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503 Cards in this Set

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Classification tibial inter condylar eminence

Myers and McKeever


1. Minimal/no displacement


2. Elevated anteriorly with posterior hinge


3. Complete displacement



Goal: reduce fragment as well as possible



M&M recommend ORIF for 2&3

Describe technique for ORIF displaced tibial eminence #'s

Medial para patellar approach


Expose tibial spine, meniscus & ACL & removal any entrapment


Nonabsorbable suture or wire through distal ACL brought out distalky through holes drilled in prox tibial epiphysis avoiding physis



Arthroscopy technically challenging, use ACL guide



Account & Willis - ligament based or # based methods of fixation



Mal/non united #'s cause pain/instability & lack of extension



ORIF or femoral notch plasty

Complications of SH3/4 distal tibia

Growth arrest


Peripheral growth arrest from avulsion of perichondral ring


Resultant angular deformity

What is an osteoblasts?

Concerned with bone formation


Derived from local mesenchymal precursors


Form rows of cuboidal cells where new bone formed (trab/have radian systems)


Rich in Alk phos


Produce type 1 collagen & non village nous proteins for mineralisation bone matrix

What is an osteocytes?

OB either remains as quiescent lining cell or becomes resting osteocytes


In bone lacunae


? Function


Osteocytic osteolysis - PThH & Ca ion transport

Osteoclasts?

Principle mediators of bone resorption


Large multinucleated cells derived from Mono cytic precursors


Attracted by chemotaxis to prepared surfaces


Ruffled border is zone of attachment to bone


OC bone resorption carried out by lysosomal enzymes


Resorption surfaces - how ships lacunae

New bone formation occurs...

1. Endochondral ossification


Ossification of proliferating cartilage


Physis or bone repair


2. Membranous ossification


Direct Ossification


Sub periosteal new bone

Indications for SROM hinged prosthesis

Severe ligamentous insufficiency


Severe flexion or extension gap mismatch


Recurvatum deformity


Limb salvage procedures

What is patellar clunk syndrome?

Patella & hypertrophied synovium on undersurface of quads tendon can bind in box & cam mechanism


Hozack et al



Potential complication of PCL substituting knees

What is CMT ?

Hereditary sensory and motor neuropathy

What is patellar clunk syndrome?

Patella & hypertrophied synovium on undersurface of quads tendon can bind in box & cam mechanism


Hozack et al



Potential complication of PCL substituting knees

What is CMT ?

Hereditary sensory and motor neuropathy

Types of CMT

Hypertrophic & neuronal & infantile hypertrophic



Hypertrophic


Triple dose peripheral myelin protein. 22


Rep demyelinating & remyelination leads to nerve hyper trophy = onion bulb


Areflexia/symmetrical distal m weakness&wasting/g&S sensory loss



Neuronal


Neuropathy without demyelination


Reflexes intact/n n. Conduction velocity



Infantile hypertrophic = dejerine-sottas disease

Definition ankylosing spondylitis

HLA B27 seroneg spondyloarthropathy c SIJ & spine involvement

Definition ankylosing spondylitis

HLA B27 seroneg spondyloarthropathy c SIJ & spine involvement

Pathology in ank spond

1. Enthesitis


2. Synovitis diarthrodial synovial jnts



Enthesitis = inflammation of insertion of tendon/Lig/capsule into bone

Bamboo spine in which condition

Ank spond

Bamboo spine in which condition

Ank spond

Sero neg spondyloarthropathy

RF & ANA neg



Reiters


Ank spond


Psoriatic


Enteropathic

Bamboo spine in which condition

Ank spond

Sero neg spondyloarthropathy

RF & ANA neg



Reiters


Ank spond


Psoriatic


Enteropathic

Complications of ank spond

Spinal # (?neurol deficit - epidural haematoma)


Craniocervical instability


Pseudoarthrosis


Cauda equina (canal stenosis)


Periart arthropathy


Peripheral enthesitis


Extra skeletal manifestations



- ant uveitis


- aortitis


- pulm fibrosis


- colitis


- amyloidosis


- sarcoidosis


- prostatitis

X-ray features if ank spond hip/shoulder arthritis

Concentric jnt space narrowing


Osteophytes


Sub sclerosis


Bone ankylosis


Protrusio

Ank spond vs RA

Ank spond has...


M


Spine/SIJ/large jnt involvement


Enthesopathy


Aortitis


Uveitis


Bony fusion jnts


RF neg, HLA B27 pos

Ank spond vs RA

Ank spond has...


M


Spine/SIJ/large jnt involvement


Enthesopathy


Aortitis


Uveitis


Bony fusion jnts


RF neg, HLA B27 pos

Osteotomy levels for ank spond

C7/T1


Vert vsls mobile here, C8 n root most mobile, canal rel wide



L2


Localise conus


Closing wedge vert osteotomy

Ank spond vs RA

Ank spond has...


M


Spine/SIJ/large jnt involvement


Enthesopathy


Aortitis


Uveitis


Bony fusion jnts


RF neg, HLA B27 pos

Osteotomy levels for ank spond

C7/T1


Vert vsls mobile here, C8 n root most mobile, canal rel wide



L2


Localise conus


Closing wedge vert osteotomy

Diagnostic criteria ank spond

Modified New York classification



Clinical:


Improved LBP w exercise 3/12


Limitation spinal movement c/s planes


Decr chest expansion



Radiological:


Severe unilateral sacroilitis


Mild bilateral sacroilitis

Difference bw calcaneocavus & forefoot cavus

Midfoot cavus 2 DF calc only


Apex in tarsus



Forefoot cavus from PF 1st Ray

Difference bw calcaneocavus & forefoot cavus

Midfoot cavus 2 DF calc only


Apex in tarsus



Forefoot cavus from PF 1st Ray

Stages in Cavovarus

1. Flex cavus & 1MT


2. HF mobile, fixed 1st MT equinus


3. Fixed HF, fixed lesser toes


4. Bony changes

Difference bw calcaneocavus & forefoot cavus

Midfoot cavus 2 DF calc only


Apex in tarsus



Forefoot cavus from PF 1st Ray

Stages in Cavovarus

1. Flex cavus & 1MT


2. HF mobile, fixed 1st MT equinus


3. Fixed HF, fixed lesser toes


4. Bony changes

Coleman block test

Tests correctibility of hind foot by eliminating forefoot deformity

What is Meary's angle

Talo1st MT angle


N= 0



Cavus >30

What is Meary's angle

Talo1st MT angle


N= 0



Cavus >30

Calc pitch

Line be 1st MT head to inf calc and plantar surface calc



N= <20


>30 AbN


Hind foot DF & Calcaneovarus

Describe anterior drawer for ankle instability

Foot PF 10


Anchor foot, with other hand translate tibia anteriorly


+'ve if pain or translation 3mm+



CFL plays no role



Talar tilt


Tests CFL


0-23 N

Describe anterior drawer for ankle instability

Foot PF 10


Anchor foot, with other hand translate tibia anteriorly


+'ve if pain or translation 3mm+



CFL plays no role



Talar tilt


Tests CFL


0-23 N

Signs of syndesmotic widening

1. Clear space (med border fib & lay border post fib1cm above plafond) 5mm or less on AP & mortise


2. Overlap fib & ant tib tubercle


10mm AP, 1mm mortise


3. (Stress films under GA)

Classification for talar dome OCD

Bernt & Hardy X-ray


1. Subchondral compression # w no visible frag


2. Partially attached osteochondral fragment


3. Fragment detached, undisplaced


4. Detached & displaced fragment



MRI


1. Undisplaced, stable


2. Undisplaced but unstable


3. Displaced, congruent with reduction


4. Displaced & incongruent with reduction


Classification OCD knee

Clanton & De Lee (same as B&H)


1. Stable, attached, undisplaced


2. Stable, partially detached, undisplaced


3. Unstable, completely detached, undisplaced


4. Unstable, completely detached, displaced



MRI (hefty)


1. Small signal change w'out margins


2. Clear margins, no fluid


3. Partially visible fluid


4. Completely visible fluid


5. Frag/detachment/displaced


ie loose

Arthroscopic classification of OCD knee

Guhl



2components


- integrity of cartilage


- stability



4components


1. Intact


2. Early separation


3. Partially detached


4. Crater with loose bodies

Poor prognostic features of OCD

Size > 2cm


Fluid behind lesion Hefti 4


Displaced


Cartilage breach

Pathogenesis lumbar canal stenosis

Degenerative disc disease -> rel hyper mobility of facet jnts -> ligamentous hyper trophy -> n root compression & venous congestion & HT -> intermittent neurogenic claudication

Define lateral recess

Lat border dura to med border pedicle

Classification for bone defects

Rand



1. Focal metaphyseal defect, intact cortical rim


2. Ext meta defect, intact rim


3. Combined meta & cortical defect

Differentiate bw CMF, CB, CS

CMF & CB both neoplasms of incomplete cartilage differentiation


CMF meta, no hyaline cart matrix, lack mitotic activity (<1/hpf)


CB Epi, chicken wire calcification


CS: hyaline cartilage matrix, Tumour permeation & mititic activity

Sagittal balancing principles

Changing distal femur only affects ext


Changing femiral component size only affects flexion


Changing prox tibia/insert affects flex & ext

Classification of myelopathy

Nurick classification



0 N or root symptoms


1 signs cord compression, N gait


2 gait difficulties, fully employed


3 gait diff prevent employment, walks unassisted


4 walks w assistance


5 WC or bed bound

Classification of myelopathy

Ranawat



1 pain, no neuro deficit


2 subj weakness, hype reflexes, dyssthesias


3 obj weakness, long tract signs, ambulatory


4 + non ambulatory

Causes of positive trendelenburg test

1. Pain in WB


2. Weak hip abductors


3. Shortened femoral neck


4. Subluxed/dislocated hip

Acquired dislocation hip after 1 yr due to

Sepsis


Muscle imbalance


Persistent traumatic dislocation



Less commonly


Charcot


TB

N neck shaft angle at birth

160


Decr to 125 later


<120 = coxa vara

Congenital coxa vara

Rare developmental disorder infancy/early childhood


Defect endochondral ossification medial calcar


When starts walking -bends into coxa vara



Hilgenreiners epi angle


N 25-35


>60 -> valgus osteotomy

Acquired coxa valga

'Mechanical' femoral neck shortening or overgrowth GT


Causes abd m at disadvantage



Childhood: rickets , bone dystrophies, perthes


Adolescence: epiphysioloysis


Any age bone softening: osteomalacia, FD, path #, infection, # mal union, Paget's

