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50 Cards in this Set

  • Front
  • Back
Primary lesion
First lesion to appear on skin in response to a causative agent-usually has a recognizable structure
Secondary lesion
Changes can occur in primary lesion and result in this type of lesion. These changes occur in epidermal layer and caused by several factors including scratching,rubbing,meds,treatments,client med condition or disease process
4 types of wound exudate
serous
sanguineous
serosanguineous
purulent
serous drainage
clear,watery plasma
sanguineous drainage
red
serosanguineous drainage
mixture of serous and sanguineous drainage,consists of plasma and red blood cells and is pink and watery
purulent drainage
pus, a protein-rich liquid product of the liquefaction of necrotic tissue. It is made up of cells and cellular debris and is usually caused by an infection. It is thick and yellow, green, tan or brown
Drainage from surgical wound
Initially sanguineous. Over several days, it changes to sersanguineous and then serous
Pressure ulcer
any lesion caused by ischemia from unrelieved pressure that leads to necrosis of underlying tissues. Develop when soft tissure is compressed between a bony prominence and an external surface.
How to prevent pressure ulcer
monitor client
cleanse the skin
increase circulation
decrease microorganisms
remove excess moisture
decrease friction and excoriation
provide nutrition
position client properly
prevent shear
decrease pressure
open wound
disruption or break in skin
penetration by sharp object or instrument
closed wound
trauma caused by blow with blunt object
clean wound
wound free of infectious organisms
surgical incision not entering or affected by secretions from respiratory,gastrointestinal or genitourinary tracts
contaminated wound
wound with microorganisms
penetration of skin by dirt bacteria
penetrating wound
wound with break through epidermis,dermis and underlying tissues; may enter organs
penetration by object or instrument (usually accidental)
abrasion
superficial injury caused by rubbing or scraping of skin against another surface
friction injury resulting from fall or rubbing against bed linens
laceration
open wound with jagged edges
penetration of skin by sharp object
contusion
closed wound; may be swollen, discolored and painful
blunt trauma, such as being hit by object
partial-thickness wound
extends through the first layer of skin (epidermis) and into but not through the second layer of skin (dermis)
full-thickness wound
extend through both epidermis and dermis and may involve subcutaneous tissue, muscle and possibly bone
3 phases of wound healing
inflammatory phase
proliferative or reconstruction phase
maturation or remodeling phase
Inflammatory phase of wound healing
begins at time of tissue injury and lasts 3-4 days
inflammation characterized by heat,erythema,edema, pain
leukocytes migrate to wound
monocytes migrate to wound & clean bed of bacteria and and cellular debris
Proliferative or reconstruction phase of wound healing
lasts 4-21 days
collagen fills wound bed, new blood vessels develop, granulation tissue formed by fibroblasts
Maturation or remodeling phase of wound healing
much maturation occurs by 3-4 weeks, maximum may not occur until 2 years
phase characterized by reorganization of collagen fibers, wound remodeling and maturation of tissues to approximate skin's original strength
3 types of wound healing
primary intention
secondary intention
tertiary intention
primary intention
clinician approximates wound edges or margins and secures them using sutures, staples, or steri-strips
occurs in the first 14 days of injury
wound bed free of hematoma,debris, or exudate, then fills in with minimal granulation tissue, and the scar is usually thin or flat
secondary intention
prolonged
skin or wound edges can't be approximated, as in a pressure ulcer or a wound that is large or infected
all dead and infectious tissue must be removed and the wound must fill with granulation tissue.
wound contraction plays a greater role
generally involves greater tissue loss, higher risk for infection, prolonged healing and a large scar
tertiary intention
delayed or primary closure
occurs in wounds that may be contaminated, infected or draining exudate.
May be left open intentionally for 3-5 days to let healing begin by allowing contaminated or infected matter to drain out.
Once infection clears & granulation has begun,wound closed with sutures,staples or steri-strips
Stages of pressure ulcers
Stages I-IV
Stage I pressure ulcer
observable, pressure-related alteration of intact skin whose indicators as compared with adjacent or opposite area of body, may include changes in one or more of the following:
skin temperature
tissue consistency (firm/boggy)
sensation
appears as defined area of persistent redness in lightly pigmented skin
Stage II pressure ulcer
Partial-thickness skin loss involving the epidermis,dermis or both. Ulcer is superficial and presents as abrasion, blister or shallow crater
Stage III pressure ulcer
full-thickness skin loss involving damage or necrosis of subcutaneous tissue that may extend to but not through underlying fascia. Present clinically as a deep crater with or w/o undermining of adjacent tissues
Stage IV pressure ulcer
Full-thickness skin loss with extensive destruction, tissue necrosis or damage to muscle,bone or supporting structures (tendon,joint capsule)
Developmental factors in wound healing
infants and children heal faster than older adults
older adults may be less mobile and have fragile,slowly healing skin
As a person ages, wound healing phase can be prolonged because of decresed inflammatory responses.
Changes in vascular,immune and respiratory systems can impair wound healing
Psyiological factors in wound healing
Immunosuppression
Incontinence
Hypoxemia
Diabetes
Neurological impairment
procedures
Medications
Infection
Hemorrhage
Fistula
Dehiscence and Evisceration
Environmental factors in wound healing
Client environment should support clean,dry intact condition
Skin integrity affected by skin dryness, friction from bed linens, moisture from incontenince, perpiration,emesis and wound drainage.
hemorrhage
bleeding from wound bed or site
fistula
abnormal passage between two internal organs or between and organ and external skin surface.
Forms because healing tissues don't close.
dehiscence
separation of wound edges
evisceration
protusion of internal organ such as a bowel loop through the incision. Wound eviscerations are medical emegencies
What to do for evisceration
1.stay calm

