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50 Cards in this Set
- Front
- Back
Primary lesion
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First lesion to appear on skin in response to a causative agent-usually has a recognizable structure
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Secondary lesion
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Changes can occur in primary lesion and result in this type of lesion. These changes occur in epidermal layer and caused by several factors including scratching,rubbing,meds,treatments,client med condition or disease process
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4 types of wound exudate
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serous
sanguineous serosanguineous purulent |
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serous drainage
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clear,watery plasma
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sanguineous drainage
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red
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serosanguineous drainage
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mixture of serous and sanguineous drainage,consists of plasma and red blood cells and is pink and watery
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purulent drainage
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pus, a protein-rich liquid product of the liquefaction of necrotic tissue. It is made up of cells and cellular debris and is usually caused by an infection. It is thick and yellow, green, tan or brown
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Drainage from surgical wound
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Initially sanguineous. Over several days, it changes to sersanguineous and then serous
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Pressure ulcer
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any lesion caused by ischemia from unrelieved pressure that leads to necrosis of underlying tissues. Develop when soft tissure is compressed between a bony prominence and an external surface.
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How to prevent pressure ulcer
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monitor client
cleanse the skin increase circulation decrease microorganisms remove excess moisture decrease friction and excoriation provide nutrition position client properly prevent shear decrease pressure |
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open wound
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disruption or break in skin
penetration by sharp object or instrument |
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closed wound
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trauma caused by blow with blunt object
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clean wound
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wound free of infectious organisms
surgical incision not entering or affected by secretions from respiratory,gastrointestinal or genitourinary tracts |
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contaminated wound
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wound with microorganisms
penetration of skin by dirt bacteria |
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penetrating wound
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wound with break through epidermis,dermis and underlying tissues; may enter organs
penetration by object or instrument (usually accidental) |
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abrasion
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superficial injury caused by rubbing or scraping of skin against another surface
friction injury resulting from fall or rubbing against bed linens |
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laceration
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open wound with jagged edges
penetration of skin by sharp object |
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contusion
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closed wound; may be swollen, discolored and painful
blunt trauma, such as being hit by object |
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partial-thickness wound
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extends through the first layer of skin (epidermis) and into but not through the second layer of skin (dermis)
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full-thickness wound
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extend through both epidermis and dermis and may involve subcutaneous tissue, muscle and possibly bone
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3 phases of wound healing
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inflammatory phase
proliferative or reconstruction phase maturation or remodeling phase |
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Inflammatory phase of wound healing
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begins at time of tissue injury and lasts 3-4 days
inflammation characterized by heat,erythema,edema, pain leukocytes migrate to wound monocytes migrate to wound & clean bed of bacteria and and cellular debris |
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Proliferative or reconstruction phase of wound healing
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lasts 4-21 days
collagen fills wound bed, new blood vessels develop, granulation tissue formed by fibroblasts |
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Maturation or remodeling phase of wound healing
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much maturation occurs by 3-4 weeks, maximum may not occur until 2 years
phase characterized by reorganization of collagen fibers, wound remodeling and maturation of tissues to approximate skin's original strength |
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3 types of wound healing
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primary intention
secondary intention tertiary intention |
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primary intention
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clinician approximates wound edges or margins and secures them using sutures, staples, or steri-strips
occurs in the first 14 days of injury wound bed free of hematoma,debris, or exudate, then fills in with minimal granulation tissue, and the scar is usually thin or flat |
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secondary intention
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prolonged
skin or wound edges can't be approximated, as in a pressure ulcer or a wound that is large or infected all dead and infectious tissue must be removed and the wound must fill with granulation tissue. wound contraction plays a greater role generally involves greater tissue loss, higher risk for infection, prolonged healing and a large scar |
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tertiary intention
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delayed or primary closure
occurs in wounds that may be contaminated, infected or draining exudate. May be left open intentionally for 3-5 days to let healing begin by allowing contaminated or infected matter to drain out. Once infection clears & granulation has begun,wound closed with sutures,staples or steri-strips |