Acquired coxa valga

'Mechanical' femoral neck shortening or overgrowth GT


Causes abd m at disadvantage



Childhood: rickets , bone dystrophies, perthes


Adolescence: epiphysioloysis


Any age bone softening: osteomalacia, FD, path #, infection, # mal union, Paget's

PFFD classification

Aitken


A gap in femoral neck or subtroch region


B dysplasia & shortening


C absent femoral head


D a genesis of entire prox femur & acet


50% have distal anomalies

Acquired coxa valga

'Mechanical' femoral neck shortening or overgrowth GT


Causes abd m at disadvantage



Childhood: rickets , bone dystrophies, perthes


Adolescence: epiphysioloysis


Any age bone softening: osteomalacia, FD, path #, infection, # mal union, Paget's

PFFD classification

Aitken


A gap in femoral neck or subtroch region


B dysplasia & shortening


C absent femoral head


D a genesis of entire prox femur & acet


50% have distal anomalies

Treatment PFFD

A&B - mech fn hip jnt


Subtroch osteotomy


Bone grafts


LL



C&D


Amputation


Van Ness

Acquired coxa valga

'Mechanical' femoral neck shortening or overgrowth GT


Causes abd m at disadvantage



Childhood: rickets , bone dystrophies, perthes


Adolescence: epiphysioloysis


Any age bone softening: osteomalacia, FD, path #, infection, # mal union, Paget's

PFFD classification

Aitken


A gap in femoral neck or subtroch region


B dysplasia & shortening


C absent femoral head


D a genesis of entire prox femur & acet


50% have distal anomalies

Treatment PFFD

A&B - mech fn hip jnt


Subtroch osteotomy


Bone grafts


LL



C&D


Amputation


Van Ness

DD transient Synovitis hip

Perthes


SCFE


TB Synovitis


Juvenile chronic arthritis & ank spond

DD of perthes

Transient Synovitis


Morquio's


Cretinism


MED


SSD


Gauchers


Esp if bilateral

NH perthes as described by Herring 1994

A small % have a v diff course w recurrent loss of motion, pain, eventual poor outcome. Most children have mod prob in active phase & then improve steadily, eventually having a satisfactory outcome

Interpretation of positive trendelenburg

Pain: centre over hip to decr Abd pull, decr JRF



Pivot: sub/dislocated hip, femoral neck shortening



Power: Abd weakness

What is Baumann's angle?

Humeral capitellar angle


Reliably predicts carrying angle after reduction


N = 85-89


>5 degrees cf other side unacceptable

What is Baumann's angle?

Humeral capitellar angle


Reliably predicts carrying angle after reduction


N = 85-89


>5 degrees cf other side unacceptable

Schmorls nodes

Small radio lucent defects in subchondral bone, Central/axial disc protrusions

Scheuermann def

Ant very body wedging 5 degrees in 3consecutive vertebrae


Overall kyphosis angle >40

Scheuermann def

Ant very body wedging 5 degrees in 3consecutive vertebrae


Overall kyphosis angle >40

DD scheuermanns

Postural


Infective: discitis , OM, TB spondylitis


SED: look for defects in other joints

Klippel feil syndrome


Defn

Multiple ABN segments cervical spine


Failure of F or S 3-8 weeks gestation

Klippel feil syndrome


Defn

Multiple ABN segments cervical spine


Failure of F or S 3-8 weeks gestation

Associated conditions w Klippel Feil

Cong scoliosis


Sprengels


Renal aplasia


Mirror motions synkinesis


Cong heart disease


Cong C stenosis


Brainstem


Basilar invagination


AAI


Adj level disease

Sprengel deformity

In descended scapula


Small, rel wide, med rotated


Ass: K-F, kidney, diastematomyelia, scoliosis

Classification for bone stock in TKR revision

1. Contained metaphyseal defect: BG, TKR



2. Damaged metaphysis


A one condyle B both


Cement <1cm, augment <2cm, BG



3. Def metaphysis +/- collaterals +/- quads


1 bulk structural allograft


2. Tumour prosthesis/custom


3. Tantalum cones meta filling


4. Mesh/impaction graft

What is stress shielding

Describes phenomenon of proximal femoral bone density loss observed over time in the presence of solidly fixed implant

Tarsal coalition

****


Calcaneonavicular under 12 two thirds

Retro peritoneal lipomas - consider what in DD?

Adrenal myelolipoma


Angio myelolipoma


Liposarcoma

Features of lipoma to suggest malignant transformation

Thick or nodular septations


Evidence of invasion


Non fatty soft tissue component



<11% have intra substance calcifications

Def tarsal coalition

Congenital fibrous, cartilaginous or bony connection bw 2 + tarsal bones due to failure segmentation

Defn Blounts

Progressive pathological genu Varum centred at the knee

Describe N bowing pattern in children from birth

At birth- genu Varum


14m - neutral


3 - peak genu valgum approx 10 degrees


7- physiologic adult genu valgum approx 5-7 degrees

Features differentiating Blounts from N

N - symmetrical flare distal F/prox T



Blounts -


Often bilateral, asymmetrical


Progressive


Varus deformity focused at the prox tibia


Often severe


Sharp angular deformity


Metaphyseal beaking


Varus thrust

Angle measurement in Blounts

Drennan angle


Angle subtended by line joining metaphyseal beaks & line along lat border tibia



>16 degrees = 95% chance of progression


<10 = 95% chance natural resolution

Aetiology of blounts

Mechanical overload in genetically susceptible individuals



Dyschondrosis in medial prox tibial metaphysis

Blounts angular measurement

Drennan angle



Line connecting metaphyseal beaks


Line perpendicular to long axis tibia


Angle subtended bw 2 lines

Findings in quadrilateral space syndrome

Compression of axillary n (&post circumflex humeral artery)


Vague pain, worse on abd/ER


Deltoid & teres minor weakness/wasting



Supra scapular n compression: weak SST & IST

What is a lipoma

Benign tumour of mature adipocytes which is the most common soft tissue tumour

What is a HAGL lesion?

Humeral avulsion GH ligament


Specifically IGH ligament

What is is acromiale

Failure of fusion of adjacent ossification centres



Simulates acromion #

What is is acromiale

Failure of fusion of adjacent ossification centres



Simulates acromion #

Acromion ossification centres

4


Preacromion


Meso


Meta


Basiacromion - fuses by 12


All others by 22-25



60% bilateral

Components in lump exam

Site


Size


Consistency


Margin


Multiplicity


Tenderness


Trans illuminates

Diabetic ulcer classification

Wagner


0 skin intact, foot at risk


1 superficial ulcer


2 deeper f/t extension


3 deep abscess or OM


4 partial gangrene forefoot


5 extensive gangrene

Operation for interdigital neuroma

Neurectomy with nerve burial & release trans interMT ligament

Ankle instability study

Kerkhoffs Cochrane 2002 meta analysis


Early WB


- greater %


- reduced time


To return to work/sports

Definition scheuermann's disease

Wedging of more than 5 degrees in 3 adjacent vertebra & overall kyphosis angle >40

Definition of complete spinal cord injury

No voluntary anal contraction


AND 0/5 distal motor


AND 0/2 distal sensory scores ( no periianal sensation)


AND intact bulbocavernosis reflex



(If BC reflex absent - could be in spinal shock)

Definition of complete spinal cord injury

No voluntary anal contraction


AND 0/5 distal motor


AND 0/2 distal sensory scores ( no periianal sensation)


AND intact bulbocavernosis reflex



(If BC reflex absent - could be in spinal shock)

X-ray changes childhood discitis

Loss of lumbar lordosis


1/52 disc space narrowing


1-3/52 end plate narrowing


3/52 sawtooth erosion


Longstanding: scalloping


Vert Magna: resolved infections

Complications & incidence in laminectomy & discectomy

10% recurrence


5% arachnoid iris


2% infection


1% DVT


0.2% cauda equina


0.1% mortality

Complications & incidence in laminectomy & discectomy

10% recurrence


5% arachnoid iris


2% infection


1% DVT


0.2% cauda equina


0.1% mortality

Which provocative test is highly specific but not Sn for C cord compression or myelopathy

Lhermitte provocative manouver



Electric shock like sensations that pass through extremities when neck is flexed



Also in MS

Neurological level of injury is

The most caudal level of spinal cord that has N motor & sensory function



Ie c5 tetra plexus has N c5 function

Pt with hip flexion deformity & ank spond at increased risk of what postop THR

Anterior dislocation

Definition of Sn & Sp

Sensitivity = how good a test is at finding something if it's actually there


Or how often test will correctly identify a positive amongst all positives



Specificity = how accurate a test is against false positives


Or % of times test will correctly identify negative result

Causes of spinal canal stenosis

Cong vert dysplasia eg achondroplasia


Chronic disc protrusion


Facet jnt arthritis/hyper trophy


Paget's


Spondylolisthesis


Ligamentous flavin hypertrophy

Causes of spinal canal stenosis

Cong vert dysplasia eg achondroplasia


Chronic disc protrusion


Facet jnt arthritis/hyper trophy


Paget's


Spondylolisthesis


Ligamentous flavin hypertrophy

Canal stenosis measurements

On CT - more accurate


AP mid Sagittal <11mm


Interpedicular transverse <16mm

Classification for freiburgs infraction

Smillie


1. Subchondral # on MRI


2. Dorsal collapse of articular surface


3. Collapse dorsal MT head, plantar intact


4. Collapse entire head, jnt narrow


5. Severe arthritic changes

Classification lateral mass #

Lamina & pedicle #


Kotani


A. Separation


B. Comminution


C. Split


D. Traumatic spondylolysis

Diagnostic criteria for DISH

Flowing ossification AL aspect 4 contiguous vertebrae


Preservation disc height


Rel absence significant degenerative changes


Absence facet jnt ankylosis/SIJ ankylosis, sclerosis or intra articular Osseous fusion

