflex abnormalities for Brown-Sequard syndrome
ipsilateral hyporeflexia at level of lesion
ipsilateral hyperreflexia below
motor abnormalities for anterior cord syndrome
LMN weakness at level of lesion
sensory abnormalities for anterior cord syndrome
pain/temperature hypesthesia
(preserved vibration/proprioception/fine touch)
reflex abnormalities for anterior cord syndrome
hyporeflexia at level of lesion
motor abnormalities for central cord syndrome
weakness at level of lesion
sensory abnormalities for central cord syndrome
cape hypesthesia for pain/temperature
reflex abnormalities for central cord syndrome
hyporeflexia at level of lesion
main cause of anterior cord syndrome
ischemia from ASA insufficiency
symptoms common to cauda equina and conus medullaris syndromes
flaccid LE paralysis
saddle anesthesia
urinary retention
sphincter hypotonia
diminished AJs
LMN findings
2 features of conus medullaris not present in cauda equina
UMN findings
symmetric deficits
2 features of cauda equina not present in conus medullaris
asymmetric deficits
anterior choroidal stroke infarcts ___ (2)
it pw ___ (3)
GPi
posterior limb of internal capsule
contralateral hemiparesis
contralateral hypesthesia
homonymous hemianopia