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40 Cards in this Set

  • Front
  • Back
what is in telencephalon
cerebral hemisphere
cerebral cortex
subcortical white matter
basal ganglia
basal forebrain nuclei
What is in diencephalon
thalamus
hypothalamus
epithalamus
what is in mesencephalon
cerebral peduncles
midbrain tectum
midbrain tegmentum
what is in metencephalon
pons
cerebellum
what is in myelencephalon
medulla
function of parietal operculum
secondary sensory cortex
where is the primary visual cortex
in the occipital lobe along the banks calcarine sulcus
what is primary auditory cortex composed of
transverse gyri of heschl located in the slyvian fissure
how is the visual cortex organized
retinotopic fashion
how is primary auditory cortex organized
tonotopic
layers of neocortex, main connections
I molecular layer - dendrites and axons from other layers
II small pyramidal layer- cortical-cortical connections
III medium pyramidal layer cortical-cortical connections
IV granual layer- Recieves input from thalmus
V large pyramidal layer- sends outputs to subcortical structures
VI polymorphic layer - sends outputs to Thalmus
define ataxia, derived from what lesion
disorders in balance and coordination, cerebellum lesion
lesions in basal ganglia cause what type of movement and comparable to what disease
hypokinetic movement- parkinsons
hyperkinetic movement- huntingtons
monosynaptic reflex: what receptors involved, pathway
muscle spindle -> inhibatory interneuron -> motor neuron
where does the reticular formation extend
central portions from medulla to midbrain
caudal portions of reticular formation in medulla and lower pons involve?
motor and autonomic functions
rostral reticular formation in upper pons and midbrain plays a important role in?
regulating level of consciousness
lesions effecting pontomesencephalic reticular formation casue?
lethargy and coma
locations of limbic system
medial and anterior temporal lobe
anterior insula
inferior medial frontal lobes
cingulate gyri
hippocampal formation
amygdala (located within medial temporal lobe)
several nuceli medial thalamus, basal ganglia, septal area, and brainstem
what is fornix
paired arch-shaped white mattter connecting hippocampal formation to hypothalamus and septal nuclei
lesions to lymbic system cause?
deficits in the consolidation on innediate recall into longer-term memories
difficult forming new memories
epileptic seizures beigin with empotion
wernicke's area location
left side on superior temporal gyrus
lesions in wernicke's area casue
deficits in language comprehension (receptive or sensory aphasia)
location of Broca's area
frontal lobe covering pars triangularis and pars operculi
lesions in Broca's area
deficits in production of language
with sparing of language comprehension (expressive or motor aphasia)
finger agnosia
inability to identify fingers by name
lesions in parietal lobe
diffculty in calculations
right-left confusion
finger agnosia
difficulty with written language
Grestmann's syndrome- location of lesion, group symptoms
lesions in parietal lobe

diffculty in calculations
right-left confusion
finger agnosia
difficulty with written language
these group of symptoms are called what:
diffculty in calculations
right-left confusion
finger agnosia
difficulty with written language
Grestmann's syndrome
apraxia
abnormalities in motor conceptualization, planning, and execution
lesions in non-dominant parietal lobe
distortion of perceived space and neglect of contralateral side (hemineglect)
anosognosia
unawareness of a deficit on a side
frontal release signs:
caused by lesion where
"primitive reflexes" grasp, root, suck, snout reflex
frontal lobe
persevarate
repeat a single action over and over without moving on to the next one
lesions in frontal cortex can cause
frontal release sign
perseverate
disinhibited behaviors
magnetic gait
urinary incontinence
prospagnosia
inability to recognize faces
achromatopsia
inability to recognize colors
palinopsia
persistence or reapperance of an object
lesions in visual cortex can cause
prospagnosia
achromatopsia
palinopsia
magnetic gait
feet shuffle close to the floor