Diagnostic criteria for DISH

Flowing ossification AL aspect 4 contiguous vertebrae


Preservation disc height


Rel absence significant degenerative changes


Absence facet jnt ankylosis/SIJ ankylosis, sclerosis or intra articular Osseous fusion

Associated conditions DISH

L spinal stenosis


C spine: dysphasia/hoarseness/sleep apnoea/myelopathy


# & instability

Genetic link in CTEV

PITX1

Genetic link in CTEV

PITX1

Associated conditions with CTEV

Hand (Streeter dysplasia)


Diastrophic dysplasia


Arthrogryposis


Tibia hemimelia


Myelodysplasia


Prune belly syndrome

Genetic link in CTEV

PITX1

Associated conditions with CTEV

Hand (Streeter dysplasia)


Diastrophic dysplasia


Arthrogryposis


Tibia hemimelia


Myelodysplasia


Prune belly syndrome

CTEV contractures:

CAVE


MF Cavus


FF Adductus


Hindfoot varus


HF equinus

Defn disc protrusion & extrusion

Protrusion: dist bw edges of herniation less than edges of base



Extrusion: dist bw edges of disc material >dist at base

Classification CP spine deformity

Weinstein



GP 1: dble curves, minimal pelvic obliquity


GP 2: large TL/L curve w marked pelvic obliquity

What is Botox?

Competitive inhibitor of presynaptic cholinergic Rc with a finite lifetime (usually 2-3m)

What is Botox?

Competitive inhibitor of presynaptic cholinergic Rc with a finite lifetime (usually 2-3m)

Goal of CP spinal surgery

Obtain painless, solid fusion w well corrected well balanced spine w level pelvis



Careful assessment of family goals & risk/benefit analysis

Pelvic surgical fixation techniques in CP spinal fusion

Galveston technique


Stability, truncal balance, level pelvis


Lamina S1 to PSIS & bw tables of ileum just ant sciatic notch

Definition HO

Formation of bone in atypical, extraskeletal tissues


Occurring spontaneously or after trauma, or within 2 m of neurological injury

Classification for HO

Brooker


1. Bone islands


2. Bone spurs with >1cm gap bw opposing surfaces


3. Bone spurs w <1cm gap


4. Apparent bony ankylosis

Definition DISH

Common spinal disorder of unknown origin characterised by back pain & stiffness


Flowing ossification along AL margins at least 4 contiguous vertebrae & no changes spondyloarthropathy or degenerative spondylosis


Almost universally along R side thoracic vertebrae due to protection from aorta

Stable TL burst # with no neurological deficits


Op vs nonop

Operative Rx provided no long term benefits


Does have increased complication rate

Definition of protrusio

Diagnosis made on the bases of AP pelvis X-ray demonstrating CEA > 40 & medialisation medial wall acetabulum past ilioischial line

What is pelvic incidence

Correlates most strongly with isthmic spondylolisthesis grade



PI = PT + PS



Angle subtended by the initial line from COFH to midpt sacral endplate& a line perp to centre of sacral endplate

PI angles

Constant in childhood 47 degrees


Incr in adolescence


Constant in adulthood 57

What is diastematomyelia

Fibrous/cartilaginous/bony bar creating longitudinal cleft in spinal cord



If cord does not reunite distally = diplomyelia (true cord duplication)

Define a complete spinal injury

A complete injury has no sparing of motor or sensory function below the affected level and must have recovered from spinal shock (ie intact bulbocavernosis reflex intact) before diagnosis can be made



ASIA A

Difference bw neurogenic shock & spinal shock

Neurogenic: hypotension & rel brady in acute spinal cord injury


Circ collapse from loss of sympathetic tone



Spinal: temp loss spinal cord fn & reflex activity below level of injury


- flaccid areflexic paralysis


- Brady & hypo


- absent bulbocavernosis reflex

Difference bw neurogenic shock & spinal shock

Neurogenic: hypotension & rel brady in acute spinal cord injury


Circ collapse from loss of sympathetic tone



Spinal: temp loss spinal cord fn & reflex activity below level of injury


- flaccid areflexic paralysis


- Brady & hypo


- absent bulbocavernosis reflex

Variables which correspond to short life expectancy in or with CA mets

Multiple spinal mets


Multiple extra spinal mets


Unresectable organ mets


Critically ill


SCI (c or I/c)


Aggressive CA eg lung, osteosarc

Cervical stenosis

Absolute <10mm


Relative <13mm canal diameter

Defn cervical spondylosis

Chronic disc degeneration & facet arthropathy that can lead to


Radiculopathy


Myelopathy


Discogenic neck pain

Aetiological classification of lumbar canal stenosis

Cong or acquired



Congenital:


Achondroplasia


SED


Osteopetrosis



Acquired:


Degenerative: ddd, spondyl,kyphosis, scoliosis


Iatrogenic: fusion


Misc: Paget's, DISH, ank spond


Tumour


Infection


Trauma

Defn spinal instability w spondy

4mm translation or >10 degrees angulation compared to adjacent segment motion

OI vertebra called

Codfish vertebrae w compression #


Kyphoscoliosus

Rickets defn

Decr in Ca (+/- PO4) affecting mineralisation at epiphysis of long bones



Brittle bones


Physeal cupping/widening


LLax


Skull flat


Rachitic rosary


Dorsal kyphosis (cat back)


Long bone bowing



Gross distortion maturation zone &poorly defined zone of prov Ca

Defn rickets

Disorder of mineralisation of osteoid matrix caused by inadequate Ca & PO4

Defn rickets

Disorder of mineralisation of osteoid matrix caused by inadequate Ca & PO4

Types of rickets

Vit D def (nutritional)


Vit D dependent (type 1&2)


Renal osteodystrophy


Hypophosphatasia


Familial hypophosphatemic (Vit D Rs)

Defn rickets

Disorder of mineralisation of osteoid matrix caused by inadequate Ca & PO4

Types of rickets

Vit D def (nutritional)


Vit D dependent (type 1&2)


Renal osteodystrophy


Hypophosphatasia


Familial hypophosphatemic (Vit D Rs)

Defn osteopetrosis

Metabolic bone disease caused by defective osteoclastic resorption of immature bone

Pathophys of osteopetrosis

Inability to cause acidification in clear zone & therefore preventing bone resorption

Osteopetrosis


2 types

AR - lethal


AD - Albers Schonberg disease


Deactivation of 3 genes:


CA 2


Alpha3 subunit of vacuolar pump


Cl Chnl 7

Osteopetrosis


2 types

AR - lethal


AD - Albers Schonberg disease


Deactivation of 3 genes:


CA 2


Alpha3 subunit of vacuolar pump


Cl Chnl 7

Associated conditions osteopetrosis

# long bones


Coxa vara


CN palsies: blind & deaf


OM - impaired WBC fn



Erlenmeyer flask


Rugger Jersey spine


Bone within a bone


Block femoral metaphysis



Give IF gamma 1 beta for AD form

Disproportionate dwarfism

Achondroplasia


SED


MED


Diastrophic dysplasia


Kniests dysplasia


Metaphyseal chondrodysplasia

Disproportionate dwarfism

Achondroplasia


SED


MED


Diastrophic dysplasia


Kniests dysplasia


Metaphyseal chondrodysplasia

Proportionate dwarfism

Cleidocranial dysplasia (dysostosis)


Mucopolysaccharidoses

Defn MED

A form of disproportionate dwarfism characterised by irregular delayed ossification of multiple epiphyses



Failure of formation of secondary ossification centre (epiphysis)


AD


COMP defect ch19


Type IX collagen COL9A1/2/3 causing defect in type 2 collagen (9 is the link protein )

Defn MED

A form of disproportionate dwarfism characterised by irregular delayed ossification of multiple epiphyses



Failure of formation of secondary ossification centre (epiphysis)


AD


COMP defect ch19


Type IX collagen COL9A1/2/3 causing defect in type 2 collagen (9 is the link protein )

Difference bw MED & perthes

MED:


Bilateral & symmetrical


No metaphyseal cysts


Early acetabular changes

Defn SED

Form of disproportionate dwarfism characterised by short trunk and caused by defect in secondary ossification centre epiphysis

Paed forearm #


Acceptable...

<9 yr


Complete displacement


15 angulation


45 malrotation



>9 yr


30 malrotation


10 angulation prox


15 angulation distally

Defn DMD

DMD is an x linked rc disease of dystrophin causing progressive prox m wasting



Scoliosis behaves like NM curves w rapid progression <2 degrees/mnth -> restrictive cardiopulmonary fn


Most advocate fusion at 20-30

Defn sequestrum

Necrotic bone which has been walked off from its blood supply & presents as a nidus for chronic OM

Defn DMD

DMD is an x linked rc disease of dystrophin causing progressive prox m wasting



Scoliosis behaves like NM curves w rapid progression <2 degrees/mnth -> restrictive cardiopulmonary fn


Most advocate fusion at 20-30

Defn sequestrum

Necrotic bone which has been walked off from its blood supply & presents as a nidus for chronic OM

Defn involucrum

Layer of new bone formation outside the existing bone of OM

Subacute OM radio graphic classification

1A&B lucency


2 metaphyseal w cortical bone loss


3 diaphyseal


4 onion


5 epi


6 spinal

Joints with intra articular metaphysis

Shoulder


Hip


Elbow


Ankle

Poor prognostic indicators in Paeds septic arthritis

Age <6 m


OM


Hip jnt


Delay >4 days to presentation

Indicators in septic arthritis to differentiate bw transient Synovitis

90% chance if 3/4


WCC >12,000


Inability to WB


Fever >38.5


ESR>40


CRP>2.0



Temp then CRP best predictors

Septic arthritis is a surgical emerg bc...

