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52 Cards in this Set

  • Front
  • Back
What are the 3 phyla of helminths of veterinary importance?
1. Platyhelminthes
2. Acanthocephala
3. Nemathelminthes (p.61)
What is the common name of worms belonging to the class Nematomorpha?
Hair worms or horse-hair worms
(p.61)
What phylum and class do helminths belonging to the superfamily Gordioidea belong to?
Phylum: Nemathelminthes
Class: Nematomorpha
Superfamily: Gordioidea
(p.61)
What type of bodies to roundworms (intraphylum: nematoda) have?
* Elongate cylindrical bodies with no segmentation
* Complete digestive tract (mouth to anus)
(p.61)
Are roundworms (intraphylum: nematoda) dioecious or monoecious?
Dioecious: separately sexed individuals
(p.61)
Common name: Nematodes
Rounds
Common name:Cestoda
Tapes
Common name:Trematoda
Flukes
Common name: Acanthocephala
Thorny headed
Body type: Nematodes
Cylindrical
Body type: Cestoda
Flat
Body type: Trematoda
Flat
Body type: Acanthocephala
Cylindrical
What is the only helminth type thats segmented?
Cestoda
What are the only helminths type that have a mouth
Nematodes and Trematodes
What are the only helminths type that have a GI Tract
Nematodes and Trematodes (blind tract)
What is the only helminth type that has an anus/cloaca?
Nematode
What are the only helminths type that have a body cavity
Nematodes and Acanthocephala
What are the only helminths type that have an anterior sucker
Cestode, Trematode, and Acanthocephala
What intraphylum do the roundworms belong to
Nematoda
Body type: roundworms
Elongate cylindrical bodies with no segmentation

Dioecious
Do roundworms have a body cavity?
yes
GI tract: roundworms?
Complete (mouth to anus)
Life cycle: rounds?
direct or indirect
Which helminths have an idirect life cycle?
* Nematoda (can be direct or indirect)
* Cestoda
* Trematoda
* Acanthocephala
5 types of esophagus in rounds?
1. Rhabditiform: anterior and posterior swelling, found in soil and plant nematodes, and pre-infective larval stages of several parasitic nematodes

2. Filaform: simple tube like structure

3. Club shapes: baseball bat

4. Bulbed: prominent bulb at posterior

5. Stichosome: stacked donuts
Diff in anus in male/female rounds?
Anus: females

Cloaca: Males
Development of nematodes
* Egg through 4 larval stages to adult

L1: hatches and feeds on fecal material molts to L2 then L3

* L3 retains the cuticle of L2 for protection

* L3 is the usual Infective stage
Nematodes: on/off host?
Egg-Molt L3 in environment or IH

L3-Adult cause infection in Dh
6 migration types of nematodes
1. Local migration (usually within gut and all paratitic stages found in same organ)

2. Hapatotracheal migration (gut through vasculature to trachea back to gut)

3. Lymphatic tracheal migration (gut through lymphatics to trachea back to gut)

4. Skin tracheal migration (Skin to trachea to gut)

5. Somatic (hepatic aortic, lymphatic aortic, skin aortic)

6. Direct tissue migration (direct penetration theough organs)
Characteristics of neatodes belonging to the superfamily Rhabditoidea
* Only females are parasitic

* direct life cycle

* one of the 1st parasites occuring in young animals

* thin shelled larvated eggs or larvae oassed in feces
nematode of importance belonging to the superfamily Rhabditoidea
* Strongyloides stercoalis
(intestinal threadworm)
Hosts and zoonicity: Strongyloides stercoalis
Small intestine of man, primates, and dogs
Possibly zoonotic
Infective Stage: Strongyloides stercoalis
L3
Life cycle: Strongyloides stercoalis
DIRECT LIFE CYCLE
L1: passed in feces
L3: two options: either become infective filiform larvae which penetrate the skin OR become free living adults, which sexually reproduce

* Susceptible host: L3 undergo cutaneous tracheal migr, molt in lungs, coughed up and swallowed, where they mature in small intestine; female burrow into mucosa and produce larvated eggs, which may hatch and burrow into the mucosa, carried to the lungs, and repeat the cycle in autoinfection (watery diarrhea, abdominal distension)

* Resistant host: L3 enter older host w/stronger immunity, undergo skin aortic migration (bypass the lungs), and enter various tissues including mammary gland; mobilized/transferred during first few weeks of lactation

* Transmammary transmission: major method of trans to pups; do not undergo further migration, but establish in small intestine
Sci. name for the hookworm?
Ancylostoma caninum
What super family does Ancylostoma caninum (hookworm) belong to
Stongyloidea
Chaacteristics of superfamily: Stongyloidea
* Found in intestines, resp. tract or kidney fat

* Direct life cycle

* Well developed buccal cavity and muscular culb shaped esophagus

* females pass thin shelled segmented eggs
2 families of the superfamily Stongyloidea
1. Ancystomidae

2. Strongylidae
Method of penetration of hookworms (Ancylostoma caninum)
SKin penetration
Hosts and zoonicity
Small intestine of dogs/canids
Cutaneous Larval Migrans
Life cyle: hookworms (Ancylostoma caninum)
DIRECT
Segmented oval eggs passed in feces

Susceptible host: L3 undergo skin tracheal migration

Resistant host: skin-aortic migration; with L3 becoming hypobiotic in somatic tissues (mammary glands, etc)
Egg type: hookworms (Ancylostoma caninum)
Segmented oval eggs
Phyla and Infraphylum: (Ancylostoma caninum)
Phyla: Nemathelminthes

Infraphylum: Nematoda
Characteristics of the family Strongylidae
* Found in L. intestine or renal fat

* Large mouth surrounded by leaf crown

* Plug or blood feeders

* Direct life cycle
What are Cyathostomes?
Callective name fiven to 6 genera and approx. 36 species strongylate worms in the large intestine of equids

Phyla: Nemathelminthes

Infraphylum: Nematoda

Family: Strongylidae
Common name: Cyathostomes?
Blood worms, small strongyles
Cyathostome of importance
Strongyloidea
Stongylidae
Hosts and zoonicity: Strongyloidea
Stongylidae
Large intestine of horse
Infective stage: Strongyloidea
Stongylidae
L3
Life cycyle; Strongyloidea
Stongylidae
DIRECT, Mucosal migration

Segmented eggs passed in feces;

L1 feed on feces and develop to L3

L3 stay in pasture 1w-1m, but can survive for 8m and get consumed when horse is grazing;

L3 enter mucosa of LI and molt to L4;

Some larvae arrest development in mucosa (L3, L4) – hypobiosis;
Number of larvae returning to lumen depends on season and population of adults (L3/L4 will overwinter in mucosa layer; removal of adults stimulates activity)

L5 remain in LI to breed and feed on blood/mucosa
Cyathostomiosis syndrome
1-4yr horse, late winter/early spring, after deworming, loosing body condition, non-febrile, diarrhea;
Larvae acquired in one grazing season become adults the next season