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94 Cards in this Set
- Front
- Back
5 frontal lobe syndromes
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orbitofrontal
frontal convexity medial frontal massive lesion Broca's aphasia |
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orbitofrontal syndrome p/w
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disinhibition
impulsivity |
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frontal convexity syndrome p/w (2)
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apathy
aggressive behavior |
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medial frontal syndrome p/w (3)
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mutism
gait disturbance urinary incontinence |
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massive frontal lesion p/w (2)
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akinesis
abulia |
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3 parietal lobe syndromes
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Gerstmann
alexia with agraphia Balint |
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Gerstmann syndrome is caused by lesion in ___
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dominant angular or supramarginal gyrus
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Gerstmann syndrome p/w (4)
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agraphia
acalculia finger agnosia L-R confusion |
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alexia with agraphia is caused by lesion in ___
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dominant angular gyrus
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Balint syndrome is caused by lesion in ___
this happens in context of ___ |
b/l occipito-parietal cortex
watershed infarct |
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Balint syndrome p/w (4)
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optic ataxia
ocular apraxia visual inattention simultagnosia |
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3 Temporal lobe syndromes
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Prosopagnosia
Klüver-Bucy Wernicke's aphasia |
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prosopagnosia is caused by lesion in ___
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nondominant or b/l temporo-occipital cortex
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Klüver-Bucy syndrome is caused by lesion in ___
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b/l medial temporal lobes
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3 occipital lobe syndromes
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anton syndrome
palinopsia alexia without agraphia |
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anton syndrome is caused by ___ lesion and pw ___ (2)
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b/l parieto-occipital
anosagnosia preserved pupillary reflexes |
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palinopsia is caused by ___ (2) and pw ___
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occipitotemporal lesion
recovery from cortical blindness persistence of prior retinal image while gazing at another |
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alexia without agraphia is caused by ___ (2)
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dominant occpital lobe lesion
splenium of corpus callosum |
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2 deficits associated with alexia without agraphia
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R homonymous hemianopia
color anomia |
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3 syndromes of optic chiasm
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anterior chiasm syndrome
body of chiasm syndrome posterior chiasm syndrome |
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anterior chiasm syndrome pw ___ (2)
it is aka ___ |
ipsilateral monocular vision loss
contralateral superior temporal field cut junctional scotoma |
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superior temporal field cut in anterior chiasm syndrome is caused by compression of ___ aka ___
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contralateral inferior nasal fibers
Wilbrand knee |
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body of chiasm syndrome pw ___
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bitemporal hemianopia
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posterior chiasm syndrome pw ___ or ___
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bitemporal scotoma with intact peripheral field
contralateral homonymous hemianopia |
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posterior chiasm lesion causes scotoma, i.e. ___, because ___
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defect in macular vision
macular fibers decussate posteriorly |
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3 non-movement-related deficits from BG lesion
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impaired learning
abulia poor executive function |
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5 midbrain syndromes
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Claude's
Weber's Benedikt's top of basilar Parinaud's |
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Claude syndrome has ___ (2)
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CN3 palsy
contralateral ataxia |
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ataxia in Claude's syndrome is due to ___ lesion
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rubrospinal tract
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Weber's syndrome has ___ (2)
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CN3 palsy
contralateral hemiparesis |
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hemiparesis in Weber's syndrome is due to ___ lesion
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corticospinal
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Benedikt's syndrome has ___ (3)
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CN3 palsy
contralateral ataxia contralateral hemiparesis |
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Parinaud's syndrome has ___ (6)
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upgaze palsy
impaired convergence convergence retraction nystagmus light-near dissociation lid retraction skew deviation |
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lid retraction in Parinaud is aka ___
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Collier's sign
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8 pontine syndromes
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basis pontis
Millard-Gubler Foville Raymond Raymond-Cestani-Chenais Marie-Foix AICA locked-in |
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3 basis pontis syndromes
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ataxic hemiparesis
dysarthria-clumsy hand pure motor hemiparesis |
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Millard-Gubler syndrome results from lesion in ___
structures affected are ___ (3) |
lateral ventral pons
fascicles of CN6 fascicles of CN7 CS tract |
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Millard-Gubler syndrome presents with ___ (3)
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ipsilateral peripheral CN6 palsy (no conjugate gaze palsy)
ipsilateral CN7 palsy contralateral hemiparesis |
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Foville syndrome results from a ___ lesion in ___
structures affected are ___ (4) |
laterally extensive
medial ventral pons fascicles of CN6 fascicles of CN7 CS tract PPRF |
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Foville syndrome presents with ___ (3)
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ipsilateral CN6 palsy (with conjugate gaze palsy)
ipsilateral CN7 palsy contralateral hemiparesis |
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Raymond syndrome results from a ___ lesion in ___
structures affected are ___ (3) |
non-laterally-extensive
medial ventral pons fascicles of CN6 CS tract |
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Raymond syndrome presents with ___ (3)
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ipsilateral abducens palsy
contralateral central facial palsy contralateral hemiparesis |
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Raymond-Cestan-Chenais syndrome results from a lesion in ___
structures affected are ___ (3) |
rostral pons
medial lemniscus superior cerebellar peduncle supranuclear oculomotor tract |
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Raymond-Cestan-Chenais syndrome presents with ___ (2)
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ataxia
contralateral hypesthesia ipsilateral gaze palsy |
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Marie Foix syndrome results from a lesion in ___
structures affected are ___ (3) |
lateral pons
middle cerebellar peduncle CS tract (basis pontis?) spinothalamic tract |
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Marier Foix syndrome presents with ___ (2)
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ipsilateral ataxia
contralateral hemiparesis contralateral ST hypesthesia |
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locked-in syndrome results from a lesion in ___
structures affected are ___ (3) |
bilateral ventral pons
CS tract corticobulbar tract CN 6 fascicles |
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locked-in syndrome presents with ___ (5)