Chondrolytic effect from pus


Pressure effect from fluid contributing to risk of AVN

Action of Botox

Botulinum toxin A decr Ach levels in synaptic cleft by blocking presynaptic release of Ach peripherally

Action of Botox

Botulinum toxin A decr Ach levels in synaptic cleft by blocking presynaptic release of Ach peripherally

Action of baclofen

Acts as GABA agonist


Effects more centrally as an agonist at presynaptic GABA B away from NMJ


Primarily at spinal cord level to block mono & poly synaptic reflexes

How is poly manufactured?

Addition polymerisation occurs when a free radical added to monomer that contains C-C bond. Free radical breaks the dble C bond & occupies one of the sites. This results in another C having a free bonding site which then reacts with another free radical, and the reaction progresses

How do you improve wear characteristics of poly?

Irradiate material to increase cross linking to improve wear

How do you improve wear characteristics of poly?

Irradiate material to increase cross linking to improve wear

Creep

Time dependent deformation in response to a constant load

Stress relaxation

Time dependent decr in load reqd to maintain a material at a constant strain

American Rheumatism association criteria for Dx RA

5 out of following must be present for >6 weeks


1. Morning stiffness


2. Pain on motion or tenderness in at least 1 jnt


3. Swelling at least 1 jnt


4. Swelling of at least 1 other jnt


5. Poor mucin ppt from synovial fluid


6. Characteristic histo changes in synovium


7. Characteristic changes in nodules

Bearing surface in young F with DDH needing THR

C on C


V smooth, hard, strong, stiff, biocompatible, bioinert, does not corrode & has v low friction & wear


Linear wear rates minimal cf M on P

Classification systems of DR #

Fernandez


AO


Frykman


Melone



Fernandez: defines # according to mech of injury & useful for surgical planning

How to treat a comminuted intra articular DR #

Treat this operatively via ORIF utilising a volar Henry's approach through the bed of FCR using a variable angle locking locking plate to achieve anatomical reduction & stable fixation of the fracture fragments

What is Tinels sign

Tinels reported this eponymous sign in 1915


Repetitive percussion over the nerve causes tingling sensation in the cutaneous distribution of that nerve


And reproduces patients symptoms

What would be expected in NCS in CTS?

Incr latency


Decr amplitude & velocity

Classification of causes in CTS

Szabo classification



Anatomical: decr size, once contents



Physiological: neuropathic, inflammatory, altered fluid balance



Patterns of use: repetitive, WB w wrist extended, vibration

Classification of causes in CTS

Szabo classification



Anatomical: decr size, once contents



Physiological: neuropathic, inflammatory, altered fluid balance



Patterns of use: repetitive, WB w wrist extended, vibration

Defn blounts

Growth disorder of proximal PM tibial epiphysis secondary to combination of hereditary & developmental factors



Repetitive trauma to knee in overwt children when ambulating may overload growth plate (heuter volkmann principle)

Classification of causes in CTS

Szabo classification



Anatomical: decr size, once contents



Physiological: neuropathic, inflammatory, altered fluid balance



Patterns of use: repetitive, WB w wrist extended, vibration

Defn blounts

Growth disorder of proximal PM tibial epiphysis secondary to combination of hereditary & developmental factors



Repetitive trauma to knee in overwt children when ambulating may overload growth plate (heuter volkmann principle)

What constitutes a skeletal survey

Skull


Pelvis


Chest


C/t/L spine


Tubular bones one side AP only


Lat if asymmetry


H&F

Thurston Hollands sign?

SH2 injury with metaphyseal extension

Defn nonunion

Nonunion is the arrest of the bone healing process



US FDA


# that has failed to show progressive evidence of healing by 4-6 m after the injury

Defn nonunion

Nonunion is the arrest of the bone healing process



US FDA


# that has failed to show progressive evidence of healing by 4-6 m after the injury

Delayed union

# taking longer to show progression of healing than would normally be expected



Failure of # to unite within the expected time

Atrophic nonunion

Needs:


Apposition of biologically viable bone ends


Mechanically stable # fixation


Autologous osteoinductive bone graft application (crest is gold standard)


Healthy viable soft tissue envelope

# healing stages

McKibbin JBJS 1978


Haematoma


Inflammation


Neoangiogenesis


Soft callus


Hard callus

Callus

Woven bone formed by mineralisation of fibrocartilage matrix

Bone remodelling

In response to local stress/strain in accordance to Wolfs law

Creeping substitution

Primary bone healing


Advancing front OC


Followed by OB laying down bone matrix


N bone turnover mechanism

Healing of drill holes

Late medullary callus

Healing of drill holes

Late medullary callus

Inter fragmentary strain

Strain (motion) inversely proportional to # gap



Small gap & small motion = large strain


Large gap + small motion = small strain

Pulsed US in # healing

US is acoustic radiation at freq above human hearing. Mech energy transmitted to body as high freq pressure waves


Wolfs law



Multiple studies suggest efficacy w accelerated # healing



Heckman JBJS 1994


R DB RCT


Tibial dia #


Incr healing

Intra membranous bone formation

Essential in


-fetal development


-Fracture healing



Occurs without cartilage model



Embryonic flat bone( skull, max, mandible, clavicle, pelvis, subperiosteal long bone)


Distraction osteogenesis


Blastem bone (kids w amputations)


# healing w rigid fixation compression plating)


1 component of healing w IMN



Conditions w defects in IMO:


Cleidocranial dysplasia


CBFA1 (runx2) on chmm 6

Enchondral bone

Long physeal growth


Embryonic long bone formation


Non rigid # healing



Cartilage model


Chondrocytes produce cartilage


Absorbed by OC, bone replaces cartilage by OB


Occurs on metaphyseal side


Collagen X



Sox-9: regulates chondrogenesis


PTHrP- delays diff chondro in z of H


Hueter volkmann law

Compression across growth plate slows longitudinal growth



Tension accelerates

Groove of ranvier

Osteochondroma



Ring incr mech strength of physis

NSAIDs & # healing

Inhibit via prevent calcification of osteoid matrix



JBJS Br 2000


- significant


- 2/3 tibia # nonunion

Biofilm bacteria complex

Entity comprising bacteria in an extracellular matrix with a glycocalyx



Matrix is a vascular

Enchondral bone

Long physeal growth


Embryonic long bone formation


Non rigid # healing



Cartilage model


Chondrocytes produce cartilage


Absorbed by OC, bone replaces cartilage by OB


Occurs on metaphyseal side


Collagen X



Sox-9: regulates chondrogenesis


PTHrP- delays diff chondro in z of H


Hueter volkmann law

Compression across growth plate slows longitudinal growth



Tension accelerates

Groove of ranvier

Osteochondroma



Ring incr mech strength of physis

NSAIDs & # healing

Inhibit via prevent calcification of osteoid matrix



JBJS Br 2000


- significant


- 2/3 tibia # nonunion

Biofilm bacteria complex

Entity comprising bacteria in an extracellular matrix with a glycocalyx



Matrix is a vascular

Osteomyelitis

Cierny-mader system



Anatomy of bone involvement the subclassifies according to physiologic status of host


Define lesion & hosts ability to deal with it


Prognosis well correlated



1. IM


2. Superficial


3. Invasive localised w IM involvement


4. Invasive diffuse

General treatment of OM

Surgical debridement


Skeletal stabilisation


Soft tissue Mx


Space - dead space Mx


AB's



Goal: complete eradication of infection w preserved soft tissue envelope, healed bone segment, preserved LL & fn

Pathogenesis OM patterns with age

Truetas patterns


1. Infants: transphyseal vsls 1yr


2. Children: nutrient A meta, subperiosteal infection


3. Adult: infection spreads along shaft

Enchondral bone

Long physeal growth


Embryonic long bone formation


Non rigid # healing



Cartilage model


Chondrocytes produce cartilage


Absorbed by OC, bone replaces cartilage by OB


Occurs on metaphyseal side


Collagen X



Sox-9: regulates chondrogenesis


PTHrP- delays diff chondro in z of H


Hueter volkmann law

Compression across growth plate slows longitudinal growth



Tension accelerates

Groove of ranvier

Osteochondroma



Ring incr mech strength of physis

NSAIDs & # healing

Inhibit via prevent calcification of osteoid matrix



JBJS Br 2000


- significant


- 2/3 tibia # nonunion

Biofilm bacteria complex

Entity comprising bacteria in an extracellular matrix with a glycocalyx



Matrix is a vascular

Osteomyelitis

Cierny-mader system



Anatomy of bone involvement the subclassifies according to physiologic status of host


Define lesion & hosts ability to deal with it


Prognosis well correlated



1. IM


2. Superficial


3. Invasive localised w IM involvement


4. Invasive diffuse

General treatment of OM

Surgical debridement


Skeletal stabilisation


Soft tissue Mx


Space - dead space Mx


AB's



Goal: complete eradication of infection w preserved soft tissue envelope, healed bone segment, preserved LL & fn

Pathogenesis OM patterns with age

Truetas patterns


1. Infants: transphyseal vsls 1yr


2. Children: nutrient A meta, subperiosteal infection


3. Adult: infection spreads along shaft

Principles of Mx of intra articular #

Anatomical reduction


Early mobilisation

Conditions causing lumbrical plus

FDP avulsion


FDP transection


Too long tendon graft


Amputation through middle phalanx

Quadrigia effect

Characterised by active flexion lag in fingers adjacent to a digit with a previously injured or repaired FDP tendon



Most commonly due functional shortening of FDP


>1cm

Quadrigia effect

Characterised by active flexion lag in fingers adjacent to a digit with a previously injured or repaired FDP tendon



