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15 Cards in this Set
- Front
- Back
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seborrheic keratosis |
common, skin only benign proliferation of epidermal basal cells appear "stuck on" NOT moles treatment: can be removed for esthetics |
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dermatosis papulosa nigra |
form of seborrheic keratosis that occurs in 30% of blacks, skin only autosomal dominant multiple small dark brown-black papules scattered across zygomatic arch |
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achrocordon (skin tag) |
soft, PEDUNCULATED, small, skin-colored papules neck, eyelids, cheeks, chest treatment: removal for cosmetic purposes |
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sebaceous hyperplasia |
localized proliferation of sebaceous glands clinically: adults over 40 skin only often umbilicated small, less than 5mm treatment: excision for cosmetic purposes |
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basal cell carcinoma |
most common cancer locally invasive, slow spreading 85% on skin of head and neck etiology: chronic sun exposure clinically: pearly raised umbilicated painless papule often on face, skin only telangiectasias on edges eventual central ulceration metastases very rare less common clinicopathologic variants: pigmented BCC sclerosing BCC superficial BCC treatment: surgical excision |
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xanthelasma |
soft yellow plaques that occur mostly near inner canthus of eyelid (upper lid more than lower) etiology: half of these lesions are associated with elevated plasma lipid levels treatment: removal for cosmetic purposes treat lipid disorder as well |
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psoriasis |
onset in teens and twenties symmetrical distribution on scalp, elbows, knees etiology: activated T cells cause keratinocyte proliferation genetic component not well understood treatment: topical corticosteroids humira for psoriatic arthritis |
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actinic (solar) keratosis (AK) |
common in fair-skinned adults etiology: chronic long term sun exposure clinically: over 40 scaly plaque, white/grey/brown some peripheral erythema "feels like sandpaper" treatment: removal with excision |
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actinic cheilitis |
AK on vermillion of lower lip "sailor's lip" "farmer's lip" etiology: chronic sun exposure |
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squamous cell carcinoma of the skin |
second most common skin cancer after basal chronic exposure to sunlight causes most cases frequently begin as actinic keratoses that progress to have persistent "crusting and bleeding" treatment: surgical excision |
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effects of chronic sun exposure |
wrinkles irregular pigmentation with brown and red spots leathery, rough texture |
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cutaneous melanoma |
third most common skin cancer after BCC and SCC light skinned adults with history of sun burns are at risk proliferation of malignant melanocytes clinically: asymmetry, border irregularity color variation diameter greater than 6mm evolutionary changes treatment: excision |
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actinic lentigo (age spots, liver spots)
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90% of whites older than 70 chronic sun exposure flat, tan-brown 5mm-1cm well defined but irregular borders "big freckles" increase in melanin, not melanocytes |
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acquired melanocytic nevus (MOLE) |
benign proliferation of nevus cell (neural crest in origin) increased frequency in light skin, women, head and neck developmental stages: junctional nevus - nevus at junction of epithelium and CT clinically indistinguishable from lentigo simplex, children compound nevus - nevus cells at junction of epithelium/CT and in dermis, slightly elevated soft papule, teens and adults intradermal nevus - all nevus cells in CT, dome shaped, NO PIGMENTATION, hairs growing from in, less than 6mm |
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blue nevus |
uncommon variant of a mole second most common nevus of the oral cavity benign proliferation of melanocytes deep in CT blue due to the tyndall effect skin - hand, scalp, face oral - almost always palate treatment: none required biopsy to rule out malignant melanoma |
