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34 Cards in this Set

  • Front
  • Back
autoregulation
the control of blood flow by cerebal BV vasoconstrinction in response to increased blood pressure- this increases the resistance to blood flow
chemoreception
neurons, astrocytes and cerebral BV collaborate to respond to increased neural activity. they react to metabolites such as CO or glutamate to regulate blood flow in the brain
autonomic innervation
plays a minor role in regulating blood flow in the brain. helps system to adapt to stress and manage extremes of auto regulation range
What are the 2 arterial sources in the brain?
internal carotid= 80%
vertebral=20%
What does the internal carotid artery supply and what are it's main branches?
-supplies the telenchephlon and diencephlon
-anterior cerebral, anterior communication, middle cerebral, opthalmic, anterior choroidal & posterior commuicating arteries
anterior cerebral artery supply & ischemic effects
-supplies medial frontal and parietal lobes, corpus callosum
-Ischemia - contralateral motor and somatosensory deficits (> in legs)
anterior communicating artery
Join right and left anterior cerebral arteries
middle cerebral artery supply & ischemic effects
-lateral cerebral hemispheres (most pre/post central gyri)
-Ischemia - major motor and somatosensory deficits with ischemia, also language deficits if in dominant hemisphere
ophthalmic artery supply & ischemic effects
-orbital contents
-Ischemia - Vision loss
anterior choroidal artery supply & ischemic effects
-supplies choroid plexus (lateral ventricles), optic tract, internal capsule, thalamus
-Ischemia contralateral motor and somatosensory deficits as well as vision problems
posterior communicating artery
connects to posterior cerebral artery
lenticulostriate / lateral striate arteries
-Small branches of the middle cerebral artery supply deep structures of the diencephalon and telencephalon
perforating branches
-Smallest branches of the middle cerebral artery supply deep structures of the diencephalon and telencephalon, they are unnamed
-frequent area for stroke –-> disproportionate deficits due to thalamic connections (Internal capsule)
Vertebral arteries
-Pass through foramen magnum and join together to form basilar artery
-Supplies most of brainstem and cerebellum, as well as parts of diencephalon, occipital and temporal lobes
-Also blood supply to spinal cord
What are the branches of the vertebral arteries?
-Posterior spinal arteries (paired), supply blood for the posterior 1/3 of that half of cord
-Anterior spinal artery (unpaired), supply blood for the anterior 2/3 of cord
-Posterior inferior cerebellar artery (PICA) supply blood for the inferior cerebellar surface, 4th ventricle choroid plexus, and lateral medulla
-All 3 give off perforating branches to brainstem structures
What comes off the Basilar artery?
-2 branches
-Anterior inferior cerebral artery
-Postrior inferior cerebral artery
-posterior cerebral artery
-pontine
-labrinthine (sometimes, sometimes off AICA)
Anterior inferior cerebellar artery
(AICA), which supplies the anteriorinferrior cerebellum and caudal pons
Posterior inferior cerebellar artery
(PICA), supplies the inferior cerebellar surface, 4th ventricle choroid plexus, and lateral medulla
Posterior cerebral artery supplies what and effects of ischemia
-inferomedial occipital / temporal lobes and posterior choroid of 3rd ventricle
Ischemia - visual field losses
pontine arteries
– smaller branches of basilar artery supplies remainder of pons
Labyrinthine / internal auditory artery supplies what and effects of ischemia
– supplies inner ear
-Ischemia - results in vertigo and deafness (ipsilateral)
Cerebral arterial circle
-aka circle of willis
-interconnects internal carotid and basilar systems
-anterior communicating artery joins contralateral anterior cerbral arteries
-posterior communicating joins internal carotid to ipsilateral posterior cerebral arteries
What are the 2 types of CVAs?
ischemic and hemorrhagic strokes
ischemic stroke
caused by:Altered blood pressure
Arterial disease
Blockage
hemorrhagic stroke
caused by spontaneous rupture of small blood vessels
Causes ischemia distal to the rupture
transient ischemic attacks (TIAs)
-an ischemic event that ends relatively quickly (mins-hrs); becomes a stroke after 24 hours
-usually due to minute emboli that partially block artery then are broken down
-results in little or no neurological deficits
spinal cord ischemia
motor or sensory deficients in extremities distal to lesion site
Aneurysm
- ballon like swelling of the arterial walls
-causes deficits as it compresses structures when the swelling increases in size
-rupture can result in massive hemorrhage in the subarachnoid space
Arteriovenous malformations (AVMs)
-congenital defects where large anastomoses (communicating holes) exist between arteries and veins
-Can steal blood from adjacent b/c of low resistance
-may hemorrhage
Venous drainage of brain
-Include cerebral veins, dural sinuses, emissary veins (through skull) and venous plexuses
-Principle route is through cerebral veins that empty into dural venous sinuses
-Flow to internal jugular veins and basilar venous plexus around base of brain
What do emissary veins do?
Emissary veins connect extracranial veins with dural sinuses. Play a minor role in drainage but are clinacally important as they may spread infection into cranial cavity
Superficial cerebral veins
- these are on the surface of the cerebral hemisphere
-Superior group – empties into superior sagittal sinus
-Inferior group – empties into transverse and cavernous sinuses
deep cerebral veins
-drain internal structures
-more consistant configuration
-empty into straight sinus
-Internal cerebral vein: major deep vein
forms next to the interventricular foramen bends posteriorly
-Great cerebral vein (Great Vein of Galen)
formed by fusion of 2 internal cerebral veins
Joins with inferior sagittal sinus to form straight sinus
Superior cerebellar artery
supplies the superoanterior cerebellum, caudal midbrain, and rostral pons