2. ask colleague to obtain supplies and notify physician while you stay w/ client

3.help client into semi-fowler's position with knees slightly flexed after gatching foot of bed. This position will ease pressure on wound, prevent further tearing of the wound edges and reduce risk of further evisceration
4. cover protruding intesting with sterile dressing moistened saline to prevent wound contaminaiton and keep abdominal contents moist

5. monitor client closely, assess VS and Pulse ox readings

6. Establish intravenous access to provide fluids and prepare client for surgery

7. continue to provide emotion support
Which nutrients essential to wound healing?
Vitamins C,A,B complex
Iron
copper
zinc
Lab work to look for if client has non-healing wounds
culture-type of infection

WBC-elevated may suggest systemic infection

Prealbumin and Albumin levels-low levels indicate poor nutrional status which slows wound healing; albumin best indicator of long-term nutritional status

Radiological studies-for suspected infection of wound over bony prominence, rules out osteomyelitis. Standar radiograph or bone scan can be used to detect infection
Intervention to promote wound healing: Provide adequate nutrion
Malnutrition inhibits wound healing and may increase risk for wound infection
Wound healing depends o availability of adequate protein, vitamins, an minerals.
Intervention to promote wound healing: Cleaning wound
Wound bed must be clean and free of infection. Bacteria, devitalized tissue and exudate must be removed to promote healthy granulation tissue. Benefits of clean wound must be weighed against potential trauma to wound bed.
Intervention to promote wound healing: Maintain moist wound bed
moisture enhances wound reepithelialization, helping a wound heal faster and with less scar tissue
Intervention to promote wound healing: dressing wound
optimal dressings absorb drainage and provide aseptic environment that provides barrier against further trauma
Intervention to promote wound healing: draining wound
drains prevent fluid from collecting between surfaces of wound, which would separate wound surfaces and prevent them from growing together to heal wound
Intervention to promote wound healing: debriding wound
removes dirt of dirt, foreign matter and dead tissue from wound.removing this tissue promotes wound healig
Describe technique when doing wound culture
-Preliminary actions
-Rinse or irrigate wound thoroughly with sterile normal saline before obtaining culture (cleansing the wound before culturing will remove surface bacteria)
-Swab the entire wound bed using zigzag technique and rotating swab starting at top of wound and proceeding to bottom of wound. Swab wound edges using 10pt. coverage. (Using this technique will cover entire wound bed.
-Place swab in culture tube and send the labled specimen to lab. (Prompt delivery of swab specimen will facilitate prompt culturing of the swab.)