Most commonly due functional shortening of FDP


>1cm

Hook of hamate pull test

Hand in ulnar deviation


Flex DIPJ of RF & LF


Flexor tendons act as deforming forces on # site


Positive test = pain

Kaplans lesion

Most common IF


Complex irreducible dorsal dislocation MCPJ


MC head buttonholes into palm


Volar plate interposed bw base P1 & MC head

Classification dorsal PIP #/D

Hastings


Based on amt P2 articular surface involvement



1-stable. <30%


Dorsally based ext block splint


2. Tenuous. 30-50%


If reducible in flex, dorsally based ext block splint


3. Unstable. >50%


ORIF,hamate autograft or volar plate arthroplasty

Features Diastrophic dysplasia

Hitch hikers thumb


Club feet


Cleft palate


Cauliflower ears


Joint contractures


Dislocations


Scoliosis, C kyphosis,SBO, AAI


Quadriplegia

Femoral rollback

Progressive posterior change in the femoral-tibial contact point as the knee moves into flexion

Spot weld

Indicates stable osteointegration of an extensively porous coated implant

Bony pedestal

Bone accumulation within the canal below the tip of a mechanically loose stem



Keeps loose stem from subsiding further

Load relaxation

Time dependent


Non linear


Decrease in load

Spot weld

Indicates stable osteointegration of an extensively porous coated implant

Bony pedestal

Bone accumulation within the canal below the tip of a mechanically loose stem



Keeps loose stem from subsiding further

Load relaxation

Time dependent


Non linear


Decrease in load

Stress relaxation

Decrease in stress as tendon is subjected to constant strain over extended period

Prerequisite for efficient gait

Stable stance phase


Step length adequate


Sufficient foot clearance in swing


Swing phase foot prepositioning


Energy conservation

Planovalgus foot

Physiological


Pathological


- cong


- acquired



Cong: TACS


Tarsal coalition/acc Nav/CVT/skew foot



Acq: TRANC


Tib post/RA/arthritis/Neuro(CP,polio)/Charcot

Cavovarus foot

RINT



Residual club foot


Idiopathic


Neuro (CVA/CMT/CP/PNS/spinal cord/Spina bifida/polio)


Traumatic (tib ant rupture/mal United talar neck/compartment syndrome)

Concept of effective joint space

Well fixed cementless sockets causes expansile pattern of osteolysis



Radio lucent area starts at implant bone interface and expands



Joint fluid & wear particles flow according to pressure gradients & follow path of least resistance

Polio

Viral disease affecting anterior horn cells

Polio

Viral disease affecting anterior horn cells

Postpolio syndrome

Not a re activation


Aging phenomenon by which more nerve cells become inactive. Occurs after middle age



Treatment postpolio

When deformity overcomes functional capacity utilise standard polio surgeries: contracture release, arthrodesis & tendon transfer

Nonunion

US FDA


# occurred minimum 9 m prev & has no radio graphic signs of progression towards healing for 3 consecutive m

Tumours of vertebral body

Histiocytosis X


GCT


Chordoma


Osteosarcoma


Haemangioma


Mets


Marrow cell tumours

Tumours of vertebral body

Histiocytosis X


GCT


Chordoma


Osteosarcoma


Haemangioma


Mets


Marrow cell tumours

Tumours of posterior elements

OO


OB


ABC

Ranawat's guidelines for protrusio

1. <5mm protrusio -> no BG


2. >5mm protrusio & intact medial wall -> BG (no augmentation)


3. Uncontainable defect -> augmentation device

Frozen shoulder

Global reduction in shoulder motion associated with inflammation of capsule



No inflammatory cells


Active fibroblastic proliferation

Frozen shoulder

Global reduction in shoulder motion associated with inflammation of capsule



No inflammatory cells


Active fibroblastic proliferation

Frozen shoulder classification

Nevasier


- freezing (most painful)


- frozen


- thawed



Each 4-8 months

How do OC resorb bone?

Bind to surface of bone using integrin anchor proteins


Secrete protons & carbonic anhydrase system which dissolves HA mineral matrix


Also secrete proteolytic lysosomal enzymes which hydrolyse organic cellular components

Fixation of displaced sub capital NOF in child

Transphyseal # fixation provides the most reliable fracture stability and recommended in hip # >6 yrs despite risk of premature physeal closure

Acceptable alignment paed tibial shaft #

Children <10 any direction


Adolescent <5 in all planes

Name of curve describing physiologic LL alignment

Selenius curve


Birth 15 g varum


2 0


3-4 10


7 it's 7 degrees gvalgum

Angles in Paeds genu varum & valgum

Selenius curve tibiofenoral angle


Drennan angle >16 abN (N <11)


Meta/epi angle (N <20)

Main objectives THR

Pain relief


Stability


Mobility


Equal LL

Definition of anteversion of acetabulum

The angle the axis of acetabulum makes with the coronal plane of the body



Campbell's

Safe range in acetabular cup positioning

Lewinnek


Anteversion 15 +/- 10


Abd/inclination 40 +/- 10

Am Coll Rh arth defn

MAX RANS


Morning stiffness 1hr


Arthritis >3 areas >6/52


X-ray changes


RF


Arthritis hand >6/52


Nodules


Symmetrical arthritis

Defn strain

Deformation of a material when a given force is applied

AAOS recommendation for use of nuclear medicine imaging in Dx PJI

Nuclear imaging is an option in whom Dx has not been made & are not scheduled for Surg


WBC & Tc bone scan is most specific scanning method

Blood supply to bone

3 sources


1. High pressure nutrient arteries (endosteal circulation)


2. Meta-epiphyseal system


3. Periosteum (low pressure)

Causes acetabular dysplasia

DDH


Perthes


SCFE


NMD eg CP


Skeletal dysplasia eg MED, SED


Trauma


Infection

Symptomatic dydplasia

Asymptomatic - NH unknown


Symptomatic - poor prognosis


Untreated DDH 50% have oa at 50yrs

X-ray measurements acetabular dysplasia

Shentons line (cooperman- best prognostic line)


AI: N<20 paed


Sharps AA: adults N<42


CEA: N20-40


Sourcil/tonnis: N<10


Weinstein extrusion index: head covered/total width N<75


Tonnis angle

Measures inclination of WB zone


>10 AbN

Treatment options young symptomatic hip dysplasia

Supportive


Arthroscopy


PAO +/- femoral: congruous jnt, concentrically reduces hip


Salvage: unreduced hip & incongruent jnt


THA: established OA

Shelf

Incr WB surface by applying extra articular buttress of bone lat over Subluxed femoral head


Fibrocartilage cover

Sharps angle

N 33-38

Acet dysplasia angles

Tonnis N <10


Sharps N 33-38 (AI paeds)


CEA N 20-40


Ant CEA Lequesne (ant coverage femoral head) N>20 (<20 instability)


Weinstein extrusion index N>75

Principles of Mx of #

Obtain & maintain length, alignment & rotation

Sites of ulnar nerve compression

Cubital tunnel & guyons canal


2heads FCU/aponeurosis


Arcade of struthers


Bw osbornes Lig &MCL



Also: med head triceps, MIS, med epicondyle, fascial bands FCU, anconeus epitrochlearis, apon FDS


EXternal sources: #, med epicondyle nonunion, osteophytes, HO, tumours, ganglions

Gauchers disease

AR deficiency in B glucocerebrosidase


Accumulation sphingolipids

DDx for cubital tunnel

Local & systemic


Local: epicondylitis, R/m n entrapment, arthropathy, brachial plexus (pancoast), cervical radiculopathy


Systemic: peripheral neuropathy, mono neuritis multiplex, MS, leprosy

Larsens syndrome

Hypermobility


Multiple joint dislocations


Extra bones wrist & feet


Double ossification centre calc

Aspirin, heparin, warfarin

Aspirin irreversibly blocks TXA2 in plts, inhibiting aggregation


LMWH bind antithrombin which leads to conformational change & accelerates - thrombin (IIa) & + FXa


Warfarin - vitK dependent synthesis of Ca dep clotting factors 2,7,9,10

Lapland Cardinal line

Transverse line from 1st web space to hook of hamate, parallel to prox palmar crease


Just prox to sup palmar arch

Blood supply femoral head

Extra capsular arterial ring (MFCA, LFCA, sup & inf gluteal a)


Ascending cervical br - most impt Lat epiphyseal a


Intracapsular sub synovial ring of Chung



Also a of Lig teres, epi & meta supply

Classification subtroch #

Russell Taylor


1A- no piriformus exam, LT intact


1B- LT #


2A- piriformis ext


2B- LT #

Classification of FDP tendon avulsion

Leddy & packer


1 retraction to Palm


2 retraction to PIP


3 avulsion & # base distal phalanx

Creeping substitution

Process by which cancellous BG is incorporated. OB lay down new bone over old graft bone, which is subsequently reabsorbed

Osteoinductive

Recruit host mesenchymal cells to diff into OB for new bone formation

Osteoconductive

Form scaffold for new blood vsl ingrowth & new bone formation

The endurance limit is

The max stress under which a material will not fail regardless of how many cycles are applied to it

Osteogenic

Ability of cellular elements within graft to make new bone

X-ray features psoriatic arthritis

Pencil in cup


Jnt space narrowing


Peri articular erosions


Osteolysis


PIP/DIP ankylosis


Morningstar appearance


Asymmetric, oligoarticular


Carpus/MCPJ/PIPJ/DIPJ

Gout

Mono sodium irate crystal deposition disorder


Punched out peri articular erosions with sclerotic overhanging margins

Gout treatment

Acute:


Indomethacin 50mg tds


Colchicine (if PUD)


Or steroids if can't take above


Chronic:


Allopurinol (x oxidase inh)


Colchicine prophylaxis after recurrent attacks

Bone infarct

Interruption blood supply: intrinsic or extrinsic factors


RF's- like osteonecrosis


Smoke up a chimney


MRI central signal remains N marrow


Observe

FD

Developmental abN caused by failure of prod N bone


Associated:


McCune Albright (Coast of Maine)


Mazabraud


OFD


1% risk osteosarc or fibrosarcoma change

OFD

DDx adamantinoma


Rare form FD which prinarily affects ant cortex diaphyseal tibia, no periosteal rn