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quadriplegia
b/l facial palsy b/l abducens palsy dysarthria aphonia |
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2 medullary syndromes
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lateral
medial |
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LMS is aka ___
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Wallenberg
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8 structures affected in LMS are ___ (3-1-3-1, medial to lateral, dorsal to ventral)
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dorsal motor nucleus of 10
NTS CN8 descending autonomic fibers N Ambiguus spinal nucleus of 5 inferior cerebellar penduncle lateral spinothalamic tract |
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LMS syndrome presents with ___ (6)
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dysphagia
vertigo ataxia Horner's ipsi trigeminothalamic hypesthesia contra spinothalamic hypesthesia |
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3 structures affected in LMS are ___
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CN12 nucleus
pyramidal tract medial lemniscus |
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MMS presents with ___ (4)
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ipsilateral tongue deviation
contralateral hemiparesis contralateral ST hypesthesia contralateral DC hyepsthesia |
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crossed motor hemiparesis is aka ___ and results from a lesion in ___
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hemiplegia cruciata
inferior medulla |
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hemiplegia cruciata presents with ___
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ipsilateral arm weakness
contralateral leg weakness |
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hydroxyamphetamine distinguishes ___ from ___ order Horner's syndrome
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1st or 2nd
3rd |
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tilting head ipsi/contra to weak side worsens diplopia in CN4 palsy
this is called ___ |
ipsi
Bielschowsky's sign |
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modalities in CN7
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GSA
GVE SVA SVE |
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nervus intermedius contains ___ (3) fibers and joins with ___ fibers at ___
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GSA
GVE SVA SVE geniculate ganglion |
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8 branches of CN7 from proximal to distal
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greater petrosal
n. to stapedius n. to auricular branch of X chorda tympani posterior auricular branch n. to digastric n. to stylohyoid facial n. proper |
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CN7 deficits a/w hyperacusis localize to ___
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the part proximal to the nerve to stapedius
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Gradenigo syndrome is ___ (3)
it is caused by ___ which may result from ___ (3) |
CN6 palsy
CN7 palsy retro-orbital facial pain lesion at petrous apex OM mastoiditis nasopharyngeal Ca |
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2 kinds of aberrant regeneration after Bell's palsy
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crocodile tears
gustatory sweating |
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crocodile tears is aka ___ (2)
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gustatolacrimal reflex
Bogorad syndrome |
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9 parameters to distinguish central from peripheral vertigo
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intensity
duration position-dependency n/v direction of nystagmus nystagmus modulation by gaze fatiguability latency associated neuro sx |
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central/peripheral vertigo is more intense
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peripheral
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central/peripheral vertigo is continuous
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central
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central/peripheral vertigo is position-dependent
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peripheral
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central/peripheral vertigo has worse n/v
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peripheral
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direction of vertigo in peripheral vertigo
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horizontal, rotational
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2 features specific for central nystagmus
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can be vertical
can change direction |
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central/peripheral vertigo has nystagmus affected by fixation
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peripheral
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nystagmus in peripheral vertigo does ___ with gaze fixation
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dampens
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central/peripheral vertigo has delay before nystagmus onset on provokation
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peripheral
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4 spinal cord syndromes
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transverse myelopathy
Brown-Sequard anterior cord central cord |
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3 categories of abnormalities for SC syndromes
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motor
sensory reflexes |
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motor abnormalities for transverse myelopathy
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LMN weakness at level of lesion, UMN below
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sensory abnormalities for transverse myelopathy
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hyperesthesia/dysesthesia at level of lesion,
loss of all modalities 2-3 segments below |
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reflex abnormalities for transverse myelopathy
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hyporeflexia at level of lesion, hyperreflexia below
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motor abnormalities for Brown-Sequard syndrome
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LMN weakness at level of lesion
UMN weakness below |
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sensory abnormalities for Brown-Sequard syndrome
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ipsilateral hyperesthesia at level of lesion
ipsilateral loss of proprioception/fine touch/vibration below lesion contralateral loss of pain/temperature 2-3 segments below lesion |
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reflex abnormalities for Brown-Sequard syndrome
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ipsilateral hyporeflexia at level of lesion
ipsilateral hyperreflexia below |
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motor abnormalities for anterior cord syndrome
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LMN weakness at level of lesion
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sensory abnormalities for anterior cord syndrome
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pain/temperature hypesthesia
(preserved vibration/proprioception/fine touch) |
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reflex abnormalities for anterior cord syndrome
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hyporeflexia at level of lesion
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motor abnormalities for central cord syndrome
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weakness at level of lesion
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sensory abnormalities for central cord syndrome
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cape hypesthesia for pain/temperature
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reflex abnormalities for central cord syndrome
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hyporeflexia at level of lesion
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main cause of anterior cord syndrome
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ischemia from ASA insufficiency
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symptoms common to cauda equina and conus medullaris syndromes
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flaccid LE paralysis
saddle anesthesia urinary retention sphincter hypotonia diminished AJs LMN findings |
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2 features of conus medullaris not present in cauda equina
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UMN findings
symmetric deficits |
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2 features of cauda equina not present in conus medullaris
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asymmetric deficits
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anterior choroidal stroke infarcts ___ (2)
it pw ___ (3) |
GPi
posterior limb of internal capsule contralateral hemiparesis contralateral hypesthesia homonymous hemianopia |