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34 Cards in this Set
- Front
- Back
autoregulation
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the control of blood flow by cerebal BV vasoconstrinction in response to increased blood pressure- this increases the resistance to blood flow
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chemoreception
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neurons, astrocytes and cerebral BV collaborate to respond to increased neural activity. they react to metabolites such as CO or glutamate to regulate blood flow in the brain
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autonomic innervation
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plays a minor role in regulating blood flow in the brain. helps system to adapt to stress and manage extremes of auto regulation range
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What are the 2 arterial sources in the brain?
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internal carotid= 80%
vertebral=20% |
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What does the internal carotid artery supply and what are it's main branches?
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-supplies the telenchephlon and diencephlon
-anterior cerebral, anterior communication, middle cerebral, opthalmic, anterior choroidal & posterior commuicating arteries |
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anterior cerebral artery supply & ischemic effects
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-supplies medial frontal and parietal lobes, corpus callosum
-Ischemia - contralateral motor and somatosensory deficits (> in legs) |
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anterior communicating artery
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Join right and left anterior cerebral arteries
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middle cerebral artery supply & ischemic effects
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-lateral cerebral hemispheres (most pre/post central gyri)
-Ischemia - major motor and somatosensory deficits with ischemia, also language deficits if in dominant hemisphere |
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ophthalmic artery supply & ischemic effects
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-orbital contents
-Ischemia - Vision loss |
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anterior choroidal artery supply & ischemic effects
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-supplies choroid plexus (lateral ventricles), optic tract, internal capsule, thalamus
-Ischemia contralateral motor and somatosensory deficits as well as vision problems |
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posterior communicating artery
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connects to posterior cerebral artery
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lenticulostriate / lateral striate arteries
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-Small branches of the middle cerebral artery supply deep structures of the diencephalon and telencephalon
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perforating branches
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-Smallest branches of the middle cerebral artery supply deep structures of the diencephalon and telencephalon, they are unnamed
-frequent area for stroke –-> disproportionate deficits due to thalamic connections (Internal capsule) |
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Vertebral arteries
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-Pass through foramen magnum and join together to form basilar artery
-Supplies most of brainstem and cerebellum, as well as parts of diencephalon, occipital and temporal lobes -Also blood supply to spinal cord |
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What are the branches of the vertebral arteries?
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-Posterior spinal arteries (paired), supply blood for the posterior 1/3 of that half of cord
-Anterior spinal artery (unpaired), supply blood for the anterior 2/3 of cord -Posterior inferior cerebellar artery (PICA) supply blood for the inferior cerebellar surface, 4th ventricle choroid plexus, and lateral medulla -All 3 give off perforating branches to brainstem structures |
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What comes off the Basilar artery?
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-2 branches
-Anterior inferior cerebral artery -Postrior inferior cerebral artery -posterior cerebral artery -pontine -labrinthine (sometimes, sometimes off AICA) |
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Anterior inferior cerebellar artery
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(AICA), which supplies the anteriorinferrior cerebellum and caudal pons
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Posterior inferior cerebellar artery
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(PICA), supplies the inferior cerebellar surface, 4th ventricle choroid plexus, and lateral medulla
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Posterior cerebral artery supplies what and effects of ischemia
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-inferomedial occipital / temporal lobes and posterior choroid of 3rd ventricle
Ischemia - visual field losses |
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pontine arteries
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– smaller branches of basilar artery supplies remainder of pons
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Labyrinthine / internal auditory artery supplies what and effects of ischemia
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– supplies inner ear
-Ischemia - results in vertigo and deafness (ipsilateral) |
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Cerebral arterial circle
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-aka circle of willis
-interconnects internal carotid and basilar systems -anterior communicating artery joins contralateral anterior cerbral arteries -posterior communicating joins internal carotid to ipsilateral posterior cerebral arteries |
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What are the 2 types of CVAs?
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ischemic and hemorrhagic strokes
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ischemic stroke
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caused by:Altered blood pressure
Arterial disease Blockage |
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hemorrhagic stroke
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caused by spontaneous rupture of small blood vessels
Causes ischemia distal to the rupture |
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transient ischemic attacks (TIAs)
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-an ischemic event that ends relatively quickly (mins-hrs); becomes a stroke after 24 hours
-usually due to minute emboli that partially block artery then are broken down -results in little or no neurological deficits |
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spinal cord ischemia
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motor or sensory deficients in extremities distal to lesion site
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Aneurysm
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- ballon like swelling of the arterial walls
-causes deficits as it compresses structures when the swelling increases in size -rupture can result in massive hemorrhage in the subarachnoid space |
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Arteriovenous malformations (AVMs)
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-congenital defects where large anastomoses (communicating holes) exist between arteries and veins
-Can steal blood from adjacent b/c of low resistance -may hemorrhage |
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Venous drainage of brain
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-Include cerebral veins, dural sinuses, emissary veins (through skull) and venous plexuses
-Principle route is through cerebral veins that empty into dural venous sinuses -Flow to internal jugular veins and basilar venous plexus around base of brain |
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What do emissary veins do?
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Emissary veins connect extracranial veins with dural sinuses. Play a minor role in drainage but are clinacally important as they may spread infection into cranial cavity
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Superficial cerebral veins
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- these are on the surface of the cerebral hemisphere
-Superior group – empties into superior sagittal sinus -Inferior group – empties into transverse and cavernous sinuses |
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deep cerebral veins
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-drain internal structures
-more consistant configuration -empty into straight sinus -Internal cerebral vein: major deep vein forms next to the interventricular foramen bends posteriorly -Great cerebral vein (Great Vein of Galen) formed by fusion of 2 internal cerebral veins Joins with inferior sagittal sinus to form straight sinus |
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Superior cerebellar artery
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supplies the superoanterior cerebellum, caudal midbrain, and rostral pons
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