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10 Cards in this Set

  • Front
  • Back
what do baroreceptors in atria and great vessels do in trauma
- released in response to pain
- stop the inhibition of ACTH, ADH, and GH
Renin Secretion
- from increased symp, decreased RA perfusion, and decreased NaCl to macula densa
- does RAAS, vasoconstriction, limits Na and water
ALD secretion
- stimulated by RAAS and ACTH
- does increased resorption of Na in DT for K and H
E and NE secretion
- stiumlated by hypovolemia, pain, hypoglycemia, histamine
- does increased CO, BP, glycogenolysis
- increased ACTH, lipolysis
- decreased insulin secretion
ADH secretion
- Stimuli are pain, hypovolemia, ACTH
- does increased water resorption in DT and CD
- Splanchnic vasoconstriction
- increased glycogenolysis
GH secretion
- Stimuli are hypovolemia, hypoglycemia, tissue injury
- it increased glucose by opposing insulin
- promotes lipolysis and secretion of certain proteins
glucagon secretion
- Stimuli are hypoglycemia, sympathetic activity
- Does increased lipolysis, gluconeogenesis and glycogenolysis
- increased glucose and ketogenesis
what causes DIC in trauma
- increased locat mediators ADP and collagen get into system
- fibrinolytic system is blocked
what is the metabolic response to injury
- hyperglycemia
- FA mobilization
- increased extracellular osmolality, so edema
- increased O2 consumption
- Acidosis
what happens to O2 curve in injury
- hypoxia makes it go to the right
- DPG makes it go to the right
- Acidosis makes it go to the right
- hypothermia and anemia make it go to the left, so keep them warm and give them blood