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10 Cards in this Set
- Front
- Back
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what do baroreceptors in atria and great vessels do in trauma
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- released in response to pain
- stop the inhibition of ACTH, ADH, and GH |
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Renin Secretion
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- from increased symp, decreased RA perfusion, and decreased NaCl to macula densa
- does RAAS, vasoconstriction, limits Na and water |
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ALD secretion
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- stimulated by RAAS and ACTH
- does increased resorption of Na in DT for K and H |
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E and NE secretion
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- stiumlated by hypovolemia, pain, hypoglycemia, histamine
- does increased CO, BP, glycogenolysis - increased ACTH, lipolysis - decreased insulin secretion |
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ADH secretion
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- Stimuli are pain, hypovolemia, ACTH
- does increased water resorption in DT and CD - Splanchnic vasoconstriction - increased glycogenolysis |
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GH secretion
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- Stimuli are hypovolemia, hypoglycemia, tissue injury
- it increased glucose by opposing insulin - promotes lipolysis and secretion of certain proteins |
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glucagon secretion
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- Stimuli are hypoglycemia, sympathetic activity
- Does increased lipolysis, gluconeogenesis and glycogenolysis - increased glucose and ketogenesis |
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what causes DIC in trauma
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- increased locat mediators ADP and collagen get into system
- fibrinolytic system is blocked |
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what is the metabolic response to injury
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- hyperglycemia
- FA mobilization - increased extracellular osmolality, so edema - increased O2 consumption - Acidosis |
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what happens to O2 curve in injury
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- hypoxia makes it go to the right
- DPG makes it go to the right - Acidosis makes it go to the right - hypothermia and anemia make it go to the left, so keep them warm and give them blood |