Osteoblastic rimming


Bowing, pseudarthrosis <30%


Usually regress

Causes of bowing/deformity

Congenital


Soft bone


Dysplasia


Physeal injury


# mal union


Pagets

DDx for lump at joint line

Meniscal cyst


Ganglion


Calcific deposits in collateral ligament


Meniscal extrusion


Tumours (s: lipoma, fibroma, b:OC

Principles of recurrent patella dislocation

Repair/strengthen MPFL


Realign extensor mechanism

Ultimate tensile strength is

Highest stress observed on s-s curve


Represents highest load/stress before material fails

Nora lesion characterised by

Florid reactive periostitis


Cartilage cap component

Secondary chondrosarcoma

Paget's


FD


Enchondromatosis- olliers & maffuccis


MHE

Signs of malignant change of enchondroma

Pain in absence trauma


Older age


Extension into soft tissue

Associations w spondylolisthesis

SBOcculta


T hyper Kyphosis


Scheuermann

RF for progression spondyl

Young age at presentation


F, slip angle >10


High grade slip


Done shaped or sacrum >30

Classification adult scoliosis

DINIT


Deg


Idiopathic - non treated AIS


NM


Iatrogenic


Traumatic

Bone scan Dx pattern in sacral insuff #

H shaped

Criteria for surgical Mx nonunion lat condyle #

Flynn


Large metaphyseal fragment


Displacement <1cm from jnt surface


Open viable lat condylar physis



SC closing wedge w ulnar n transposition

Aseptic loosening of m on m articulations

Characterised by perivasc infiltrate of lymphocytes and accumulation of plasma cells

Friction

Rs to sliding motion bw 2 bodies in contact

Knee dislocation classification

Schenck


1. D w ACL or PCL intact


2. ACL & PCL torn


3. ACL, PCL & either PLC or PMC


4. ACL, PCL, PLC, PMC


5. #/dislocation knee

Excursion distance in THR

Distance the head must travel to dislocate after primary impingement has occurred


Usually half width of femoral head

Large femoral heads in THR have

Greater primary arc motion and greater excursion distance


Once head/neck ratio


Incr volumetric wear

Indications for ORIF med epi #

Incarcerated epi within jnt


Associated dislocation


High demand w high valgus stress eg pitcher

Mucopolysaccharidoses

GP of metabolic syndromes characterised by absence/malfunctioning of lysosomal enzymes which break down GAG's



Morquio - incr keratin sulfate

Features of morquio syndrome

Spine: hyperL lordosis, T kyphosis, odo ihypo w instability


Hip dysplasia, coxa vara


Genu valgum


LL


N intelligence & facies


Proportionate dwarfism

Features of morquio syndrome

Spine: hyperL lordosis, T kyphosis, odo ihypo w instability


Hip dysplasia, coxa vara


Genu valgum


LL


N intelligence & facies


Proportionate dwarfism

Dorsal PIPJ #/ D

Hastings


<30% stable


30-50% tenuous


>50% unstable


ORIF/volar plate arthroplasty/dynamic ext splint/hemi hamate osteochondral reconstruction

Features of morquio syndrome

Spine: hyperL lordosis, T kyphosis, odo ihypo w instability


Hip dysplasia, coxa vara


Genu valgum


LL


N intelligence & facies


Proportionate dwarfism

Dorsal PIPJ #/ D

Hastings


<30% stable


30-50% tenuous


>50% unstable


ORIF/volar plate arthroplasty/dynamic ext splint/hemi hamate osteochondral reconstruction

Describe hemi hamate recon

Volar approach, shot gun jnt


# prepared, measure defect, matching graft harvested from hamate, fixed to base w appropriate length screws

Classification of Glenoid #

Ideberg


1-nonangulated/displaced


2A-short/displaced >1cm


2B- angulated >40degrees

Dupuytrens classification

Woodruff


1. Early palmar disease, no contracture


2. 1 finger, MCPJ contr


3. 2 + PIPJ


4. 3+ >1 finger


5. Finger in palm

Complications dupuytrens


20 yr rv

5% CRPS


3% digital n


2% dig a/infection/haem


Wound healing probs 23%

1st CMC OA


Rationale for Rx

Current evidence supports simple traoeziectomy over more complex procedures secondary to low complication rate & similar clinical outcome

Priorities for RA hand

Fireman: save my beer (relieve pain) dog (improve fn) remote (slow progression) & wife (improve appearance)


& address compressive neuropathy

What is sauve kapandji

Creates a distal R/U arthrodesis w ulnar pseudarthrosis prox to the fusion site to allow rotation

X-ray features RA wrist

Extensive pannus


Intra carpal supination


Volar subluxation


Ulnar translation


Caput ulnae


SLAC


Carpal collapse

Causes of swan neck

Primarily-lax volar plate (trauma, RA, LL)


Secondary-imbalanced forces PIPJ ie ext>fl


(Mallet/FDS rupture/intrinsic contracture/MCPJ volar sub

Treatment swan neck

Non op - dble ring splint


Op - volar plate advancement & PIPJ balancing w central slip tenotomy(fowler) or FDS tenodesis or SORL

Freidrichs ataxia

Progressive AR syndrome of early onset w decr or absent reflexes & predominant spine cerebellar in coordination

THR dislocation

3% primary


10-15% revision



Early >1 yr - surgical factors


Late >5 yrs - Mayo 0.8%

THR dislocation

3% primary


10-15% revision



Early >1 yr - surgical factors


Late >5 yrs - Mayo 0.8%

RF for dislocation THR

F, age >70


Less experienced surgeon


NM disorder, alcohol, DDH, Rev, AVN, takedown arthrodesis, rev # NOF, troch migration

Soft tissue injury classification

Tscherne & Gotz


0. Min ST damage, indirect, simple #


1. Sup abrasion, direct, mild-mod #


2. Deep contaminated, impending Comp.syndrome, severe #


3. Ext skin contusion/crush, cmpt syndrome, Severe #

Biofilm def

Complex aggregation of micro orgs which adhere to each other on a solid substrate

Stages of biofilm

Initial attachment


Irreversible attachment


Initial maturation


Further maturation


Dispersion

Causes of irreducibility of complex dislocation MCPJ

MC head Buttonhole bw flexor tendon (ulnar) & lumbrical (radial)


Volar plate


Sup trans MC Lig


Natatory Lig


Sesamoids

Classification for swan neck

Nalebuff (treatment options)


1. Flex hyperextension PIPJ


2. Hyperextension + intrinsic tightness


3. Limited flex PIPJ in all positions MCPJ (permanent lat band dorsal subluxation)


4. Destruction PIPJ

Radial club hand associations

Sonic hedgehog


1:100,000 bilateral 50-72%


Associations: VACTERL, VATER, TAR, Fanconi, Holt-Oram

Diastematomyelia

Cong anomaly caused by persistence in neuro enteric canal by Osseous/cartilaginous or fibrous tissue causing Sagittal division of spinal cord or cauda equina

Cauda equina

Constellation of symptoms that result from compression of terminal spinal nerve roots in LS region


Bilateral leg pain/B&B dysf/saddle anaesthesia/LL sensorimotor dysfn

RF for OM/infections

Start with all the 'I'


Infection - recent haematogenous


IVDU/Inflammatory (RA)/immunocompromised(DM,smoker, malnutrition, obesity


Trauma

Acute Ptolemaic OM


Rule of 50's

50yrs


50% in L spine


50% staph aureus


50% w epidural abscess have neurological compromise


50% elevated WCC



90% ESR/CRP elevated

Ank spond

Systemic chronic autoimmune seroneg spondyloarthropathy


Characterised by HLAB27, RF neg, primarily affecting axial spine



S: syndesmophytes, spinal apo physeal jnts, SIJ, symphysis pubis, entheSitis

Causes of nonunion

MAGI


Motion


A vascularity


Infection


Gap



Motion- inadequate stability


Avascularity-open#, stripping during surg


Infection


#gap-bone loss, nailed in distraction

Causes of nonunion

MAGI


Motion


A vascularity


Infection


Gap



Motion- inadequate stability


Avascularity-open#, stripping during surg


Infection


#gap-bone loss, nailed in distraction

Components of tibial nonunion exam

Union


Infection


Deformity


Joint above & below


NV exam

Signs of infection on xray

Harris & barrack jbjs96


Radio lucent lines


Focal osteolysis & endosteal scalloping


Periosteal new bone formation


Signs of infection on xray

Harris & barrack jbjs96


Radio lucent lines


Focal osteolysis & endosteal scalloping


Periosteal new bone formation


Signs of loosening on xray

Oneill & Harris jbjs84


RL lines >2mm bone/cement


Cement/prosthesis RL line


Cement #, prosthesis migration, bone destruction, femoral stem #

FD


Underlying prob

Shepherd crook


Think cAMP looking wrist

FD


Underlying prob

Shepherd crook


Think cAMP looking wrist

Carpal coalitions

<0.2%


Associated w tarsal coalitions & UL FofF def:


- arthro multiplex cong


- bird headed dwarfism


- hand foot uterus


- FAS


- Diastrophic dwarfism


- turners

Synovial chondromatosis

Rare, benign met aplasia of synovial membrane resulting in formation of multiple intra articular cartilaginous bodies that may/not calcify, self limiting, non aggressive most common in knee & hip


3-5th decade, M3:1


Extremely rare in children


1 & 2 forms

Synovial chondromatosis

Rare, benign met aplasia of synovial membrane resulting in formation of multiple intra articular cartilaginous bodies that may/not calcify, self limiting, non aggressive most common in knee & hip


3-5th decade, M3:1


Extremely rare in children


1 & 2 forms

Synovial osteochondromatosis


Diff bw 1 & 2nd forms

Primary- less common


Secondary- setting if pre existing OA/RA/Osteonecrosis/OCD/neuropathic osteoarthropathy/TB/osteochondral #

