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44 Cards in this Set
- Front
- Back
transient global amnesia involves loss of (2) but preservation of (2)
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lost:
-- recent events -- ability to retain new info preserved: -- immediate recall -- remote memory |
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transient global amnesia episodes typically last how long?
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< 24 hours
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a classic example of crossed brainstem findings (2)
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ipsilateral cranial nerve
contralateral long tract |
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signs that indicate CNS disease but are nonlocalizing within the CNS
(7) |
hemiparesis
hemisensory loss hemiataxia unilateral hyperreflexia Babinski sign eye movement disorders of cerebral, brainstem, or cerebellar origin position sense loss exceeding loss of vibration perception |
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5 pathognomonic signs of cerebral hemisphere disease
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dementia or a confused state
abnormalities of -- mood, personality, behavior [with abnormal neuro signs] aphasia seizure homonymous hemianopsia |
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impaired rapid alternating movements have what localizing value?
can be caused by what? |
localizing value when unilateral
proprioception loss and weakness of any etiology |
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a couple signs that may occur with any encephalopathy
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postural tremor
asterixis |
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encephalopathy implies a _
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rapidly developing confusional state
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causes of encephalopathy
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metabolic
infections e.g. encephalitis nutritional disorders toxins or drugs trauma |
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radicular pain means
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pain radiated along a dermatome
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pain radiating to the shoulder may be _
and is particularly common with ___ disease |
radicular
schwannoma |
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spastic hemiparesis localizes to _____
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corticospinal tract
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main probable diagnosis of spinal cord disease, and a secondary possible diagnosis
given a 2-year history of neck pain |
structural degenerative disease
neoplasm |
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aphasia is often misinterpreted as _
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confusion
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aphasia suggests _
whereas confusion suggests (3) |
stroke
metabolic toxic hypoxic-ischemic |
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perseveration may occur in (at least) two kinds of disease
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aphasia
dementia |
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signs of aphasia include (4)
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paraphasias
dysnomia abnormal fluency poor repetition |
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three motor systems and their roles
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corticospinal -- initiation of movement
extrapyramidal -- maintenance of posture cerebellar -- coordination |
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nuclei of the extrapyramidal system
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basal ganglia nuclei:
caudate globus pallidus subthalamus putamen - - - - - - - - - in the midbrain: substantia nigra |
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atheromatous disease of the cerebral vessels may cause _ with subsequent _
and thereby it may cause a stroke |
stenosis
thrombosis |
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branches of the PCA supply (5)
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subthalamic nucleus
thalamus portions of the midbrain inferior temporal lobe occipital lobe |
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hemiballism -- description / location
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flinging irregular movement at proximal joints
esp. shoulder and hip |
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chorea affects distal vs. proximal?
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distal
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choreiform movements
description location |
rapid, jerky
distal |
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how may patients with homonymous hemianopsia present?
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may c/o visual loss in only one eye
b/c the temporal field loss is larger and more apparent to the patient |
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a treatment for hemiballism
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dopamine antagonist
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the cardinal features of diseases affecting the brainstem:
(2) |
crossed findings
specific types of abnormal eye movements |
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most forms of pathologic nystagmus occur with _ lesions
less often occur with _ lesions |
brainstem
cerebellar |
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the direction of nystagmus refers to its
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quick phase
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describe the nystagmus associated with drug toxicity
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horizontal, gaze-evoked
i.e. left-beating nystagmus on left lateral gaze nystagmus in the vertical plane may also occur with drug toxicity |
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describe the nystagmus seen in peripheral vestibular disease
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horizontal, direction-fixed nystagmus
with a slight rotatory element |
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left eight nerve lesion --> what kind of nystagmus?
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direction-fixed
contralateral (right beating) horizontal nystagmus with a slight counterclockwise rotatory element it is most prominent on right (contralateral) lateral gaze |
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nystagmus which is of equal velocity in both directions
and has no separation into quick and slow phases is known as |
pendular nystagmus
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Wallenberg syndrome
sxs |
ipsilateral face
-- pain -- reduced pain and temp -- both contralateral limbs and body -- loss of pain and temp ipsilateral horner nystagmus incoordination of ipsilateral arm leaning and veering / gait ataxia in deep lesions: dysphagia and hoarseness |
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the cause Alpert mentioned for
Wallenberg's syndrome |
vertebral occlusion
with ischemia in PICA |
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Horner's sxs
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ipsilateral:
miosis ptosis anhidrosis (warmth) |
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explanation for Horner's in Wallenberg syndrome
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sympathetics from hypothalamus traverse the dorsolateral medulla
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a couple examples of facial sensory deficits associated with Wallenberg's
and what their underlying lesion is |
absent corneal response
numbness in the mouth spinal tract and nucleus of CN V |
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a lesion that explains left heel to shin ataxia
in Wallenberg's |
left inferior cerebellar peduncle
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weakness of the left soft palate
may be seen in Wallenberg's lesion? |
nucleus ambiguus
which is motor for CN X |
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body sensory symptoms for Wallenberg's
and their underlying lesion |
Alpert: contralateral hypesthesia (decreased sensation)
Blueprints: loss of pain and temperature spinothalamic tract |
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what part of the cerebellum plays a role in slow eye movements?
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cerebellar flocculus and vermis
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_ eye movement when the head is turned is mediated by _
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slow visual persuit
vestibulo-ocular reflex |
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what part of the cerebellum plays a role in slow eye movements?
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cerebellar flocculus and vermis
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