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19 Cards in this Set

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dyskinesias
difficulty controlling voluntary movements in absence of paralysis, paresis, or apraxia. 2 types.
1.hyperkinesia and 2. hypokinesia
chorea aka huntingtons dissease
a type of hyperkinesia

spontaneous rapid random movements, due to loss of ENK striasmal neurons favors direct pathway promoting thalamic drive.
huntingtons is characterized by loss of caudate nucleus
athetosis
a type of hyperkinesia
writhing(twisting turning) movements
dystonia
a type of hyperkinesia
joint or postural freezing due to antagonistic coactivation
ex idiopathic torsional dystonia
spasmodic torticollis
ballismus
a type of hyerkinesia
rapid flailing movements of the extremities due to loss of subthalamic nucleus that increses thalamic drive
tic
a type of hyperkinesia
with spontaneous single repeated movement, so movement is not random
tourette's syndrome
a type of hyperkinesia
movements with vocalizations
hypokinesia
condition marked by paucity of movements despite motivational and muscular ability is intact, ex parkinsons(loss of dopaminergic excitation of direct path and inhibition of indirect path) --> decrese thalamic drive
cerebellar ataxia
damage to cerebellum but not paralysis or paresis. appears ipsilateraly unless it is a vermal lesion
clinical signs of cerebellar ataxia

*dysmetria
*dysdiadochokinesia
*motor decomposition
*dysarthia
ataxic gait- drunken gait
nystagmus- eye movements
hypotonia- pendulous reflexes
dysmetria- intention tremor
limb ataxia- poorly coordinated
dysdiadochokinesia- repetitive movement disorder
motor decomposition- movements broken into components
dysarthia- slow speech
3 symptoms seen in vestibulocerebellum and inner ear lesions, making the two hard to differentiate from eachother
truncal ataxia- inability to balance
nystagmus- not normal in unconcious
vertigo- sense of room spinning, often accompanied by nausea
symptoms of flocculonodular lobe damage
truncal ataxia
and nystagmus.*fast phase of nystagmus is towards the side of the lesion, slow phase away
spinocerebellar damage AKA ant lob syndrome
can be caused by malnutrition (ex alcoholism)
shows with ataxia, but NO nystagmus or vertigo
cerebralcerebellum AKA neocerebellar damage
usually unilateral
*general loss of muscle tone- hyporeflexia and pendular reflex
less muscle coordination, takes longer to change directions
dysmetria, intention tremor, dysdiadochokinesia and decomposition of movement

These symptoms also involve speech.
apraxia
inability to perform learned motor skills in absence of paralysis. caused by lesions in supplementary motor area, but can still move. just cant integrate muscle groups.
astereognosis
lesion in parietal association cortex --> to tactile amnesia inability to ID an object by touch
agraphestesia
lesion in parietal association cortex --> inability to recognize object drawn onto skin even on hand
neglect syndrome
lesion in parietal association cortex --> failure to recognize opposite side of body and its surroundings
allodynia
perception of pain from non painful stimuli