Clinical tests for TFCC

Fovea tenderness


Axial compression test


Piano key test

Clinical tests for TFCC

Fovea tenderness


Axial compression test


Piano key test

TFCC classification

Palmer


1. Traumatic


2. Degenerative


Each have A/B/C/D

Classification of multiply injured ligaments knee

Schenck - KD


KD 1 ACL +MCL/LCL


KD2 ACL + PCL


KD3M ACL + PCL +MCL


KD3L ACL + PCL +LCL


KD 4 all

Classification of multiply injured ligaments knee

Schenck - KD


KD 1 ACL +MCL/LCL


KD2 ACL + PCL


KD3M ACL + PCL +MCL


KD3L ACL + PCL +LCL


KD 4 all

Classification SNAC wrist & treatment

Watson


1 radial styloid beaking


2 radio scaphoid arthritis


3 mid carpal arthritis: SC & CL



SLAC wrist arthritis develops


@4yrs 75% had RS changes


@9yrs 60% had midcarpal ie grade 3 changes



Nonop


Op: 4CF- better strength & ROM


PRC (min age 35-40) risk of subsequent OA higher


Wrist arthrodesis

Functions of the meniscus

Transmit load across knee


Enhance articular conformity


Distribute synovial fluid across articular surface


Prevent soft tissue impingement


AP stabilisation of knee

Effect of partial meniscectomy

Resect <35% meniscus


Increases contact pressure 350%

Factors influencing meniscal repair

Location: red red


Tear pattern: long >1cm or radial to periphery


Quality: repair not indicated in macerated or degenerative tears

Causes of medial scapular winging

Based on direction top corner scapular - up & med


Med more common; usual young athlete


Serratus anterior deficit - LTN


- iatrogenic from anaesthetic, surgery


- trauma scapular #


- repetitive stretch eg weightlifter


- compression


- brachial plexus injury


- v rarely due to rhomboid dysfunction DSN injury



Can be mistaken for Parsonage Turner Syndrome in absence of trauma

Causes of medial scapular winging

Based on direction top corner scapular - up & med


Med more common; usual young athlete


Serratus anterior deficit - LTN


- iatrogenic from anaesthetic, surgery


- trauma scapular #


- repetitive stretch eg weightlifter


- compression


- brachial plexus injury


- v rarely due to rhomboid dysfunction DSN injury



Can be mistaken for Parsonage Turner Syndrome in absence of trauma

Causes of lateral scapular winging

Deficit in trapezius due to spinal acc nerve injury


Eg iatrogenic-LN dissection in post neck



Eden-Lange transfer

Causes of medial scapular winging

Based on direction top corner scapular - up & med


Med more common; usual young athlete


Serratus anterior deficit - LTN


- iatrogenic from anaesthetic, surgery


- trauma scapular #


- repetitive stretch eg weightlifter


- compression


- brachial plexus injury


- v rarely due to rhomboid dysfunction DSN injury



Can be mistaken for Parsonage Turner Syndrome in absence of trauma

Causes of lateral scapular winging

Deficit in trapezius due to spinal acc nerve injury


Eg iatrogenic-LN dissection in post neck



Eden-Lange transfer

Prognostic indicators of functional outcome following DR intra articular #

3/12: quality of reduction


<2mm step off,within 2mm height, restore carpal alignment


1yr:age&income


Michigan hand Qu scores

Blood supply to capitellum

Recurrent radial


Collateral radial


Recurrent interosseous

Flexor tendon repair

4 strand adelaide repair with 3.0 Ticron with 6.0 vicryl epi tendinous repair

Early passive motion protocols in

No op details


Soft tissue loss


2 strand repair

Early passive motion protocols in

No op details


Soft tissue loss


2 strand repair

Early active motion protocols flexor tendon repairs

Enhanced strength with reduced loss of fn


4 strand repair etc

Features of rickets

Defect in mineralisation of osteoid matrix caused by inadequate ca & phos



Rachitic rosary, codfish vert, bowing, retarded growth, dental disease, path #, waddling gait



Think hillbilly with bowed, waddling gait, poor teeth going cod fishing

Dorsal approach for foot fasciotomy

Double incision


Immediately medial to 2nd MT, immediately lateral to 4th MT

Incidence sciatic n palsy in THR

1-2% primary THR


3-4% Rev THR


5-6% DDH

Radio ulnar synostosis

Defect in longitudinal segmentation in 7th week


AD & sporadic, bilateral 60%


Associated general sk abN eg hip dysplasia, CTEV, LL, madelungs


Also syndromes: carpenter, apert, arthrogryposis, multiple exostosis

Theories for pain production in disc disease

Directly due to annulus micro tears


Nerve compression


Mechanical - incr load on post elements


Cytokines IL-1

Which approach


Transtectal trans w impacted roof #

Ext iliofemoral

Which approach


Transtectal trans w impacted roof #

Ext iliofemoral

Kocher langenbeck approach

Comm post wall #


PW & PC #


Simple PW#

Which approach


Transtectal trans w impacted roof #

Ext iliofemoral

Kocher langenbeck approach

Comm post wall #


PW & PC #


Simple PW#

Ant Column post hemiT

II approach

Pedowitz diagnostic criteria CECS

Preexercise pressure 15mmHg


1min post exercise pressure 30mmHg


5min PEP 20mmHg

Medial epicondyle #

Min displaced - slab then AROM 1/52


Irred incarceration & open # - Surg


CR: sup/valgus/WE


ORIF with ulnar n protection

Osteolysis

End result of a biological process when number of wear particles following joint replacement overwhelms body's capacity to clear them from circulation. Residual particles phagocytosis by Macrophages, release cytokines/inflamm that recruit OC to resorb bone

Blood supply patella

Desc genicular


Ant genicular


Sup/inf med & lat genicular


Form prepatellar arterial network


+ trans infrapatellar artery


= extraosseous


Also intraosseous from peripatellar anastomoses & trans infrapatellar a

Defn arthrogryposis

Generalised term for congenital Jon progressive limitation of joint movement due to soft tissue contractures affecting 2 or more joints

DD for Larsens

E-D syndrome



Larsens is similar to amyloplasia but instead of rigid joints, gross LL



This has multiple joint dislocations


Double ossification centre in calc is diagnostic

Defn arthrogryposis

Generalised term for congenital Jon progressive limitation of joint movement due to soft tissue contractures affecting 2 or more joints

DD for Larsens

E-D syndrome



Larsens is similar to amyloplasia but instead of rigid joints, gross LL



This has multiple joint dislocations


Double ossification centre in calc is diagnostic

Double ossification centre in calc is diagnostic of what

Larsens

Defn arthrogryposis

Generalised term for congenital Jon progressive limitation of joint movement due to soft tissue contractures affecting 2 or more joints

DD for Larsens

E-D syndrome



Larsens is similar to amyloplasia but instead of rigid joints, gross LL



This has multiple joint dislocations


Double ossification centre in calc is diagnostic

Double ossification centre in calc is diagnostic of what

Larsens

Defn arthrogryposis

General term for congenital, non progressive limitation in joint movement secondary to soft tissue contraction affecting 2+ joints

Defn CP

Non progressive injury to the immature brain incurred within first 2 yrs resulting in disorder of movement or posture

Ortho manifestations of CP

Contractures


#


Deformity


Hip subluxation/dislocation


Spine deformity


Foot deformity


Gait disorders

Ortho manifestations of CP

Contractures


#


Deformity


Hip subluxation/dislocation


Spine deformity


Foot deformity


Gait disorders

Classification CP

Type of motor disorder MAARS


Mixed


Athetoid


Ataxic


Rigid


Spastic

Classification CP according to limbs

Mono


Di


Tri


Hemi


Quad


Spastic pentaplegia


Cong knee dislocation

Associated:


DDH, CTEV, CVT, breech


Arthrogryposis, Larsens, downs, myelodysplasia-


Ie LLax/m imbalance

Classification coxa vara

Cong


Dev


Dysplastic


Acq


Physeal


Sub physeal

Classification coxa vara

Cong


Dev


Dysplastic


Acq


Physeal


Sub physeal

Causes of dysplastic coxa vara

OI


Rickets


Pagets


Renal


FD


Osteopetrosis


Hypothyr


Sk dysplasia: CCD, SED, Jansen Schmidt

Mode of action of cephalosporins

Bactericidal


Disrupt synthesis of peptidoglycan layer forming bacterial cell wall

Complications THR

Death 0.5-1% at 3/12


DVT Rx 10-30%


Fatal PE 0.2%


Infection 1%/3% rev


Dislocation 3%/10% rev


Sciatic n 0.5% femoral 0.1%


Vasc 0.25%


HO symptomatic 3-10%


LLD 10-20%

Complications THR

Death 0.5-1% at 3/12


DVT Rx 10-30%


Fatal PE 0.2%


Infection 1%/3% rev


Dislocation 3%/10% rev


Sciatic n 0.5% femoral 0.1%


Vasc 0.25%


HO symptomatic 3-10%


LLD 10-20%

Pedicle screw fixation starting point

Vary from level to level, pt to pt


Confluence of midpt of trans process, lat edge sup articular facet & superomed edge of mammillary process is approx entry point

AAOS clinical practice guidelines for surgical fixation DR#

Post red radial shortening >3mm


Dorsal tilt >10


Intra art stepoff >2mm

AAOS clinical practice guidelines for surgical fixation DR#

Post red radial shortening >3mm


Dorsal tilt >10


Intra art stepoff >2mm

?vit C post op ORIF DR#

Mod recommendation


500mg p/day 6/52

Extra capsular supply to femoral head

Desc br MCFA


Desc br LCFA


Asc br Sup gluteal


Inf epi gastric a



MCFA & LCFA br form extracapsular ring -> Asc cervical a


most impt lat epiphyseal a


-> sub synovial intra articular ring

Blood supply femoral head

Extracapsular


Medullary


Lig teres



Extracap: Desc Br MCFA/LCFA


Asc br sup gluteal, inf epi gastric a

Causes AVN hip

Traumatic


A traumatic - steroids/Alcohol



AS IT GRIPS 3C



Also:


IT (idio, trauma)


GRIPS (gaucher, gout , RTx, RA, infection, inflamm, pancreatitis, pregnancy, SLE, ScD, smoking


CRF, chemo, caisson

Bone graft in protrusio

Ranawat recommendations


<5mm no BG


>5mm + iliac med wall BG


Med wall def BG + cage augment/screws

Technical issues in THR in protrusio

Dislocation (insitu neck cut)


