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18 Cards in this Set

  • Front
  • Back
how do BVs run in the muscle
- they pierce the epimysium
- they run parallel to eachother in the perimysium
how do nerves run in the muscle
- they pierce the epimysium
- they course perimysim and branch into nerve fibers that run in the endomysium
what is a tendon w/ muscle fibers running the length of it?
fibers of Sharpey
tendon fibers that pierce the bone
- 2 layers of CT, 1 around a tendon, 1 around surrounding sructures, to protects the tendon
- filled w/ lubricant, like a synovial cavity
- some tendons develop sesamoid bones for the same function
why is fiber diameter important in muscles?
- it determines max strength
- it changes in mature muscles, suggesting the level of muscle use has changed
sarcomere triad
- each sarcomere has 2
- 1 transaverse tubule (infoldings b/t each myofibril and 2 terminal cisternae (lateral SR sac)
what parts of the sarcomere change?
- I band
- H zone
where are the cell bodies of motor neurons?
in the gray matter of the anterior horn
what makes the A band?
- myosin (the motor)
- C-protein, M-protein
- titin, connects to myosin at Z line (a stabilizer)
what makes up the I band?
- Actin, an intermediate filament - G is the pure form, when it binds ATP, it becomes the F form (binds Ca)
- tropomyosin - 2 a-chains complexed w/ F-actin
- Troponin - C binds Ca to remove I, letting myosin and actin interact
- an intermediate filatment that allows propagation of muscle transmission b/t sarcomeres
- connection then continues at the membrate w/ adhesion sites called costameres (where dystrophin is)
- acts as a stabilizer of integreal ion channels
- prevents membrane disruption
how is ATP in the muscle recycled?
phosphocreatine (there is 4-5X as much as ATP
ATPase in muscle
- 1st kind activated at 10X the resting concentration of Ca and makes the muscle go
- 2nd kind activated at 10X resing concentration of Ca and gets rid of Ca
how are more muscle fibers recruited
- first 20 % is type 1
- then type 2
- at very high efforts, type 2 do the most work and are first to fatigue
3 phases of muscle repair
- desruction phase w/ necrosis and hematoma
- repair phase w/ phago of necrotised tissue, new stuff from satelite cells
- remodeling phase, or maturation of regenerated myofibrils (it can work now)
- note, healing is from satellite cells (stem cells)
Myositis ossificans
- prolif of bone and carilage w/in SkM (non-cancer)
- usually after a contusion of quads
- becomes non-painful, but bone remains
- if painful, you can remove it, but only when it matures (1-2 yrs)