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Stages of pressure ulcers
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Stages I-IV
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Stage I pressure ulcer
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observable, pressure-related alteration of intact skin whose indicators as compared with adjacent or opposite area of body, may include changes in one or more of the following:
skin temperature tissue consistency (firm/boggy) sensation appears as defined area of persistent redness in lightly pigmented skin |
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Stage II pressure ulcer
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Partial-thickness skin loss involving the epidermis,dermis or both. Ulcer is superficial and presents as abrasion, blister or shallow crater
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Stage III pressure ulcer
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full-thickness skin loss involving damage or necrosis of subcutaneous tissue that may extend to but not through underlying fascia. Present clinically as a deep crater with or w/o undermining of adjacent tissues
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Stage IV pressure ulcer
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Full-thickness skin loss with extensive destruction, tissue necrosis or damage to muscle,bone or supporting structures (tendon,joint capsule)
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Developmental factors in wound healing
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infants and children heal faster than older adults
older adults may be less mobile and have fragile,slowly healing skin As a person ages, wound healing phase can be prolonged because of decresed inflammatory responses. Changes in vascular,immune and respiratory systems can impair wound healing |
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Psyiological factors in wound healing
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Immunosuppression
Incontinence Hypoxemia Diabetes Neurological impairment procedures Medications Infection Hemorrhage Fistula Dehiscence and Evisceration |
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Environmental factors in wound healing
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Client environment should support clean,dry intact condition
Skin integrity affected by skin dryness, friction from bed linens, moisture from incontenince, perpiration,emesis and wound drainage. |
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hemorrhage
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bleeding from wound bed or site
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fistula
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abnormal passage between two internal organs or between and organ and external skin surface.
Forms because healing tissues don't close. |
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dehiscence
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separation of wound edges
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evisceration
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protusion of internal organ such as a bowel loop through the incision. Wound eviscerations are medical emegencies
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What to do for evisceration
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1.stay calm
2. ask colleague to obtain supplies and notify physician while you stay w/ client 3.help client into semi-fowler's position with knees slightly flexed after gatching foot of bed. This position will ease pressure on wound, prevent further tearing of the wound edges and reduce risk of further evisceration 4. cover protruding intesting with sterile dressing moistened saline to prevent wound contaminaiton and keep abdominal contents moist 5. monitor client closely, assess VS and Pulse ox readings 6. Establish intravenous access to provide fluids and prepare client for surgery 7. continue to provide emotion support |
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Which nutrients essential to wound healing?
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Vitamins C,A,B complex
Iron copper zinc |
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Lab work to look for if client has non-healing wounds
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culture-type of infection
WBC-elevated may suggest systemic infection Prealbumin and Albumin levels-low levels indicate poor nutrional status which slows wound healing; albumin best indicator of long-term nutritional status Radiological studies-for suspected infection of wound over bony prominence, rules out osteomyelitis. Standar radiograph or bone scan can be used to detect infection |
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Intervention to promote wound healing: Provide adequate nutrion
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Malnutrition inhibits wound healing and may increase risk for wound infection
Wound healing depends o availability of adequate protein, vitamins, an minerals. |
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Intervention to promote wound healing: Cleaning wound
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Wound bed must be clean and free of infection. Bacteria, devitalized tissue and exudate must be removed to promote healthy granulation tissue. Benefits of clean wound must be weighed against potential trauma to wound bed.
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Intervention to promote wound healing: Maintain moist wound bed
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moisture enhances wound reepithelialization, helping a wound heal faster and with less scar tissue
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Intervention to promote wound healing: dressing wound
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optimal dressings absorb drainage and provide aseptic environment that provides barrier against further trauma
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Intervention to promote wound healing: draining wound
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drains prevent fluid from collecting between surfaces of wound, which would separate wound surfaces and prevent them from growing together to heal wound
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Intervention to promote wound healing: debriding wound
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removes dirt of dirt, foreign matter and dead tissue from wound.removing this tissue promotes wound healig
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Describe technique when doing wound culture
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-Preliminary actions
-Rinse or irrigate wound thoroughly with sterile normal saline before obtaining culture (cleansing the wound before culturing will remove surface bacteria) -Swab the entire wound bed using zigzag technique and rotating swab starting at top of wound and proceeding to bottom of wound. Swab wound edges using 10pt. coverage. (Using this technique will cover entire wound bed. -Place swab in culture tube and send the labled specimen to lab. (Prompt delivery of swab specimen will facilitate prompt culturing of the swab.) |