Med wall defect: rim fit, mesh & BG


Acetabular fixation: cup/cage


Femoral offset: needs incr


LL: incr


Sciatic n: often close

Teardrop

Radiological landmark that delineates the true floor of the pelvis


Lat limb: inf aspect ant wall acetabulum


Med limb: jn obt canal + a/inf portion quad plate

Classify bipartate patella

Saupe


50% bilateral


Ass N-P syndrome

Causes of paed equinovarus foot

CP spastic hemi


Duchenne


Residual CTEV


Tibial hemimelia

Classify tibial tubercle #

Ogden


1. # secondary oss centre near PT insertion


2. Propagates to prox jn with primary oss centre


3. Extends post to cross primary odd centre


4. Entire prox tib physis


5. Periosteal avulsion of ext mech from SOC

Prox tibia physeal closure order

Post to ant


Prox to distal

At risk w prox tibial tubercle #

Recurrent ant Tib artery

Classification met adductus

Bleck


HBL 2/3


Then 3/4


The 4/5 & unable to abduct forefoot beyond ML

Secondary causes CVT

Primary


Secondary:


Arthrogryposis


Myelomeningocoele


Sacral agenesis


DDH


Multiple pterygium syndrome

Small separated fragment of bone from growth plate in SH2

Thurston holland sign

McCune Albright

Unilat polyostotic FD


Cafe au lait (CofMaine)


Precocious puberty


Oncogenic osteomalacia FGF23

Indications for surgery in DM Charcot foot

RIP


Recurrent ulcerations


Infection


Pain

Osteoinductive BG

Ability of graft to induce or recruit pluripotent mesenchymal stem cells that differentiate into bone forming OB & chondroblasts

Osteoinductive BG

Ability of graft to induce or recruit pluripotent mesenchymal stem cells that differentiate into bone forming OB & chondroblasts

Osteoconductive BG

Refers to scaffolding


Provides environment for new bone apposition by supporting host cap ingrowth, perivasc tissue, OPG cells

Osteoinductive BG

Ability of graft to induce or recruit pluripotent mesenchymal stem cells that differentiate into bone forming OB & chondroblasts

Osteoconductive BG

Refers to scaffolding


Provides environment for new bone apposition by supporting host cap ingrowth, perivasc tissue, OPG cells

Osteogenic BG

By fresh cancellous BG contains viable donor Osteocytes or precursors which promote primary bone formation

Creeping substitution

Cancellous BG


Concomitant OB deposition of new bone osteoid & Osteoclast resorption of necrotic donor trabeculae

Standard surgical procedure for degenerative spondylolisthesis

Decompression laminectomy and partial medial facetectomy & instrumented fusion

CP is a non progressive permanent disorder of movement & posture sustained from a brain injury before the age of 2 yrs

Most common


Spastic diplegia or hemiplegia 65%


Higher in premature & Low birth Wt children

Aetiology of CP

Prenatal 44%


TORCH infections


Drugs/ETOH


Perinatal 27%


Hypoxia/prematurity/LBW


Postnatal 5%


Meningitis/head inj/immersion

Mercer Rangs stages of CP

1. Dynamic contracture


2. Fixed musculotendinous contractures


3. Bone & jnt involvement

NF

Inherited AD disorder of neural crest origin with skeletal/skin/ophthal abN

NF2

Acoustic schwannoma

Diagnostic criteria NF1

NIH: 2+/7 (62AO2LO1st)


6+ cafe au lait


2+ NF or 1 plexiform NF


Axillary/inguinal freckling


Optic glioma


2+ lisch nodules


Osseous lesion


1st degree relative

Treatment options tibia pseudarthrosis

IMN & BG


Vasc BG


Distraction osteogenesis


Rarely amputation

DD hemihypertrophy

Idiopathic


NF1


Beckwith Weidemann syndrome (cong o'growth syndrome)


Klippel-

Causes of hemihypertrophy

Idiopathic


Proteus


Beck with Weidemann


Klippel trenaunay weber syndrome


NF1

Causes of torticollis

CINB


Congenital muscular torticollis 80%


Inflammatory


Neurogenic eg Klippel feil


Bony malformation

Clinical test for glom us tumour

Love test


Application pinhead pressure over suspected area resulting in exquisite tenderness

Frozen shoukder

Idiopathic inflammatory condition characterised by progressive shoulder pain & dysfunctipn due to tightening capsulligamentous structures which spontaneously resolves

Frozen shoulder classification

Freezing: painful, capsular adhesions


Frozen: decr pain inc stiffness, capsular contractures


Thawing: decr stiffness

Frozen shoulder classification

Freezing: painful, capsular adhesions


Frozen: decr pain inc stiffness, capsular contractures


Thawing: decr stiffness

ER typically decr in Frozen shoulder


DD

Frozen shoulder


OA


Unreduced posterior dislocation

Most Sn indicator of Frozen shoukder

Pain on forced ER

Is Frozen shoulder inflammatory

Yes


Clear progression from perivasc at mono inflammatory infiltrates to reactive capsular fibrosis

5 main components of revision hip Surg

Previous Surg info


Classify bone defects


Extraction equipment


Implantation equipment


BG

Pulm effects of IMN

Pulm failure from pulm injury not from method of fracture fixation

Indications shoulder arthrodesis

Chronic infection GHJ


Stabilisation in paralytic disorders


Post traumatic brachial plexus palsy


Salvage failed GHJA


Arthritic disease unsuitable for Arthroplasty/young pt


Stabilisation after resection for neoplasia

Indications shoulder arthrodesis

Chronic infection GHJ


Stabilisation in paralytic disorders


Post traumatic brachial plexus palsy


Salvage failed GHJA


Arthritic disease unsuitable for Arthroplasty/young pt


Stabilisation after resection for neoplasia

CI shoulder arthrodesis

I elbow arthrodesis


C shoulder arthrodesis


Paralysis scapular stabilisers


Charcot arthropathy

Position of shoulder arthrodesis

10-15 FF & Abd


45 IR

Goals in treatment calc #

1. Restore congruency post facet 2. Restore height (Bohler) 3. Reduction width calc 4. Decompress subfib space for perineal tendons 5. Realignment tub into valgus 6. Reduction CCJ if #

Goals in treatment calc #

1. Restore congruency post facet 2. Restore height (Bohler) 3. Reduction width calc 4. Decompress subfib space for perineal tendons 5. Realignment tub into valgus 6. Reduction CCJ if #

Checks for post femoral IMN

Occult neck #


Symmetry of lower limb lengths & limb segments length


Symmetry of rotation: passive & active


Examine knee ligs for concomitant injury


Palpate thigh/calf - ensure soft


How to make AB impregnated cement nail

40F chest tube for 10mm nail


Humeral nail guide wire 2.5mm ball tip


2bags PMMA cement 2g Vanc + 2.4g tobra

DR # acceptable

AAOS clinical guidelines 2009


- radial height N11mm


Loss <3mm


- radial inclination N 22 deg


>10 degrees


- volar tilt N 11


<10 dorsal tilt


- articular surface


<2mm step


-DRUJ stability & congruence

Screw home

IR femur in last 15 degrees extension

At vertical, r'ship of mech vs anatomical axis

Mech is 3 deg Valgus


Ana is 9 deg Valgus


So ANA is 6 deg Valgus rel to mech axis

Qangle N values

15 ext


8 flex

Discoid meniscus classification

Watanabe


1. Incomplete coverage


2. Complete coverage


3. Wrisberg variant

Discoid meniscus classification

Watanabe


1. Incomplete coverage


2. Complete coverage


3. Wrisberg variant

Diff bw Weisberg variant discoid meniscus

1&2 have intact meniscotibial ligs


Wrisberg doesn't therefore moves abN & often symptomatic even if not torn

6 general indications for meniscal repair

Complete long tear >10mm


Tear w/in peripheral 1/3 or <3mm meniscocapsular jn


Unstable tear displaced by probe


Tear without secondary deg/deformity


Tear in active pt


Tear identified during Lig stab procedure

MRI appearance bloody effusion

High T1


Low T2

Criteria for Dx exercise induced CCS

>30mmHg at 1 min post exercise


>20 at 5 mins


>15 at 15 mins

4 risk factors for infection in TKR

RA


M


Inc INR


Compromised skin

Risk factors peri prosthetic # in TKR

Osteoporosis


Osteonecrosis


Osteolysis


Notching


Stress shielding

Classification distal femoral peri prosthetic #

Lewis & Rorabeck


1. Undisplaced, stable prosthesis


2. Displ, stable


3. Displ, loose prosthesis

Classification distal femoral peri prosthetic #

Lewis & Rorabeck


1. Undisplaced, stable prosthesis


2. Displ, stable


3. Displ, loose prosthesis

Prox tibial peri prosthetic #

Felix, Stuart & hanssen


1. Plateau


2. Adj stem


3. Dist to prosthesis


4. Tib tub


Subclassify A-C desc rel stability of prosthesis



Like Vanc...

3 ways to protect ext mech

Quads snip


V Y quadricepsplasty


Tub osteotomy

Salvage options for disrupted ext mech

SemiT autograft


Allograft

3 Surg indications for constrained hinge

All ligs gone


Knee resection


Hyperextension instability eg polio

Atlas/axis space occupied by cord

Steele's rule of thirds


1/3 cord


1/3 dens


1/3 space

Treatment acute sag band rupture

MCPJ ext splint

Treatment acute sag band rupture

MCPJ ext splint

Sag band rupture >2/52 treatment

Ext centralisation procedure

Therapeutic measures for perthes

3 primary obj


- improved mobility


- weight relief


- improved containment

5 causes kyphosis

SANTI


Scheuermanns


Ank spond


NF


Trauma eg compression #


Iatrogenic (post laminectomy)

Indications for Surg in TL burst #

Unstable


>50% Loh


>50% canal compromise


>30 degrees kyphosis


Incomplete/prog neuro deficit

Arthrofibrosis

The excessive fibroblastic tissue healing response that results in loss of motion esp after knee trauma or surgery