Oral Manifestations Of Sexually Transmitted Diseases Test 2.5

Front 1

etiology of syphilis?

Back 1

Treponema pallidum

– An anaerobic spirochete

Front 2

two types of syphilis?

Back 2

congenital and acquired

Front 3

CongenitalSyphilis two ways?

Back 3

1. Acquired in utero, but not before the fourth month

2. Oral Lesions

Front 4

3 types of developmental teeth/tissue disorders from congenital syphilis

Back 4

hutchinson's incisors, mulberry molars, soft tissue

Front 5

what are Hutchinson’sincisors?

Back 5

screwdriver shaped incisors caused by syphilis during tooth development

Front 6

what are mulberry molars?

Back 6

malformedfirst molars, with small nodules of enamel occlusally vs cusps

Front 7

what is soft tissue?

Back 7

Gummas,glossitis, mucous patches, rhagades (fissuring and scarring in the commisures)

Front 8

two types of acquired syphilis?

Back 8

primary and secondary

Front 9

primary syphilis is from? transmitted how?

Back 9

Follows sexual activity that involves direct contact with a syphiliticlesion. The organism can be transmittedby vaginal, anal or oral sex.

Front 10

primary lesion appears when? appears where and how? heals?

Back 10

usuallya genital chancre that appears three weeks after exposure. It appears at the site of entry, and isusually firm, round , small and painless. Spontaneous healing in 3-6 weeks

Front 11

how will primary syphilis progress?

Back 11

Will progress to secondarystage without treatment

Front 12

secondary syphilis Symptoms

Back 12

Sorethroat, malaise, fever, chills, macular cutaneous eruptions

Front 13

a. Secondary lesion description?

Back 13

mucouspatches

Front 14

Secondary lesion appears? lesion begins when?borders are?

Back 14

When lesion appears grayisherosions usually occur on the tongue, lips, tonsils.- Lesions begin 5-6 weeks after chancre disappearsand are very infective to dental clinicians.-Borders are often irregular and resemble “snailtracks”.

Front 15

secondary syphilis progression?

Back 15

Will progress to tertiarystage without treatment

Front 16

TertiarySyphilis Manifestations

Back 16

– Manifests years later in the CNS, CVS, skeleton, skin and almost anyorgan

- Gummas

Front 17

Tertiarylesion Description

Back 17

circumscribedlesions consisting of inflammation and rubber-like necrosis.

Front 18

Tertiarylesion Location

Back 18

On palate, may progress to perforation

Front 19

Syphiliticglossitis

Back 19

chronicinflammation of the tongue

Front 20

Diagnosis of Acquired Syphilis 2 things

Back 20

a. Darkfield microscopy toidentify spirochetes b. Blood test to detectsyphilitic antibodies (VDRL)

Front 21

Treatmentof Acquired Syphilis

Back 21

a. Penicillin – single doseif condition is less than 1 yearduration

b. Penicillin – larger doseneeded for more established disease

c. PCN allergy – alternateantibiotics

Front 22

Gonorrhea cause?

Back 22

infectionby neisseria gonorrhoeae after sexual contact.

Front 23

incubation period of gonorrhea

Back 23

1 to 4 weeks

Front 24

gonorrhea symptoms for males

Back 24

sudden and frequent painfulurination, mucopurulent discharge, fever

Front 25

gonorrhea symptoms for females

Back 25

symptomsnot as evident but ultimately more destructive

Front 26

gonorrhea symptoms for newborns

Back 26

Opthalmianeotorum – Gonoccocal eye infection ina newborn

Front 27

gonorrhea oral lesions appear

Back 27

painfulwhite, yellow or gray patches which slough off leaving bleeding points. Observe on gingiva, tongue and soft palate

Front 28

treatment for gonorrhea

Back 28

1.penicillin

2.tetracycline

3.erythromycin ointmentin eyes of newborn

Front 29

AcquiredImmunodeficiency Syndrome cause (4)

Back 29

1. Virus-Human ImmunodeficiencyVirus (HIV), which is classified as a retrovirus.

2. Parasitic nature-Unlikebacteria, viruses are parasites that can only reproduce within living cells.75-100% of those infectedwith HIV will get AIDS

3. CD4 T helper lymphocytes-keycells in immune system function of fighting microbial infection, are infectedwith HIV

4. Opportunistic infections andtumors-- Defects in T cells (# or function) = + susceptibility to opportunistic infections and tumors

Front 30

Epidemiology of HIV

Back 30

1. homosexual men

2. IV drug users

3. Heterosexual contact

4. Children of infected mothers

5. Recipients of bloodtransfusion, blood components or tissue

6. Unidentified risk

Front 31

transmission of HIV (4)

Back 31

1. Sexual contact with infected persons 2. exposure to infected Blood products 3. Mother to newborn through the placenta – 4. Only blood, semen, vaginal secretions andbreast milk have been proven to transmit the virus. There is no evidence that HIV is transmittedthrough saliva.

Front 32

Oral Manifestations of HIV 12

Back 32

Candidiasis,Kaposi’ssarcoma,Hairyleukoplakia,Angularcheilitis,NonHodgkin’s lymphoma,HIVrelated periodontal disease,Oralwarts,HerpesZoster,NUG, Petechiae,Recurrent pathos ulcers,Herpessimplex

Front 33

Candidiasis

-early sign?

-most common in?

-seen where in mouth?

Back 33

-Earlysign of immune system deficiency.

-Mostcommon fungal infection seen in HIV patients.

-Palate, buccal and labial mucosa, dorsum of the tongue

Front 34

Kaposi’ssarcoma most common?

-common areas?

-appears?

Back 34

• Most common malignancy found in AIDS patients (20%)

• Palate and gingiva most common areas.

• Appears as a red or blue patch witch enlarges to a blue/purplemass. May ulcerate and bleed.

Front 35

Hairyleukoplakia predictor of?

- appears?

- thought to be caused by what?

Back 35

-Significantpredictor from HIV to AIDS, usually within three years.

-Shaggy white appearance on lateral borders oftongue.

-Painless and thought to becaused by Epstein Barr Virus.

Front 36

Angularcheilitis usually observed in?

- when present in young person what is suspected?

-appears as?

Back 36

– Usually observed in the elderly

– When present in a young person, suspect HIV infection

– Appears as fissures or ulcers radiating from the commissures

Front 37

NonHodgkin’s lymphoma most common type of?

-can occur ?

- what is the most common site?

- what can be seen radiographically?

Back 37

• -Most common type of lymphoma associated with AIDS

• Can occur anywhere in the mouth.

-The palate is the most common site.

• See bone destruction radiographically

Front 38

HIVrelated periodontal disease more severe than?

- resembles?

- does not respond to ?

- more rapid?

Back 38

– -More severe than non-AIDS patients

– Resembles NUG

– Does not respond to conventional therapy

– More rapid progression

Front 39

Oralwarts cause?

Back 39

HumanPapilloma Virus

Front 40

indicator disease

Back 40

1. PCP-Pneumocystis carinii pneumonia 2. Candidiasisof the esophagus, trachea, or lungs 3. Cytomegalovirusretinitis with vision loss 4. Generalized Herpes simplex/herpes zosterlesions lasting more than a month 5. Neurological disorders/dementia 6. Opportunistic infections with laboratoryevidence of HIV infection

Front 41

treatment of HIV

Back 41

1.AZT

2. Acyclovir

3. radiation and chemotherapy for kaposi’s