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23 Cards in this Set

  • Front
  • Back
ST epithelial cells that secrete HCL & intrinsic factor
parietal cells
functions of HCl (2)
-digestion of food (activates pepsin)
-barrier against micro-organisms
functions of the digestive system (6)
1. food digestion
2. nutrient absorption
3. delivery of nutrition to blood for distribution
4. excretion of waste
5. secretion of hormones
6. fluid & electrolyte balance
GERD =
Gastro-Esophageal Reflux Disease
reflux of acid from the ST to esophagus
GERD
LES =
Lower Esophageal Sphincter
causes decrease in effectiveness in closure causing ST acid in lower esophagus
irritation/pressure change in LES
many GERD sufferers have a ____ of the ST through the diaphragm
herniation (hiatal hernia)
hypochlorhydria =
lack of ST acid
hyperchlorhydria =
high levels of ST acid
erosion of the mucosal lining of ST
gastric ulcer
erosion of 1st part of SI
duodenal ulcer
common causes of peptic ulcers (3)
1. H. pylori bacteria
2. increased HCl secretion
3. inadequate mucosal protection against ST acid
common contributing factors with peptic ulcers
-emotional stress
-alcohol
-smoking
-food allergies
-milk consumption
-manual labor
-genetic predisposition
-low fiber diet
growth of abnormally large breasts in males
gynecomastia
milky nipple discharge unassociated with breast feeding
galactorrhea
these drugs neutralize HCl in ST over period of weeks, giving time for ulcer to heal; relieve burning sensation of heartburn; Sx relieving/not curative
antacids
3 families of antacids
-Aluminum
-Magnesium
-Calcium
common SE's of antacids (mainly from overuse)
-diarrhea/constipation
-electrolyte imbalances
-acid rebound
-inadequate digestion
-more prone to infection
another use for antacids
replacement therapy for minerals
used in conjunction with other treatments to TX H.Pylori
Bismuth
bismuth effects on poop
will turn feces black
mechanisms of bismuth in the treatment of H.Pylori (2)
-antimicrobial
-helps to form mucosal lining for protection